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Journal of Medicinal Chemistry, ISSN 0022-2623, 08/2012, Volume 55, Issue 16, pp. 7193 - 7207
Journal Article
Cardiovascular Diabetology, ISSN 1475-2840, 11/2013, Volume 12, Issue 1, pp. 158 - 158
Background: Endoplasmic reticulum (ER) stress is considered one of the mechanisms contributing to reactive oxygen species (ROS)-mediated cell apoptosis. In... 
Diabetic cardiomyopathy | Oxidative stress | Apoptosis | Endoplasmic reticulum stress | MITOCHONDRIAL OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | ER STRESS | PREVENTION | SUPPRESSION | RAT MODEL | MYOCARDIAL APOPTOSIS | CELL-DEATH | HEART | ENDOCRINOLOGY & METABOLISM | MICE | CONTRIBUTES | Free Radical Scavengers - pharmacology | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | eIF-2 Kinase - metabolism | eIF-2 Kinase - drug effects | Activating Transcription Factor 6 - genetics | Activating Transcription Factor 6 - drug effects | Gene Knockdown Techniques | Membrane Proteins - metabolism | Diabetes Mellitus, Experimental - metabolism | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Endoplasmic Reticulum Stress - drug effects | Membrane Proteins - genetics | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Rats | Rats, Sprague-Dawley | Activating Transcription Factor 6 - metabolism | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Acetylcysteine - pharmacology | Protein-Serine-Threonine Kinases - drug effects | Glucose - metabolism | Membrane Proteins - drug effects | Myocytes, Cardiac - metabolism | Signal Transduction - physiology | Apoptosis - physiology | Endoplasmic Reticulum Stress - physiology | Studies | Kinases | Rodents | Animal models
Journal Article
Molecular Cancer, ISSN 1476-4598, 05/2017, Volume 16, Issue 1, pp. 91 - 13
Background: Nutrient deprivation, hypoxia, radiotherapy and chemotherapy induce endoplasmic reticulum (ER) stress, which activates the so-called unfolded... 
Eukaryotic translation initiation factor-2α kinase 3/protein kinase RNA-like endoplasmic reticulum kinase | Multidrug resistance related protein 1 | Chemoresistance | Endoplasmic reticulum stress | Unfolded protein response | INDUCED APOPTOSIS | ER STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | Eukaryotic translation initiation factor-2 alpha kinase 3/protein kinase RNA-like endoplasmic reticulum kinase | DOWN-REGULATION | COLON-CANCER CELLS | NRF2 | METABOLISM | ONCOLOGY | ANTICANCER DRUGS | MULTIDRUG-RESISTANCE | EXPRESSION | Colonic Neoplasms - genetics | Endoplasmic Reticulum Stress - drug effects | Unfolded Protein Response - genetics | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | Humans | eIF-2 Kinase - antagonists & inhibitors | HT29 Cells | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Cell Death - genetics | Signal Transduction - drug effects | Colonic Neoplasms - pathology | Multidrug Resistance-Associated Proteins - genetics | NF-E2-Related Factor 2 - genetics | Mice | Cell Death - drug effects | Doxorubicin - administration & dosage | eIF-2 Kinase - genetics | Drugs | Cell culture | Transcription factors | Profiling | Transcription | Colorectal cancer | Homeostasis | Activation | Drug resistance | Drug development | Kinases | Cancer therapies | Proteins | Antioxidants | Cell growth | Pathways | Protein folding | Penicillin | Xenografts | Nutrients | Stress response | Protein transport | Adaptation | Stresses | Enzymes | Deprivation | Cell survival | Tumor cells | Mortality | Multidrug resistance | Cloning | Radiation therapy | Gene expression | Stressing | Ribonucleic acid--RNA | Survival | Stress | Membrane proteins | Chemotherapy | Protein kinase | Cell death | Hypoxia | Disruption | Endoplasmic reticulum | Transporter | Tumors | Cancer | Apoptosis
Journal Article
Circulation Research, ISSN 0009-7330, 10/2007, Volume 101, Issue 9, pp. 919 - 927
Carbon monoxide (CO), a reaction product of the cytoprotective heme oxygenase (HO)-1, is antiapoptotic in a variety of models of cellular injury, but the... 
Carbon monoxide | Heme oxygenase-1 | Apoptosis | Endoplasmic reticulum stress | SURVIVAL | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | carbon monoxide | apoptosis | DEATH | heme oxygenase-1 | PERK | INDUCTION | endoplasmic reticulum stress | ELEMENTS | NRF2 | GENES | CHOP | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Gene Expression Regulation, Enzymologic - drug effects | Transcription Factor CHOP - genetics | Heme Oxygenase-1 - metabolism | MAP Kinase Signaling System - physiology | Apoptosis - drug effects | eIF-2 Kinase - metabolism | Humans | Oxidative Stress - physiology | Gene Expression Regulation, Enzymologic - physiology | Carbon Monoxide - metabolism | DNA-Binding Proteins - metabolism | Heme Oxygenase-1 - genetics | Carbon Monoxide - pharmacology | Enzyme Induction - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Umbilical Veins - cytology | Endoplasmic Reticulum - enzymology | Cells, Cultured | Nuclear Proteins - metabolism | Regulatory Factor X Transcription Factors | Activating Transcription Factor 6 - metabolism | MAP Kinase Signaling System - drug effects | Endothelial Cells - cytology | NF-E2-Related Factor 2 - metabolism | RNA, Small Interfering | Transcription Factors | Apoptosis - physiology | Enzyme Induction - physiology | Endothelial Cells - enzymology | Endothelial Cells - drug effects
Journal Article
Journal Article
Journal Article
Journal Article
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Journal Article
Circulation, ISSN 0009-7322, 04/2014, Volume 129, Issue 13, pp. 1397 - 1406
Journal Article