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1. Full Text Chromosomal aberration frequency in lymphocytes predicts the risk of cancer: results from a pooled cohort study of 22 358 subjects in 11 countries
by Bonassi, Stefano and Norppa, Hannu and Ceppi, Marcello and Strömberg, Ulf and Vermeulen, Roel and Znaor, Ariana and Cebulska-Wasilewska, Antonina and Fabianova, Eleonora and Fucic, Alexandra and Gundy, Sarolta and Hansteen, Inger-Lise and Knudsen, Lisbeth E and Lazutka, Juozas and Rossner, Pavel and Sram, Radim J and Boffetta, Paolo
Carcinogenesis (New York), ISSN 0143-3334, 6/2008, Volume 29, Issue 6, pp. 1178 - 1183
Life Sciences & Biomedicine | Oncology | Science & Technology | Biological and medical sciences | Carcinogenesis, carcinogens and anticarcinogens | Medical sciences | Tumors | Genetic Predisposition to Disease | Risk Assessment | Humans | Middle Aged | Risk Factors | Male | Incidence | Neoplasms - genetics | Lymphocytes | Chromosome Aberrations | Aged, 80 and over | Adult | Female | Aged | Neoplasms - epidemiology | Cohort Studies | Index Medicus | Molecular Epidemiology | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
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2. Full Text Familial Hemophagocytic Lymphohistiocytosis Type 5 (FHL-5) Is Caused by Mutations in Munc18-2 and Impaired Binding to Syntaxin 11
by zur Stadt, Udo and Rohr, Jan and Seifert, Wenke and Koch, Florian and Grieve, Samantha and Pagel, Julia and Strauß, Julia and Kasper, Brigitte and Nürnberg, Gudrun and Becker, Christian and Maul-Pavicic, Andrea and Beutel, Karin and Janka, Gritta and Griffiths, Gillian and Ehl, Stephan and Hennies, Hans Christian
American journal of human genetics, ISSN 0002-9297, 2009, Volume 85, Issue 4, pp. 482 - 492
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Fundamental and applied biological sciences. Psychology | General aspects. Genetic counseling | Medical genetics | Biological and medical sciences | Molecular and cellular biology | Genetics of eukaryotes. Biological and molecular evolution | Medical sciences | Lymphohistiocytosis, Hemophagocytic - pathology | Munc18 Proteins - genetics | Humans | Child, Preschool | Munc18 Proteins - metabolism | Genotype | Infant | Male | Chromosome Mapping | Exocytosis | Chromosomes, Human, Pair 19 | Qa-SNARE Proteins - metabolism | Qa-SNARE Proteins - genetics | Female | Polymorphism, Single Nucleotide | Mutation | Lymphohistiocytosis, Hemophagocytic - genetics | SNARE Proteins - metabolism | Usage | Gene mutations | Lymphocytes | Analysis | Inflammation | Genetic aspects | Research | Single nucleotide polymorphisms | Proteins | Genotype & phenotype | Genetic disorders | Binding sites | Index Medicus
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3. Full Text Subtelomere FISH analysis of 11 688 cases: an evaluation of the frequency and pattern of subtelomere rearrangements in individuals with developmental disabilities
by Ravnan, J B and Tepperberg, J H and Papenhausen, P and Lamb, A N and Hedrick, J and Eash, D and Ledbetter, D H and Martin, C L
Journal of medical genetics, ISSN 0022-2593, 06/2006, Volume 43, Issue 6, pp. 478 - 489
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Cardiology. Vascular system | General aspects. Genetic counseling | Biological and medical sciences | Medical sciences | Medical genetics | Genetic Testing | Humans | Child, Preschool | In Situ Hybridization, Fluorescence | Infant | Male | Developmental Disabilities - genetics | Telomere - chemistry | Phenotype | Adolescent | Chromosome Aberrations | Adult | Female | Retrospective Studies | Child | Developmental Disabilities - diagnosis | Telomeres | Usage | Analysis | Child development deviations | Development and progression | Genetic aspects | Research | In situ hybridization | Developmental disabilities | Studies | Confidence intervals | Genotype & phenotype | Laboratories | Intellectual disabilities | Cloning | Cytogenetics | Genomes | Index Medicus | subtelomere | variant | mental retardation | fluorescence in situ hybridisation | Original | telomere
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4. Full Text Primary renal well‐differentiated neuroendocrine tumour (carcinoid): next‐generation sequencing study of 11 cases
by Pivovarcikova, Kristyna and Agaimy, Abbas and Martinek, Petr and Alaghehbandan, Reza and Perez‐Montiel, Delia and Alvarado‐Cabrero, Isabel and Rogala, Joanna and Kuroda, Naoto and Rychly, Boris and Gasparov, Slavko and Michalova, Kvetoslava and Michal, Michal and Hora, Milan and Pitra, Tomas and Tuckova, Inna and Laciok, Simon and Mareckova, Jana and Hes, Ondrej
Histopathology, ISSN 0309-0167, 07/2019, Volume 75, Issue 1, pp. 104 - 117
kidney | well‐differentiated neuroendocrine tumour | carcinoid | next‐generation sequencing | primary | well-differentiated neuroendocrine tumour | next-generation sequencing | Pathology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Immunohistochemistry | Kidney Neoplasms - genetics | Humans | Middle Aged | Male | Kidney Neoplasms - metabolism | Loss of Heterozygosity | Antigens, CD - genetics | Carcinoid Tumor - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Cell Differentiation | Cadherins - genetics | Neuroendocrine Tumors - pathology | Neuroendocrine Tumors - metabolism | Proto-Oncogene Proteins - genetics | DNA-Binding Proteins - genetics | Neuroendocrine Tumors - genetics | Carcinoid Tumor - pathology | Carcinoid Tumor - genetics | Kidney Neoplasms - pathology | Aged | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | Mutation | Gene mutations | Analysis | Tumors | Synaptophysin | Kidneys | Insulinoma | Thalassemia | Myc protein | Chromosome 3 | E-cadherin | Heterozygosity | CD99 antigen | Angiogenesis | Daxx protein | Protein X | Loss of heterozygosity | Genetic analysis | VHL protein | CD56 antigen | Neuroendocrine tumors | Index Medicus
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5. Full Text Fetal fraction in maternal plasma cell‐free DNA at 11–13 weeks' gestation: relation to maternal and fetal characteristics
by Ashoor, G and Syngelaki, A and Poon, L. C. Y and Rezende, J. C and Nicolaides, K. H
Ultrasound in obstetrics & gynecology, ISSN 0960-7692, 01/2013, Volume 41, Issue 1, pp. 26 - 32
trisomy 21 | cell‐free DNA | non‐invasive prenatal testing | fetal fraction | first‐trimester screening | cell-free DNA | first-trimester screening | non-invasive prenatal testing | Acoustics | Life Sciences & Biomedicine | Technology | Radiology, Nuclear Medicine & Medical Imaging | Obstetrics & Gynecology | Science & Technology | Gynecology. Andrology. Obstetrics | Biological and medical sciences | Medical sciences | Chromosome aberrations | Medical genetics | Pregnancy | Pregnancy Trimester, First - blood | Fetal Blood - chemistry | Humans | Risk Factors | Alleles | Maternal Serum Screening Tests - standards | Adult | Female | Male | Sequence Analysis, DNA - methods | DNA - blood | Chorionic gonadotropin | Pregnant women | Analysis | DNA | Glycoproteins | Chromosomes | Index Medicus
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6. Full Text Long-term results of St Jude Total Therapy Studies 11, 12, 13A, 13B, and 14 for childhood acute lymphoblastic leukemia
by Pui, C.H and Pei, D and Sandlund, J.T and Ribeiro, R.C and Rubnitz, J.E and Raimondi, S.C and Onciu, M and Campana, D and Kun, L.E and Jeha, S and Cheng, C and Howard, S.C and Metzger, M.L and Bhojwani, D and Downing, J.R and Evans, W.E and Relling, M.V
Leukemia, ISSN 0887-6924, 02/2010, Volume 24, Issue 2, pp. 371 - 382
B-lineage ALL | Chemotherapy | CNS relapse | T-lineage ALL | Leukemia | Prognostic factors | Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | Prognosis | Follow-Up Studies | Humans | Child, Preschool | Infant | Male | Neoplasm Recurrence, Local - pathology | Neoplasm, Residual | Precursor Cell Lymphoblastic Leukemia-Lymphoma - therapy | Time Factors | Neoplasms, Second Primary - therapy | Female | Neoplasms, Second Primary - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Child | Injections, Spinal | Neoplasm Recurrence, Local - therapy | Risk Factors | Immunophenotyping | Survival Rate | Treatment Outcome | Combined Modality Therapy | Cranial Irradiation | Remission Induction | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Adolescent | Chromosome Aberrations | Neoplasm Recurrence, Local - genetics | Neoplasms, Second Primary - pathology | Care and treatment | Physiological aspects | Research | Acute lymphocytic leukemia | T cells | Health aspects | Risk factors | Cancer | Index Medicus | leukemia | prognostic factors | chemotherapy
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7. Full Text The human cadherin 11 is a pro-apoptotic tumor suppressor modulating cell stemness through Wnt/Β-catenin signaling and silenced in common carcinomas
by Li, L and Ying, J and Li, H and Zhang, Y and Shu, X and Fan, Y and Tan, J and Cao, Y and Tsao, S.W and Srivastava, G and Chan, A.T.C and Tao, Q
Oncogene, ISSN 0950-9232, 08/2012, Volume 31, Issue 34, pp. 3901 - 3912
cadherin | carcinoma | methylation | tumor suppressor gene | CDH11 | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Cadherins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Epithelial-Mesenchymal Transition - genetics | Wnt Proteins - metabolism | Promoter Regions, Genetic - genetics | Gene Knockdown Techniques | DNA Methylation | Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Tumor Suppressor Proteins - genetics | Neoplastic Stem Cells - pathology | Cell Membrane - metabolism | Cadherins - genetics | Chromosomes, Human, Pair 16 - genetics | Cell Line | Tumor Suppressor Proteins - metabolism | Signal Transduction | HCT116 Cells | Comparative Genomic Hybridization - methods | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | Microscopy, Confocal | Cell Line, Tumor | CpG Islands - genetics | Neoplasms - pathology | Index Medicus | Carcinoma | Wnt protein | Epithelial cells | catenin | Fetuses | Multiple primary tumors | AKT protein | Tumor cell lines | Cadherin | Gene deletion | Stress | epigenetics | Fibers | Demethylation | Actin | Stem cells | DNA methylation | Tumor suppressor genes | genomics | chromosome 16 | Cell migration | Apoptosis | CpG islands | Tumors | Original
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