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Journal Article
Nature (London), ISSN 1476-4687, 2017, Volume 547, Issue 7662, pp. 227 - 231
The regenerative capacity of the adult mammalian heart is limited, because of the reduced ability of cardiomyocytes to progress through mitosis(1). Endogenous... 
PRESSURE-OVERLOAD | GENE | ADULT HEART REGENERATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOUSE MODEL | MUSCLE | DUCHENNE MUSCULAR-DYSTROPHY | MICE | Protein-Serine-Threonine Kinases - deficiency | Phosphorylation | Cell Proliferation | Glycoproteins - metabolism | Dystroglycans - metabolism | Male | Phosphoproteins - metabolism | Heart Failure - prevention & control | Multiprotein Complexes - metabolism | Dystrophin - deficiency | Mice, Inbred mdx | Cardiomyopathies | Glycoproteins - deficiency | Multiprotein Complexes - deficiency | Dystrophin - metabolism | Protein-Serine-Threonine Kinases - metabolism | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Organ Size | Heart Failure - genetics | Pressure | Multiprotein Complexes - chemistry | Animals | Dystrophin - genetics | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Adaptor Proteins, Signal Transducing - metabolism | Heart | Genes | Homeostasis | Genomes | Kinases | Muscular dystrophy | Cell growth | Actin | Duchenne's muscular dystrophy | Cell cycle | Extracellular matrix | Heart diseases | Dystrophin | Deoxyribonucleic acid--DNA | Heart failure | Dystroglycan | Cardiomyocytes | Glycoproteins | Studies | Yes-associated protein | Regeneration | Point mutation | Cytoskeleton | Mutation | Dystrophy
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2007, Volume 357, Issue 26, pp. 2677 - 2686
Journal Article
Neuromuscular disorders : NMD, ISSN 0960-8966, 10/2017, Volume 27, p. S244
Journal Article
Human molecular genetics, ISSN 0964-6906, 12/2014, Volume 23, Issue 25, pp. 6697 - 6711
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 03/2006, Volume 26, Issue 11, pp. 2841 - 2851
The dystrobrevins (alpha DB and beta DB) bind directly to dystrophin and are components of a transmembrane dystrophin - glycoprotein complex (DGC) that links... 
GABA receptor | Retina | Behavior | ERG | Dystrophin | Muscular dystrophy | DYSTROPHIN-GLYCOPROTEIN COMPLEX | MOUSE | UTROPHIN-DEFICIENT MICE | dystrophin | DYSTROGLYCAN | NEUROSCIENCES | GABA(A) RECEPTOR SUBTYPES | MDX MICE | PROTEIN COMPLEX | SKELETAL-MUSCLE | muscular dystrophy | retina | DUCHENNE MUSCULAR-DYSTROPHY | behavior | NEUROMUSCULAR-JUNCTIONS | Muscular Dystrophy, Animal - genetics | Hippocampus - chemistry | Muscular Dystrophy, Animal - physiopathology | Multiprotein Complexes | Male | Prosencephalon - chemistry | Receptors, GABA-A - deficiency | Neuromuscular Junction - chemistry | Purkinje Cells - physiology | Behavior, Animal | Dystrophin - physiology | Dystrophin-Associated Proteins - genetics | Retina - ultrastructure | Dystrophin - deficiency | Muscle, Skeletal - chemistry | Mice, Inbred mdx | Ataxia - genetics | Dystrophin-Associated Proteins - physiology | Neuromuscular Junction - pathology | Prosencephalon - pathology | Psychomotor Performance | Synapses - physiology | Mice, Inbred C57BL | Retina - physiopathology | Dystrophin-Associated Proteins - deficiency | Genotype | Cerebellum - physiopathology | Hippocampus - pathology | Muscular Dystrophy, Animal - psychology | Purkinje Cells - chemistry | Retina - chemistry | Mice, Knockout | Animals | Dystrophin - genetics | Receptors, GABA-A - physiology | Mice | Muscle, Skeletal - pathology | Receptor Aggregation
Journal Article
Neuromuscular Disorders, ISSN 0960-8966, 2016, Volume 26, pp. S160 - S160
Journal Article