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Journal Article
Nucleic Acids Research, ISSN 0305-1048, 09/2013, Volume 41, Issue 16, pp. 7589 - 7605
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 01/2014, Volume 53, Issue 1, pp. 7 - 18
Journal Article
Molecular Cell, ISSN 1097-2765, 08/2018, Volume 71, Issue 4, pp. 621 - 628.e4
FANCA is a component of the Fanconi anemia (FA) core complex that activates DNA interstrand crosslink repair by monoubiquitination of FANCD2. Here, we report... 
FANCA | strand exchange | DNA double-strand break repair | Fanconi anemia | single-strand annealing | UBIQUITINATION | COMPLEX | RESECTION | REPLICATION PROTEIN-A | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISTINCT | HUMAN RAD52 PROTEIN | ANEMIA PATHWAY | LIGASE RFWD3 | HOMOLOGOUS RECOMBINATION | ACCUMULATION | CELL BIOLOGY | Epithelial Cells - metabolism | Baculoviridae - metabolism | Humans | Baculoviridae - genetics | Moths | DNA Breaks, Double-Stranded | Fanconi Anemia Complementation Group A Protein - genetics | Fanconi Anemia Complementation Group A Protein - metabolism | Fanconi Anemia Complementation Group Proteins - metabolism | Fanconi Anemia Complementation Group G Protein - genetics | Rad52 DNA Repair and Recombination Protein - metabolism | DNA End-Joining Repair | DNA Mismatch Repair | Cloning, Molecular | Osteoblasts - cytology | Epithelial Cells - cytology | Recombinant Proteins - metabolism | Gene Expression | Recombinational DNA Repair | Genetic Vectors - chemistry | Fanconi Anemia Complementation Group Proteins - genetics | Genetic Vectors - metabolism | Fanconi Anemia Complementation Group G Protein - metabolism | Recombinant Proteins - genetics | DNA - metabolism | Rad52 DNA Repair and Recombination Protein - genetics | DNA - genetics | Animals | Cell Line, Tumor | Osteoblasts - metabolism | Yuan (China) | Medical colleges | Annealing | DNA | Genetic research | Molecular biology | Fanconi's anemia | DNA double strand break repair
Journal Article
Cell, ISSN 0092-8674, 03/2013, Volume 152, Issue 6, pp. 1344 - 1354
Journal Article
The EMBO Journal, ISSN 0261-4189, 11/2009, Volume 28, Issue 21, pp. 3413 - 3427
Homologous recombination (HR) and non‐homologous end joining (NHEJ) represent distinct pathways for repairing DNA double‐strand breaks (DSBs). Previous work... 
ataxia telangiectasia | Artemis | homologous recombination | double‐strand breaks | non‐homologous end joining | Double-strand breaks | Ataxia telangiectasia | Homologous recombination | Non-homologous end joining | BIOCHEMISTRY & MOLECULAR BIOLOGY | double-strand breaks | MAMMALIAN-CELLS | CELL BIOLOGY | REPLICATION FORKS | IN-VIVO | non-homologous end joining | GENE CONVERSION | DIRECTED REPAIR | CELL-CYCLE | END RESECTION | IONIZING-RADIATION | DSB REPAIR | SISTER-CHROMATID EXCHANGES | Humans | DNA Breaks, Double-Stranded - radiation effects | DNA-Binding Proteins - metabolism | Heterochromatin - metabolism | Gene Deletion | Tumor Suppressor Proteins - genetics | DNA Repair Enzymes - metabolism | Cell Cycle Proteins - genetics | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Rad51 Recombinase - metabolism | DNA Repair - drug effects | Tumor Suppressor Proteins - metabolism | G2 Phase - radiation effects | Cell Cycle Proteins - metabolism | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Replication Protein A - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | DNA Helicases | Apoptosis Regulatory Proteins | BRCA2 Protein - metabolism | Fibroblasts - radiation effects | G1 Phase - radiation effects | Endonucleases | HeLa Cells | Proteins | Molecular biology | Genetic recombination | DNA repair | Radiation
Journal Article