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Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
1997, Lung biology in health and disease, ISBN 9780824798178, Volume 102., xxiii, 855
Book
Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 8/2011, Volume 68, Issue 2, pp. 445 - 455
The natural flavonoid fisetin was recently identified as a lead compound that stabilizes endothelial cell microtubules... 
Fisetin | Lewis lung carcinoma | Angiogenesis | Antitumour activity | Cyclophosphamide | Medicine & Public Health | Cancer Research | Oncology | Cytotoxicity | EA·hy 926 endothelial cells | Pharmacology/Toxicology | Flavonoid | EA•hy 926 endothelial cells | APOPTOSIS | ANTIINFLAMMATORY ACTIVITY | CELL-CYCLE ARREST | PROLIFERATION | FLUOROURACIL | CANCER | IN-VITRO | ONCOLOGY | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | EA.hy 926 endothelial cells | INHIBITORS | NIH 3T3 Cells | Cyclophosphamide - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Tubulin Modulators - pharmacology | Humans | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Antineoplastic Agents, Alkylating - pharmacology | Flavonoids - adverse effects | Antineoplastic Agents, Alkylating - administration & dosage | Cyclophosphamide - adverse effects | Cyclophosphamide - therapeutic use | Flavonoids - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Angiogenesis Inhibitors - administration & dosage | Antineoplastic Agents, Phytogenic - administration & dosage | Angiogenesis Inhibitors - therapeutic use | Flavonoids - administration & dosage | Tubulin Modulators - administration & dosage | Female | Flavonoids - pharmacology | Antineoplastic Agents, Phytogenic - therapeutic use | Angiogenesis Inhibitors - adverse effects | Antineoplastic Agents, Phytogenic - adverse effects | Cell Line | Cell Survival - drug effects | Tubulin Modulators - adverse effects | Mice, Inbred C57BL | Angiogenesis Inhibitors - pharmacology | Tubulin Modulators - therapeutic use | Carcinoma, Lewis Lung - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Cell Movement - drug effects | Animals | Tumor Burden - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Cyclophosphamide - pharmacology | Carcinoma, Lewis Lung - pathology | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Alkylating - adverse effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | Endothelial Cells - drug effects | Antimitotic agents | Flavonoids | Flavones | Lung cancer | Bioflavonoids | Accountants | Drug therapy, Combination | Universities and colleges | Antineoplastic agents | Endothelium | Tumors | Index Medicus | cytology | Antineoplastic Agents, Phytogenic | pathology | Cell Proliferation | Endothelial Cells | Tubulin Modulators | Neovascularization, Pathologic | fisetin | administration & dosage | pharmacology | flavonoid | Carcinoma, Lewis Lung | Antineoplastic Agents, Alkylating | cytotoxicity | drug therapy | Cell Survival | angiogenesis | Antineoplastic Combined Chemotherapy Protocols | drug effects | Tumor Burden | Angiogenesis Inhibitors | EA.hy 926 | Cell Cycle | antitumour activity | adverse effects | therapeutic use | Cell Movement
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 511, Issue 7509, pp. 312 - 318
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 4, p. e35685
We have previously shown that mesenchymal stem cells (MSC) improve function upon integration in ischemic myocardium... 
MIGRATION | SURVIVAL | BAD | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | H9C2 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INHIBIT APOPTOSIS | CHEMOKINE RECEPTORS | STROMAL CELLS | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Apoptosis - drug effects | Caspase 3 - metabolism | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Mesenchymal Stromal Cells - cytology | Caspase 3 - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - drug effects | Endothelial Cells - metabolism | Cytokines - secretion | Mesenchymal Stromal Cells - metabolism | Chemotaxis - drug effects | Mesenchymal Stromal Cells - secretion | Phosphatidylinositol 3-Kinases - genetics | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Dogs | Mice | Primary Cell Culture | Cytokines - biosynthesis | Cytokines - pharmacology | Endothelial Cells - drug effects | Cytokines | Comparative analysis | Vascular endothelial growth factor | Stem cells | Apoptosis | Heart | Phosphorylation | Leukocyte migration | Mesenchyme | Intracellular signalling | AKT protein | Kinases | Macrophages | Caspase-3 | Proteins | Angiogenesis | Signal transduction | Mitochondria | Cell growth | Ischemia | Rodents | Cell cycle | Tumor necrosis factor-TNF | Conditioning | Localization | Cardiology | Growth factors | Extracellular signal-regulated kinase | Caspase | Cardiomyocytes | Inflammation | Ribonucleic acid--RNA | Chemotaxis | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Inhibitors | γ-Interferon | Myocardium | Hypoxia | Interferon | Laboratory animals | Cell migration | Monocyte chemoattractant protein 1 | RNA | Ribonucleic acid
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 40, pp. 12516 - 12521
Human pluripotent stem cell-based in vitro models that reflect human physiology have the potential to reduce the number of drug failures in clinical trials and offer a cost-effective approach... 
Organoid | Toxicology | Differentiation | Tissue engineering | Machine learning | toxicology | HUMAN NEOCORTEX | tissue engineering | DEVELOPMENTAL NEUROTOXICITY | differentiation | HUMAN BRAIN | MULTIDISCIPLINARY SCIENCES | CLASSIFICATION | FATTY-ACIDS | machine learning | CANCER | organoid | IN-VITRO | HUMAN CEREBRAL-CORTEX | MICROGLIA | GENE-EXPRESSION | Embryonic Stem Cells - metabolism | Microglia - metabolism | Embryonic Stem Cells - cytology | Humans | Brain - growth & development | Support Vector Machine | Neural Stem Cells - cytology | Xenobiotics - pharmacology | Brain - metabolism | Neurogenesis - genetics | Mesenchymal Stromal Cells - cytology | Xenobiotics - classification | Gene Expression Regulation, Developmental | Cell Differentiation | Neurogenesis - drug effects | Culture Media, Serum-Free - pharmacology | Gene Ontology | Polyethylene Glycols - pharmacology | Microglia - cytology | Mesenchymal Stromal Cells - drug effects | Brain - cytology | Pluripotent Stem Cells - cytology | Tissue Engineering - methods | Microglia - drug effects | Endothelial Cells - metabolism | Cells, Cultured | Neural Stem Cells - drug effects | Mesenchymal Stromal Cells - metabolism | Cell Communication - genetics | Macrophages - cytology | Pluripotent Stem Cells - metabolism | Macrophages - metabolism | Embryonic Stem Cells - drug effects | Endothelial Cells - cytology | Models, Biological | Pluripotent Stem Cells - drug effects | Cell Communication - drug effects | Macrophages - drug effects | Hydrogels - pharmacology | Neural Stem Cells - metabolism | Endothelial Cells - drug effects | Biological Sciences
Journal Article
PloS one, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e46842
Journal Article
Nature cell biology, ISSN 1476-4679, 2015, Volume 17, Issue 8, pp. 994 - 1003
The use of human pluripotent stem cells for in vitro disease modelling and clinical applications requires protocols that convert these cells into relevant adult cell types... 
DIRECTED DIFFERENTIATION | IN-VIVO | MOUSE | DEFINITIVE ENDODERM | GROWTH-FACTOR | HUMAN BLASTOCYSTS | STROMAL CELLS | WNT | CULTURE | LINES | CELL BIOLOGY | Coculture Techniques | Humans | Endothelial Cells - transplantation | Glycogen Synthase Kinase 3 beta | Cell Lineage - drug effects | Dose-Response Relationship, Drug | Human Umbilical Vein Endothelial Cells - physiology | Transfection | Time Factors | Gene Expression Regulation, Developmental | Transcription, Genetic | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Myocytes, Smooth Muscle - transplantation | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Cell Line | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - enzymology | Induced Pluripotent Stem Cells - transplantation | Myocytes, Smooth Muscle - physiology | Gene Expression Profiling - methods | Muscle, Smooth, Vascular - transplantation | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Muscle, Smooth, Vascular - cytology | Metabolomics - methods | Bone Morphogenetic Protein 4 - pharmacology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Mice, Inbred NOD | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | Endothelial Cells - drug effects | Usage | Cell research | Growth | Stem cells | Muscle cells | Research | Cell differentiation | Endothelium
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 22, pp. 9944 - 9949
Actomyosin contractility affects cellular organization within tissues in part through the generation of mechanical forces at sites of cell—matrix and cell—cell contact... 
Cell growth | Microinjections | Epithelial cells | Bowties | Cell adhesion | Cadherins | Adherens junctions | Focal adhesions | Physiological regulation | Endothelial cells | Mechanotransduction | Traction force | Myosin | Adherens junction | PDMS | ACTIVATION | mechanotransduction | MULTIDISCIPLINARY SCIENCES | myosin | EXTENSION | E-CADHERIN | FOCAL ADHESIONS | GASTRULATION | traction force | adherens junction | RAC | ENDOTHELIAL-CELLS | GROWTH | RHO | REQUIRE | Myosin Type II - physiology | rhoA GTP-Binding Protein - administration & dosage | Cadherins - metabolism | Mechanotransduction, Cellular - drug effects | Humans | Green Fluorescent Proteins - genetics | rhoA GTP-Binding Protein - physiology | rhoA GTP-Binding Protein - genetics | Mechanotransduction, Cellular - physiology | Antigens, CD - genetics | Antigens, CD - metabolism | Endothelial Cells - ultrastructure | Thrombin - pharmacology | Lysophospholipids - pharmacology | rac GTP-Binding Proteins - physiology | Cadherins - genetics | Endothelial Cells - physiology | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Cells, Cultured | Recombinant Proteins - genetics | rho GTP-Binding Proteins - physiology | Recombinant Proteins - administration & dosage | Sphingosine - pharmacology | Biomechanical Phenomena | Sphingosine - analogs & derivatives | Adherens Junctions - physiology | Adherens Junctions - drug effects | Adherens Junctions - ultrastructure | Amino Acid Substitution | Endothelial Cells - drug effects | Junctional complexes (Epithelium) | Cell junctions | Properties | Biological Sciences | Physical Sciences
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, pp. e0147198 - e0147198
.... Attention has focused on the identification and relative contribution of tissue specific stem cells to the regeneration process... 
SKELETAL-MUSCLE | SATELLITE CELL NICHE | STEM-CELLS | ADULT MUSCLE | MACROPHAGES | MYOGENESIS | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | SELF-RENEWAL | PROLIFERATION | TEMPLATE DNA STRANDS | Barium Compounds - toxicity | Chlorides - toxicity | Muscle, Skeletal - injuries | Cytokines - physiology | Necrosis | Muscle Development | Cold Injury - pathology | Muscle, Skeletal - drug effects | Satellite Cells, Skeletal Muscle - physiology | Freezing - adverse effects | Models, Animal | Myoblasts - physiology | Elapid Venoms - toxicity | Macrophages - physiology | Green Fluorescent Proteins - analysis | Vascular Endothelial Growth Factor Receptor-2 - analysis | Mice, Inbred C57BL | Cobra Cardiotoxin Proteins - toxicity | Mice, Transgenic | Muscle, Skeletal - physiology | Regeneration - physiology | Animals | Fibrosis | Stem Cells - physiology | Mice | Muscle, Skeletal - pathology | Neovascularization, Physiologic | Regeneration - immunology | Cold Injury - physiopathology | Cycles | Barium | Animal models | Transplants & implants | Transgenic | Stem cell transplantation | Confocal microscopy | Infections | Confocal | Epidemiology | Regeneration (physiology) | Cell growth | Histopathology | Rodents | Cell cycle | Gangrene | Basal lamina | Injuries | Immune system | Cytokines | Tissue engineering | Organs | Muscles | Blood vessels | Cell division | Inflammation | Skeletal muscle | Barium chloride | Regeneration | Musculoskeletal system | Microscopy | Experimental design | Stem cells | Infiltration | Chemokines | Index Medicus | Green Fluorescent Proteins | Cobra Cardiotoxin Proteins | Macrophages | Cellular Biology | Vascular Endothelial Growth Factor Receptor-2 | Freezing | Stem Cells | Life Sciences | Cold Injury | Barium Compounds | Chlorides | Myoblasts | Elapid Venoms | Muscle, Skeletal | Satellite Cells, Skeletal Muscle
Journal Article