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Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 14, pp. 1785 - 1822
In 2016, the American College of Cardiology published the first expert consensus decision pathway (ECDP) on the role of non-statin therapies for low-density... 
PCSK9 inhibitors | lipids | ASCVD | bile acid sequestrants | ezetimibe | LDL-C | statins | cholesterol | dyslipidemia | ACC Expert Consensus Decision Pathway | FAMILIAL HYPERCHOLESTEROLEMIA | CARDIAC & CARDIOVASCULAR SYSTEMS | EFFICACY | SAFETY | HEART-FAILURE | SCIENTIFIC STATEMENT | REDUCING LIPIDS | DOUBLE-BLIND | EVOLOCUMAB | ROSUVASTATIN | Sequestering Agents - pharmacology | United States | Humans | Coronary Artery Disease - prevention & control | Enzyme Inhibitors - pharmacology | Ezetimibe - pharmacology | Cholesterol, LDL - analysis | Hypercholesterolemia - drug therapy | Consensus | Medication Therapy Management - organization & administration | Hypercholesterolemia - diagnosis | Anticholesteremic Agents - pharmacology | Cardiology - methods | Chemoprevention - methods | Decision-making | Usage | Task forces | Comorbidity | Low density lipoproteins | Coronary heart disease | Risk factors | Cardiovascular agents | Deoxycholic acid | Prevention | Hypercholesterolemia | Blood cholesterol | Cardiac patients | Diabetes | Universities and colleges | Cardiology | Statins | Lipoproteins (low density) | Clinical trials | Risk | Cardiovascular disease | Genetic screening | Atherosclerosis | Inhibition | Medical research | Risk groups | Decision making | Health risks | Subtilisin | Disease control | Patients | Low density lipoprotein | Cholesterol | Algorithms | Kexin | Inhibitors | Acids | Arteriosclerosis | Diagnostic systems | Cardiovascular diseases | Risk management
Journal Article
Journal Article
FEBS Letters, ISSN 0014-5793, 12/2015, Volume 589, Issue 24, pp. 3998 - 4009
Homocysteine (Hcy) is an independent risk factor for atherosclerosis, but the underlying molecular mechanisms are not known. We investigated the effects of Hcy... 
DNA methylation | Foam cell | Homocysteine | Atherosclerosis | Fatty acid-binding protein 4 | atherosclerosis | homocysteine | free Cholesterol | DNMT1 | S-adenosylhomocysteine | AZC | azacytidine | S-adenosylmethionine | FABP4 | fatty acid-binding protein 4 | DNA methyltransferases 1 | HHcy | Hyperhomocysteinemia | ApoE | SAH | total Cholesterol | apolipoprotein E knockout | Cholesteryl Ester | SAM | Hcy | HYPERHOMOCYSTEINEMIA | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | BINDING PROTEIN 4 | ADIPOGENESIS | CELL BIOLOGY | BIOPHYSICS | DISEASE | PPAR-GAMMA | DIFFERENTIATION | EXPRESSION | Diet - adverse effects | Cholesterol - blood | Humans | Hyperhomocysteinemia - metabolism | Male | Monocytes - metabolism | Aorta - metabolism | Fatty Acid-Binding Proteins - metabolism | Apolipoproteins E - metabolism | Atherosclerosis - etiology | DNA Methylation | Hyperhomocysteinemia - physiopathology | Monocytes - pathology | Foam Cells - pathology | Aorta - enzymology | Foam Cells - metabolism | Genes, Reporter | Repressor Proteins - metabolism | Methionine - poisoning | Promoter Regions, Genetic | Hyperhomocysteinemia - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Repressor Proteins - genetics | Foam Cells - enzymology | Cholesterol - metabolism | Hyperhomocysteinemia - etiology | Fatty Acid-Binding Proteins - genetics | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Monocytes - enzymology | Animals | Apolipoproteins E - genetics | Cell Line, Tumor
Journal Article
Journal of Internal Medicine, ISSN 0954-6820, 03/2014, Volume 275, Issue 3, pp. 296 - 303
Recent trials of anti‐amyloid agents have not produced convincing improvements in clinical outcome in Alzheimer's disease; however, the reason for these poor... 
tau | amyloid | cell biology | genetics | Alzheimer's disease | Cell biology | Genetics | Tau | Amyloid | THERAPEUTICS | COMMON VARIANTS | COMPLEMENT ACTIVATION | DEPOSITION | ANTIBODY | BETA-PEPTIDE | MECHANISMS | AMYLOID PRECURSOR PROTEIN | APOLIPOPROTEIN-E | MEDICINE, GENERAL & INTERNAL | A-BETA | Brain - metabolism | Amyloid - genetics | Amyloid beta-Peptides - antagonists & inhibitors | Genetic Predisposition to Disease | Immunotherapy - methods | Amyloid - metabolism | Early Medical Intervention | Humans | Alzheimer Disease - metabolism | Alzheimer Disease - genetics | Genetic research | Amyloidosis | Cells | Brain | Neurosciences | cholesterol transport | Alzheimer disease | cholesterol synthesis | immunoglobulin G1 antibody | genetic association | bapineuzumab | follow up | solanezumab | phase 3 clinical trial (topic) | cellular distribution | phase 1 clinical trial (topic) | innate immunity | amyloid precursor protein | presenilin | Immunotherapy | Amyloid beta-Peptides | priority journal | Notch3 receptor | Neurovetenskaper | clusterin | cerebrospinal fluid level | clinical pathway | signal transduction | avagacestat | cholesterol metabolism | randomized controlled trial (topic) | risk factor | article | genetic variability | phase 2 clinical trial (topic) | pathogenesis | semagacestat | genetic analysis | complement classical pathway | apolipoprotein E | gamma secretase inhibitor | nonhuman | drug clearance
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2013, Volume 33, Issue 11, pp. 2481 - 2490
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2014, Volume 16, Issue 4, pp. 357 - 366
The YAP and TAZ mediators of the Hippo pathway ( hereafter called YAP/TAZ) promote tissue proliferation and organ growth. However, how their biological... 
ORGAN GROWTH | CHOLESTEROL | YAP/TAZ | TRANSCRIPTION | HIPPO PATHWAY | STATINS | CELL BIOLOGY | Phosphorylation - physiology | Cell Proliferation | Active Transport, Cell Nucleus - physiology | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Drosophila Proteins - metabolism | Phosphoproteins - metabolism | Sterol Regulatory Element Binding Proteins - genetics | Breast Neoplasms - metabolism | Drosophila melanogaster - metabolism | RNA Interference | Tumor Suppressor Proteins - genetics | HEK293 Cells | Trans-Activators - genetics | Female | Transcription, Genetic | Hydroxymethylglutaryl-CoA Reductases, NAD-Dependent - metabolism | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | HCT116 Cells | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Sterol Regulatory Element Binding Proteins - metabolism | Transcription Factors - genetics | Mevalonic Acid - metabolism | Polyisoprenyl Phosphates - metabolism | Transcription Factors - metabolism | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | rho GTP-Binding Proteins - metabolism | Trans-Activators - metabolism | Mice | Polyisoprenyl Phosphates - biosynthesis | Pyridines - pharmacology | RNA, Small Interfering | Drosophila Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Cell metabolism | Genetic research | Genetic aspects | Research | Cellular control mechanisms
Journal Article
Journal Article