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Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 22, pp. 9944 - 9949
Actomyosin contractility affects cellular organization within tissues in part through the generation of mechanical forces at sites of cell—matrix and cell—cell contact... 
Cell growth | Microinjections | Epithelial cells | Bowties | Cell adhesion | Cadherins | Adherens junctions | Focal adhesions | Physiological regulation | Endothelial cells | Mechanotransduction | Traction force | Myosin | Adherens junction | PDMS | ACTIVATION | mechanotransduction | MULTIDISCIPLINARY SCIENCES | myosin | EXTENSION | E-CADHERIN | FOCAL ADHESIONS | GASTRULATION | traction force | adherens junction | RAC | ENDOTHELIAL-CELLS | GROWTH | RHO | REQUIRE | Myosin Type II - physiology | rhoA GTP-Binding Protein - administration & dosage | Cadherins - metabolism | Mechanotransduction, Cellular - drug effects | Humans | Green Fluorescent Proteins - genetics | rhoA GTP-Binding Protein - physiology | rhoA GTP-Binding Protein - genetics | Mechanotransduction, Cellular - physiology | Antigens, CD - genetics | Antigens, CD - metabolism | Endothelial Cells - ultrastructure | Thrombin - pharmacology | Lysophospholipids - pharmacology | rac GTP-Binding Proteins - physiology | Cadherins - genetics | Endothelial Cells - physiology | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Cells, Cultured | Recombinant Proteins - genetics | rho GTP-Binding Proteins - physiology | Recombinant Proteins - administration & dosage | Sphingosine - pharmacology | Biomechanical Phenomena | Sphingosine - analogs & derivatives | Adherens Junctions - physiology | Adherens Junctions - drug effects | Adherens Junctions - ultrastructure | Amino Acid Substitution | Endothelial Cells - drug effects | Junctional complexes (Epithelium) | Cell junctions | Properties | Biological Sciences | Physical Sciences
Journal Article
Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 8/2011, Volume 68, Issue 2, pp. 445 - 455
The natural flavonoid fisetin was recently identified as a lead compound that stabilizes endothelial cell microtubules... 
Fisetin | Lewis lung carcinoma | Angiogenesis | Antitumour activity | Cyclophosphamide | Medicine & Public Health | Cancer Research | Oncology | Cytotoxicity | EA·hy 926 endothelial cells | Pharmacology/Toxicology | Flavonoid | EA•hy 926 endothelial cells | APOPTOSIS | ANTIINFLAMMATORY ACTIVITY | CELL-CYCLE ARREST | PROLIFERATION | FLUOROURACIL | CANCER | IN-VITRO | ONCOLOGY | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | EA.hy 926 endothelial cells | INHIBITORS | NIH 3T3 Cells | Cyclophosphamide - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Tubulin Modulators - pharmacology | Humans | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Antineoplastic Agents, Alkylating - pharmacology | Flavonoids - adverse effects | Antineoplastic Agents, Alkylating - administration & dosage | Cyclophosphamide - adverse effects | Cyclophosphamide - therapeutic use | Flavonoids - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Angiogenesis Inhibitors - administration & dosage | Antineoplastic Agents, Phytogenic - administration & dosage | Angiogenesis Inhibitors - therapeutic use | Flavonoids - administration & dosage | Tubulin Modulators - administration & dosage | Female | Flavonoids - pharmacology | Antineoplastic Agents, Phytogenic - therapeutic use | Angiogenesis Inhibitors - adverse effects | Antineoplastic Agents, Phytogenic - adverse effects | Cell Line | Cell Survival - drug effects | Tubulin Modulators - adverse effects | Mice, Inbred C57BL | Angiogenesis Inhibitors - pharmacology | Tubulin Modulators - therapeutic use | Carcinoma, Lewis Lung - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Cell Movement - drug effects | Animals | Tumor Burden - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Cyclophosphamide - pharmacology | Carcinoma, Lewis Lung - pathology | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Alkylating - adverse effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | Endothelial Cells - drug effects | Antimitotic agents | Flavonoids | Flavones | Lung cancer | Bioflavonoids | Accountants | Drug therapy, Combination | Universities and colleges | Antineoplastic agents | Endothelium | Tumors | Index Medicus | cytology | Antineoplastic Agents, Phytogenic | pathology | Cell Proliferation | Endothelial Cells | Tubulin Modulators | Neovascularization, Pathologic | fisetin | administration & dosage | pharmacology | flavonoid | Carcinoma, Lewis Lung | Antineoplastic Agents, Alkylating | cytotoxicity | drug therapy | Cell Survival | angiogenesis | Antineoplastic Combined Chemotherapy Protocols | drug effects | Tumor Burden | Angiogenesis Inhibitors | EA.hy 926 | Cell Cycle | antitumour activity | adverse effects | therapeutic use | Cell Movement
Journal Article
Circulation research, ISSN 0009-7330, 06/2013, Volume 113, Issue 1, pp. 40 - 51
.... However, its role in the regulation of smooth muscle cell (SMC) function is unknown. OBJECTIVE:To elucidate the role of miR-126 secreted by endothelial cells (ECs... 
Smooth muscle cell | Shear stress | Endothelial cell | Extracellular miR-126 | Atherosclerosis | atherosclerosis | CIRCULATING MICRORNAS | APOPTOSIS | extracellular miR-126 | CARDIAC & CARDIOVASCULAR SYSTEMS | smooth muscle cell | endothelial cell | NEOINTIMAL HYPERPLASIA | PROLIFERATION | MECHANISMS | shear stress | MIR-126 | PERIPHERAL VASCULAR DISEASE | CARDIOVASCULAR-DISEASES | HEMATOLOGY | INSULIN-RECEPTOR SUBSTRATE-1 | EXPRESSION | Human Umbilical Vein Endothelial Cells | MicroRNAs - therapeutic use | Coculture Techniques | Humans | Recombinant Fusion Proteins - physiology | Culture Media, Conditioned - pharmacology | Hemorheology | Ligation | Neointima | Insulin Receptor Substrate Proteins - biosynthesis | Argonaute Proteins - physiology | Insulin Receptor Substrate Proteins - genetics | Myocytes, Smooth Muscle - cytology | Cell Line | Forkhead Transcription Factors - biosynthesis | Argonaute Proteins - genetics | Cell Culture Techniques - instrumentation | Paracrine Communication | Endothelial Cells - metabolism | Gene Expression Regulation | Gene Silencing | Genes, bcl-2 | Forkhead Transcription Factors - genetics | Muscle, Smooth, Vascular - cytology | Proto-Oncogene Proteins c-bcl-2 - biosynthesis | Animals | Umbilical Arteries - cytology | Carotid Artery, Common - pathology | Mice | MicroRNAs - genetics | MicroRNAs - physiology | Forkhead Box Protein O3
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 4, p. e35685
We have previously shown that mesenchymal stem cells (MSC) improve function upon integration in ischemic myocardium... 
MIGRATION | SURVIVAL | BAD | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | H9C2 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INHIBIT APOPTOSIS | CHEMOKINE RECEPTORS | STROMAL CELLS | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Apoptosis - drug effects | Caspase 3 - metabolism | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Mesenchymal Stromal Cells - cytology | Caspase 3 - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - drug effects | Endothelial Cells - metabolism | Cytokines - secretion | Mesenchymal Stromal Cells - metabolism | Chemotaxis - drug effects | Mesenchymal Stromal Cells - secretion | Phosphatidylinositol 3-Kinases - genetics | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Dogs | Mice | Primary Cell Culture | Cytokines - biosynthesis | Cytokines - pharmacology | Endothelial Cells - drug effects | Cytokines | Comparative analysis | Vascular endothelial growth factor | Stem cells | Apoptosis | Heart | Phosphorylation | Leukocyte migration | Mesenchyme | Intracellular signalling | AKT protein | Kinases | Macrophages | Caspase-3 | Proteins | Angiogenesis | Signal transduction | Mitochondria | Cell growth | Ischemia | Rodents | Cell cycle | Tumor necrosis factor-TNF | Conditioning | Localization | Cardiology | Growth factors | Extracellular signal-regulated kinase | Caspase | Cardiomyocytes | Inflammation | Ribonucleic acid--RNA | Chemotaxis | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Inhibitors | γ-Interferon | Myocardium | Hypoxia | Interferon | Laboratory animals | Cell migration | Monocyte chemoattractant protein 1 | RNA | Ribonucleic acid
Journal Article
PloS one, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e46842
Journal Article
Cell stem cell, ISSN 1934-5909, 2017, Volume 20, Issue 4, pp. 490 - 504.e5
.... Here, we used comparison of induced pluripotent stem cell-derived endothelial cells (iPSC-ECs) from three families with unaffected mutation carriers... 
cell survival | bone morphogenetic protein receptor 2 | unaffected mutation carrier | cell adhesion | penetrance | cell signaling | transcriptomic analysis | pulmonary arterial hypertension | endothelial dysfunction | induced pluripotent stem cell-derived endothelial cell | MIGRATION | PATHOGENESIS | ANGIOGENESIS | GENE | INTEGRIN | ARTERIAL-HYPERTENSION | MOUSE | RECEPTOR | DYSFUNCTION | EXPRESSION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Humans | Hypertension, Pulmonary - prevention & control | Base Sequence | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Cell Survival - drug effects | Bone Morphogenetic Protein Receptors, Type II - genetics | Induced Pluripotent Stem Cells - drug effects | Endothelial Cells - metabolism | Neovascularization, Physiologic - genetics | Hypertension, Pulmonary - genetics | Cell Adhesion - drug effects | Mutation - genetics | Bone Morphogenetic Protein 4 - pharmacology | Gene Editing | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Sequence Analysis, RNA | Signal Transduction - drug effects | Endothelial Cells - cytology | Heterozygote | Hypertension, Pulmonary - pathology | Smad Proteins - metabolism | Endothelial Cells - drug effects | Pulmonary arterial hypertension | induced pluripotent stem cell derived endothelial cell
Journal Article
Journal Article