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Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 81, pp. 62 - 70
Abstract Age-associated cardiovascular diseases are at least partially ascribable to vascular cell senescence. Replicative senescence (RS) and stress-induced... 
Cardiovascular | Vascular aging | miRNA | Senescence | Mitochondria | lncRNA | Endothelial cells | LncRNA | MiRNA | CARDIAC & CARDIOVASCULAR SYSTEMS | LONG NONCODING RNAS | TELOMERASE | P16(INK4A) | CELLULAR SENESCENCE | CANCER | FAMILY | CELL BIOLOGY | DNA | GENE-EXPRESSION | PROTEINS | BINDING | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Molecular Sequence Data | Male | MicroRNAs - metabolism | Aorta - metabolism | RNA - genetics | G2 Phase Cell Cycle Checkpoints - radiation effects | G2 Phase Cell Cycle Checkpoints - drug effects | Ultraviolet Rays | Aging - genetics | Mitochondria - genetics | Cellular Senescence | Base Sequence | Human Umbilical Vein Endothelial Cells - cytology | Myocytes, Smooth Muscle - radiation effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | RNA - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Mitochondria - metabolism | RNA, Long Noncoding - genetics | Animals | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | MicroRNAs - genetics | Aorta - cytology | Human Umbilical Vein Endothelial Cells - radiation effects | RNA, Long Noncoding - metabolism | Aging - metabolism | Analysis | Mitochondrial DNA | Endothelium
Journal Article
Journal Article
Journal Article
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 08/2018, Volume 101, pp. 64 - 73
Accumulation of senescent endothelial cells can contribute to endothelium dysfunction. Suppression of MTOR signaling has been shown to delay senescence but the... 
Human endothelial cell | Rapamycin | MicroRNA-107 | Mammalian target of rapamycin | Cellular senescence | MAMMALIAN TARGET | ACTIVATION | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | VEGF EXPRESSION | PTEN | CELL-CYCLE ARREST | PROLIFERATION | SUPPRESSION | CELL BIOLOGY | PATHWAY | Phosphatases | MicroRNA | Surface active agents | Albumin | Sulfates | Protein binding | Endothelium
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 04/2004, Volume 94, Issue 6, pp. 768 - 775
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 11/2017, Volume 92, pp. 18 - 25
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2009, Volume 297, Issue 5, pp. 1673 - 1684
Medial artery calcification, which does not accompany lipid or cholesterol deposit, preferentially occurs in elderly population, but its underlying mechanisms... 
Runt-related transcription factor-2 | Statin | runt-related transcription factor-2 | TRANSCRIPTION FACTORS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PHOSPHATE-INDUCED CALCIFICATION | CELLULAR SENESCENCE | IN-VITRO | statin | II COLLAGEN | BONE-MATRIX PROTEINS | ARTERIAL CALCIFICATION | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | EXPRESSION | GLA PROTEIN | Calcinosis - genetics | Tunica Media - pathology | Age Factors | Muscle, Smooth, Vascular - metabolism | Humans | Alkaline Phosphatase - metabolism | Myocytes, Smooth Muscle - pathology | Cellular Senescence - drug effects | Cell Transdifferentiation - genetics | Aorta - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Glucuronidase - deficiency | rho-Associated Kinases - antagonists & inhibitors | RNA Interference | rho-Associated Kinases - metabolism | Tunica Media - metabolism | Cell Transdifferentiation - drug effects | Calcinosis - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Extracellular Matrix Proteins - metabolism | Muscle, Smooth, Vascular - drug effects | Calcium-Binding Proteins - metabolism | Cellular Senescence - genetics | Collagen Type I - metabolism | Osteoblasts - drug effects | Cells, Cultured | Gene Expression Regulation | Aging - pathology | Mice, Knockout | Aorta - pathology | Osteoblasts - pathology | Muscle, Smooth, Vascular - pathology | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Glucuronidase - genetics | Calcinosis - prevention & control | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Calcinosis - pathology | Osteoblasts - metabolism | Aging - metabolism | Apoptosis | Physiological aspects | Calcification | Aging | Transcription factors | Vascular smooth muscle | Research
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 16, pp. C67 - C68
Objectives To study the mechanism of the transforming growth factor-β(TGF-β) in Ginsenoside Rb1 against the replicative senescence of human umbilical vein... 
Proteins | Protein expression | Senescence | Endothelial cells
Journal Article