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endothelial-to-mesenchymal transition (151) 151
index medicus (136) 136
humans (80) 80
animals (70) 70
endothelium (67) 67
fibrosis (58) 58
stem cells (54) 54
endothelial cells (46) 46
expression (43) 43
endothelial to mesenchymal transition (41) 41
cells, cultured (39) 39
tgf-beta (36) 36
cell biology (33) 33
signal transduction (32) 32
male (31) 31
cardiac & cardiovascular systems (30) 30
cells (30) 30
article (29) 29
cardiac fibrosis (29) 29
inflammation (29) 29
mice (29) 29
angiogenesis (27) 27
endothelial cells - metabolism (27) 27
activation (26) 26
endothelial cells - pathology (25) 25
biochemistry & molecular biology (24) 24
epithelial-mesenchymal transition (24) 24
growth-factor-beta (24) 24
contributes (23) 23
mesenchyme (23) 23
to-mesenchymal transition (23) 23
proteins (22) 22
disease (21) 21
epithelial-mesenchymal transition - drug effects (21) 21
fibroblasts (21) 21
physiology (20) 20
progenitor cells (20) 20
atherosclerosis (19) 19
oxidative stress (19) 19
cancer (18) 18
endothelial cell (18) 18
gene expression (18) 18
rats (18) 18
analysis (17) 17
collagen (17) 17
medicine, research & experimental (17) 17
disease models, animal (16) 16
mechanisms (16) 16
endmt (15) 15
heart (15) 15
in-vitro (15) 15
female (14) 14
hypoxia (14) 14
pathogenesis (14) 14
pharmacology & pharmacy (14) 14
smooth-muscle-cells (14) 14
actins - metabolism (13) 13
apoptosis (13) 13
cardiovascular (13) 13
differentiation (13) 13
oncology (13) 13
peripheral vascular disease (13) 13
progression (13) 13
transformation (13) 13
transforming growth factors (13) 13
bone morphogenetic proteins (12) 12
development and progression (12) 12
endothelial cells - drug effects (12) 12
epithelial-to-mesenchymal transition (12) 12
kinases (12) 12
micrornas (12) 12
phenotype (12) 12
respiratory system (12) 12
smooth muscle (12) 12
cardiovascular system (11) 11
cell proliferation (11) 11
dysfunction (11) 11
endothelial cells - cytology (11) 11
endothelial cells - physiology (11) 11
endothelial‐to‐mesenchymal transition (11) 11
epithelial-mesenchymal transition - physiology (11) 11
extracellular-matrix (11) 11
growth factors (11) 11
hypertension (11) 11
mice, inbred c57bl (11) 11
pulmonary hypertension (11) 11
tgf-β (11) 11
transforming growth factor beta1 - pharmacology (11) 11
autophagy (10) 10
cell differentiation (10) 10
endothelial dysfunction (10) 10
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pathology (10) 10
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biophysics (9) 9
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Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 01/2019, Volume 73, Issue 2, pp. 190 - 209
Endothelial to mesenchymal transition (EndMT) is a process whereby an endothelial cell undergoes a series of molecular events that lead to a change in... 
cardiovascular | EndMT | endothelial to mesenchymal transition | Endothelial to mesenchymal transition
Journal Article
INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE, ISSN 1107-3756, 10/2017, Volume 40, Issue 4, pp. 1009 - 1018
Corneal diseases exhibit a high prevalence and are prone to cause blindness; furthermore, maintaining the morphology and ionic transporter expression in... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | cell junctions | morphology | CYTOSKELETON | RHO-KINASE | CELL-ADHESION | thiazovivin | ionic transporter expression | epithelial-to-mesenchymal transition | FAMILY | endothelial-to-mesenchymal transition | GTPASES | CULTURE-CONDITIONS | ENHANCEMENT | human corneal endothelial cells | Cornea - surgery | Sodium-Potassium-Exchanging ATPase - genetics | Cell Proliferation | Epithelial Cells - metabolism | Cadherins - agonists | Cadherins - metabolism | Epithelial Cells - drug effects | Humans | Middle Aged | Actins - metabolism | Epithelial-Mesenchymal Transition - drug effects | Male | Antigens, CD - genetics | rho-Associated Kinases - antagonists & inhibitors | Actins - genetics | Antigens, CD - metabolism | Cornea - drug effects | Snail Family Transcription Factors - genetics | rho-Associated Kinases - metabolism | Snail Family Transcription Factors - antagonists & inhibitors | Adult | Cornea - cytology | Female | Zonula Occludens-1 Protein - metabolism | Cadherins - genetics | Epithelial Cells - cytology | Zonula Occludens-1 Protein - agonists | Zonula Occludens-1 Protein - genetics | Corneal Transplantation | Snail Family Transcription Factors - metabolism | Signal Transduction | Gene Expression Regulation | rho-Associated Kinases - genetics | Pyrimidines - pharmacology | Cornea - metabolism | Sodium-Potassium-Exchanging ATPase - metabolism | Actins - antagonists & inhibitors | Cadherins - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Primary Cell Culture
Journal Article
Drug Design, Development and Therapy, ISSN 1177-8881, 08/2015, Volume 9, pp. 4599 - 4611
Background: Relaxin (RLX) can prevent cardiac fibrosis. We aimed to investigate the possible mechanism and signal transduction pathway of RLX inhibiting... 
Endothelial to mesenchymal transition | Myocardial fibrosis | Transforming growth factor β | Relaxin | Notch | VITRO | CELLS | CHEMISTRY, MEDICINAL | transforming growth factor beta | relaxin | endothelial to mesenchymal transition | FIBROBLASTS | myocardial fibrosis | HEART | IN-VIVO | PHARMACOLOGY & PHARMACY | DIFFERENTIATION | STRESS | HORMONE RELAXIN | EXPRESSION | PROGRESSION | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Epithelial-Mesenchymal Transition - drug effects | Male | Isoproterenol | Relaxin - pharmacology | Cardiomyopathies - physiopathology | Myocardium - metabolism | Disease Models, Animal | Cardiomyopathies - drug therapy | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Dipeptides - pharmacology | Ventricular Function, Left - drug effects | Cells, Cultured | Cardiomyopathies - pathology | Myocardium - pathology | Receptor, Notch1 - metabolism | Rats, Sprague-Dawley | Cell Movement - drug effects | Collagen - metabolism | Animals | Signal Transduction - drug effects | Cardiomyopathies - metabolism | Fibrosis | Human Umbilical Vein Endothelial Cells - pathology | Receptor, Notch1 - antagonists & inhibitors | Endothelial Cells - pathology | Endothelial Cells - drug effects | Cellular signal transduction | Models | Properties | Health aspects | Heart diseases | Vimentin | Heart | Cell culture | Collagen (type I) | Mesenchyme | Transforming growth factor | Mobility | Kinases | Proteins | Signal transduction | Veins | Rodents | Fibroblasts | Growth factors | Level (quantity) | Endothelial cells | Inhibitors | Collagen | Morphology | Stem cells | Myocardium | Hypoxia | Transduction | Notch protein | Microvasculature | Laboratory animals | Immunofluorescence | Umbilical vein | Cell migration
Journal Article
Developmental Biology, ISSN 0012-1606, 10/2019
Journal Article
The Journal of Pathology, ISSN 0022-3417, 03/2019, Volume 247, Issue 3, pp. 333 - 346
Endothelial‐to‐mesenchymal transition (EndMT) has been unveiled as a common cause for a multitude of human pathologies, including cancer and cardiovascular... 
endothelial‐to‐mesenchymal transition | fibroblast | tumor necrosis factor alpha | inflammation | transforming growth factor beta | endothelial cell | vascular calcification | osteoblast | bone morphogenetic protein | pulmonary arterial hypertension | c‐Jun N‐terminal kinase | endothelial-to-mesenchymal transition | c-Jun N-terminal kinase | CELLS | OSTEOBLAST DIFFERENTIATION | SARCOMA-LIKE TUMORS | ATHEROSCLEROSIS | PATHOLOGY | CHALLENGES | INHIBITION | ONCOLOGY | CARDIOVASCULAR-DISEASE | TGF-BETA RECEPTOR | MUTATIONS | EXPRESSION | Interleukin-1beta - pharmacology | Osteogenesis - physiology | Epithelial-Mesenchymal Transition - physiology | Humans | Osteogenesis - drug effects | Cells, Cultured | Epithelial-Mesenchymal Transition - drug effects | Down-Regulation - drug effects | Vascular Calcification - pathology | Mice, Inbred C3H | Down-Regulation - physiology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Cell Differentiation - drug effects | Inflammation Mediators - pharmacology | Vascular Calcification - physiopathology | Endothelium, Vascular - pathology | Signal Transduction - physiology | Bone Morphogenetic Protein Receptors, Type II - physiology | Endothelial Cells - physiology | Calcification | Bone morphogenetic proteins | Inflammation | Analysis | Stem cells | Endothelium | Calcification (ectopic) | Animal models | Mesenchyme | Transforming growth factor-b | Interleukin | Differentiation (biology) | Risk factors | Signal transduction | Biomedical materials | Ischemia | Mineralization | Animal tissues | Aorta | Tumor necrosis factor-TNF | Biocompatibility | Growth factors | Sensitizing | Bone morphogenetic protein receptor type II | Cytokines | Health risks | Tumor necrosis factor-α | Risk analysis | Cell differentiation | Patients | Endothelial cells | Signaling | Signal processing | Biomarkers | Transduction | Cardiovascular diseases | Cancer | Original Papers | Original Paper
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2018, Volume 315, Issue 5, pp. H1293 - H1303
Journal Article
CELLS, ISSN 2073-4409, 12/2015, Volume 4, Issue 4, pp. 631 - 652
The pathophysiology of diabetic nephropathy (DN), one of the most serious complications in diabetic patients and the leading cause of end-stage renal disease... 
MYOFIBROBLAST TRANSITION | endothelial-to-mesenchymal transition (EndoMT) | TGF-BETA | epithelial-to-mesenchymal transition (EMT) | ANGIOTENSIN-II | E-CADHERIN | renal fibrosis | FIBROBLASTS | CELL BIOLOGY | CELL COMMUNICATION | TRANSFORMING GROWTH-FACTOR-BETA-1 | HIGH GLUCOSE | EXPRESSION | diabetic nephropathy
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 06/2017, Volume 42, Issue 1, pp. 357 - 372
Endothelial-to-mesenchymal transition (EndMT) plays significant roles under various pathological conditions including cardiovascular diseases, fibrosis, and... 
Endothelial-to-mesenchymal transition | MALAT1 | SMAD3 | Endothelial progenitor cells | miR-145 | TGFBR2 | Receptors, Transforming Growth Factor beta - genetics | Endothelial Progenitor Cells - cytology | MicroRNAs - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - chemistry | Humans | Transforming Growth Factor beta1 - metabolism | Collagen Type III - metabolism | Epithelial-Mesenchymal Transition - drug effects | MicroRNAs - metabolism | Smad3 Protein - metabolism | Smad3 Protein - genetics | Receptor, Transforming Growth Factor-beta Type II | RNA Interference | Base Sequence | 3' Untranslated Regions | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - pharmacology | Endothelial Progenitor Cells - metabolism | Collagen Type I - metabolism | Signal Transduction | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | RNA, Long Noncoding - genetics | Down-Regulation - drug effects | Sequence Alignment | Receptors, Transforming Growth Factor beta - metabolism | Smad3 Protein - chemistry | MicroRNAs - genetics | Protein-Serine-Threonine Kinases - chemistry | Antagomirs - metabolism | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | RNA, Small Interfering - metabolism | Studies | Liver cancer | Cell growth | MicroRNAs | Rodents | Smooth muscle | Metastasis | Growth factors | Cell adhesion & migration
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 10/2017, Volume 21, Issue 10, pp. 2359 - 2369
Journal Article