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Journal of Neuroscience, ISSN 0270-6474, 09/2001, Volume 21, Issue 17, pp. 6933 - 6939
Inflammatory pain, characterized by a decrease in mechanical nociceptive threshold (hyperalgesia), arises through actions of inflammatory mediators, many of... 
Epinephrine | Protein kinase A | Nociceptor | β-adrenergic receptor | Pain | Mitogen and extracellular signal-regulated kinase kinase | Ras | Extracellular signal-regulated kinase | Mitogen-activated protein kinase | Protein kinase Cε | G-protein | Hyperalgesia | nociceptor | extracellular signal-regulated kinase | pain | C TRANSLOCATION | MAP KINASE | mitogen and extracellular signal-regulated kinase kinase | DEPENDENT PROTEIN-KINASE | epinephrine | NEUROSCIENCES | SENSORY NEURONS | NERVE GROWTH-FACTOR | mitogen-activated protein kinase | protein kinase C epsilon | beta-adrenergic receptor | NEURITE OUTGROWTH | MECHANICAL HYPERALGESIA | INFLAMMATORY PAIN | hyperalgesia | B-RAF | protein kinase A | Hyperalgesia - chemically induced | ras Proteins - metabolism | Heterotrimeric GTP-Binding Proteins - metabolism | Male | Neurons - cytology | Ganglia, Spinal - cytology | Ganglia, Spinal - physiopathology | Dose-Response Relationship, Drug | Heterotrimeric GTP-Binding Proteins - antagonists & inhibitors | Nociceptors - physiopathology | Isoenzymes - metabolism | Protein Kinase C - metabolism | Neurons - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Neurons - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Adrenergic beta-2 Receptor Antagonists | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Rats | Protein Kinase C - antagonists & inhibitors | Enzyme Activation - drug effects | Receptors, Adrenergic, beta-2 - metabolism | Hyperalgesia - physiopathology | Animals | Signal Transduction - drug effects | Mice | Ganglia, Spinal - drug effects | Pain Measurement | Nociceptors - drug effects | Isoenzymes - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 | Crosses, Genetic | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Protein Kinase C-epsilon | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 02/2014, Volume 63, Issue 2, pp. 514 - 525
The number and activity of brown adipocytes are linked to the ability of mammals to resist body fat accumulation. In some conditions, certain white adipose... 
OBESITY | GENE | ENDOCRINOLOGY & METABOLISM | MUSCLE | RECEPTOR | PROLIFERATION | DIFFERENTIATION | EXPRESSION | FAT-CELL | EXERCISE | ADIPOSE-TISSUE | MAP Kinase Signaling System - physiology | Nitriles - pharmacology | Adipose Tissue, White - metabolism | Caenorhabditis elegans Proteins - metabolism | Adipocytes - cytology | Male | Adipocytes - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | Recombinant Proteins | Extracellular Signal-Regulated MAP Kinases - genetics | Pichia - metabolism | Membrane Transport Proteins - genetics | Dietary Fats | Membrane Transport Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Butadienes - pharmacology | Fibronectins - pharmacology | Fibronectins - administration & dosage | Mice, Inbred C57BL | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Imidazoles - pharmacology | 3T3-L1 Cells | Rats, Sprague-Dawley | Animals | Adipocytes - metabolism | Pichia - genetics | Protein Binding | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Fibronectins - genetics | Mice | Pyridines - pharmacology | Mutation | Mitochondrial Uncoupling Proteins | Caenorhabditis elegans Proteins - genetics | Adipose Tissue, White - drug effects | Physiological research | Cellular signal transduction | Research | Identification and classification | Membrane proteins | Proteins | Homeostasis | Diabetes | Kinases | Genes | Cells | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Natural Products, ISSN 0163-3864, 04/2013, Volume 76, Issue 4, pp. 664 - 671
Six new sesquiterpenoids, aristoyunnolins A-F (1-6), an artifact of isolation [7-O -ethyl madolin W (7)], and 12 known analogues were isolated from stems of... 
PATHWAYS | MANSHURIENSIS | CHEMISTRY, MEDICINAL | MOLLISSIMA | ROOTS | PHARMACOLOGY & PHARMACY | CONSTITUENTS | STEM | PLANT SCIENCES | Sesquiterpenes - isolation & purification | MAP Kinase Signaling System - physiology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Drugs, Chinese Herbal - pharmacology | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - metabolism | Mitogen-Activated Protein Kinase 3 - pharmacology | Flavonoids - pharmacology | Molecular Structure | Plant Stems - chemistry | Phosphorylation - drug effects | Extracellular Signal-Regulated MAP Kinases - physiology | Drugs, Chinese Herbal - isolation & purification | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Sesquiterpenes - chemistry | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Blotting, Western | Aristolochia - chemistry | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinases - physiology | Signal Transduction - drug effects | Drugs, Chinese Herbal - chemistry | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Sesquiterpenes - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Endocrinology, ISSN 0013-7227, 10/2017, Volume 158, Issue 10, pp. 3676 - 3683
We previously showed that thyrotropin (TSH)/insulinlike growth factor (IGF)-1 receptor cross-talk appears to be involved in Graves' orbitopathy (GO)... 
THYROID-CELLS | STIMULATION | DISEASE | GROWTH | ENDOCRINOLOGY & METABOLISM | ORBITAL FIBROBLASTS | TSH | HYPERTHYROIDISM | GRAVES OPHTHALMOPATHY PATHOGENESIS | EXPRESSION | HUMAN THYROTROPIN RECEPTOR | Graves Ophthalmopathy | Receptor, IGF Type 1 - metabolism | Insulin-Like Growth Factor I - pharmacology | Receptor Cross-Talk | Receptors, Thyrotropin - metabolism | Humans | Thyroid Epithelial Cells - metabolism | Mitogen-Activated Protein Kinase 1 - drug effects | Symporters - metabolism | Mitogen-Activated Protein Kinase 3 - drug effects | Hyaluronic Acid - secretion | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Thyrotropin - pharmacology | Fibroblasts - drug effects | HEK293 Cells | Symporters - drug effects | Phosphorylation - drug effects | Thyroid Epithelial Cells - drug effects | Fibroblasts - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Insulin-like growth factor I | Pathogenesis | Activation | Insulin-like growth factors | Kinases | Synergism | Pertussis toxin | Proteins | Pertussis | Synergistic effects | Hyaluronan | Receptors | Fibroblasts | Inhibition | Thyroid | Iodides | Secretion | Extracellular signal-regulated kinase | Cultures | Thyroid-stimulating hormone receptors | Signaling | Thyroid-stimulating hormone | Sodium | Thyrocytes | Kidney transplantation | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2011, Volume 286, Issue 50, pp. 42981 - 42991
Background: Listeria monocytogenes is an intracellular pathogen that invades the host cytoplasm. Results: Cells deficient in TLR2 and NOD2 show defective... 
ADAPTIVE IMMUNITY | CROHN-DISEASE | EARLY PHASE | CRYOPYRIN INFLAMMASOME | EPITHELIAL-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HOST-DEFENSE | INTRACELLULAR PATHOGENS | INFECTION | GENOME-WIDE ASSOCIATION | INNATE IMMUNITY | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Cell Line | Microscopy, Electron, Transmission | Toll-Like Receptor 2 - genetics | Cells, Cultured | Nod2 Signaling Adaptor Protein - genetics | Signal Transduction - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Listeria monocytogenes - immunology | Macrophages - cytology | Toll-Like Receptor 2 - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Blotting, Western | Autophagy - immunology | Listeria monocytogenes - pathogenicity | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Nod2 Signaling Adaptor Protein - metabolism | Mice, Mutant Strains | Listeriosis - immunology | Mice | Macrophages - immunology | Index Medicus | Toll-Like Receptor 2 | Signal Transduction | Receptor-Interacting Protein Serine-Threonine Kinases | Listeria monocytogenes | Listeriosis | Nod2 Signaling Adaptor Protein | Extracellular Signal-Regulated MAP Kinases | Macrophages | Autophagy | Life Sciences | Development Biology | Toll-like receptors (TLR) | Immunology | Nod-like receptors (NLR) | ERK
Journal Article
Cancer Research, ISSN 0008-5472, 12/2009, Volume 69, Issue 24, pp. 9228 - 9235
Journal Article
Journal Article
Biological Psychiatry, ISSN 0006-3223, 02/2011, Volume 69, Issue 3, pp. 218 - 227
Background: Activation of the extracellular signal-regulated kinase (ERK) in the striatum is crucial for long-term behavioral alterations induced by drugs of... 
Src family kinases | Cocaine | NR2B containing-NMDA receptor | dopamine | extracellular signal-regulated kinase (ERK) | glutamate
Journal Article
Journal Article
11/2013
Nicotine Addiction is a chronic relapsing disorder. Relapse risk persists despite years of abstinence. Drug-associated cues have been demonstrated to induce... 
0347 | Extracellular-signal Regulated Kinase (ERK) | incubation of nicotine craving | cAMP Response Element Binding Protein (CREB) | nicotine addiction | 0419 | Fos
Dissertation
Cancer Biology & Therapy, ISSN 1538-4047, 05/2019, Volume 20, Issue 5, pp. 653 - 665
Background: Neuroblastoma is the commonest malignancy in neonates. Long non-coding RNA (lncRNA) RNA component of mitochondrial RNA processing endoribonuclease... 
neuroblastoma | microRNA-206 | extracellular signal-regulated kinases | TACR1 | RMRP | CELLS | APOPTOSIS | ACTIVATION | NEUROKININ-1 RECEPTOR | PROLIFERATION | CHILDHOOD | ANTITUMORAL ACTION | L-733,060 | ONCOLOGY | SUBSTANCE-P | EXPRESSION
Journal Article
The Journal of allergy and clinical immunology, 07/2019
15-Lipoxygenase 1 (15LO1) is expressed in airway epithelial cells in patients with type 2-high asthma in association with eosinophilia. Chronic rhinosinusitis... 
Index Medicus | Abridged Index Medicus
Journal Article
Journal Article