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The Journal of Physiology, ISSN 0022-3751, 05/2004, Volume 556, Issue 3, pp. 983 - 1000
Muscular adaptation to physical exercise has previously been described as a repair process following tissue damage. Recently, evidence has been published to... 
INDUCED INJURY | PHYSIOLOGY | IMMUNOREACTIVITY | ECCENTRIC EXERCISE | REGENERATION | LEUKEMIA INHIBITORY FACTOR | DENERVATED HUMAN MUSCLE | SATELLITE CELLS | STRENGTH | FACTOR-I | EXPRESSION | NEUROSCIENCES | Immunohistochemistry | Granulocytes - cytology | Cytokines - analysis | Humans | Middle Aged | DNA-Binding Proteins - analysis | Ki-67 Antigen - metabolism | Male | Muscle, Skeletal - metabolism | Proteins - analysis | Pain - metabolism | fas Receptor - metabolism | Fascia - chemistry | Oxygen Consumption - physiology | fas Receptor - analysis | Antigens, CD - metabolism | Antigens, CD - analysis | Running - physiology | C-Reactive Protein - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator | CD11b Antigen - analysis | Receptors, Cytokine - metabolism | Testosterone - blood | C-Reactive Protein - analysis | Hormones - blood | Leukocytes - chemistry | Interleukin-6 - metabolism | Lymphocytes - metabolism | CD56 Antigen - analysis | Lymphocytes - cytology | Insulin-Like Growth Factor I - analysis | Muscle, Skeletal - physiology | CD3 Complex - metabolism | Leukemia Inhibitory Factor Receptor alpha Subunit | Regression Analysis | Pain - physiopathology | Receptors, Cytokine - analysis | Adolescent | Antigens, Differentiation, Myelomonocytic - analysis | Creatine Kinase - metabolism | Transcription Factors - analysis | Leukocytes - metabolism | Insulin-Like Growth Factor I - metabolism | Receptors, OSM-LIF | Interleukin-6 - analysis | Monocytes - cytology | Receptors, Cell Surface - analysis | Monocytes - metabolism | Receptors, Aryl Hydrocarbon - analysis | DNA-Binding Proteins - metabolism | Testosterone - metabolism | Flow Cytometry | Interleukin-6 - blood | Exercise Test - methods | Muscle, Skeletal - chemistry | Fascia - metabolism | Heart Rate - physiology | Receptors, Aryl Hydrocarbon - metabolism | Adult | Female | Leukocyte Count | Leukemia Inhibitory Factor | Isometric Contraction - physiology | Cytokines - blood | Leukocytes - cytology | CD56 Antigen - metabolism | Creatine Kinase - blood | Cytokines - metabolism | Receptors, Cell Surface - metabolism | Transcription Factors - metabolism | CD3 Complex - analysis | Ki-67 Antigen - analysis | Proteins - metabolism | Hormones - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | CD11b Antigen - metabolism | Growth Substances - metabolism | Pain - diagnosis | Index Medicus | Research Papers | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, pp. e0128269 - e0128269
Although B cell depletion therapy (BCDT) is effective in a subset of rheumatoid arthritis (RA) patients, both mechanisms and biomarkers of response are poorly... 
SYSTEMIC-LUPUS-ERYTHEMATOSUS | RITUXIMAB THERAPY | PREDICT | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | DISEASE | TISSUE | SUBSETS | IDENTIFICATION | EXPRESSION | Humans | Middle Aged | Receptors, Complement 3d - immunology | Male | Immunoglobulin D - immunology | fas Receptor - analysis | Rituximab - therapeutic use | fas Receptor - immunology | Ki-67 Antigen - immunology | Tumor Necrosis Factor-alpha - immunology | Female | B-Lymphocytes - pathology | Interleukin-10 - analysis | Antirheumatic Agents - therapeutic use | Immunoglobulin D - analysis | Biomarkers - analysis | Arthritis, Rheumatoid - pathology | Tumor Necrosis Factor-alpha - analysis | Receptors, Complement 3d - analysis | Arthritis, Rheumatoid - therapy | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Ki-67 Antigen - analysis | Aged | Arthritis, Rheumatoid - immunology | Interleukin-10 - immunology | Lymphocyte Depletion - methods | Rheumatoid factor | Arthritis | Systemic lupus erythematosus | B cells | Comparative analysis | Biological markers | CD27 antigen | Lymphocyte receptors | Therapy | Multiple sclerosis | Populations | Disease | Immunoglobulin D | Blood | Immunology | Peripheral blood | Cell cycle | Interleukin 1 | Bone marrow | Tumor necrosis factor-TNF | Bioindicators | Pretreatment | Lupus | Immunological memory | Memory cells | Cytokines | Repopulation | Secretion | CD95 antigen | Abnormalities | Rheumatology | T cell receptors | Inflammation | Patients | Medicine | Depletion | Lymphocytes B | Tumor necrosis factor | Rheumatoid arthritis | Interleukin 10 | CD20 antigen | Biomarkers | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2003, Volume 125, Issue 2, pp. 437 - 443
Background & Aims: The pathogenesis of nonalcoholic steatohepatitis (NASH) remains poorly understood. Although apoptosis is a common mechanism of liver injury,... 
PATHOGENESIS | FIBROSIS | HEPATITIS | PATHWAY | FATTY LIVER-DISEASE | MOUSE | INJURY | RECEPTOR | DEATH | GASTROENTEROLOGY & HEPATOLOGY | DAMAGE | Immunohistochemistry | In Situ Nick-End Labeling | Fatty Liver - metabolism | Fatty Liver - pathology | Humans | Middle Aged | Fatty Liver - therapy | Hepatocytes - pathology | Male | fas Receptor - analysis | Adult | Female | Aged | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 2017, Volume 109, Issue 6, p. djw283
Background: Pancreatic cancer is one of the cancers where anti-PD-L1/PD-1 immunotherapy has been unsuccessful. What confers pancreatic cancer resistance to... 
TARGET GENES | ANTI-PD-L1 ANTIBODY | T-CELL IMMUNITY | ONCOLOGY | IFN-GAMMA | METHYLTRANSFERASE ACTIVITY | FACTOR-KAPPA-B | PROMOTES | TUMOR MICROENVIRONMENT | CHECKPOINT BLOCKADE | HISTONE | Neoplasm Transplantation | Myeloid-Lymphoid Leukemia Protein - metabolism | Tumor Escape | Pancreatic Neoplasms - metabolism | Epigenesis, Genetic | Humans | Programmed Cell Death 1 Receptor - analysis | Antibodies, Monoclonal - therapeutic use | Histone-Lysine N-Methyltransferase - analysis | RNA, Messenger - metabolism | Histone-Lysine N-Methyltransferase - antagonists & inhibitors | Immunotherapy | Female | Indoles - pharmacology | Myeloid-Lymphoid Leukemia Protein - analysis | Promoter Regions, Genetic | Histone-Lysine N-Methyltransferase - genetics | Carcinoma - immunology | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Programmed Cell Death 1 Receptor - metabolism | Pancreatic Neoplasms - genetics | Down-Regulation - drug effects | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Piperazines - pharmacology | Tumor Microenvironment - immunology | B7-H1 Antigen - metabolism | T-Lymphocytes, Cytotoxic - chemistry | Animals | Histone-Lysine N-Methyltransferase - metabolism | Myeloid-Lymphoid Leukemia Protein - genetics | Pancreatic Neoplasms - immunology | Cell Line, Tumor | Carcinoma - genetics | Carcinoma - therapy | Indoles - therapeutic use | B7-H1 Antigen - analysis | Mice | Carcinoma - metabolism | Fas Ligand Protein - genetics | DNA Methylation - drug effects | Myeloid-Lymphoid Leukemia Protein - antagonists & inhibitors | Pancreatic Neoplasms - therapy | Care and treatment | Pancreatic cancer | Genetic aspects | Diagnosis | Gene expression | Health aspects | Membrane proteins | Index Medicus
Journal Article
Journal Article
Brazilian Journal of Medical and Biological Research, ISSN 0100-879X, 2014, Volume 47, Issue 8, pp. 662 - 669
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 11/2006, Volume 12, Issue 22, pp. 6808 - 6816
Purpose: The purpose of the present study was to evaluate granulocyte macrophage colony-stimulating factor (GM-CSF)–secreting tumor cell immunotherapy in... 
regulatory T cells | dendritic cells | Cancer immunotherapy | GM-CSF | VEGF | LUNG-CANCER | COLONY-STIMULATING FACTOR | DENDRITIC CELLS | CLINICAL-TRIAL | ONCOLOGY | TUMOR VACCINE | IN-VIVO | MEDIATED GENE-TRANSFER | DIFFERENTIATION | AUGMENTS ANTITUMOR IMMUNITY | Recombinant Proteins - therapeutic use | Immunotherapy - methods | Cell Count | Gene Expression Regulation, Neoplastic | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Granulocyte-Macrophage Colony-Stimulating Factor - therapeutic use | fas Receptor - analysis | Granulocyte-Macrophage Colony-Stimulating Factor - secretion | Receptors, Vascular Endothelial Growth Factor - metabolism | T-Lymphocytes, Regulatory - cytology | Neoplasms, Experimental - mortality | Receptors, Vascular Endothelial Growth Factor - genetics | Tumor Cells, Cultured | Neoplasms, Experimental - secretion | Colonic Neoplasms - therapy | Lymphocytes, Tumor-Infiltrating - cytology | Recombinant Proteins - metabolism | Melanoma, Experimental - therapy | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Mice, Transgenic | Treatment Outcome | Combined Modality Therapy | Recombinant Proteins - genetics | Fas Ligand Protein - physiology | T-Lymphocytes, Regulatory - drug effects | Animals | Survival Analysis | Carcinoma - therapy | Dendritic Cells - cytology | Mice | Mice, Inbred BALB C | Apoptosis | Genetic Therapy - methods | Index Medicus
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 07/2016, Volume 34, Issue 19, pp. 2294 - 2302
Journal Article