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Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 12/2009, Volume 187, Issue 6, pp. 761 - 772
Selective degeneration and death of one or more classes of neurons is the defining feature of human neurodegenerative disease. Although traditionally viewed as... 
Motor neurons | Nervous system diseases | Reviews | Neurodegenerative diseases | Neurons | Astrocytes | Motor neuron disease | Huntington disease | Parkinson disease | Amyotrophic lateral sclerosis | Prion diseases | CU/ZN-SUPEROXIDE-DISMUTASE | GLUTAMATE TRANSPORTER EAAT2 | MOTOR-NEURON DEGENERATION | SLOW DISEASE PROGRESSION | MONOAMINE-OXIDASE-B | LINKED SOD1 MUTANTS | AMYOTROPHIC-LATERAL-SCLEROSIS | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSGENIC MOUSE MODEL | GENOME-WIDE ASSOCIATION | CELL BIOLOGY | Neurons - pathology | Axonal Transport | Superoxide Dismutase - genetics | Capillaries - pathology | T-Lymphocytes - enzymology | Humans | Astrocytes - pathology | Stress, Physiological | Nerve Degeneration - genetics | Astrocytes - enzymology | Endoplasmic Reticulum - pathology | Cell Death | Superoxides - metabolism | Microglia - pathology | Amyotrophic Lateral Sclerosis - enzymology | T-Lymphocytes - pathology | Superoxide Dismutase - metabolism | Endoplasmic Reticulum - enzymology | Nerve Degeneration - enzymology | Amyotrophic Lateral Sclerosis - genetics | Microglia - enzymology | Capillaries - enzymology | Nerve Degeneration - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Neurons - enzymology | Glutamic Acid - metabolism | Mutation | Superoxide Dismutase-1 | Care and treatment | Physiological aspects | Superoxide dismutase | Genetic aspects | Disease susceptibility | Research | Health aspects | Index Medicus
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 01/2015, Volume 78, pp. 202 - 212
The transcription factor Nrf2 regulates the basal and inducible expression of a battery of cytoprotective genes. Whereas numerous Nrf2-inducing small molecules... 
Brusatol | Free radicals | Hepatocyte | Nrf2 | Toxicity | Keap1 | Chemical stress | Cell defense | OXIDATIVE STRESS | ACTIVATION | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN-SYNTHESIS INHIBITION | CANCER | TRANSCRIPTION FACTOR NRF2 | DEFENSE PATHWAY | ACETAMINOPHEN | ENDOCRINOLOGY & METABOLISM | DEGRADATION | RNA, Small Interfering - genetics | NF-E2-Related Factor 2 - antagonists & inhibitors | Oxidative Stress | Apoptosis - drug effects | Humans | Hepatocytes - metabolism | Autophagy | Quassins - pharmacology | Hepatocytes - cytology | Carcinoma, Hepatocellular - drug therapy | Liver Neoplasms - pathology | NF-E2-Related Factor 2 - genetics | Real-Time Polymerase Chain Reaction | Hepatocytes - drug effects | Oxidation-Reduction | RNA, Messenger - genetics | Cells, Cultured | Liver Neoplasms - drug therapy | Reverse Transcriptase Polymerase Chain Reaction | Brucea - chemistry | Blotting, Western | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Proliferation - drug effects | Mice | Carcinoma, Hepatocellular - metabolism | Chemical inhibitors | Metabolites | Proteolysis | Health aspects | Quinone | Cells | Index Medicus | Gclm, glutamate–cysteine ligase regulatory subunit | NEM, N-ethylmaleimide | NAPQI, N-acetyl-p-benzoquinone imine | IAA, iodoacetamide | Nrf2, nuclear factor erythroid 2-related factor 2 | Original Contribution | Keap1, Kelch-like ECH-associated protein 1 | DNCB, 2,4-dinitrochlorobenzene | Nqo1, NAD(P)H dehydrogenase quinone 1 | CDDO-Me, methyl-2-cyano-3,12-dioxooleana-1, 9dien-28-oate | HIF-1α, hypoxia-inducible factor 1α | RT-qPCR, real-time quantitative PCR | AMC, 7-amino-4-methylcoumarin
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 11/2008, Volume 28, Issue 10, pp. 1989 - 2002
It has been suggested that accumulation of beta‐amyloid (Aβ) peptide triggers neurodegeneration, at least in part, via glutamate‐mediated excitotoxicity in... 
neurodegeneration | Alzheimer’s disease | β‐amyloid‐internalization | tau phosphorylation | neuroprotection | glutamate | Tau phosphorylation | Neuroprotection | β-amyloid-internalization | Glutamate | Neurodegeneration | Alzheimer's disease | NMDA RECEPTOR | CHANNEL BLOCKER MEMANTINE | beta-amyloid-internalization | ALZHEIMERS-DISEASE | NEUROSCIENCES | CELL-DEATH | CHOLINERGIC NEURONS | MOUSE MODEL | A-BETA | GLUTAMATERGIC SYSTEM | TRANSGENIC MICE | Phosphotransferases - metabolism | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Peptide Fragments - toxicity | Caspase 3 - metabolism | tau Proteins - metabolism | Cyclic AMP Response Element-Binding Protein - drug effects | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Neuroprotective Agents - pharmacology | Up-Regulation - physiology | Caspase 3 - drug effects | Neurons - metabolism | Phosphorylation - drug effects | tau Proteins - drug effects | Neurons - drug effects | Alzheimer Disease - physiopathology | Memantine - pharmacology | Amyloid beta-Peptides - toxicity | Cells, Cultured | Alzheimer Disease - drug therapy | Rats | Excitatory Amino Acid Antagonists - pharmacology | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Extracellular Fluid - drug effects | Phosphotransferases - drug effects | Up-Regulation - drug effects | Amyloid beta-Peptides - antagonists & inhibitors | Extracellular Fluid - metabolism | Animals | Peptide Fragments - antagonists & inhibitors | Alzheimer Disease - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Glutamic Acid - metabolism | Enzyme Activation - physiology | Index Medicus
Journal Article
Journal Article