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Rheumatology, ISSN 1462-0324, 04/2018, Volume 57, Issue suppl_3
Journal Article
Rheumatology, ISSN 1462-0324, 04/2018, Volume 57, Issue suppl_3
Journal Article
Nature, ISSN 0028-0836, 08/2011, Volume 476, Issue 7361, pp. 458 - 462
Glucocorticoids are released in response to stressful experiences and serve many beneficial homeostatic functions. However, dysregulation of glucocorticoids is... 
ANTIDEPRESSANTS | FOREBRAIN | GLUCOCORTICOID-RECEPTOR | MULTIDISCIPLINARY SCIENCES | NEURONS | RATS | ANIMAL-MODEL | ASTROCYTES | LESIONS | BRAIN | TRANSGENIC MICE | Depression - physiopathology | Male | Receptors, Glucocorticoid - metabolism | Hippocampus - drug effects | Receptors, Glucocorticoid - analysis | Depression - drug therapy | Dexamethasone - pharmacology | Dentate Gyrus - drug effects | Glucocorticoids - metabolism | Neurogenesis - radiation effects | Behavior, Animal - drug effects | Neurogenesis - drug effects | Stress, Physiological - drug effects | Swimming | Hypothalamo-Hypophyseal System - physiology | Corticosterone - secretion | Pituitary-Adrenal System - physiology | Glucocorticoids - secretion | Stress, Physiological - physiology | Hypothalamo-Hypophyseal System - drug effects | Restraint, Physical - psychology | Pituitary-Adrenal System - drug effects | Mice, Inbred C57BL | Restraint, Physical - physiology | Behavior, Animal - physiology | Mice, Transgenic | Hippocampus - cytology | Dentate Gyrus - cytology | Animals | Dentate Gyrus - physiology | Neurogenesis - physiology | Glucocorticoids - pharmacology | Corticosterone - metabolism | Mice | Hippocampus - physiology | Corticosterone - analysis | Murders & murder attempts | Antidepressants | Rodents | Histology | Behavior | Kinases | Laboratory animals | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 42, pp. 17035 - 17040
Journal Article
2001, Milestones in drug therapy., ISBN 3764360593, x, 205
Book
PLoS Biology, ISSN 1544-9173, 10/2015, Volume 13, Issue 10, pp. 1 - 16
Sepsis, an exaggerated systemic inflammatory response, remains a major medical challenge. Both hyperinflammation and immunosuppression are implicated as causes... 
NK CELLS | TUMOR NECROSIS FACTOR | IFN-GAMMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | ENDOTOXIN TOLERANCE | GAMMA-INTERFERON | BIOLOGY | SEPTIC SHOCK | RECEPTOR | MICE | T-CELLS | Antibodies, Neutralizing - administration & dosage | Receptors, Glucocorticoid - antagonists & inhibitors | Spleen - immunology | Dendritic Cells - immunology | Dendritic Cells - pathology | Male | Shock, Septic - prevention & control | Receptors, Glucocorticoid - metabolism | Spleen - drug effects | Shock, Septic - immunology | Shock, Septic - metabolism | Receptors, Glucocorticoid - agonists | CD11c Antigen - metabolism | Dose-Response Relationship, Drug | Interleukin-12 - blood | CD11c Antigen - genetics | Glucocorticoids - metabolism | Antibodies, Neutralizing - therapeutic use | Interleukin-12 - antagonists & inhibitors | Dendritic Cells - drug effects | Female | Spleen - pathology | Dendritic Cells - metabolism | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Specific Pathogen-Free Organisms | Immunity, Innate - drug effects | Mice, Inbred C57BL | Cells, Cultured | Glucocorticoids - blood | Mice, Transgenic | Interleukin-12 - metabolism | Mice, Knockout | Animals | Glucocorticoids - antagonists & inhibitors | Signal Transduction - drug effects | Spleen - metabolism | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Receptors, Glucocorticoid - genetics | Glucocorticoids - agonists | Crosses, Genetic | Index Medicus | Medical research | Flow cytometry | Statistical analysis | Cytokines | Dendritic cells | Rodents | Mortality | Sepsis | Software | Inflammation | Experiments
Journal Article
Journal Article
2006, Handbook of experimental pharmacology, ISBN 9783540258759, Volume 172, viii, 442
Molecular chaperones are involved in a wide variety of essential cellular processes in living cells. A subset of molecular chaperones have been initially... 
Molecular chaperones | Physiological effect | Proteins | Medical Biochemistry | Biomedicine | Pharmacology/Toxicology | Cell Biology
Book
Psychoneuroendocrinology, ISSN 0306-4530, 2013, Volume 38, Issue 9, pp. 1675 - 1687
Summary Considerable evidence suggests that cannabinoids modulate the behavioral and physiological response to stressful events. We have recently shown that... 
Endocrinology & Metabolism | Psychiatry | Contextual fear extinction | Cannabinoids | Post traumatic stress disorder | Glucocorticoid receptors | Single prolonged stress | CB1 receptors | PSYCHIATRY | BASOLATERAL AMYGDALA | CONDITIONED FEAR | ACTH RESPONSE | SINGLE-PROLONGED STRESS | NEUROSCIENCES | INFRALIMBIC CORTEX | ENHANCE EXTINCTION | GLUCOCORTICOID-RECEPTOR | ENDOCRINOLOGY & METABOLISM | CENTRAL-NERVOUS-SYSTEM | MEMORY RECONSOLIDATION | FAST FEEDBACK | Amygdala - physiopathology | Amygdala - drug effects | Receptors, Glucocorticoid - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - physiology | Prefrontal Cortex - physiopathology | Receptors, Glucocorticoid - biosynthesis | Extinction, Psychological - drug effects | Fear - drug effects | Male | Hippocampus - drug effects | Fear - physiology | Freezing Reaction, Cataleptic - drug effects | Receptor, Cannabinoid, CB1 - physiology | Prefrontal Cortex - drug effects | Piperidines - pharmacology | Hypothalamo-Hypophyseal System - physiopathology | Extinction, Psychological - physiology | Pyrazoles - pharmacology | Pituitary-Adrenal System - physiopathology | Microinjections | Receptor, Cannabinoid, CB2 - drug effects | Models, Psychological | Morpholines - pharmacology | Rats | Rats, Sprague-Dawley | Cannabinoids - pharmacology | Naphthalenes - pharmacology | Animals | Benzoxazines - pharmacology | Receptor, Cannabinoid, CB1 - drug effects | Receptors, Glucocorticoid - genetics | Freezing Reaction, Cataleptic - physiology | Mifepristone - pharmacology | Electroshock | Hippocampus - physiopathology | Receptors, Glucocorticoid - physiology | Stress, Psychological - physiopathology | Phenols | Corticosteroids | Psychic trauma | Analysis | Post-traumatic stress disorder | Index Medicus | PTSD | Neurotransmitters | Emotional Regulation | Amygdala | Glucocorticoids | Memory | Receptor Binding | Hallucinogenic Drugs | Conditioned Emotional Responses | Animal Models | Hippocampus | Prefrontal Cortex
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 38, pp. 25593 - 25601
Glucocorticoids are important regulators of lipid homeostasis, and chronically elevated glucocorticoid levels induce hypertriglyceridemia, hepatic steatosis,... 
OBESITY | PLASMA | GENE | CHROMATIN | ANGIOGENESIS | DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERLIPIDEMIA | MICE | TRANSCRIPTIONAL REGULATION | PROTEIN-4 | Transcription, Genetic - drug effects | Hypertriglyceridemia - genetics | Humans | Dexamethasone - adverse effects | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fatty Liver - chemically induced | Adipose Tissue - metabolism | Angiopoietin-like 4 Protein | Dexamethasone - pharmacology | Glucocorticoids - genetics | Glucocorticoids - metabolism | Response Elements - genetics | Lipoprotein Lipase - metabolism | Glucocorticoids - adverse effects | Fatty Liver - genetics | Angiopoietins - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Liver - metabolism | Rats | Hypertriglyceridemia - metabolism | Mice, Knockout | Triglycerides - metabolism | Adipose Tissue, White | Hypertriglyceridemia - chemically induced | Animals | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dexamethasone - metabolism | Glucocorticoids - pharmacology | Mice | Transcription, Genetic - genetics | Triglycerides - genetics | Angiopoietins - genetics | Index Medicus | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling | Transcription | Dermatologi och venereologi | Dermatology and Venereal Diseases | Dexamethasone/adverse effects/metabolism/pharmacology | Hypertriglyceridemia/chemically induced/genetics/metabolism | Receptors | Adipose Tissue/metabolism | Liver/metabolism | Tumor | Adipose Tissue | Cell Line | Fatty Liver/chemically induced/genetics/metabolism | Triglycerides/genetics/ metabolism | Response Elements/genetics | Angiopoietins/genetics/ metabolism | Knockout | Lipoprotein Lipase/genetics/metabolism | White | Hepatocytes/metabolism | Genetic/drug effects/genetics | Glucocorticoids/adverse effects/genetics/ metabolism/pharmacology | Glucocorticoid/genetics/ metabolism
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 10/2015, Volume 414, Issue C, pp. 120 - 131
Transient early-life perturbations in glucocorticoids (GC) are linked with cardiovascular disease risk in later life. Here the impact of early life... 
Heart | Development | Zebrafish | Echocardiography | Glucocorticoids | VENTRICLE | RECEPTOR EXPRESSION | INJURY | MODEL | LEVEL | CELL BIOLOGY | CARDIAC-HYPERTROPHY | DISEASE | ENDOCRINOLOGY & METABOLISM | RESISTANCE | EXPOSURE | Heart - physiopathology | Heart - embryology | Gene Expression Regulation, Developmental - drug effects | Dexamethasone - adverse effects | Embryo Culture Techniques | Zebrafish - embryology | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Glucocorticoids - metabolism | Somatomedins - genetics | Heart - drug effects | Myocytes, Cardiac - metabolism | Receptors, Glucocorticoid - administration & dosage | Heart Function Tests - drug effects | Zebrafish Proteins - genetics | Ventricular Myosins - genetics | Corticosteroids | Gene expression | Analysis | Steroids | Index Medicus | HPA, hypothalamic-pituitary-adrenal | Dex, Dexamethasone | plb, phospholamban | igf1, insulin-like growth factor 1 | Mo, morpholino | glucocorticoid receptor knockdown | ef1α, elongation factor 1 alpha | fgf, fibroblast growth factor | gapdh, glyceraldehyde 3-phosphate dehydrogenase | H&E, hematoxylin and eosin | nkx2.5, homeobox protein Nkx-2.5 | qRT-PCR, quantitative real-time polymerase chain reaction | GCs, Glucocorticoids | ryr, ryanodine receptor | vmhc, ventricular myosin heavy chain | GFP, green fluorescent protein | GC, Glucocorticoid | UPL, universal probes library | PBS, phosphate buffered saline | serca, sarco | GR | mef2c, myosin enhancer factor 2c | pdgf, platelet derived growth factor | Tg (CMLC2:GFP), transgenic cardiomyosin light chain 2: green fluorescent protein | endoplasmic reticulum Ca2+-ATPase | gata4, gata binding factor 4 | HPI, hypothalamic-pituitary-interrenal | GR, glucocorticoid receptor (various, see gene nomenclature below) | hpf, hours post fertilisation | PFA, paraformaldehyde | IOD, inflow: outflow distance
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 02/2014, Volume 171, Issue 3, pp. 735 - 745
Background and PurposeThe COX-2/PGE2 pathway in hypoxic cancer cells has important implications for stimulation of inflammation and tumourigenesis. However,... 
hypoxia | glucocorticoid receptor | hypoxia‐inducible factor‐1 | cyclooxygenase‐2 | glucocorticoid‐induced leucine zipper | glucocorticoid-induced leucine zipper | cyclooxygenase-2 | hypoxia-inducible factor-1 | DEXAMETHASONE | PROTEIN | ACETYLATION | RECEPTOR | FACTOR-KAPPA-B | FACTOR-I | CANCER | INHIBITION | GENE | PHARMACOLOGY & PHARMACY | Dexamethasone - antagonists & inhibitors | Receptors, Glucocorticoid - antagonists & inhibitors | Humans | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Receptors, Glucocorticoid - metabolism | Alveolar Epithelial Cells - immunology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Receptors, Glucocorticoid - agonists | Cell Hypoxia | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Dexamethasone - pharmacology | RNA Interference | Cyclooxygenase 2 - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cell Line | Cyclooxygenase 2 - chemistry | Transcription Factors - antagonists & inhibitors | Alveolar Epithelial Cells - metabolism | Alveolar Epithelial Cells - drug effects | Transcription Factors - genetics | Hormone Antagonists - pharmacology | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Cell Movement - drug effects | Animals | Glucocorticoids - antagonists & inhibitors | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | Glucocorticoids - pharmacology | Genes, Reporter - drug effects | Mice | Transcription Factors - agonists | Index Medicus
Journal Article
Journal Article
15.