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humans (17) 17
oncology (11) 11
dcc receptor (9) 9
male (8) 8
tumor suppressor proteins - genetics (7) 7
immunohistochemistry (6) 6
tumor suppressor proteins - metabolism (6) 6
colorectal cancer (5) 5
female (5) 5
frequent loss (5) 5
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mutations (5) 5
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receptors, cell surface (5) 5
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aged (4) 4
animals (4) 4
cancer (4) 4
cell adhesion molecules - genetics (4) 4
dcc (4) 4
dcc protein (4) 4
deleted in colorectal cancer (4) 4
deleted in colorectal cancer gene (4) 4
expression (4) 4
genetic aspects (4) 4
loss of heterozygosity (4) 4
netrin-1 (4) 4
prognosis (4) 4
surgery (4) 4
adhesion (3) 3
allelic loss (3) 3
cell adhesion molecules - metabolism (3) 3
cell biology (3) 3
colon (3) 3
colon cancer (3) 3
colonic neoplasms - genetics (3) 3
colonic neoplasms - metabolism (3) 3
colorectal neoplasms - genetics (3) 3
colorectal-cancer (3) 3
cytoskeletal proteins - metabolism (3) 3
deleted in colorectal carcinoma gene (3) 3
development and progression (3) 3
gastroenterology & hepatology (3) 3
genes, dcc - genetics (3) 3
genes, tumor suppressor (3) 3
index medicus (3) 3
medicine & public health (3) 3
messenger-rna (3) 3
mice (3) 3
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pathology (3) 3
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sensitivity and specificity (3) 3
survival analysis (3) 3
tumor cells, cultured (3) 3
tumor progression (3) 3
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adenocarcinoma (2) 2
adolescent (2) 2
adult (2) 2
akt, protein kinase b (2) 2
alleles (2) 2
antibodies, monoclonal - immunology (2) 2
apoptosis (2) 2
axon guidance (2) 2
beta catenin (2) 2
biomarkers, tumor - metabolism (2) 2
blotting, southern - standards (2) 2
blotting, western (2) 2
cadherins - metabolism (2) 2
cancers (2) 2
carcinoma (2) 2
cell differentiation - genetics (2) 2
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chromosome deletion (2) 2
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colon adenoma (2) 2
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Journal Article
British Journal of Cancer, ISSN 0007-0920, 02/1998, Volume 77, Issue 3, pp. 466 - 471
Using a bacterial fusion protein, a deleted colorectal carcinoma (DCC)-specific monoclonal antibody (MAb) 127-22 was established. Although MAb 127-22 reacted... 
Colon cancer | Deleted colorectal carcinoma | Metastasis | Tumour-suppressor gene | colon cancer | TUMOR-SUPPRESSOR GENES | tumour-suppressor gene | HETEROZYGOSITY | P53 | deleted colorectal carcinoma | MESSENGER-RNA | ONCOLOGY | metastasis | MUTATIONS | TISSUES | FREQUENT LOSS | DELETIONS | DECREASED EXPRESSION | PROGRESSION
Journal Article
Cancer, ISSN 0008-543X, 01/1999, Volume 85, Issue 2, pp. 453 - 464
BACKGROUND Although frequent loss of the tumor suppressor gene deleted in colon carcinoma (DCC) has been demonstrated in endometrial carcinoma, an alteration... 
gene deleted in colon carcinoma (DCC) | endometrial carcinoma | endometrial hyperplasia | estrogen receptor | progesterone receptor | Endometrium | Estrogen receptor | Endometrial hyperplasia | Gene deleted in colon carcinoma (DCC) | Progesterone receptor | Endometrial carcinoma
Journal Article
Journal Article
Journal of Histochemistry & Cytochemistry, ISSN 0022-1554, 5/2019, Volume 67, Issue 5, pp. 335 - 349
Netrin-1 is a potent axonal and neuronal guidance cue in the developing nervous system. Netrin-1 functions are mediated by its receptors, such as deleted in... 
choline acetyltransferase (ChAT) | deleted in colorectal cancer (DCC) | enteric nervous system (ENS) | Netrin-1 | neuronal nitric oxide synthase (nNOS) | LOCALIZATION | PERIPHERAL-NERVE REGENERATION | AXON GUIDANCE | RECEPTOR | GLIAL-CELLS | CELL BIOLOGY | GUINEA | PROTEINS | EXPRESSION | PLEXUS
Journal Article
European Journal of Cell Biology, ISSN 0171-9335, 2006, Volume 85, Issue 8, pp. 769 - 783
The deleted in colorectal cancer (DCC) gene encodes a 170- to 190-kDa protein of the Immunoglobulin superfamily. Firstly identified as a tumor suppressor gene... 
tumor suppressor | Deleted in colorectal cancer | ERM proteins | Cell migration | Cell adhesion | PROTEIN-KINASE-C | NETRIN RECEPTOR | TYROSINE PHOSPHORYLATION | CELL BIOLOGY | IN-VITRO | NF2 TUMOR-SUPPRESSOR | EPITHELIAL-CELLS | CORTICAL ACTIN ORGANIZATION | cell adhesion | COLORECTAL-CANCER | deleted in colorectal cancer | cell migration | GENE-PRODUCT | Immunohistochemistry | Colonic Neoplasms - genetics | Immunoprecipitation | Desmosomes - ultrastructure | Humans | Actins - metabolism | Recombinant Fusion Proteins - physiology | Extracellular Matrix - metabolism | Molecular Sequence Data | Recombinant Fusion Proteins - metabolism | Colonic Neoplasms - metabolism | Transfection - methods | Tumor Suppressor Proteins - genetics | Cytoskeletal Proteins - metabolism | Microfilament Proteins - metabolism | Receptors, Cell Surface - physiology | Neurofibromin 2 - metabolism | Recombinant Fusion Proteins - biosynthesis | Amino Acid Sequence | Desmosomes - metabolism | DCC Receptor | Tumor Suppressor Proteins - metabolism | Receptors, Cell Surface - metabolism | Cytoskeleton - ultrastructure | Microscopy, Electron | Blotting, Western | HT29 Cells | Tumor Suppressor Proteins - physiology | Cell Adhesion - physiology | Colonic Neoplasms - pathology | Cytoskeleton - metabolism | Protein Binding | Models, Genetic | Receptors, Cell Surface - genetics | Immunoglobulins | Colon cancer | Actin | Phospholipases | Muscle proteins | Protein kinases | Incendiary weapons | Tumors
Journal Article
Digestive Diseases and Sciences, ISSN 0163-2116, 12/2006, Volume 51, Issue 12, pp. 2213 - 2219
Since significant neoplasia after initial colonoscopy is low, we conducted this pilot study to compare the predictive role for colorectal neoplasia recurrence... 
Biochemistry, general | Medicine & Public Health | Colonic effluent | Hepatology | Gastroenterology | Monoclonal antibodies | Oncology | Deleted in colon cancer | Adnab-9 | Transplant Surgery | DCC PROTEIN | POPULATION | MARKER | colonic effluent | RISK | COLON-CANCER | EFFLUENT | MONOCLONAL-ANTIBODY ADNAB-9 | NETRIN-1 | SURVEILLANCE | deleted in colon cancer | GASTROENTEROLOGY & HEPATOLOGY | COLONOSCOPIC POLYPECTOMY | monoclonal antibodies | Predictive Value of Tests | Adenoma - genetics | Colorectal Neoplasms - genetics | Humans | Middle Aged | Male | Antigens, Tumor-Associated, Carbohydrate - immunology | Colorectal Neoplasms - diagnosis | Adenoma - metabolism | Neoplasm Recurrence, Local - immunology | Colonoscopy | Sensitivity and Specificity | Tumor Suppressor Proteins - genetics | Biomarkers, Tumor - metabolism | Retrospective Studies | Antibodies, Monoclonal - immunology | Colorectal Neoplasms - metabolism | Adenoma - diagnosis | DCC Receptor | Tumor Suppressor Proteins - metabolism | Enzyme-Linked Immunosorbent Assay | Neoplasm Recurrence, Local - metabolism | Receptors, Cell Surface - metabolism | Neoplasm Recurrence, Local - diagnosis | Receptors, Cell Surface - immunology | Tumor Suppressor Proteins - immunology | Disease Progression | Antigens, Tumor-Associated, Carbohydrate - metabolism | Pilot Projects | Antigens, Tumor-Associated, Carbohydrate - genetics | Aged | Biomarkers, Tumor - genetics | Antibodies, Monoclonal - metabolism | Biomarkers, Tumor - immunology | Receptors, Cell Surface - genetics
Journal Article
Diseases of the Colon & Rectum, ISSN 0012-3706, 4/2001, Volume 44, Issue 4, pp. 549 - 557
Journal Article
Journal of Biomedical Science, ISSN 1021-7770, 9/2002, Volume 9, Issue 6, pp. 716 - 720
We examined the expression of the putative tumor suppressor gene deleted in colorectal carcinoma (DCC) in human colon adenoma tissues and cell lines. One... 
Colon adenoma | Biomedicine | Deleted in colorectal carcinoma gene | Biomedicine general | Tumor progression
Journal Article
Journal of Clinical Pathology, ISSN 0021-9746, 2001, Volume 54, Issue 9, pp. 684 - 688
Background-Coeliac sprue is a chronic disease, in which there is a characteristic mucosal lesion of the small intestine and impaired nutrient absorption, which... 
β-catenin | Coeliac disease | E-cadherin | Deleted in colon carcinoma gene | P53 | beta-catenin | DCC PROTEIN | ADENOCARCINOMA | SMALL-INTESTINE | CELL-PROLIFERATION | SMALL-BOWEL | COLON | PATHOLOGY | deleted in colon carcinoma gene | p53 | CELIAC-DISEASE | coeliac disease | REDUCED EXPRESSION | Immunohistochemistry | Celiac disease | Pathology | Duodenum | Physiological aspects | Research | Gene expression
Journal Article
Diseases of the Colon & Rectum, ISSN 0012-3706, 2/2003, Volume 46, Issue 2, pp. 192 - 202
PURPOSE: Adjuvant therapy, either preoperatively or postoperatively, and modifications of surgery have been used to try to improve outcome of surgery for... 
Medicine | Genetic markers | Rectal cancer | Deleted in colorectal cancer gene | Thymidylate synthase | Adjuvant therapy | p53 | DCC PROTEIN | SURGERY | rectal cancer | PATIENT SELECTION | adjuvant therapy | thymidylate synthase | RESIDUAL DISEASE | CURATIVE SURGERY | deleted in colorectal cancer gene | RADIATION-THERAPY | RECTOSIGMOID CARCINOMA | CHEMORADIATION | PROGNOSTIC-SIGNIFICANCE | MUTATIONS | RADIOTHERAPY | genetic markers | GASTROENTEROLOGY & HEPATOLOGY | Cell Adhesion Molecules - genetics | Adenocarcinoma - pathology | Prognosis | Humans | Middle Aged | Male | Receptors, Cell Surface | Immunoenzyme Techniques | Tumor Suppressor Protein p53 - genetics | Polymorphism, Single-Stranded Conformational | Adenocarcinoma - metabolism | Tumor Suppressor Proteins - genetics | Polymerase Chain Reaction | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Rectal Neoplasms - pathology | Genes, DCC - genetics | Genes, Tumor Suppressor | DCC Receptor | Tumor Suppressor Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Thymidylate Synthase - metabolism | Survival Rate | Combined Modality Therapy | Genes, p53 - genetics | Rectal Neoplasms - therapy | Cell Adhesion Molecules - metabolism | Thymidylate Synthase - genetics | Rectal Neoplasms - mortality | Adenocarcinoma - therapy | Aged | Rectal Neoplasms - metabolism | Neoplasm Staging | Adenocarcinoma - mortality
Journal Article
Journal Article