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Blood, ISSN 0006-4971, 07/2017, Volume 130, Issue 2, pp. 167 - 175
Langerhans cell histiocytosis (LCH) and Erdheim-Chester disease (ECD) are rare histiocytic disorders induced by somatic mutation of MAPK pathway genes.... 
MANAGEMENT | DENDRITIC CELLS | NEOPLASMS | DISTINCT | MONOCYTES | SUBSETS | NOTCH | MUTATIONS | MATURATION | HEMATOLOGY | BLOOD | Antigens, CD - immunology | Dendritic Cells - immunology | Histiocytosis, Langerhans-Cell - diagnosis | Histiocytosis, Langerhans-Cell - genetics | Humans | Lectins, C-Type - immunology | Dendritic Cells - pathology | Hematopoietic Stem Cells - pathology | Erdheim-Chester Disease - immunology | Male | Receptors, Notch - genetics | Monocytes - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Hematopoietic Stem Cells - immunology | Histiocytosis, Langerhans-Cell - pathology | Bone Marrow Cells - immunology | Monocytes - pathology | Adult | Female | Cell Differentiation | Foam Cells - pathology | Glycoproteins - genetics | Transforming Growth Factor beta - immunology | Antigens, CD1 - immunology | Diagnosis, Differential | Erdheim-Chester Disease - genetics | Gene Expression | Bone Marrow Cells - pathology | Immunophenotyping | Lipopolysaccharide Receptors - immunology | Histiocytosis, Langerhans-Cell - immunology | Erdheim-Chester Disease - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Glycoproteins - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | Transforming Growth Factor beta - genetics | Proto-Oncogene Proteins B-raf - genetics | Proto-Oncogene Proteins B-raf - immunology | Alleles | Erdheim-Chester Disease - diagnosis | Lipopolysaccharide Receptors - genetics | Foam Cells - immunology | Antigens, CD1 - genetics | Mutation | Receptors, Notch - immunology | Myeloid Neoplasia
Journal Article
Journal Article
Journal Article
Clinica Chimica Acta, ISSN 0009-8981, 09/2013, Volume 424, pp. 245 - 252
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2012, Volume 122, Issue 9, pp. 3114 - 3126
Atherosclerosis is a chronic inflammatory disease of large and medium-sized arteries characterized by leukocyte accumulation in the vessel wall. Both innate... 
MEDICINE, RESEARCH & EXPERIMENTAL | REDUCES ATHEROSCLEROSIS | ATHEROGENESIS | DENDRITIC CELLS | INTERFERON-GAMMA | IMMUNE-RESPONSES | LYMPHOCYTE RECRUITMENT | LOW-DENSITY-LIPOPROTEIN | PLAQUE-FORMATION | FOAM CELLS | TRANSGENIC MICE | Cell Proliferation | Leukocyte Common Antigens - metabolism | Apolipoproteins E - deficiency | Coculture Techniques | Dendritic Cells - immunology | Vasculitis - pathology | Dendritic Cells - physiology | Male | Aorta - metabolism | CD11 Antigens - metabolism | CD4-Positive T-Lymphocytes - physiology | Cell Tracking | Vasculitis - immunology | Adventitia - metabolism | CD4-Positive T-Lymphocytes - immunology | Antigen-Presenting Cells - physiology | Diet, High-Fat | Female | Lipoproteins, LDL - metabolism | Plaque, Atherosclerotic - pathology | Dendritic Cells - metabolism | Macrophages - immunology | Macrophages - physiology | Antigen-Presenting Cells - metabolism | Atherosclerosis - pathology | Atherosclerosis - immunology | Cytokines - metabolism | Tissue Culture Techniques | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Cell Communication | Aorta - immunology | Antigen-Presenting Cells - immunology | Mice, Knockout | Aorta - pathology | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Adventitia - pathology | Mice | Cell Movement
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2016, Volume 36, Issue 2, pp. 274 - 284
OBJECTIVE—Apolipoprotein A-I (apoA-I) has been shown to possess several atheroprotective functions, including inhibition of inflammation. Protease-secreting... 
endothelial cells | chymase | inflammatory | proteases | mast cell | apolipoprotein A-I | carboxyl-terminal cleavage | PROTEOLYTIC INACTIVATION | HUMAN APOA-I | FOAM CELLS | HIGH-DENSITY-LIPOPROTEIN | HDL | CHOLESTEROL EFFLUX | ENDOTHELIAL-CELLS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | TNF-ALPHA | HEMATOLOGY | BINDING | Apolipoprotein A-I - pharmacology | Reactive Oxygen Species - metabolism | Chymases - metabolism | Coculture Techniques | Humans | NF-kappa B - metabolism | Atherosclerosis - enzymology | Transendothelial and Transepithelial Migration | Proteolysis | Inflammation Mediators - metabolism | Neutrophils - metabolism | Foam Cells - metabolism | Protein Structure, Tertiary | Mast Cells - immunology | Atherosclerosis - immunology | Cytokines - metabolism | Neutrophil Activation | Signal Transduction | Endothelial Cells - metabolism | Neutrophils - immunology | Inflammation - immunology | Apolipoprotein A-I - metabolism | Cholesterol - metabolism | Mast Cells - enzymology | Cell Adhesion | Mast Cells - drug effects | Peptides - pharmacology | Endothelial Cells - immunology | Cell Line, Tumor | Foam Cells - immunology | Inflammation - prevention & control | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Atherosclerosis - prevention & control | Endothelial Cells - drug effects | 10030 | 10195 | 10189 | 10188 | Basic Sciences
Journal Article
SAE technical paper series, Volume 980965.
The energy dissipation behavior of foams depending on changes of loading rate input has been investigated and simulated to provide and guide a safe car... 
Foams | Occupant protection | Plastics
eJournal
Vascular Pharmacology, ISSN 1537-1891, 01/2019, Volume 112, pp. 54 - 71
During atherosclerosis, the gradual accumulation of lipids into the subendothelial space of damaged arteries results in several lipid modification processes... 
Non-coding RNAs | Protease | Stem/progenitor cells | Atherosclerosis | Genetic animal model | Cardiovascular disease | Foam cell | Genome wide association studies | Macrophages | Endothelial cells | Vascular smooth muscle cells | Pharmacological inhibition | MACROPHAGE SCAVENGER RECEPTORS | INTERCELLULAR-ADHESION MOLECULE-1 | COLONY-STIMULATING FACTOR | PROMOTES COLLAGEN ACCUMULATION | LOW-DENSITY-LIPOPROTEIN | MARROW-DERIVED CELLS | REVERSE CHOLESTEROL TRANSPORT | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | CLASS-A OVEREXPRESSION | SIGNAL-TRANSDUCTION PATHWAY | Atherosclerosis - pathology | Genetic Predisposition to Disease | Atherosclerosis - drug therapy | Apoptosis - drug effects | Atherosclerosis - genetics | Humans | Risk Factors | Cholesterol - metabolism | Molecular Targeted Therapy | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Foam Cells - drug effects | Cardiovascular Agents - pharmacology | Necrosis | Phenotype | Animals | Signal Transduction - drug effects | Foam Cells - pathology | Foam Cells - metabolism | Medical research | Medical colleges | RNA | Genomics | Endothelium | Proteins | Proteases | Analysis | Medicine, Experimental | Blood lipids | Cardiovascular diseases | Transformation | Target recognition | Lipids | Smooth muscle | Cell surface | Accumulation | Arteries | Cell adhesion molecules | Phagocytes | Signal transduction | Receptors | Extravasation | Scavenger receptors | Damage accumulation | Efflux | Streak | Therapeutic applications | Muscles | Pharmacology | Inflammation | Ribonucleic acid--RNA | Progenitor cells | Cholesterol | Chemokine receptors | Esterification | Monocytes | Lipoproteins | Researchers | Cell death | Arteriosclerosis | Stem cells | Cells (biology) | Non-coding RNA
Journal Article