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PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e84934
Objectives: Fibrosis is characterized by excessive tissue remodeling resulting from altered expression of various growth factors, cytokines and proteases. We... 
ENZYME-IMMUNOASSAY | GELATINASE-A | MARKER | LAMININ | MULTIDISCIPLINARY SCIENCES | LIVER FIBROSIS | SEQUENCE | UNIQUE | EXTRACELLULAR-MATRIX | SERUM | PEPTIDE | Collagen Type IV - metabolism | Autoantigens - metabolism | Enzyme-Linked Immunosorbent Assay | Humans | Rats | Matrix Metalloproteinase 12 - metabolism | Carbon Tetrachloride - toxicity | Liver Cirrhosis - chemically induced | Basement Membrane - physiopathology | Animals | Analysis of Variance | Mass Spectrometry | Liver Cirrhosis - metabolism | Pulmonary Fibrosis - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Lung diseases, Obstructive | Liver diseases | Cytokines | Proteases | Collagen | Liver | Carbon tetrachloride | Fibrosis | Monoclonal antibodies | Mass spectrometry | Enzyme-linked immunosorbent assay | Antigenic determinants | Matrix metalloproteinases | Pathogenesis | Critical care | Chains | Biology | Matrix metalloproteinase | Assaying | Remodeling | Fragmentation | Degradation | Proteins | Ethics | Extracellular matrix | Tumor necrosis factor-TNF | Chronic obstructive pulmonary disease | Collagen (type IV) | Bioindicators | Metalloproteinase | Growth factors | Enzymes | Lung diseases | Organs | Fragments | Mass spectroscopy | Epitopes | Patients | Pulmonary fibrosis | Obstructive lung disease | Biomarkers | Laboratory animals | Immunoassays
Journal Article
Journal Article
Journal Article
Human Mutation, ISSN 1059-7794, 04/2015, Volume 36, Issue 4, pp. 463 - 473
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0138372
The functional crosstalk between angiotensin II (Ang II) and tumor necrosis factor (TNF)-alpha has been shown to cause adverse left ventricular remodeling and... 
CTGF EXPRESSION | NADPH OXIDASE | INDUCED HYPERTENSION | XANTHINE-OXIDASE | NAD(P)H OXIDASE | CARDIAC RESTRICTED OVEREXPRESSION | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | GENE-EXPRESSION | BLOOD-PRESSURE | TRANSGENIC MICE | Tumor Necrosis Factor-alpha - metabolism | MAP Kinase Signaling System - physiology | Oxidative Stress - physiology | Collagen Type III - metabolism | NADPH Oxidases - metabolism | Male | NF-kappa B - metabolism | Fibrosis - metabolism | RNA, Messenger - metabolism | Myocardium - metabolism | Blood Pressure - drug effects | Blood Pressure - physiology | p38 Mitogen-Activated Protein Kinases - metabolism | Heart - physiopathology | Angiotensin II - pharmacology | Collagen Type I - metabolism | Ventricular Remodeling - physiology | Hypertension - physiopathology | Hypertension - metabolism | Animals | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Receptor, Angiotensin, Type 1 - metabolism | Heart - drug effects | Signal Transduction - physiology | Mice | Fibrosis - pathology | Oxidative Stress - drug effects | Transforming Growth Factor beta - metabolism | Ventricular Remodeling - drug effects | Connective Tissue Growth Factor - metabolism | Cardiomegaly - metabolism | Oxidases | Hypertension | Heart | Oxidative stress | RNA | Heart enlargement | Transforming growth factors | Necrosis | Tumor necrosis factor | Collagen | Angiotensin | Bone morphogenetic proteins | Tumors | Veterinary colleges | Reactive oxygen species | Collagen (type I) | Crosstalk | Body weight | Collagens | mRNA | Kinases | NAD(P)H oxidase | Infusion | Signal transduction | Etanercept | Tumor necrosis factor-TNF | Blood pressure | Angiotensin II | Heart diseases | NF-κB protein | Collagen (type III) | MAP kinase | Connective tissue growth factor | JNK protein | Tumor necrosis factor-α | Gene expression | Signaling | Nitric oxide | Fibrosis | Ventricle | Hypertrophy
Journal Article
Matrix Biology, ISSN 0945-053X, 01/2017, Volume 57-58, pp. 29 - 44
Journal Article
Nan fang yi ke da xue xue bao = Journal of Southern Medical University, ISSN 1673-4254, 07/2006, Volume 26, Issue 7, p. 981
To investigate collagen type I alpha 1 (COL1A1) and alpha 2 (COL1A2) gene polymorphisms in Chinese and their relationship with bone mineral density. Totalling... 
Bone Density | Lumbar Vertebrae - metabolism | Gene Frequency | Humans | Middle Aged | Genotype | Male | Polymorphism, Genetic | Collagen Type I - genetics | Polymorphism, Restriction Fragment Length | China | Adult | Female | Aged | Collagen - genetics
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e22447
Background: Although enzyme replacement therapy (ERT) is available for several lysosomal storage disorders, the benefit of this treatment to the skeletal... 
MECHANISM | GENE | MULTIDISCIPLINARY SCIENCES | ARYLSULFATASE-B | JOINT DISEASE | BONE | IDENTIFICATION | Bone and Bones - pathology | Chondrocytes - pathology | Enzyme Replacement Therapy | Anti-Inflammatory Agents - pharmacology | Humans | Mucopolysaccharidosis VI - pathology | Motor Activity - drug effects | Rats | Trachea - drug effects | Bone and Bones - drug effects | Mucopolysaccharidosis VI - therapy | Trachea - pathology | Cartilage, Articular - pathology | Chondrocytes - drug effects | Gait - drug effects | Animals | Anti-Inflammatory Agents - therapeutic use | Cartilage, Articular - drug effects | Mucopolysaccharidosis VI - physiopathology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Trachea - abnormalities | Enzymes | Medical research | Tumor necrosis factor | Collagen | Medicine, Experimental | Mucopolysaccharidosis | Gene therapy | Health aspects | Biopharmaceutics | Therapy | Animal models | Motor activity | Disorders | N-acetyl-galactosamine | Clinical trials | Lysosomal storage diseases | Systematic review | Cartilage | Angiogenesis | Bone growth | Computed tomography | Rodents | Computer architecture | Toll-like receptors | Ceramide | Bones | TRANCE protein | Trachea | Recombinant | Cytokines | Poly(ADP-ribose) polymerase | Cystic fibrosis | Inflammation | Cartilage (articular) | TLR4 protein | Tumor necrosis factor-α | Crohns disease | Medicine | Serum levels | Rheumatoid arthritis | Chondrocytes | Biomarkers | Skeletal system | Combined treatment | Apoptosis
Journal Article