X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (4617) 4617
Publication (576) 576
Book Chapter (83) 83
Book Review (83) 83
Book / eBook (25) 25
Conference Proceeding (15) 15
Dissertation (9) 9
Web Resource (2) 2
Magazine Article (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (3471) 3471
index medicus (1712) 1712
immunology (1701) 1701
animals (1527) 1527
complement (1513) 1513
female (965) 965
male (932) 932
complement activation (909) 909
adult (619) 619
proteins (614) 614
biochemistry & molecular biology (605) 605
activation (536) 536
middle aged (500) 500
mice (473) 473
inflammation (441) 441
complement system proteins - immunology (420) 420
analysis (399) 399
protein binding (370) 370
article (325) 325
complement system proteins - metabolism (317) 317
molecular sequence data (315) 315
complement c4 - genetics (309) 309
amino acid sequence (307) 307
aged (304) 304
research (301) 301
innate immunity (293) 293
complement system (286) 286
factor-h (279) 279
alternative pathway (275) 275
research article (275) 275
genes (263) 263
disease (260) 260
complement activation - immunology (256) 256
expression (252) 252
cell biology (251) 251
alleles (244) 244
genetic aspects (240) 240
transplantation (240) 240
microbiology (237) 237
decay-accelerating factor (234) 234
binding (231) 231
health aspects (230) 230
immune system (225) 225
antibodies (220) 220
major histocompatibility complex (219) 219
base sequence (217) 217
adolescent (215) 215
risk factors (213) 213
system (211) 211
c4b-binding protein (210) 210
physiological aspects (210) 210
membrane cofactor protein (209) 209
medicine (203) 203
complement c4b - metabolism (201) 201
systemic-lupus-erythematosus (200) 200
lectins (197) 197
complement c4b (195) 195
complement c4 - metabolism (191) 191
complement system proteins - genetics (191) 191
multidisciplinary sciences (190) 190
child (188) 188
pathology (188) 188
systemic lupus erythematosus (188) 188
c-reactive protein (187) 187
classical pathway (187) 187
medicin och hälsovetenskap (186) 186
polymorphism, genetic (186) 186
medical and health sciences (185) 185
enzyme-linked immunosorbent assay (184) 184
hemolytic-uremic syndrome (184) 184
complement c3 - metabolism (183) 183
medicine, research & experimental (183) 183
mannose-binding lectin (181) 181
protein (181) 181
membrane attack complex (178) 178
genetics & heredity (177) 177
surgery (177) 177
binding sites (172) 172
haplotypes (172) 172
allergy and immunology (169) 169
immunity, innate (169) 169
complement c3b - metabolism (168) 168
complement system proteins - physiology (168) 168
immune response (168) 168
gene expression (167) 167
hematology (162) 162
infectious diseases (162) 162
mutation (162) 162
pathogenesis (162) 162
rheumatology (162) 162
lupus erythematosus, systemic - immunology (160) 160
deficiency (159) 159
autoimmunity (153) 153
cells (153) 153
pathway (153) 153
graft rejection - immunology (150) 150
identification (148) 148
macular degeneration (148) 148
complement factor h (147) 147
immunity (147) 147
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (4647) 4647
French (16) 16
Russian (15) 15
German (4) 4
Japanese (2) 2
Portuguese (2) 2
Spanish (2) 2
Hungarian (1) 1
Polish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23991
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, p. e27546
Resistance to complement mediated killing, or serum resistance, is a common trait of pathogenic bacteria. Rck is a 17 kDa outer membrane protein encoded on the... 
C4BP | SERUM RESISTANCE | YERSINIA-ENTEROCOLITICA | MECHANISM | ESCHERICHIA-COLI | BIOLOGY | GENE RCK | IDENTIFICATION | COB-BINDING PROTEIN | TYPHIMURIUM VIRULENCE PLASMID | ENTERICA | Virulence - drug effects | Bacterial Outer Membrane Proteins - immunology | Complement Inactivator Proteins - metabolism | Sodium Chloride - pharmacology | Humans | Complement C4b-Binding Protein - metabolism | Salmonella Infections - immunology | Escherichia coli - immunology | Complement Inactivator Proteins - immunology | Salmonella Infections - metabolism | Salmonella - immunology | Flow Cytometry | Bacterial Outer Membrane Proteins - metabolism | Complement C4b - metabolism | Complement C4b - antagonists & inhibitors | Escherichia coli - metabolism | Protein Binding | Adult | Complement C4b-Binding Protein - immunology | Complement Activation - drug effects | Heparin - pharmacology | Salmonella | Drug resistance in microorganisms | Lectins | Bacteria | Virulence (Microbiology) | Cells | Protein binding | Flow cytometry | Salts | Laboratories | Virulence | Complement | Biology | Assaying | Alternative pathway | Mammalian cells | Lipopolysaccharides | Recruitment | Streptococcus infections | Proteins | Complement factor H | Immunology | E coli | Coding | Killing | Pathogens | C4b-binding protein | Chromatography | Bacteriology | Mutants | Membrane proteins | Cytometry | Inhibitors | Plasmids | Heparin | Complement component C4b-binding protein | Ionic interactions | Basic Medicine | Other Basic Medicine | Andra medicinska och farmaceutiska grundvetenskaper | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2001, Volume 31, Issue 9, pp. 2771 - 2780
Human complement regulators are important targets for pathogenic microorganisms. In one such interaction, Bordetella pertussis binds human C4b‐binding protein... 
C4b‐binding protein | Bacteria | Binding site | Complement | Bordetella pertussis | C4b-binding protein | complement component C4 | complement component C4b-binding protein
Journal Article
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2017, Volume 198, Issue 6, pp. 2330 - 2340
Respiratory tract infections are one of the leading causes of mortality worldwide urging better understanding of interactions between pathogens causing these... 
C4B-BINDING PROTEIN | OBSTRUCTIVE PULMONARY-DISEASE | STREPTOCOCCUS-PNEUMONIAE | EPITHELIAL-CELLS | PSEUDOMONAS-AERUGINOSA | RICH REPEAT PROTEIN | EXTRACELLULAR-MATRIX | HEPARAN-SULFATE PROTEOGLYCANS | IMMUNOLOGY | NF-KAPPA-B | COMPLEMENT INHIBITOR | Cell Line | Complement Inactivating Agents - metabolism | Antibody-Dependent Cell Cytotoxicity | Myofibroblasts - immunology | Humans | Moraxella (Branhamella) catarrhalis - immunology | Glycoproteins - metabolism | Complement Inactivating Agents - antagonists & inhibitors | Immunity, Innate | Bacterial Adhesion | Host-Pathogen Interactions | Respiratory Mucosa - pathology | Immune Evasion | Respiratory Mucosa - immunology | Respiratory Tract Infections - immunology | Moraxellaceae Infections - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Phagocytosis | Complex formation | Epithelial cells | Clinical isolates | Proline | Innate immunity | Complement | Infections | Leucine | Macrophages | Immunity | Streptococcus infections | Proteins | Respiratory tract | Phagocytes | Arginine | Bronchoalveolar lavage | Respiratory tract diseases | Extracellular matrix | Bacteria | Membrane attack complex | Alveoli | Killing | Pathogens | Opsonization | Neutrophils | C4b-binding protein | Blood vessels | Bronchus | Leukocytes (neutrophilic) | Monocytes | Lungs | Resists | Sensitivity enhancement | Complement component C4b-binding protein | Basic Medicine | Immunologi inom det medicinska området | Medical and Health Sciences | Medicin och hälsovetenskap | Immunology in the medical area | Medicinska och farmaceutiska grundvetenskaper
Journal Article
PLoS One, ISSN 1932-6203, 2008, Volume 3, Issue 8, pp. e2886 - e2886
Background: During myocardial infarction reduced blood flow in the heart muscle results in cell death. These dying/dead cells have been reported to bind... 
BIOLOGY | Immunohistochemistry | Inflammation - pathology | Acute Disease | Heart Diseases - metabolism | Complement C3 - metabolism | Humans | Complement C4b-Binding Protein - metabolism | Cardiomegaly - pathology | Myocardium - pathology | Myocardial Infarction - metabolism | Inflammation - metabolism | Aortic Valve Stenosis - pathology | Flow Cytometry | Myocardial Infarction - pathology | Myocardium - metabolism | Aortic Valve Stenosis - metabolism | Myocardial Infarction - physiopathology | Cadaver | Cardiomegaly - metabolism | Medical research | C-reactive protein | Cell death | Analysis | Medicine, Experimental | Research | Cardiovascular diseases | Blood proteins | Protein binding | Myocardial infarction | Immunoglobulin M | Heart | Complement component C3b | Heart attacks | Laboratories | Complement | Proteins | Windows (intervals) | Plasma proteins | Rodents | Heart diseases | Immune system | Heart failure | Cardiac muscle | Mortality | Neutrophils | C4b-binding protein | Cardiomyocytes | Leukocytes (neutrophilic) | Inflammation | Coronary artery disease | Blood flow | Complement activation | Inhibitors | Complement component C3 | Coronary vessels | Infiltration | Infarction | Leukocytes (granulocytic) | Complement component C4b-binding protein | Binding sites | Apoptosis | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Journal of Proteomics, ISSN 1874-3919, 12/2013, Volume 94, pp. 337 - 345
Mitral regurgitation (MR) is a common valvular lesion frequently caused by mitral valve prolapse (MVP). Surgical intervention in MVP patients with significant... 
Mitral regurgitation | Biomarkers | Platelet basic protein | Mitral valve prolapse | Complement component C4b | Haptoglobin | COMPLEMENT | ACTIVATION | PLATELET-FUNCTION | BIOCHEMICAL RESEARCH METHODS | PLASMA PROTEOME | MECHANISMS | IDENTIFICATION | DISEASE | COLORECTAL-CANCER CELLS | ECHOCARDIOGRAPHY | EXPRESSION | Proteins | Mitral valve insufficiency | Biological markers | Proteomics
Journal Article