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Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2004, Volume 172, Issue 1, pp. 567 - 576
The signaling mechanism by which the anti-inflammatory cytokine IL-10 mediates suppression of proinflammatory cytokine synthesis remains largely unknown.... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | DNA-BINDING | HUMAN NEUTROPHILS | TYROSINE PHOSPHORYLATION | INTERLEUKIN-10 RECEPTOR | GENE-EXPRESSION | KAPPA-B-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | MONONUCLEAR PHAGOCYTES | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2005, Volume 99, Issue 2, pp. 267 - 277
When ovarian carcinoma is diagnosed in stage I, up to 90% of patients can be cured with surgery and currently available chemotherapy. At present, less than 25%... 
HK6 | Claudin 3 | Tumor markers | HK10 | Ovarian carcinoma | CA125 | Osteopontin | FACTOR VEGF | DIAGNOSIS | tumor markers | osteopontin | ovarian carcinoma | SERUM | HUMAN TISSUE KALLIKREINS | OBSTETRICS & GYNECOLOGY | BREAST-CANCER | claudin 3 | ONCOLOGY | GENE-EXPRESSION | PATIENT SURVIVAL | CARCINOMA | ENDOTHELIAL GROWTH-FACTOR | Epididymal Secretory Proteins - biosynthesis | Immunohistochemistry | Vascular Endothelial Growth Factor A - biosynthesis | Epithelial Cells - metabolism | Kallikreins - biosynthesis | Humans | Membrane Glycoproteins - biosynthesis | Middle Aged | Antigens, Neoplasm - biosynthesis | GPI-Linked Proteins | Ovarian Neoplasms - pathology | beta-Defensins | Sialoglycoproteins - biosynthesis | Antigens - blood | CA-125 Antigen - biosynthesis | CA-19-9 Antigen - biosynthesis | Antigens, Neoplasm - blood | Female | Kallikreins - blood | Ovarian Neoplasms - metabolism | CA-19-9 Antigen - blood | Membrane Proteins - blood | Cystadenoma - metabolism | CA-125 Antigen - blood | Vascular Endothelial Growth Factor A - blood | Membrane Glycoproteins - blood | Ovarian Neoplasms - blood | Sialoglycoproteins - blood | Epithelial Cells - pathology | Glycoproteins - blood | Cystadenoma - blood | Claudin-3 | Mucin-1 | Mucins - blood | Biomarkers, Tumor - blood | Glycoproteins - biosynthesis | Mucins - biosynthesis | Membrane Proteins - biosynthesis | Epididymal Secretory Proteins - metabolism | Antigens - biosynthesis | Neoplasm Staging | Biomarkers, Tumor - biosynthesis | Antigens | Chemotherapy | Oncology, Experimental | Flexible response (Strategy) | Research | Universities and colleges | Deterrence (Strategy) | Cancer | Ovarian cancer
Journal Article
Diabetologia, ISSN 0012-186X, 2/2007, Volume 50, Issue 2, pp. 348 - 358
Journal Article
Experimental Cell Research, ISSN 0014-4827, 01/2015, Volume 330, Issue 2, pp. 358 - 370
There are lines of evidence demonstrating that NEDD9 (Cas-L, HEF-1) plays a key role in the development, progression, and metastasis of breast cancer cells. We... 
NEDD9 | Breast cancer | Chondroitin sulfate | Growth | Migration | CD44 | MOLECULAR-INTERACTIONS | MATRIX | ANTIBODY GD3G7 | PROTEOGLYCANS | METASTASIS | INTEGRIN | HYALURONAN SYNTHESIS | CELL BIOLOGY | HUMAN-MELANOMA INVASION | ONCOLOGY | BINDING | PROGRESSION | Up-Regulation | Cell Proliferation | Humans | Membrane Glycoproteins - biosynthesis | Spheroids, Cellular - pathology | Vesicular Transport Proteins - biosynthesis | Phosphoproteins - metabolism | Chondroitin ABC Lyase - pharmacology | Syndecan-2 - biosynthesis | Chondroitin Sulfates - biosynthesis | Versicans - biosynthesis | Sulfotransferases - biosynthesis | Female | Tumor Cells, Cultured | Antibodies, Monoclonal - immunology | N-Acetylgalactosaminyltransferases - biosynthesis | Phosphoproteins - biosynthesis | Down-Regulation | Proteoglycans - biosynthesis | Proteoglycans - metabolism | Antigens - metabolism | Hyaluronan Receptors - biosynthesis | Chondroitin ABC Lyase - metabolism | Breast Neoplasms - pathology | Neoplasm Metastasis - pathology | Fluorescent Antibody Technique | Syndecan-1 - biosynthesis | Antigens - biosynthesis | Adaptor Proteins, Signal Transducing - biosynthesis | Adaptor Proteins, Signal Transducing - metabolism | Cell Movement | Enzymes | Methylcellulose | Analysis | Stem cells | Physiological aspects | Development and progression | Sulfates | Chondroitin | Protein expression | Cell growth | Metastasis | Cell adhesion & migration
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 04/2007, Volume 115, Issue 16, pp. 2178 - 2187
Background - Recent clinical studies have suggested a major protective role for the antioxidant enzyme glutathione peroxidase-1 ( GPx1) in diabetes-associated... 
Antioxidants | Aorta | Free radicals | Cardiovascular diseases | Diabetes mellitus | Atherosclerosis | atherosclerosis | INTIMA-MEDIA THICKNESS | free radicals | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | antioxidants | CARDIOVASCULAR COMPLICATIONS | diabetes mellitus | FACTOR-KAPPA-B | cardiovascular diseases | REACTIVE OXYGEN | ENDOTHELIAL-CELLS | VASCULAR INFLAMMATION | PERIPHERAL VASCULAR DISEASE | aorta | PLATELET ACTIVATION | HEMATOLOGY | CORONARY-ARTERY-DISEASE | GLYCATION END-PRODUCTS | Superoxide Dismutase - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Membrane Glycoproteins - biosynthesis | Male | Aorta - metabolism | Vascular Endothelial Growth Factor A - genetics | Tyrosine - analogs & derivatives | NADPH Oxidases - genetics | Diabetes Mellitus, Experimental - complications | Glutathione Peroxidase - deficiency | NADPH Oxidases - biosynthesis | Receptor for Advanced Glycation End Products | Atherosclerosis - pathology | Macrophages - pathology | Oxidation-Reduction | Tyrosine - biosynthesis | Isoenzymes - genetics | Immediate-Early Proteins - biosynthesis | Glutathione Peroxidase - genetics | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Immediate-Early Proteins - genetics | NF-kappa B - biosynthesis | Fibrosis | Glutathione Peroxidase - physiology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Receptors, Immunologic - genetics | Inflammation - enzymology | Vascular Endothelial Growth Factor A - biosynthesis | Superoxide Dismutase - biosynthesis | Glutathione - metabolism | Streptozocin | Intercellular Signaling Peptides and Proteins - biosynthesis | Atherosclerosis - etiology | Atherosclerosis - enzymology | Receptors, Immunologic - biosynthesis | Diabetic Angiopathies - complications | Diabetic Angiopathies - enzymology | Vascular Cell Adhesion Molecule-1 - genetics | Aortic Diseases - enzymology | Sinus of Valsalva - pathology | Aortic Diseases - pathology | Hyperlipoproteinemia Type II - complications | Mice, Inbred C57BL | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Glutathione Peroxidase - biosynthesis | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | NF-kappa B - genetics | Apolipoproteins E - genetics | Inflammation - genetics | Isoenzymes - biosynthesis | Aortic Diseases - etiology | Connective Tissue Growth Factor | Hyperlipoproteinemia Type II - genetics | Animal models in research | Physiological aspects | Development and progression | Peroxidase | Research | Apolipoproteins | Glutathione
Journal Article
Journal Article