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glucococorticoids (3) 3
glucocorticoids - pharmacology (3) 3
animals (2) 2
cell biology (2) 2
cells, cultured (2) 2
dexamethasone - pharmacology (2) 2
glucococorticoid (2) 2
hpa axis (2) 2
humans (2) 2
index medicus (2) 2
inflammation (2) 2
life sciences (2) 2
neurosciences (2) 2
oxidative stress (2) 2
phosphorylation (2) 2
pituitary-adrenal axis (2) 2
steroid hormones (2) 2
1 null mice (1) 1
11 beta hydroxysteroid dehydrogenase (1) 1
11β hydroxysteroid dehydrogenase (1) 1
2, neural precursor cell expressed, developmentally down-regulated protein 4-2 (1) 1
5'-flanking region (1) 1
activated protein-kinase (1) 1
adrenal glands (1) 1
advanced basic science (1) 1
advertising executives (1) 1
af9, all-1 fused gene from chromosome 9 (1) 1
aging (1) 1
airway epithelium (1) 1
alpha-1-acid glycoprotein gene (1) 1
alzheimer's disease (1) 1
alzheimers-disease (1) 1
alzheimer’s disease (1) 1
amyloid-beta (1) 1
annexin (1) 1
anti-inflammatory agents - adverse effects (1) 1
anti-inflammatory agents - pharmacology (1) 1
anxieties (1) 1
article (1) 1
associated inflammatory response in pneumonia (1) 1
astrocyte (1) 1
astrocytes - cytology (1) 1
astrocytes - drug effects (1) 1
astrocytes - metabolism (1) 1
avp (1) 1
biochemistry & molecular biology (1) 1
c-reactive protein (1) 1
cachexia (1) 1
calcium (1) 1
calcium - analysis (1) 1
calcium - metabolism (1) 1
calcium signaling - drug effects (1) 1
calcium-binding proteins (1) 1
cancer cachexia (1) 1
cancer-patients (1) 1
care and treatment (1) 1
catabolism (1) 1
cd2, cluster of differentiation 2 (1) 1
cell cycle proteins - metabolism (1) 1
cells (1) 1
central nervous system depressants (1) 1
channel (1) 1
channel dexamethasone (1) 1
circadian-rhythm (1) 1
cognitive ability (1) 1
colony-stimulating factor (1) 1
cortex (1) 1
corticosteroids (1) 1
corticosterone (1) 1
corticotropin-releasing factor (1) 1
corticotropin-releasing-factor (1) 1
crh (1) 1
cytokines (1) 1
cytosol - chemistry (1) 1
cytosol - drug effects (1) 1
cytosol - metabolism (1) 1
dehydrogenases (1) 1
dependent mechanism (1) 1
depression (1) 1
depression, mental (1) 1
dexamethasone (1) 1
dexamethasone treatment (1) 1
dexamethasone-suppression test (1) 1
df, degrees of freedom (1) 1
dissociated glucocorticoid (1) 1
dna-binding (1) 1
dot1a, disruptor of telomeric silencing alternative splice variant a (1) 1
drug resistance (1) 1
dual specificity phosphatase (1) 1
enac, epithelial na+ channel (1) 1
endocrinology & metabolism (1) 1
energy resources (1) 1
enzyme inhibitors - pharmacology (1) 1
epithelial na (1) 1
epithelial na+ channel dexamethasone (1) 1
epithelial sodium channels - genetics (1) 1
epithelial sodium-channel (1) 1
etiology (1) 1
excitatory amino acids (1) 1
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1. Full Text The regulation of muscle mass by endogenous glucocorticoids
by Braun, Theodore P and Marks, Daniel L
Frontiers in Physiology, ISSN 1664-042X, 2015, Volume 6, p. 12
Glucocorticoids are highly conserved fundamental regulators of energy homeostasis. In response to stress in the form of perceived danger or acute inflammation,... 
Protein synthesis | Glucococorticoids | Wasting | Cachexia | Catabolism | Muscle | Skeletal | HPA-axis | wasting | CANCER CACHEXIA | OXIDATIVE STRESS | DNA-BINDING | PHYSIOLOGY | glucococorticoids | skeletal | cachexia | protein synthesis | 1 NULL MICE | catabolism | SKELETAL-MUSCLE | CORTICOTROPIN-RELEASING FACTOR | PROTEIN-DEGRADATION | muscle | GENE-EXPRESSION | PITUITARY-ADRENAL AXIS | DEPENDENT MECHANISM | Muscle, Skeletal
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2. Full Text Glucocorticoid Resistance: Is It a Requisite for Increased Cytokine Production in Depression? A Systematic Review and Meta-Analysis
by Perrin, AJ and Horowitz, MA and Roelofs, J and Zunszain, PA and Pariante, CM
FRONTIERS IN PSYCHIATRY, ISSN 1664-0640, 06/2019, Volume 10, p. 423
Background: Glucocorticoid resistance-reduced function of the glucocorticoid receptor (GR)-is seen in many depressed patients. It is argued that this... 
C-REACTIVE PROTEIN | glucococorticoids | PSYCHIATRY | CIRCADIAN-RHYTHM | glucocorticoid resistance | MAJOR DEPRESSION | MEDICAL ILLNESS | PLASMA INTERLEUKIN-6 | CANCER-PATIENTS | DEXAMETHASONE-SUPPRESSION TEST | inflammation | cytokines | depression | HPA AXIS | PITUITARY-ADRENAL AXIS | PERICARDIAL ADIPOSE-TISSUE | Medical research | Care and treatment | Corticosteroids | Cytokines | Depression, Mental | Medicine, Experimental | Research | Usage | Drug resistance
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3. Full Text Is AD a Stress-Related Disorder? Focus on the HPA Axis and Its Promising Therapeutic Targets
by Canet, G and Hernandez, C and Zussy, C and Chevallier, N and Desrumaux, C and Givalois, L
FRONTIERS IN AGING NEUROSCIENCE, ISSN 1663-4365, 09/2019, Volume 11
Alzheimer's disease (AD) is a progressive neurodegenerative disorder that has important health and economic impacts in the elderly. Despite a better... 
stress-related disorder | AVP (arginine vasopressin) | VASOPRESSIN 1B RECEPTOR | glucococorticoids | HPA axis (hypothalamus-pituitary-adrenal) | ALZHEIMERS-DISEASE | CRH (corticotropin-releasing hormone) | TAU PATHOLOGY | NEUROSCIENCES | GERIATRICS & GERONTOLOGY | CORTICOTROPIN-RELEASING-FACTOR | MEMORY CONSOLIDATION | AMYLOID-BETA | 11 beta hydroxysteroid dehydrogenase | MOUSE MODEL | NEUROHYPOPHYSEAL HORMONES | Alzheimer's disease | IMPROVES COGNITIVE FUNCTION | PARKINSONS-DISEASE | Advertising executives | Nervous system diseases | Stress (Psychology) | Health aspects | Oxidative stress | Neurosciences | Phosphorylation | Dehydrogenases | Glucocorticoids | Adrenal glands | Memory | Menopause | Cognitive ability | Homeostasis | Hypothalamus | Hormones | Risk factors | Proteins | Energy resources | Senile plaques | Neurodegeneration | Etiology | Aging | Cortex (prefrontal) | Neurodegenerative diseases | Secretion | Therapeutic applications | Hypothalamic-pituitary-adrenal axis | Pathology | Neurofibrillary tangles | Anxieties | Pituitary | Excitatory amino acids | Steroid hormones | Geriatrics | Alzheimer’s disease
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4. Full Text Anti-inflammatory functions of glucocorticoid-induced genes
by Clark, Andrew R
Molecular and Cellular Endocrinology, ISSN 0303-7207, 2007, Volume 275, Issue 1, pp. 79 - 97
There is a broad consensus that glucocorticoids (GCs) exert anti-inflammatory effects largely by inhibiting the function of nuclear factor κB (NFκB) and... 
Post-transcriptional | Dissociated glucocorticoid | Dual specificity phosphatase | Tristetraprolin | Inflammation | Transrepression | Glucococorticoid | Transactivation | Glucocorticoid-induced leucine zipper | Annexin | Nuclear factor κB | dual specificity phosphatase | TYROSINE AMINOTRANSFERASE GENE | ACTIVATED PROTEIN-KINASE | COLONY-STIMULATING FACTOR | INDUCED LEUCINE-ZIPPER | nuclear factor kappa B | transrepression | dissociated glucocorticoid | PHOSPHOENOLPYRUVATE CARBOXYKINASE GENE | MESSENGER-RNA STABILITY | post-transcriptional | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | glucocorticoid-induced leucine zipper | inflammation | INNATE IMMUNE-RESPONSES | annexin | ENDOCRINOLOGY & METABOLISM | glucococorticoid | ALPHA-1-ACID GLYCOPROTEIN GENE | tristetraprolin | NF-KAPPA-B | Transcription, Genetic - drug effects | NF-kappa B - antagonists & inhibitors | Animals | Anti-Inflammatory Agents - adverse effects | NF-kappa B - genetics | Anti-Inflammatory Agents - pharmacology | Humans | Glucocorticoids - pharmacology | Inflammation Mediators - antagonists & inhibitors | Glucocorticoids - adverse effects
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5. Full Text Glucocorticoids reduce intracellular calcium concentration and protects neurons against glutamate toxicity
by Suwanjang, Wilasinee and Holmström, Kira M and Chetsawang, Banthit and Abramov, Andrey Y
Cell Calcium, ISSN 0143-4160, 2013, Volume 53, Issue 4, pp. 256 - 263
Abstract Glucocorticoids are steroid hormones which act through the glucocorticoid receptor. They regulate a wide variety of biological processes. Two... 
Advanced Basic Science | Neuron | Dexamethasone | Calcium | Corticosterone | Astrocyte | Glucococorticoid | Glutamate | MECHANISMS | THYMOCYTES | HIPPOCAMPAL-NEURONS | CELL BIOLOGY | CHANNEL | DEXAMETHASONE TREATMENT | RECEPTORS | Astrocytes - cytology | Astrocytes - drug effects | Calcium - metabolism | Cells, Cultured | Cytosol - drug effects | Rats | Neurons - cytology | Calcium - analysis | Rats, Sprague-Dawley | Cytosol - chemistry | Animals | Dexamethasone - pharmacology | Calcium Signaling - drug effects | Glucocorticoids - pharmacology | Cytosol - metabolism | Neurons - metabolism | Glutamic Acid - toxicity | Neurons - drug effects | Astrocytes - metabolism | Methyl aspartate | Neurosciences | Neurons | Central nervous system depressants | Calcium-binding proteins | Steroid hormones | Index Medicus
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6. Full Text Glucocorticoids can activate the α-ENaC gene promoter independently of SGK1
by McTavish, Niall and Getty, Jennet and Burchell, Ann and Wilson, Stuart M
Biochemical Journal, ISSN 0264-6021, 10/2009, Volume 423, Issue 2, pp. 189 - 197
The role of SGK1 (serum- and glucocorticoid-induced protein kinase 1) in the glucocorticoid induction of alpha-ENaC (epithelial Na+ channel alpha subunit) gene... 
Serum- and glucocorticoid-induced protein kinase 1 (SGK1) | channel (ENaC) dexamethasone | Phosphoinositide 3-kinase (PI3K | Reporter gene | Airway epithelium | Epithelial Na | CELLS | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | airway epithelium | SERUM | EPITHELIAL SODIUM-CHANNEL | INDUCIBLE PROTEIN-KINASE | LUNG LIQUID | serum- and glucocorticoid-induced protein kinase 1 (SGK 1) | RESPONSE ELEMENT | phosphoinositide 3-kinase (PI3K) | epithelial Na+ channel (ENaC) dexamethasone | 5'-FLANKING REGION | NA+ TRANSPORT | reporter gene | EXPRESSION | Stress, Physiological - physiology | Immediate-Early Proteins - physiology | Protein-Serine-Threonine Kinases - physiology | Humans | Transcriptional Activation - drug effects | Cell Cycle Proteins - metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Intracellular Signaling Peptides and Proteins - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Promoter Regions, Genetic - drug effects | Immediate-Early Proteins - metabolism | Dexamethasone - pharmacology | Epithelial Sodium Channels - genetics | Transfection | Glucocorticoids - pharmacology | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | PI3K, phosphoinositide 3-kinase | Nedd-4 | PIM1 and 3, provirus integration site for Moloney murine leukaemia 1 and 3 | SGK1, serum- and glucococorticoid-induced protein kinase | Dot1a, disruptor of telomeric silencing alternative splice variant a | Af9, ALL-1 fused gene from chromosome 9 | df, degrees of freedom | CD2, cluster of differentiation 2 | GRE, glucocorticoid response element | 2, neural precursor cell expressed, developmentally down-regulated protein 4-2 | serum- and glucocorticoid-induced protein kinase 1 (SGK1) | ENaC, epithelial Na+ channel | NDRG1, N-myc downstream regulated gene 1 | TORC, target of rapamycin complex
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7. Full Text Pulmonary Infections in Patients on Chronic Glucocorticoid Treatment
by Rañó, Ana and Agustí, Carlos
01/2009, ISBN 0470319577, 22
This chapter contains sections titled: Introduction Associated inflammatory response in pneumonia Glucocorticoids: Mechanisms of action Glucocorticoid therapy... 
glucococorticoid therapy in mild immunosuppressive clinical settings | Pneumocystis jirovecii pneumonia (PJP) | pulmonary infections in patients on chronic GC treatment | glucocorticoids ‐ mechanisms of action | glucocorticoids in immunocompromised patients | fungal pneumonia | associated inflammatory response in pneumonia | viral pneumonia | Glucocorticoids in immunocompromised patients | Glucococorticoid therapy in mild immunosuppressive clinical settings | Viral pneumonia | Associated inflammatory response in pneumonia | Pulmonary infections in patients on chronic GC treatment | Fungal pneumonia | Glucocorticoids - mechanisms of action
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