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1. Full Text The oestrogen receptor alpha-regulated lncRNA NEAT1 is a critical modulator of prostate cancer
by Chakravarty, Dimple and Sboner, Anea and Nair, Sujit S and Giannopoulou, Eugenia and Li, Ruohan and Hennig, Sven and Mosquera, Juan Miguel and Pauwels, Jonathan and Park, Kyung and Kossai, Myriam and MacDonald, Theresa Y and Fontugne, Jacqueline and Erho, Nicholas and Vergara, Ismael A and Ghadessi, Mercedeh and Davicioni, Elai and Jenkins, Robert B and Palanisamy, Nallasivam and Chen, Zhengming and Nakagawa, Shinichi and Hirose, Tetsuro and Bander, Neil H and Beltran, Himisha and Fox, Archa H and Elemento, Olivier and Rubin, Mark A
Nature Communications, ISSN 2041-1723, 11/2014, Volume 5, Issue 1, pp. 5383 - 5383
The androgen receptor (AR) plays a central role in establishing an oncogenic cascade that drives prostate cancer progression. Some prostate cancers escape... 
ANDROGEN RECEPTOR | NUCLEAR-BODIES | MEMBRANE ANTIGEN | LONG NONCODING RNA | CHROMATIN | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION PROFILES | RADICAL PROSTATECTOMY | INCREASED SURVIVAL | PROGRESSION | REVEALS | Prostatic Neoplasms - metabolism | Epigenesis, Genetic | Humans | Male | RNA, Long Noncoding - genetics | Disease Progression | Adenocarcinoma - metabolism | Estrogen Receptor alpha - genetics | Prostatic Neoplasms - genetics | Sequence Analysis, RNA | Chromatin Immunoprecipitation | Cell Line, Tumor | Estrogen Receptor alpha - metabolism | Adenocarcinoma - genetics | RNA, Long Noncoding - metabolism | Index Medicus
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2. Full Text An oestrogen-receptor-α-bound human chromatin interactome
by Fullwood, Melissa J and Liu, Mei Hui and Pan, You Fu and Liu, Jun and Xu, Han and Mohamed, Yusoff Bin and Orlov, Yuriy L and Velkov, Stoyan and Ho, Andrea and Mei, Poh Huay and Chew, Elaine G. Y and Huang, Phillips Yao Hui and Welboren, Willem-Jan and Han, Yuyuan and Ooi, Hong Sain and Ariyaratne, Pramila N and Vega, Vinsensius B and Luo, Yanquan and Tan, Peck Yean and Choy, Pei Ye and Wansa, K. D. Senali Abayratna and Zhao, Bing and Lim, Kar Sian and Leow, Shi Chi and Yow, Jit Sin and Joseph, Roy and Li, Haixia and Desai, Kartiki V and Thomsen, Jane S and Lee, Yew Kok and Karuturi, R. Krishna Murthy and Herve, Thoreau and Bourque, Guillaume and Stunnenberg, Hendrik G and Ruan, Xiaoan and Cacheux-Rataboul, Valere and Sung, Wing-Kin and Liu, Edison T and Wei, Chia-Lin and Cheung, Edwin and Ruan, Yijun
Nature, ISSN 0028-0836, 11/2009, Volume 462, Issue 7269, pp. 58 - 64
Genomes are organized into high-level three-dimensional structures, and DNA elements separated by long genomic distances can in principle interact... 
FOXA1 | ELEMENTS | HUMAN GENOME | CHIP | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | GENES | CHROMOSOME CONFORMATION | DEFINES | BINDING-SITES | REVEALS | Cell Line | Chromatin - metabolism | Cross-Linking Reagents | Reproducibility of Results | Humans | Transcriptional Activation | Promoter Regions, Genetic - genetics | Sequence Analysis, DNA | Formaldehyde | Genome, Human - genetics | Chromatin Immunoprecipitation | Protein Binding | Estrogen Receptor alpha - metabolism | Transcription, Genetic | Binding Sites | Chromatin - genetics | Index Medicus
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3. Full Text Emergence of constitutively active estrogen receptor-α mutations in pretreated advanced estrogen receptor-positive breast cancer
by Jeselsohn, Rinath and Yelensky, Roman and Buchwalter, Gilles and Frampton, Garrett and Meric-Bernstam, Funda and Gonzalez-Angulo, Ana Maria and Ferrer-Lozano, Jaime and Perez-Fidalgo, Jose A and Cristofanilli, Massimo and Goḿez, Henry and Arteaga, Carlos L and Giltnane, Jennifer and Balko, Justin M and Cronin, Maureen T and Jarosz, Mirna and Sun, James and Hawryluk, Matthew and Lipson, Doron and Otto, Geoff and Ross, Jeffrey S and Dvir, Addie and Soussan-Gutman, Lior and Wolf, Ido and Rubinek, Tamar and Gilmore, Lauren and Schnitt, Stuart and Come, Steven E and Pusztai, Lajos and Stephens, Philip and Brown, Myles and Miller, Vincent A
Clinical Cancer Research, ISSN 1078-0432, 04/2014, Volume 20, Issue 7, pp. 1757 - 1767
Purpose: We undertook this study to determine the prevalence of estrogen receptor (ER) alpha ( ESR1) mutations throughout the natural history of... 
ONCOLOGY | POSTMENOPAUSAL WOMEN | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | MCF-7 Cells | Breast Neoplasms - pathology | Neoplasms, Hormone-Dependent - pathology | Humans | Female | Estrogen Receptor alpha - biosynthesis | High-Throughput Nucleotide Sequencing | Mutation | Neoplasm Staging | Neoplasms, Hormone-Dependent - genetics | Index Medicus | ESR1 mutations | breast cancer | estrogen receptor | endocrine resistance
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4. Full Text Oestrogen receptor alpha in pulmonary hypertension
by Wright, Audrey F and Ewart, Marie-Ann and Mair, Kirsty and Nilsen, Margaret and Dempsie, Yvonne and Loughlin, Lynn and MacLean, Margaret R
Cardiovascular Research, ISSN 0008-6363, 05/2015, Volume 106, Issue 2, pp. 206 - 216
Aims Pulmonary arterial hypertension (PAH) occurs more frequently in women with mutations in bone morphogenetic protein receptor type 2 (BMPR2) and... 
Oestrogen | BMPR2 | Oestrogen receptor alpha | Serotonin | Pulmonary hypertension | SEROTONIN TRANSPORTER | CARDIAC & CARDIOVASCULAR SYSTEMS | DISEASE MANAGEMENT | BONE MORPHOGENETIC PROTEIN | HYPOXIA | ARTERIAL-HYPERTENSION | CARDIAC-HYPERTROPHY | SMOOTH-MUSCLE-CELLS | MICE | GENDER-DIFFERENCES | GENE-TRANSCRIPTION | Bone Morphogenetic Protein Receptors, Type II - genetics | Humans | Cell Proliferation - physiology | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Bone Morphogenetic Protein Receptors, Type II - metabolism | Serotonin Plasma Membrane Transport Proteins - metabolism | Animals | Estrogen Receptor alpha - metabolism | Lung - metabolism | Mice | Hypertension, Pulmonary - pathology | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Index Medicus | Original
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5. Full Text Disinfection Byproducts Bind Human Estrogen Receptor-[alpha]
by Holmes, Breanne E and Smeester, Lisa and Fry, Rebecca C and Weinberg, Howard S
Environmental Toxicology and Chemistry, ISSN 0730-7268, 05/2019, Volume 38, Issue 5, p. 956
Disinfection byproducts are formed during most drinking water treatment and presently number >800, some of which are implicated in human health outcomes... 
Medical research | Estrogen | Medicine, Experimental | Phenols | Infection control
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6. Full Text MicroRNA-221/222 negatively regulates estrogen receptor α and is associated with tamoxifen resistance in breast cancer
by Zhao, Jian-Jun and Lin, Jianhong and Yang, Hua and Kong, William and He, Lili and Ma, Xu and Coppola, Domenico and Cheng, Jin Q
Journal of Biological Chemistry, ISSN 0021-9258, 11/2008, Volume 283, Issue 45, pp. 31079 - 31086
A search for regulators of estrogen receptor alpha(ER alpha) expression has yielded a set of microRNAs (miRNAs) for which expression is specifically elevated... 
SURVIVAL | MIR-222 | METASTASIS | INHIBITION | MESSENGER-RNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN EXPRESSION | PROLIFERATION | MICRORNAS | CELL-LINES | ER-ALPHA | Antineoplastic Agents, Hormonal - pharmacology | Apoptosis - drug effects | Neoplasm Proteins - biosynthesis | Humans | 3' Untranslated Regions - genetics | 3' Untranslated Regions - metabolism | MicroRNAs - metabolism | RNA, Neoplasm - metabolism | Breast Neoplasms - metabolism | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Tamoxifen - pharmacology | Cell Line, Tumor | RNA, Neoplasm - genetics | Female | Estrogen Receptor alpha - biosynthesis | MicroRNAs - genetics | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Drug Resistance, Neoplasm - drug effects | Index Medicus | Molecular Basis of Cell and Developmental Biology
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7. Full Text Androgen receptor inhibits estrogen receptor-α activity and is prognostic in breast cancer
by Peters, Amelia A and Buchanan, Grant and Ricciardelli, Carmela and Bianco-Miotto, Tina and Centenera, Margaret M and Harris, Jonathan M and Jindal, Shalini and Segara, Davendra and Jia, Li and Moore, Nicole L and Henshall, Susan M and Birrell, Stephen N and Coetzee, Gerhard A and Sutherland, Robert L and Butler, Lisa M and Tilley, Wayne D
Cancer Research, ISSN 0008-5472, 08/2009, Volume 69, Issue 15, pp. 6131 - 6140
There is emerging evidence that the balance between estrogen receptor-alpha (ER alpha) and androgen receptor (AR) signaling is a critical determinant of growth... 
RESPONSE ELEMENT | CELLS | ONCOLOGY | THYROID-HORMONE | DNA RECOGNITION | TRANSACTIVATION | TAMOXIFEN | MEDROXYPROGESTERONE ACETATE | GROWTH | LIGAND-BINDING DOMAIN | EXPRESSION | Prognosis | Response Elements | Signal Transduction | DNA, Neoplasm - metabolism | Humans | Receptors, Androgen - metabolism | Transcriptional Activation | Cercopithecus aethiops | Models, Molecular | Estrogen Receptor alpha - antagonists & inhibitors | Breast Neoplasms - metabolism | Animals | Estrogen Receptor alpha - genetics | Receptors, Androgen - genetics | Breast Neoplasms - pathology | Receptors, Androgen - biosynthesis | Cell Line, Tumor | Carcinoma, Ductal, Breast - pathology | Estrogen Receptor alpha - metabolism | Estrogen Receptor alpha - biosynthesis | COS Cells | Carcinoma, Ductal, Breast - metabolism | Index Medicus
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8. Full Text Estrogen Receptor-Alpha Promotes Alternative Macrophage Activation during Cutaneous Repair
by Campbell, Laura and Emmerson, Elaine and Williams, Helen and Saville, Charis R and Krust, Andrée and Chambon, Pierre and Mace, Kimberly A and Hardman, Matthew J
Journal of Investigative Dermatology, ISSN 0022-202X, 09/2014, Volume 134, Issue 9, pp. 2447 - 2457
Efficient local monocyte/macrophage recruitment is critical for tissue repair. Recruited macrophages are polarized toward classical (proinflammatory) or... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MIGRATION INHIBITORY FACTOR | OXIDE SYNTHASE ISOFORMS | IN-VIVO | NITRIC-OXIDE | MICE | EXPRESSION | VENOUS LEG ULCERS | ER-ALPHA | BETA | DERMATOLOGY | Estrogens - pharmacology | Ovariectomy | Mice, Inbred C57BL | Macrophages, Peritoneal - immunology | Estrogen Receptor alpha - immunology | Macrophages - cytology | Estrogen Receptor beta - immunology | Wound Healing - immunology | Macrophages, Peritoneal - cytology | Mice, Knockout | Estrogen Receptor beta - metabolism | Signal Transduction - immunology | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Estrogen Receptor alpha - metabolism | Estrogens - metabolism | Macrophages, Peritoneal - metabolism | Macrophages - immunology | Nitric Oxide Synthase Type II - metabolism | Index Medicus
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9. Full Text Evidence for a Mitochondrial Regulatory Pathway Defined by Peroxisome Proliferator–Activated Receptor-γ Coactivator-1α, Estrogen-Related Receptor-α, and Mitofusin 2
by Francesc X. Soriano and Marc Liesa and Daniel Bach and David C. Chan and Manuel Palacín and Antonio Zorzano
Diabetes, ISSN 0012-1797, 06/2006, Volume 55, Issue 6, pp. 1783 - 1791
Evidence for a Mitochondrial Regulatory Pathway Defined by Peroxisome Proliferator–Activated Receptor-γ Coactivator-1α, Estrogen-Related Receptor-α, and... 
COX-II, cytochrome c oxidase subunit-II | Mfn2, mitofusin 2 | PGC, peroxisome proliferator-activated receptor-γ coactivator | MEF2, myocyte enhancer factor-2 | Errα, estrogen-related receptor-α | 2A GENE | HEPATIC GLUCONEOGENESIS | BIOGENESIS | ERR-ALPHA | PGC-1-ALPHA | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | PGC-1 | TRANSCRIPTIONAL COACTIVATOR | ENERGY-METABOLISM | HUMAN SKELETAL-MUSCLE | GTP Phosphohydrolases | Gene Expression - drug effects | Rats, Wistar | Humans | Mitochondria, Muscle - metabolism | Male | Muscle, Skeletal - metabolism | Mitochondrial Proteins - genetics | Muscle, Skeletal - cytology | RNA, Messenger - metabolism | Heat-Shock Proteins - genetics | Transfection | Chromatin Immunoprecipitation | Mitochondrial Proteins - metabolism | Muscle, Skeletal - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Dioxoles - pharmacology | Estrogen Receptor alpha - metabolism | Electrophoretic Mobility Shift Assay | Membrane Proteins - metabolism | Cold Temperature | Membrane Proteins - genetics | Heat-Shock Proteins - metabolism | RNA, Messenger - genetics | Cells, Cultured | Rats | Signal Transduction - genetics | Transcription Factors - genetics | Blotting, Western | Transcription Factors - metabolism | Animals | Estrogen Receptor alpha - genetics | Signal Transduction - drug effects | Protein Binding | Signal Transduction - physiology | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Mice | HeLa Cells | Index Medicus | Abridged Index Medicus
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10. Full Text Neuroprotection mediated through estrogen receptor-α in astrocytes
by Rory D. Spence and Mary E. Hamby and Elizabeth Umeda and Noriko Itoh and Sienmi Du and Amy J. Wisdom and Yuan Cao and Galyna Bondar and Jeannie Lam and Yan Ao and Francisco Sandoval and Silvie Suriany and Michael V. Sofroniew and Rhonda R. Voskuhl
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2011, Volume 108, Issue 21, pp. 8867 - 8872
Estrogen has well-documented neuroprotective effects in a variety of clinical and experimental disorders of the CNS, including autoimmune inflammation,... 
Spinal cord | Nervous system diseases | Astrocytes | Neurons | Estrogens | Central nervous system | Central nervous system diseases | Estrogen receptors | Ligands | Inflammation | Astrogliosis | Conditional knockout | Multiple sclerosis | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | ER-BETA | multiple sclerosis | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC IMPLICATIONS | astrogliosis | MULTIPLE-SCLEROSIS | DISEASE | REACTIVE ASTROCYTES | SPINAL-CORD-INJURY | BRAIN INJURY | RAT-BRAIN | conditional knockout | EXPRESSION | Neurons - pathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Astrocytes - pathology | Cells, Cultured | Neurodegenerative Diseases - prevention & control | Estrogen Receptor alpha - deficiency | Mice, Knockout | Animals | Neuroprotective Agents - pharmacology | Inflammation - prevention & control | Mice | Estrogen Receptor alpha - physiology | Neuroprotection | Animal models | Stroke | Neurodegenerative diseases | Gene deletion | Experimental allergic encephalomyelitis | Sex hormones | Injuries | Immune system | Index Medicus | Biological Sciences
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11. Full Text The Cholesterol Metabolite 27-Hydroxycholesterol Promotes Atherosclerosis via Proinflammatory Processes Mediated by Estrogen Receptor Alpha
by Umetani, Michihisa and Umetani, Junko and Ghosh, Pritam and Ishikawa, Tomonori and Ahmed, Mohamed and Mineo, Chieko and Shaul, Philip W
Cell Metabolism, ISSN 1550-4131, 07/2014, Volume 20, Issue 1, pp. 172 - 182
Oxysterols are cholesterol metabolites that serve multiple functions in lipid metabolism, including as liver X receptor (LXR) ligands. 27-hydroxycholesterol... 
ADHESION | SIGNALING PATHWAYS | HEART-DISEASE | DNA-BINDING | LIVER X RECEPTORS | INFLAMMATION | TRANSCRIPTION | ENDOCRINOLOGY & METABOLISM | NF-KAPPA-B | INTERLEUKIN-6 GENE-EXPRESSION | STEROL 27-HYDROXYLASE | CELL BIOLOGY | Steroid Hydroxylases - metabolism | Apolipoproteins E - deficiency | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Apolipoproteins E - metabolism | Cytochrome P450 Family 7 | RNA Interference | Estrogen Receptor alpha - metabolism | Female | Cytokines - genetics | Steroid Hydroxylases - genetics | Macrophages - immunology | Cell Line | Atherosclerosis - pathology | NF-KappaB Inhibitor alpha | Cytokines - metabolism | Endothelial Cells - metabolism | Inflammation | Cell Adhesion - drug effects | Cholesterol - metabolism | Hydroxycholesterols - pharmacology | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Estrogen Receptor alpha - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Endothelial Cells - cytology | Apolipoproteins E - genetics | Steroid Hydroxylases - deficiency | Hydroxycholesterols - metabolism | Macrophages - drug effects | Mice | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Metabolites | Atherosclerosis | Estrogen | Cytochrome P-450 | Physiological aspects | Phenols | Cholesterol | Prevention | Blood circulation disorders | Liver | Endothelium | Index Medicus
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12. Full Text MMTV-Espl1 transgenic mice develop aneuploid, estrogen receptor alpha (ER)-positive mammary adenocarcinomas
by Mukherjee, M and Ge, G and Zhang, N and Edwards, D.G and Sumazin, P and Sharan, S.K and Rao, P.H and Medina, D and Pati, D
Oncogene, ISSN 0950-9232, 11/2014, Volume 33, Issue 48, pp. 5511 - 5522
Separase, a protease encoded by the ESPL1 gene, cleaves the chromosomal cohesin during mitosis. Separase protein and transcripts are overexpressed in a wide... 
aneuploidy | mammary tumor animal model | cohesin | Espl1 | Separase | p53 | MOLECULAR CHARACTERIZATION | CELLS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLONAL EVOLUTION | HUMAN BREAST-CANCER | TUMORS | CHROMOSOMAL INSTABILITY | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | TUMORIGENESIS | Immunohistochemistry | Separase - genetics | Adenocarcinoma - pathology | Mice, Inbred C57BL | Aneuploidy | In Situ Hybridization, Fluorescence | Mice, Transgenic | Mammary Neoplasms, Experimental - metabolism | Blotting, Western | Mammary Neoplasms, Experimental - genetics | Adenocarcinoma - metabolism | Animals | Comparative Genomic Hybridization | Fluorescent Antibody Technique | Mammary Neoplasms, Experimental - pathology | Mammary Tumor Virus, Mouse | Female | Adenocarcinoma - genetics | Estrogen Receptor alpha - biosynthesis | Mice | Separase - metabolism | Adenocarcinoma | Receptors | Proteases | Estrogen | Causes of | Genetic aspects | Breast cancer | Gene expression | Health aspects | Transgenic animals | Neurons | Rodents | Adenoviruses | Cancer | Index Medicus | Chromosomal segregation | Animal model | Cohesin | Mammary Cancer
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13. Full Text Cooperative interaction between retinoic acid receptor-α and estrogen receptor in breast cancer
by Ross-Innes, Caryn S and Stark, Rory and Holmes, Kelly A and Schmidt, Dominic and Spyrou, Christiana and Russell, Roslin and Massie, Charlie E and Vowler, Sarah L and Eldridge, Matthew and Carroll, Jason S
Genes and Development, ISSN 0890-9369, 01/2010, Volume 24, Issue 2, pp. 171 - 182
Retinoic acid receptor-alpha (RAR alpha) is a known estrogen target gene in breast cancer cells. The consequence of RAR alpha induction by estrogen was... 
Estrogen receptor | Retinoic acid receptor-a | Chromatin | Breast cancer | Transcription | retinoic acid receptor-alpha | transcription | estrogen receptor | CYCLIN D1 | NUCLEAR RECEPTORS | C-MYC | DEVELOPMENTAL BIOLOGY | chromatin | CELL-LINES | CELL BIOLOGY | GENE | RESPONSE ELEMENTS | GENETICS & HEREDITY | BINDING-SITES | TRANSCRIPTION FACTOR | EXPRESSION | ACUTE PROMYELOCYTIC LEUKEMIA | Receptors, Estrogen - metabolism | Humans | Gene Expression Regulation, Neoplastic | Protein Binding | Female | Ligands | Receptors, Retinoic Acid - metabolism | DNA - metabolism | Estrogens - metabolism | Breast Neoplasms - metabolism | Index Medicus | Research Paper | retinoic acid receptor-α
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