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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 134, Issue 2, pp. 382 - 389.e4
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 3, pp. 708 - 718.e5
Background & Aims Little is known about factors that regulate intestinal epithelial differentiation; microbial recognition receptors such as Toll-like receptor... 
Gastroenterology and Hepatology | Development | Microbiota | Pediatric Gastrointestinal Disorder | Mouse Model | BILE-ACID | STEM-CELLS | BACTERIA | RECOGNITION | SELF-RENEWAL | BETA | PATHOGENESIS | IMMATURE | GASTROENTEROLOGY & HEPATOLOGY | TLR4 | EXPRESSION | Intestine, Small - pathology | Receptors, Notch - metabolism | Enterocolitis, Necrotizing - genetics | Enterocolitis, Necrotizing - metabolism | Humans | Enterocolitis, Necrotizing - microbiology | Goblet Cells - microbiology | Transfection | Organoids | RNA Interference | Toll-Like Receptor 4 - deficiency | Cell Differentiation | Enterocolitis, Necrotizing - pathology | Infant, Newborn | Disease Models, Animal | Animals, Newborn | Cell Line | Signal Transduction | Enterocolitis, Necrotizing - prevention & control | Infant Formula | Tissue Culture Techniques | Intestine, Small - microbiology | Mice, Inbred C57BL | Rats | Toll-Like Receptor 4 - genetics | Bile Acids and Salts - metabolism | Hypoxia - complications | Goblet Cells - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Animals | Enterocolitis, Necrotizing - etiology | Mice | Goblet Cells - pathology | Intestine, Small - metabolism | Antigen-antibody reactions | Enterocolitis, Pseudomembranous | Enterocolitis, Neonatal necrotizing | RNA | Analysis | Fluorescence | Mitogens | Deoxycholic acid | Bile acids | Microbiota (Symbiotic organisms) | Gastrointestinal diseases | Cell differentiation
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2017, Volume 139, Issue 4, pp. 1111 - 1126.e4
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0171579
Background Some oral probiotics have been shown to prevent necrotizing enterocolitis (NEC) and decrease mortality effectively in preterm very low birth weight... 
LACTIS BB12 SUPPLEMENTATION | BIFIDOBACTERIUM-BREVE | LOW-BIRTH-WEIGHT | LATE-ONSET SEPSIS | INTESTINAL MICROBIOTA | MULTIDISCIPLINARY SCIENCES | DOUBLE-BLIND | PROPHYLACTIC PROBIOTICS | PRETERM INFANTS | LACTOBACILLUS-REUTERI | ORAL SUPPLEMENTATION | Probiotics - administration & dosage | Infant, Premature, Diseases - prevention & control | Enterocolitis, Necrotizing - prevention & control | Infant, Premature, Diseases - microbiology | Infant, Very Low Birth Weight | Humans | Enterocolitis, Necrotizing - mortality | Enterocolitis, Necrotizing - microbiology | Infant, Premature, Diseases - mortality | Randomized Controlled Trials as Topic | Enterocolitis, Necrotizing - epidemiology | Probiotics - classification | Infant, Premature | Probiotics - therapeutic use | Infant, Premature, Diseases - epidemiology | Infant, Newborn | Prevention | Infants (Newborn) | Probiotics | Enterocolitis, Pseudomembranous | Usage | Enterocolitis, Neonatal necrotizing | Birth weight, Low | Research | Health aspects | Neonates | Pediatrics | Bias | Clinical trials | Necrotizing enterocolitis | Infants | Gestation | Saccharomyces | Enterocolitis | Reduction | Newborn babies | Supplementation | Medical research | Mortality | Dietary supplements | Community colleges | Meta-analysis | Medicine | Studies | Babies | Hospitals | Collaboration | Sepsis | Birth weight
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e83304
Necrotizing enterocolitis (NEC) is the most devastating intestinal disease affecting preterm infants. In addition to being associated with short term mortality... 
EMPIRICAL ANTIBIOTIC-TREATMENT | COMMUNITY | FLORA | MULTIDISCIPLINARY SCIENCES | FECAL MICROFLORA | PRETERM INFANTS | OUTCOMES | Intestines - drug effects | Enterocolitis, Necrotizing - drug therapy | Humans | Enterocolitis, Necrotizing - microbiology | Male | Biodiversity | Case-Control Studies | Anti-Bacterial Agents - therapeutic use | Microbiota | Female | RNA, Ribosomal, 16S | Infant, Newborn | Intestines - pathology | Bifidobacterium - classification | Infant, Premature, Diseases - microbiology | Bifidobacterium - genetics | Risk Factors | Infant, Premature, Diseases - etiology | Infant, Premature, Diseases - drug therapy | Intestines - microbiology | Enterocolitis, Necrotizing - etiology | Infant, Premature | Environment | Anti-Bacterial Agents - pharmacology | Metagenome | Cluster Analysis | Enterocolitis, Pseudomembranous | Microbiota (Symbiotic organisms) | Enterocolitis, Neonatal necrotizing | Antibiotics | RNA | Infants (Premature) | Gastrointestinal diseases | Analysis | Mortality | Klebsiella | Neonates | Pediatrics | Dysbacteriosis | Intensive care | rRNA 16S | Pathogenesis | Necrotizing enterocolitis | Infants | Gestation | Enterocolitis | Epidemiology | Microorganisms | Newborn babies | Intestine | Diagnosis | Feces | Public health | Milk | Complications | Environmental factors | Morbidity | Feeding | Medicine | Studies | Babies | Birth weight | Colonization | Breast milk
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, p. e0161635
Zonulin protein is a newly discovered modulator which modulates the permeability of the intestinal epithelial barrier by disassembling intercellular tight... 
PATHOGENESIS | TIGHT JUNCTIONS | EXPERIMENTAL NECROTIZING ENTEROCOLITIS | EPITHELIAL-CELLS | PERMEABILITY | MULTIDISCIPLINARY SCIENCES | ZONULIN | MICE | DYSFUNCTION | INTERLEUKIN-6 | PATHOPHYSIOLOGY | Caco-2 Cells | Severity of Illness Index | Tight Junctions - metabolism | Ileum - pathology | Intestinal Mucosa - metabolism | Cytokines - metabolism | Cholera Toxin - metabolism | Enterocytes - metabolism | Enterocolitis, Necrotizing - metabolism | Humans | Ileum - metabolism | Rats | Enterocolitis, Necrotizing - microbiology | Intestinal Mucosa - microbiology | Permeability | Animals | Biomarkers | Bifidobacterium - physiology | Enterocolitis, Necrotizing - pathology | Tight Junctions - pathology | Disease Models, Animal | Intestinal Mucosa - pathology | Cellular proteins | Enterocolitis, Pseudomembranous | Care and treatment | Bifidobacterium | Enterocolitis, Neonatal necrotizing | Development and progression | Protein biosynthesis | Genetic aspects | Health aspects | Neonates | Necrotizing enterocolitis | Enterocolitis | Kinases | Small intestine | Lipopolysaccharides | Interleukin 6 | Proteins | Intestine | Rodents | Tumor necrosis factor-TNF | Pretreatment | Localization | Tight junctions | Secretion | Inflammation | Tumor necrosis factor-α | Ileum | Zonula occludens-1 protein | Monolayers | In vivo methods and tests | Position (location) | Dismantling | Electrical junctions | Integrity
Journal Article