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Journal of Neuroimmunology, ISSN 0165-5728, 2015, Volume 284, pp. 18 - 29
Abstract Dopamine receptors have been described in T-cells, however their signalling pathways coupled remain unknown. Since cAMP and ERKs play key roles... 
Neurology | Allergy and Immunology | Knockout mice | Th1 differentiation | Neuroimmunology | TCR signalling | T-cell activation | Dopamine receptors | IMMUNITY | DENDRITIC CELLS | NEURODEGENERATION | PROLIFERATION | IMMUNOLOGY | NEUROSCIENCES | IN-VITRO | CD4+AND CD8+T CELLS | SECRETION | TH1 | CYTOTOXICITY | LYMPHOCYTES | T-Lymphocytes - physiology | Receptors, Dopamine D3 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Dose-Response Relationship, Drug | Receptors, Dopamine D3 - genetics | Cell Differentiation - genetics | Lymphocyte Activation - genetics | Phosphorylation - genetics | Dopamine Agents - pharmacology | T-Lymphocytes - drug effects | Phosphorylation - drug effects | Cell Differentiation - physiology | Cyclic AMP - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Signal Transduction - genetics | Enzyme Activation - drug effects | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Ionomycin - pharmacology | Receptors, Dopamine D5 - metabolism | Mice | Receptors, Dopamine D5 - genetics | Enzyme Activation - genetics | CD4 Antigens - metabolism
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 665 - 680
Cellular lipid metabolism has been linked to immune responses; however, the precise mechanisms by which de novo fatty acid synthesis can regulate inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | GLUCOSE-METABOLISM | ATP-CITRATE LYASE | UCP2 | SEPTIC SHOCK | CELL-GROWTH | CRITICALLY-ILL PATIENTS | INTENSIVE INSULIN THERAPY | CANCER | MITOCHONDRIAL UNCOUPLING PROTEINS | Fatty Acid Synthase, Type I - genetics | Fatty Acid Synthase, Type I - biosynthesis | Lipids - genetics | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Interleukin-1beta - genetics | Lipids - biosynthesis | Sepsis - pathology | Interleukin-1beta - metabolism | Mitochondrial Proteins - metabolism | Sepsis - metabolism | Macrophages - pathology | Down-Regulation - genetics | Inflammasomes - genetics | Sepsis - genetics | Mice, Knockout | Carrier Proteins - genetics | Enzyme Induction - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Sepsis - chemically induced | Ion Channels - metabolism | Caspase 1 - genetics | Sepsis - therapy | Mice | Uncoupling Protein 2 | Interleukin-18 - genetics | Interleukin-18 - metabolism | Cellular proteins | Immune response | Synthesis | Sepsis | Development and progression | Lipids | Genetic aspects | Properties | Enzymes | Cytokines | Pathogenesis | Mortality | Glucose | Metabolism | Gene expression | Fatty acids | Proteins | Studies | Metabolites | Consent | Rodents
Journal Article
Journal Article
2010, 2nd ed., Methods in molecular biology, ISBN 9781607617945, Volume 661., xiv, 529
Book
Nature Medicine, ISSN 1078-8956, 11/2008, Volume 14, Issue 11, pp. 1256 - 1263
Journal Article