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1. Biological response modifiers
: interferons, double-stranded RNA and 2́,5́-oligoadenylates
by Müller, Werner E. G and Schröder, H. C
1994, Progress in molecular and subcellular biology, ISBN 9783540572855, Volume 14., xiv, 275
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2. Full Text Small Ubiquitin-Like Modifier 3–Modified Proteome Regulated by Brain Ischemia in Novel Small Ubiquitin-Like Modifier Transgenic Mice: Putative Protective Proteins/Pathways
by Yang, Wei and Sheng, Huaxin and Thompson, J Will and Zhao, Shengli and Wang, Liangli and Miao, Pei and Liu, Xiaozhi and Moseley, M Arthur and Paschen, Wulf
Stroke, ISSN 0039-2499, 04/2014, Volume 45, Issue 4, pp. 1115 - 1122
BACKGROUND AND PURPOSE—Small ubiquitin-like modifier (SUMO) conjugation is a post-translational modification associated with many human diseases.... 
brain ischemia | SUMO | proteomics | HETEROGENEOUS NUCLEAR RIBONUCLEOPROTEINS | IDENTIFICATION | SUMOYLATION | CLINICAL NEUROLOGY | CONJUGATION | SUMO proteins | transgenic mice | PERIPHERAL VASCULAR DISEASE | GLOBAL CEREBRAL-ISCHEMIA | PROTEINS | Proteome - genetics | RNA Processing, Post-Transcriptional | Ischemic Attack, Transient - genetics | Small Ubiquitin-Related Modifier Proteins - metabolism | Ubiquitins - genetics | Brain Ischemia - genetics | Brain Ischemia - metabolism | Mice, Transgenic | Brain Ischemia - physiopathology | Stroke - physiopathology | Stroke - genetics | Small Ubiquitin-Related Modifier Proteins - genetics | Stroke - metabolism | Animals | SUMO-1 Protein - genetics | Mass Spectrometry | Proteomics | Mice | SUMO-1 Protein - metabolism | Ischemic Attack, Transient - metabolism | Proteome - metabolism | Ubiquitins - metabolism | Ischemic Attack, Transient - physiopathology
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3. Full Text An international initiative to identify genetic modifiers of cancer risk in BRCA1 and BRCA2 mutation carriers: The Consortium of Investigators of Modifiers of BRCA1 and BRCA2 (CIMBA)
by Chenevix-Trench, Georgia and Milne, Roger L and Antoniou, Antonis C and Couch, Fergus J and Easton, Douglas F and Goldgar, David E and CIMBA
Breast Cancer Research, ISSN 1465-5411, 04/2007, Volume 9, Issue 2, pp. 104 - 104
BRCA1 and BRCA2 mutations exhibit variable penetrance that is likely to be accounted for, in part, by other genetic factors among carriers. However, studies... 
BREAST-CANCER | POLYGLUTAMINE REPEAT | MAMMOGRAPHIC DENSITY | RAD51 | ONCOLOGY | RECEPTOR CAG REPEAT | SUSCEPTIBILITY | LENGTH | SINGLE-NUCLEOTIDE POLYMORPHISM | AIB1 GENE | ASSOCIATION | Genetic Predisposition to Disease | Gene Frequency | Humans | Breast Neoplasms - etiology | Risk | International Cooperation | Breast Neoplasms - genetics | Neoplasm Proteins | Alleles | Genes, BRCA2 | Heterozygote | Genes, BRCA1 | Mutation | Gene mutations | Oncology, Experimental | Genomics | Genetic research | Genetic aspects | Research | Health aspects | Risk factors | Cancer | Medical and Health Sciences | Medicin och hälsovetenskap | Genes | international cooperation | BRCA1 | Klinisk medicin | heterozygote | Breast Neoplasms | BRCA2 | genetics | genetic predisposition | human | tumor suppressor gene | tumor protein | article | gene frequency | Clinical Medicine | mutation | breast tumor | allele | risk | Cancer and Oncology | Cancer och onkologi
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4. Full Text Genetic modifiers of cystic fibrosis-related diabetes
by Blackman, Scott M and Commander, Clayton W and Watson, Christopher and Arcara, Kristin M and Strug, Lisa J and Stonebraker, Jaclyn R and Wright, Fred A and Rommens, Johanna M and Sun, Lei and Pace, Rhonda G and Norris, Sarah A and Durie, Peter R and Drumm, Mitchell L and Knowles, Michael R and Cutting, Garry R
Diabetes, ISSN 0012-1797, 10/2013, Volume 62, Issue 10, pp. 3627 - 3635
Diabetes is a common age-dependent complication of cystic fibrosis (CF) that is strongly influenced by modifier genes. We conducted a genome-wide association... 
TRANSMEMBRANE CONDUCTANCE REGULATOR | ENDOCRINOLOGY & METABOLISM | BETA-CELL | LUNG-DISEASE SEVERITY | MELLITUS | SLC26A9 | SECRETION | LIVER-DISEASE | ASSOCIATION | EXPRESSION | GLUCOSE-TOLERANCE | RNA-Binding Proteins - genetics | United States - epidemiology | Genetic Predisposition to Disease | Genome-Wide Association Study | Diabetes Mellitus, Type 2 - genetics | Humans | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Genotype | Male | Cystic Fibrosis - epidemiology | Diabetes Mellitus, Type 2 - epidemiology | Cyclin-Dependent Kinase 5 - genetics | tRNA Methyltransferases | Cystic Fibrosis - genetics | Adolescent | Antiporters - genetics | Adult | Female | Polymorphism, Single Nucleotide | Odds Ratio | Cystic fibrosis | Genetic aspects | Diabetes | Risk factors | Original Research
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5. Full Text Genetic modifiers of Huntington's disease
by Gusella, James F and MacDonald, Marcy E and Lee, Jong‐Min
Movement Disorders, ISSN 0885-3185, 09/2014, Volume 29, Issue 11, pp. 1359 - 1365
Huntington's disease (HD) is a devastating neurodegenerative disorder that directly affects more than 1 in 10,000 persons in Western societies but, as a family... 
genetic association | HTT | Huntington's disease | huntingtin | genetic modifier | Genetic modifier | Huntingtin | Genetic association | MODIFYING AGE | PGC-1-ALPHA | EXPANDED CAG REPEAT | CLINICAL NEUROLOGY | AGE-OF-ONSET | PREDICT-HD | VENEZUELAN KINDREDS | EXPANSION | KNOCK-IN MICE | TRINUCLEOTIDE REPEAT LENGTH | PROGRESSION | Huntington Disease - therapy | Huntingtin Protein | Humans | Huntington Disease - genetics | Trinucleotide Repeats - genetics | Nerve Tissue Proteins - genetics | Genetic Therapy - methods | Genetic research
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6. Full Text Modifiers of C9orf72 dipeptide repeat toxicity connect nucleocytoplasmic transport defects to FTD/ALS
by Jovičič, Ana and Mertens, Jerome and Boeynaems, Steven and Bogaert, Elke and Chai, Noori and Yamada, Shizuka B and Paul, Joseph W and Sun, Shuying and Herdy, Joseph R and Bieri, Gregor and Kramer, Nicholas J and Gage, Fred H and Van Den Bosch, Ludo and Robberecht, Wim and Gitler, Aaron D
Nature Neuroscience, ISSN 1097-6256, 08/2015, Volume 18, Issue 9, pp. 1226 - 1229
C9orf72 mutations are the most common cause of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). Dipeptide repeat proteins (DPRs) produced... 
HUMAN NEURONS | IN-VITRO | C9FTD/ALS | EXPANSIONS | ALS | FUS MUTATIONS | PATHOLOGY | PROTEINS | HEXANUCLEOTIDE REPEAT | NEUROSCIENCES | RNA FOCI | Frontotemporal Dementia - genetics | Yeasts | DNA Repeat Expansion - physiology | Active Transport, Cell Nucleus - physiology | Amyotrophic Lateral Sclerosis - genetics | Humans | Cells, Cultured | Frontotemporal Dementia - metabolism | Proteins - genetics | Animals | Cell Nucleus - metabolism | Proteins - metabolism | Cell Nucleus - genetics | Gene Deletion | Amyotrophic Lateral Sclerosis - metabolism | C9orf72 Protein | Mice | Dipeptides - metabolism | Dipeptides - genetics | Gene mutations | Amyotrophic lateral sclerosis | Genetic aspects | Research | Frontotemporal dementia | Health aspects | Risk factors | Index Medicus
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7. Full Text Epigenetic modulators, modifiers and mediators in cancer aetiology and progression
by Feinberg, Andrew P and Koldobskiy, Michael A and Göndör, Anita
Nature Reviews Genetics, ISSN 1471-0056, 05/2016, Volume 17, Issue 5, pp. 284 - 299
This year is the tenth anniversary of the publication in this journal of a model suggesting the existence of 'tumour progenitor genes'. These genes are... 
EPITHELIAL-MESENCHYMAL TRANSITION | CHROMATIN REMODELING GENES | EMBRYONIC STEM-CELLS | ACUTE LYMPHOBLASTIC-LEUKEMIA | GENETICS & HEREDITY | NUCLEAR LAMINA INTERACTIONS | CHRONIC LYMPHOCYTIC-LEUKEMIA | ACUTE MYELOID-LEUKEMIA | HISTONE-MODIFYING GENES | RECURRENT SOMATIC MUTATIONS | REPRESSIVE COMPLEX 2 | DNA Methylation | Animals | Neoplasms - etiology | Epigenesis, Genetic | Humans | Stem Cells - pathology | Mutation - genetics | Neoplasms - pathology | Disease Progression | Gene mutations | Genes | Disease susceptibility | Genetic aspects | Properties | Carcinogenesis | Identification and classification | Health aspects
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8. Full Text Pluronic block copolymers: Evolution of drug delivery concept from inert nanocarriers to biological response modifiers
by Batrakova, Elena V and Kabanov, Alexander V
Journal of Controlled Release, ISSN 0168-3659, 2008, Volume 130, Issue 2, pp. 98 - 106
Polymer nanomaterials have sparked a considerable interest as vehicles used for diagnostic and therapeutic agents; research in nanomedicine has not only become... 
Pluronic | Nanocarrier | Biological response modifiers | Drug delivery system | Multidrug resistance | multidrug resistance | MICROVESSEL ENDOTHELIAL-CELLS | nanocarrier | RESISTANCE-ASSOCIATED PROTEIN | SURFACE-ACTIVE AGENTS | BLOOD-BRAIN-BARRIER | CHEMISTRY, MULTIDISCIPLINARY | P-GLYCOPROTEIN | biological response modifiers | OXYETHYLENE OXYPROPYLENE POLYMERS | BREAST-CANCER CELLS | pluronic | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | drug delivery system | EFFLUX TRANSPORTER ACTIVITY | MULTIDRUG-RESISTANCE | Neoplasms - metabolism | Propylene Glycols - pharmacology | Immunologic Factors - chemistry | Nanoparticles - chemistry | Humans | Polyethylene Glycols - chemistry | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Drug Carriers - chemistry | Neoplasms - drug therapy | Animals | Propylene Glycols - chemistry | Neoplasms - pathology | Immunologic Factors - pharmacology | Polyethylene Glycols - pharmacology | Drugs | Drug delivery systems | Genetic research | Drug resistance | Gene expression | Block copolymers | Vehicles
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9. Full Text The Expanding Universe of Ubiquitin and Ubiquitin-Like Modifiers
by Richard D. Vierstra
Plant Physiology, ISSN 0032-0889, 9/2012, Volume 160, Issue 1, pp. 2 - 14
Proteins | Yeasts | Enzymes | Ubiquitins | Proteomics | UPDATES | Amino acids | Gene expression regulation | Sulfur | Plant cells | Transfer RNA | UBIQUITYLATED PROTEINS | RIBOSOMAL-PROTEINS | EMBRYO DEVELOPMENT | CRYSTAL-STRUCTURE | AFFINITY PURIFICATION | FUNCTIONAL-ANALYSIS | PROTEASOME SYSTEM | PROTEOMIC ANALYSIS | CONJUGATION SYSTEM | ARABIDOPSIS CULLIN ATCUL1 | PLANT SCIENCES | Adaptation, Physiological | Autophagy-Related Protein 12 | Arabidopsis Proteins - genetics | Protein Structure, Secondary | Ubiquitins - genetics | Stress, Physiological | Ubiquitin-Protein Ligases - metabolism | Autophagy | Arabidopsis - metabolism | Protein Transport | Arabidopsis - genetics | Arabidopsis Proteins - metabolism | Cell Nucleus - metabolism | Sumoylation | Transcription, Genetic | Ubiquitin-Protein Ligases - genetics | Ubiquitins - metabolism | Evolution, Molecular | Ubiquitin | Protein research | Genetic aspects | Research | Plant physiology
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10. Full Text Long non-coding RNAs and chromatin modifiers
: Their place in the epigenetic code
by Marchese, Francesco P and Huarte, Maite
Epigenetics, ISSN 1559-2294, 01/2014, Volume 9, Issue 1, pp. 21 - 26
The emergence of long non-coding RNAs (lncRNAs) has shaken up our conception of gene expression regulation, as lncRNAs take prominent positions as components... 
long noncoding RNA | histone modification | chromatin | epigenetics | gene expression | Epigenetics | Histone modification | Chromatin | Gene expression | Long noncoding RNA | RECRUITMENT | ELEMENTS | HUMAN GENOME | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | ANTISENSE | GENETICS & HEREDITY | GENE-EXPRESSION | X-CHROMOSOME INACTIVATION | CONFORMATION | DIFFERENTIATION | REPRESSIVE COMPLEX 2 | Epigenesis, Genetic - physiology | Chromatin - metabolism | Gene Expression | Animals | Potassium Channels, Voltage-Gated - metabolism | Humans | DNA Restriction-Modification Enzymes - metabolism | RNA, Long Noncoding - genetics | RNA, Long Noncoding - metabolism | X Chromosome - genetics | DNA-Binding Proteins | Review
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