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Journal of the National Cancer Institute, ISSN 0027-8874, 10/2010, Volume 102, Issue 19, pp. 1496 - 1512
Background The cell surface proteoglycan, chondroitin sulfate proteoglycan 4 (CSPG4), is a potential target for monoclonal antibody (mAb)-based immunotherapy... 
STEM-CELLS | MELANOMA-ASSOCIATED ANTIGEN | MDA-MB-435 CELLS | ONCOLOGY | PHOSPHORYLATION | GENE-EXPRESSION PATTERNS | HMW-MAA | ANTIIDIOTYPIC MONOCLONAL-ANTIBODIES | CHONDROITIN SULFATE PROTEOGLYCAN | M14 MELANOMA | NG2 NULL MOUSE | Lung Neoplasms - drug therapy | Immunotherapy - methods | Apoptosis - drug effects | Breast Neoplasms - immunology | Humans | Immunoblotting | Lung Neoplasms - pathology | Transplantation, Heterologous | Antineoplastic Agents - therapeutic use | Breast Neoplasms - metabolism | Pleural Effusion - pathology | Cell Movement - immunology | Chondroitin Sulfate Proteoglycans - drug effects | Signal Transduction - immunology | Flow Cytometry | Biomarkers, Tumor - metabolism | Female | Antineoplastic Agents - pharmacology | Disease Models, Animal | Cell Survival - drug effects | Antibodies, Monoclonal - pharmacology | Membrane Proteins - immunology | Cell Survival - immunology | Reverse Transcriptase Polymerase Chain Reaction | Breast Neoplasms - drug therapy | Chondroitin Sulfate Proteoglycans - immunology | Cell Movement - drug effects | Lung Neoplasms - immunology | Animals | Apoptosis - immunology | Confounding Factors (Epidemiology) | Signal Transduction - drug effects | Breast Neoplasms - pathology | Melanoma - immunology | Melanoma - drug therapy | Cell Line, Tumor | Membrane Proteins - drug effects | Aged | Cell Proliferation - drug effects | Mice | Care and treatment | Immunotherapy | Development and progression | Breast cancer | Genetic aspects | Research | Metastasis | Health aspects | Gene expression | Tumors | Index Medicus | Cell proliferation | Animal models | Cell survival | Proteoglycans | Statistical analysis | Tumor cells | Lung | Blood vessels | Pleural effusion | Regression analysis | Cell surface | Metastases | Nodules | Signal transduction | Xenografts | Monoclonal antibodies | Antitumor activity | Chondroitin sulfate | Microenvironments | Cell migration | Apoptosis
Journal Article
European Urology, ISSN 0302-2838, 2017, Volume 72, Issue 1, pp. 142 - 150
Journal Article
Cancer Research, ISSN 0008-5472, 12/2011, Volume 71, Issue 24, pp. 7410 - 7422
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2016, Volume 139, Issue 4, pp. 916 - 927
Chondroitin sulfate proteoglycan 4 (CSPG4) has been identified as a highly promising target antigen for immunotherapy of triple-negative breast cancer (TNBC).... 
medical biotechnology | human cytolytic fusion protein | microtubule‐associated protein tau | triple negative breast cancer | immunotherapy | Medical biotechnology | Microtubule-associated protein tau | Immunotherapy | Triple negative breast cancer | Human cytolytic fusion protein | microtubule-associated protein tau | APOPTOSIS | DRUG-DELIVERY | DEATH | NUCLEAR TRANSLOCATION | IN-VITRO | THERAPY | IMMUNOTOXIN | ONCOLOGY | CHONDROITIN SULFATE PROTEOGLYCAN | ENDONUCLEASE-G | EXPRESSION | Recombinant Fusion Proteins - isolation & purification | Caspase 9 - metabolism | Chondroitin Sulfate Proteoglycans - genetics | Recombinant Fusion Proteins - pharmacology | Humans | Caspase 3 - metabolism | tau Proteins - metabolism | Molecular Targeted Therapy | Triple Negative Breast Neoplasms - drug therapy | Biomarkers, Tumor | Tubulin - metabolism | Single-Chain Antibodies - genetics | tau Proteins - genetics | Triple Negative Breast Neoplasms - pathology | Female | Membrane Proteins - metabolism | Disease Models, Animal | Cell Survival - drug effects | Gene Expression | Membrane Proteins - genetics | Chondroitin Sulfate Proteoglycans - metabolism | Protein Transport | Xenograft Model Antitumor Assays | Antibodies, Monoclonal - genetics | Animals | Triple Negative Breast Neoplasms - metabolism | Biomarkers | Cell Line, Tumor | Protein Binding | Recombinant Fusion Proteins - genetics | Mice | Cell Cycle - drug effects | Viral antibodies | Antigens | Nucleases | Epidermal growth factor | Estrogen | Antibodies | Breast cancer | Progesterone | Sulfates | Proteins | Tumors | Index Medicus
Journal Article
Contraception, ISSN 0010-7824, 2017, Volume 95, Issue 6, pp. 592 - 601
Journal Article
Cancer Letters, ISSN 0304-3835, 10/2014, Volume 352, Issue 2, pp. 228 - 235
Highlights • Binding of a CSPG4-specific immunotoxin to rhabdomyosarcoma cells is demonstrated. • The IT is rapidly internalized into RMS cells and efficiently... 
Hematology, Oncology and Palliative Medicine | Immunotoxin | CSPG4 | Immunotherapy | Rhabdomyosarcoma | MOLECULAR CHARACTERIZATION | NUDE-MICE | MELANOMA-ASSOCIATED ANTIGEN | SINGLE-CHAIN IMMUNOTOXIN | BREAST-CANCER | ONCOLOGY | ALVEOLAR RHABDOMYOSARCOMA | ANTIBODY-BASED IMMUNOTHERAPY | CHONDROITIN SULFATE PROTEOGLYCAN | FACTOR-I RECEPTOR | EXPRESSION | Apoptosis - drug effects | Humans | Rhabdomyosarcoma - metabolism | Rhabdomyosarcoma - pathology | Immunotoxins - pharmacology | Exotoxins - metabolism | Dose-Response Relationship, Drug | Single-Chain Antibodies - metabolism | Time Factors | Immunotoxins - metabolism | Inhibitory Concentration 50 | Membrane Proteins - metabolism | Rhabdomyosarcoma - immunology | ADP Ribose Transferases - metabolism | Cell Survival - drug effects | Immunotoxins - immunology | Single-Chain Antibodies - pharmacology | Chondroitin Sulfate Proteoglycans - metabolism | Membrane Proteins - immunology | Exotoxins - pharmacology | Chondroitin Sulfate Proteoglycans - immunology | Bacterial Toxins - metabolism | Bacterial Toxins - pharmacology | ADP Ribose Transferases - pharmacology | Cell Line, Tumor | Protein Binding | Virulence Factors - metabolism | Single-Chain Antibodies - immunology | Virulence Factors - pharmacology | Tumors | Antigens | Pediatrics | Flow cytometry | Melanoma | Breast cancer | Metastasis | Cancer therapies | Patients | Proteins | Studies | Biopsy | Medical prognosis | Chondroitin sulfate | Mutation | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 12/2017, Volume 121, Issue 12, pp. 1346 - 1359
RATIONALE:Arrhythmogenic cardiomyopathy is caused primarily by mutations in genes encoding desmosome proteins. Ventricular arrhythmias are the cardinal and... 
arrhythmogenic right ventricular dysplasia | death, sudden | desmosomes | neuroglia | desmoplakins | CARDIAC & CARDIOVASCULAR SYSTEMS | CONDUCTION SYSTEM | INTERCALATED DISKS | SCN5A MUTATIONS | DILATED CARDIOMYOPATHY | RECESSIVE MUTATION | HEART-MUSCLE CELLS | HYPERTROPHIC CARDIOMYOPATHY | WOOLLY HAIR | PERIPHERAL VASCULAR DISEASE | CHONDROITIN SULFATE PROTEOGLYCAN | HEMATOLOGY | RIGHT-VENTRICULAR CARDIOMYOPATHY | Keratosis - genetics | Humans | Cells, Cultured | Syndrome | Arrhythmogenic Right Ventricular Dysplasia - genetics | Myocytes, Cardiac - pathology | Keratinocytes - pathology | Phenotype | Animals | Desmoplakins - genetics | Keratinocytes - metabolism | Keratosis - metabolism | Myocytes, Cardiac - metabolism | Mice | Mutation | Desmoplakins - metabolism | Arrhythmogenic Right Ventricular Dysplasia - metabolism | Proteoglycans - genetics | Antigens - genetics | Cardiac arrhythmia | Conduction | Phenotypes | Baldness | Transcription | Cardiomyopathy | Gene regulation | Keratinocytes | Gene deletion | Alopecia | Fibrillation | Clonal deletion | Skin | Chondroitin sulfate | Sulfate | Ventricle | Heart diseases | Catenin | Keratosis | Index Medicus | Mechanisms | Neuroglial cells | Cardiocutaneous syndrome | Desmoplakin | Sudden death | Animal Models of Human Disease | desmosome | arrhythmogenic right ventricular cardiomyopathy | Myocardial Biology | Arrhythmias | desmosome cardiomyopathy
Journal Article
Experimental Dermatology, ISSN 0906-6705, 07/2018, Volume 27, Issue 7, pp. 769 - 778
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 01/2018, Volume 24, Issue 5, pp. 1 - 15
Journal Article
Immunologic Research, ISSN 0257-277X, 8/2011, Volume 50, Issue 2, pp. 294 - 302
PLX4032 is a BRAF-selective inhibitor shown to be efficacious in the treatment of melanomas presenting with the BRAFV600E mutation. However, favorable... 
Allergology | Signaling pathways | Immunology | Medicine & Public Health | Melanoma | Internal Medicine | CSPG4-specific mAb | BRAF inhibitor resistance | Medicine/Public Health, general | BRAF inhibitor PLX4032 | SURVIVAL | COLLAGEN | PLX4032 | ACQUIRED-RESISTANCE | SENSITIVITY | NG2 PROTEOGLYCAN | IMMUNOLOGY | ANTIGEN HMW-MAA | THERAPY | PATHWAY | IMMUNOTHERAPY | Chondroitin Sulfate Proteoglycans - genetics | Membrane Proteins - genetics | Antibodies, Monoclonal - pharmacology | Humans | Chondroitin Sulfate Proteoglycans - antagonists & inhibitors | Membrane Proteins - immunology | Down-Regulation - drug effects | Mutation - genetics | Sulfonamides - pharmacology | Chondroitin Sulfate Proteoglycans - immunology | Drug Synergism | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Membrane Proteins - antagonists & inhibitors | Signal Transduction - drug effects | Melanoma - immunology | Proto-Oncogene Proteins B-raf - genetics | Cell Line, Tumor | Indoles - pharmacology | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Antibodies, Monoclonal - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Viral antibodies | Gene mutations | Analysis | Monoclonal antibodies | Antibodies | Sulfates | Cells | Cancer | Index Medicus | Signal transduction | Cell survival | Proteoglycans | Data processing | Remission | Chondroitin sulfate | Longevity | Mutation | Cell migration
Journal Article