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1. Full Text Inflammation
by Weiss, Ursula
Nature, ISSN 0028-0836, 07/2008, Volume 454, Issue 7203, pp. 427 - 427
Inflammation is the body's immediate response to damage to its tissues and cells by pathogens, noxious stimuli such as chemicals, or physical injury. 
MULTIDISCIPLINARY SCIENCES | Inflammation - pathology | Inflammation - therapy | Chronic Disease | Humans | Inflammation - physiopathology | Autoimmune diseases | Tissue | Inflammatory diseases
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2. Full Text Nonresolving Inflammation
by Nathan, Carl and Ding, Aihao
Cell, ISSN 0092-8674, 2010, Volume 140, Issue 6, pp. 871 - 882
Nonresolving inflammation is a major driver of disease. Perpetuation of inflammation is an inherent risk because inflammation can damage tissue and necrosis... 
MEDIATED INFLAMMATION | IMMUNE-SYSTEM | ANTIINFLAMMATORY PROPERTIES | CHRONIC GRANULOMATOUS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | APOPTOTIC CELLS | NITRIC-OXIDE | AIRWAY INFLAMMATION | ACUTE LUNG INJURY | DEPENDENT CLEARANCE | PULMONARY-FIBROSIS | CELL BIOLOGY | Inflammation Mediators - immunology | Inflammation - therapy | Animals | Humans | Inflammation - immunology | Macrophages - immunology | Inflammation - physiopathology | Proteins | Drivers | Switches | Inflammatory response | Lipids | Damage | Macrophages | Stimuli
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3. Chronic inflammation
: mechanisms and regulation
by Miyasaka, Masayuki and Takatsu, Kiyoshi
2016, ISBN 9784431560685
Immunological aspects | Treatment | Chronic diseases | Inflammation
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4. Inflammation, lifestyle, and chronic diseases
: the silent link
by Aggarwal, Bharat Bhushan and Krishnan, Sunil and Guha, Sushovan
2011, ISBN 1439839891
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5. Full Text Immunity, Inflammation, and Cancer
by Grivennikov, Sergei I and Greten, Florian R and Karin, Michael
Cell, ISSN 0092-8674, 2010, Volume 140, Issue 6, pp. 883 - 899
Inflammatory responses play decisive roles at different stages of tumor development, including initiation, promotion, malignant conversion, invasion, and... 
PROMOTE TUMOR-GROWTH | EPITHELIAL-MESENCHYMAL TRANSITION | T-CELL RESPONSES | TGF-BETA | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHRONIC LYMPHOCYTIC-LEUKEMIA | COLITIS-ASSOCIATED CANCER | NECROSIS-FACTOR-ALPHA | MYELOID CELLS | NF-KAPPA-B | CELL BIOLOGY | Cell Transformation, Neoplastic - immunology | Animals | Neoplasms - immunology | Humans | Inflammation - immunology | Immune System - cytology | Inflammation - physiopathology | NF-κB | cytokines | immunity | inflammation | STAT3 | Cancer
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6. Full Text The inflammation theory of disease. the growing realization that chronic inflammation is crucial in many diseases opens new avenues for treatment
by Hunter, Philip
EMBO Reports, ISSN 1469-221X, 11/2012, Volume 13, Issue 11, pp. 968 - 970
  Scientific studies indicating that chronic inflammation is responsible for many types of diseases are discussed. Further research on this phenomenon might... 
CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Disease - etiology | Inflammation | Chronic Disease | Humans | Immunology | Infectious diseases | Epidemiology | Inflammatory diseases | Science and Society
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7. Full Text Inflammation and Colon Cancer
by Terzić, Janoš and Grivennikov, Sergei and Karin, Eliad and Karin, Michael
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 6, pp. 2101 - 2114.e5
The connection between inflammation and tumorigenesis is well-established and in the last decade has received a great deal of supporting evidence from genetic,... 
Gastroenterology and Hepatology | Inflammation | Cytokines | Immunity | Colon Cancer | CHRONIC INTESTINAL INFLAMMATION | COLITIS-ASSOCIATED CANCER | SEVERE CROHNS-DISEASE | EPITHELIAL-MESENCHYMAL TRANSITION | TUMOR-NECROSIS-FACTOR | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | KINASE-I-EPSILON | TOLL-LIKE RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | GENOME-WIDE ASSOCIATION | Colonic Neoplasms - genetics | Colonic Neoplasms - prevention & control | Colonic Neoplasms - drug therapy | Colorectal Neoplasms - genetics | Humans | Inflammatory Bowel Diseases - immunology | Gene Expression Regulation, Neoplastic | Colon - immunology | Cell Transformation, Neoplastic - genetics | Colonic Neoplasms - immunology | Colorectal Neoplasms - drug therapy | Inflammation Mediators - metabolism | Inflammatory Bowel Diseases - genetics | Anti-Inflammatory Agents - therapeutic use | Inflammatory Bowel Diseases - complications | Anticarcinogenic Agents - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Genetic Predisposition to Disease | Cytokines - metabolism | Risk Assessment | Signal Transduction | Neoplasm Invasiveness | Risk Factors | Colorectal Neoplasms - prevention & control | Colonic Neoplasms - secondary | Cell Transformation, Neoplastic - immunology | Animals | Colorectal Neoplasms - immunology | Colorectal Neoplasms - secondary | Colon - microbiology | Dendritic cells | Measles-mumps-rubella vaccine | Biological response modifiers | Sulfates | Risk factors | Dextran | Prostaglandins | Colon cancer | Gastrointestinal diseases | Vascular endothelial growth factor | Growth factors | Tumors
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8. Full Text Purinergic Signaling during Inflammation
by Eltzschig, Holger K and Sitkovsky, Michail V and Robson, Simon C
The New England Journal of Medicine, ISSN 0028-4793, 12/2012, Volume 367, Issue 24, pp. 2322 - 2333
Receptors for ATP and ADP and adenosine exert various effects. ATP and ADP signaling is mainly proinflammatory, and adenosine signaling is mainly... 
Neoplasms - metabolism | Inflammation - metabolism | Neoplasms - therapy | Humans | Immunotherapy | Signal Transduction - physiology | Acute Lung Injury - metabolism | Blood Platelets - physiology | Inflammatory Bowel Diseases - metabolism | Purine Nucleotides - metabolism | Purines | Control | Chronic diseases | Causes of | Cellular signal transduction | Inflammation | Ion channels | Research | Chemical properties | Health aspects
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9. Full Text Intestinal Inflammation and Cancer
by Ullman, Thomas A and Itzkowitz, Steven H
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 6, pp. 1807 - 1816.e1
Patients with ulcerative colitis and Crohn's disease are at increased risk for developing colorectal cancer (CRC). Chronic inflammation is believed to promote... 
Gastroenterology and Hepatology | Adaptive Immunity | IBD | Animal Model | Neoplasia | Intestine | Innate Immunity | NITRIC-OXIDE SYNTHASE | MICROSATELLITE INSTABILITY | COLITIS-ASSOCIATED CARCINOGENESIS | BOWEL-DISEASE | COLON-CANCER | CHRONIC ULCERATIVE-COLITIS | K-RAS GENE | EPITHELIAL-CELLS | COLORECTAL-CANCER | GASTROENTEROLOGY & HEPATOLOGY | PRIMARY SCLEROSING CHOLANGITIS | Colitis, Ulcerative - complications | Oxidative Stress | Colitis, Ulcerative - metabolism | Humans | Risk Factors | Crohn Disease - metabolism | Gastrointestinal Tract - microbiology | Immunity, Innate | Crohn Disease - complications | Colonic Neoplasms - physiopathology | Bacterial Infections - complications | Animals | Colonic Neoplasms - pathology | Crohn Disease - drug therapy | Anti-Inflammatory Agents - therapeutic use | Colitis, Ulcerative - drug therapy | Colonic Neoplasms - etiology | Development and progression | Ursodiol | Crohn's disease | Inflammation | DNA binding proteins | Sulfates | Dextran | Colon cancer | Analysis | Nitric oxide | DNA | Genetically modified organisms | Tumor proteins | Ulcerative colitis | Cancer
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