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Science, ISSN 0036-8075, 11/2001, Volume 294, Issue 5548, pp. 1945 - 1948
Neurotrophins are growth factors that promote cell survival, differentiation, and cell death. They are synthesized as proforms that can be cleaved... 
Hair | Receptors | Odors | HEK293 cells | Neurons | Antibodies | Ligands | Reports | PC12 cells | Endothelial cells | Apoptosis | APOPTOSIS | COEXPRESSION | NEUROTROPHIC FACTOR | BIOSYNTHESIS | MULTIDISCIPLINARY SCIENCES | RECEPTOR | DEATH | EXPRESSION | BINDING | SMOOTH-MUSCLE CELLS | NERVE GROWTH-FACTOR | Nerve Growth Factor - secretion | Brain-Derived Neurotrophic Factor - secretion | Apoptosis - drug effects | Protein Precursors - chemistry | Humans | Protein Precursors - secretion | Neurons - cytology | Nerve Growth Factors - metabolism | Receptors, Nerve Growth Factor - metabolism | Nerve Growth Factors - pharmacology | Brain-Derived Neurotrophic Factor - pharmacology | Nerve Growth Factor - chemistry | Inhibitory Concentration 50 | Furin | Brain-Derived Neurotrophic Factor - metabolism | Subtilisins - metabolism | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Fibrinolysin - metabolism | Cell Line | Cell Survival - drug effects | Nerve Growth Factor - metabolism | Nerve Growth Factors - secretion | Nerve Growth Factor - pharmacology | Rats | Nerve Growth Factors - chemistry | Receptor, Nerve Growth Factor | Protein Precursors - metabolism | Protein Precursors - pharmacology | Brain-Derived Neurotrophic Factor - chemistry | Animals | Receptor, trkA - metabolism | Cell Differentiation - drug effects | Mice | Protein Processing, Post-Translational | Matrix Metalloproteinases - metabolism | Growth factors | Neurotropin | Research | Brain | Nervous system | Cellular biology | Cells | matrix metalloproteinases | Index Medicus
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 06/2019, Volume 234, Issue 6, pp. 9525 - 9534
Journal Article
Autophagy, ISSN 1554-8627, 05/2012, Volume 8, Issue 5, pp. 812 - 825
Our study first proposed that curcumin could protect human endothelial cells from the damage caused by oxidative stress via autophagy. Furthermore, our results... 
autophagy | endothelial cell | FOXO1 | oxidative stress | curcumin | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Oxidative stress | Endothelial cell | Curcumin | Autophagy | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Protein Transport - drug effects | Autophagy - drug effects | Gene Knockdown Techniques | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Nucleus - metabolism | Forkhead Transcription Factors - metabolism | Protective Agents - pharmacology | Protein Binding - drug effects | Cytoprotection - drug effects | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Beclin-1 | Hydrogen Peroxide - toxicity | rab GTP-Binding Proteins - metabolism | Cell Survival - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Curcumin - pharmacology | Down-Regulation - drug effects | Ubiquitin-Activating Enzymes - metabolism | Apoptosis Regulatory Proteins - metabolism | Up-Regulation - drug effects | Acetylation - drug effects | Autophagy-Related Protein 7 | Human Umbilical Vein Endothelial Cells - enzymology | Signal Transduction - drug effects | Models, Biological | Human Umbilical Vein Endothelial Cells - pathology | Forkhead Box Protein O1 | Oxidative Stress - drug effects | Cell Nucleus - drug effects | CELL BIOLOGY | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/1998, Volume 273, Issue 46, pp. 30336 - 30343
Journal Article
BLOOD, ISSN 0006-4971, 09/2011, Volume 118, Issue 10, pp. 2906 - 2917
Endothelial cells (ECs) in blood vessels under formation are stabilized by the recruitment of pericytes, both in normal tissues and during angiogenesis in... 
ANTIANGIOGENIC THERAPY | APOPTOSIS | INHIBITION | ANGIOGENESIS | VINBLASTINE | GROWTH-FACTOR | PDGF-B | TUMOR VASCULATURE | HEMATOLOGY | CANCER | CARCINOMA | Islets of Langerhans - drug effects | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Neovascularization, Pathologic | Endothelium, Vascular - drug effects | Melanoma, Experimental - metabolism | Gene Expression Profiling | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | In Situ Hybridization | Islets of Langerhans - metabolism | Pericytes - pathology | Biomarkers, Tumor - metabolism | GTPase-Activating Proteins - physiology | Autocrine Communication | Proto-Oncogene Proteins c-sis - genetics | Islets of Langerhans - pathology | Proto-Oncogene Proteins c-sis - metabolism | Signal Transduction | Cell Survival | Mice, Inbred C57BL | Pericytes - metabolism | RNA, Messenger - genetics | Cells, Cultured | Melanoma, Experimental - pathology | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Blotting, Western | Proteins - genetics | Animals | Melanoma, Experimental - genetics | Proteins - metabolism | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Biomarkers, Tumor - genetics | Mice | Vinblastine - pharmacology | Antineoplastic Agents, Phytogenic - pharmacology | Apoptosis | Cell Movement | Index Medicus | Abridged Index Medicus | Vascular Biology
Journal Article
by Han, J and Pan, XY and Xu, Y and Xiao, Y and An, Y and Tie, L and Pan, Y and Li, XJ
AUTOPHAGY, ISSN 1554-8627, 05/2012, Volume 8, Issue 5, pp. 812 - 825
Our study first proposed that curcumin could protect human endothelial cells from the damage caused by oxidative stress via autophagy. Furthermore, our results... 
autophagy | PHOSPHORYLATION | ACETYLATION | endothelial cell | CARDIAC MYOCYTES | DEATH | MECHANISMS | FOXO TRANSCRIPTION FACTORS | MATURATION | FOXO1 | CELL BIOLOGY | MAINTENANCE | METABOLISM | DEGRADATION | oxidative stress | curcumin
Journal Article
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 05/2004, Volume 109, Issue 17, pp. 2058 - 2067
Background - Myocardial ischemia provides a potent stimulus to angiogenesis, and the mobilization and differentiation of endothelial progenitor cells (EPCs)... 
Nitric oxide synthase | C-reactive protein | Cells | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | MARKERS | apoptosis | ISCHEMIA | VASCULAR SMOOTH-MUSCLE | DIABETES-MELLITUS | cells | CORONARY COLLATERAL VESSELS | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | nitric oxide synthase | NEOVASCULARIZATION | HEMATOLOGY | C-Reactive Protein - antagonists & inhibitors | Neovascularization, Physiologic - drug effects | Apoptosis - drug effects | Humans | Male | Receptors, Cytoplasmic and Nuclear - biosynthesis | Nitric Oxide Synthase - genetics | C-Reactive Protein - pharmacology | Enzyme Induction - drug effects | Nitric Oxide Synthase - biosynthesis | Thiazolidinediones - pharmacology | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Cardiovascular Diseases - etiology | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Nitric Oxide Synthase Type III | Nitric Oxide - physiology | Transcription Factors - biosynthesis | Inflammation | Receptors, Cytoplasmic and Nuclear - agonists | Receptors, Cytoplasmic and Nuclear - genetics | Recombinant Proteins - pharmacology | Transcription Factors - genetics | Gene Expression Regulation - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Transcription Factors - agonists | Endothelial Cells - drug effects | Index Medicus | Abridged Index Medicus
Journal Article