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Lancet Neurology, The, ISSN 1474-4422, 2016, Volume 15, Issue 8, pp. 811 - 819
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2013, Volume 34, Issue 4, pp. 1159 - 1169
Journal Article
Brain Pathology, ISSN 1015-6305, 03/2010, Volume 20, Issue 2, pp. 459 - 467
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e13949 - e13949
Journal Article
ROMANIAN JOURNAL OF LEGAL MEDICINE, ISSN 1221-8618, 04/2017, Volume 25, Issue 1, pp. 31 - 36
Authors present two cases of sudden and unexpected death of the young siblings with the anamnesis of more unexplained deaths of their relatives (both parents)... 
sudden death | hereditary amyloidosis | transtyretin mutation | MEDICINE, LEGAL | cerebral angiopathy | BRAIN
Journal Article
Bangladesh Journal of Pharmacology, ISSN 1991-007X, 11/2013, Volume 8, Issue 4
The computational identification of missense mutation in CST3 (CYSTATIN 3 or CYSTATIN C) gene has been done in the present study. The missense mutations in the... 
In silico | Cerebral amyloid angiopathy | Genomi tool | Missense mutation | Hereditary | CST3 gene
Journal Article
Journal Article
Brain Pathology, ISSN 1015-6305, 07/2018, Volume 28, Issue 4, pp. 495 - 506
Hereditary cerebral hemorrhage with amyloidosis‐Dutch type (HCHWA‐D) is an early onset hereditary form of cerebral amyloid angiopathy (CAA) pathology, caused... 
TGFβ | amyloid β mutation E22Q | postmortem human brain tissue | cerebral amyloid angiopathy | phospho‐SMAD2/3 granules | hereditary cerebral hemorrhage with amyloidosis‐Dutch type | hereditary cerebral hemorrhage with amyloidosis-Dutch type | phospho-SMAD2/3 granules | phospho-SMAD2 | SMALL VESSEL DISEASE | PROTEIN | ALZHEIMERS-DISEASE | PATHOLOGY | NEUROSCIENCES | COLOCALIZES | CLINICAL NEUROLOGY | amyloid beta mutation E22Q | 3 granules | MICROVASCULAR DEGENERATION | TRANSFORMING GROWTH-FACTOR-BETA-1 | ANGIOPATHY | TGF beta | EXPRESSION | TRANSGENIC MICE | TGF-BETA-1 | Up-Regulation | Phosphorylation | Frontal Lobe - metabolism | Signal Transduction | Humans | Middle Aged | Smad2 Protein - metabolism | Cerebral Amyloid Angiopathy, Familial - metabolism | Cerebral Amyloid Angiopathy, Familial - pathology | Male | Smad3 Protein - metabolism | Occipital Lobe - pathology | Occipital Lobe - blood supply | Aged, 80 and over | Female | Aged | Frontal Lobe - blood supply | Transforming Growth Factor beta - metabolism | Frontal Lobe - pathology | Occipital Lobe - metabolism | Immunohistochemistry | Brain | Deregulation | Pathogenesis | Transforming growth factor-a | Transgenic mice | Blood vessels | Gene expression | Hemorrhage | Lobes | Pathology | Cerebral amyloid angiopathy | Autopsy | Smad2 protein | Fibrosis | Biomarkers | β-Amyloid | Amyloidosis | Microvasculature | Occipital lobes | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 8345 - 7
The assembly and deposition of amyloid beta protein (A beta) is a fundamental event during the early stages of Alzheimer's disease (AD) and cerebral amyloid... 
NERVOUS-SYSTEM | GM1 GANGLIOSIDE | DUTCH TYPE | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | INSECT CELLS | HEREDITARY CEREBRAL-HEMORRHAGE | MODEL | MELANOGASTER | EXPRESSION | PARKINSONS-DISEASE | Cerebral amyloid angiopathy | Molecular modelling | Insects | Transgenes | Gangliosides | Alzheimer's disease
Journal Article
APMIS, ISSN 0903-4641, 08/2016, Volume 124, Issue 8, pp. 639 - 648
Hereditary gelsolin amyloidosis ( HGA ) is a dominantly inherited systemic disease reported worldwide. HGA is characterized by ophthalmological, neurological,... 
collagen | arterial wall structure | elastic fibers | AGel amyloid angiopathy | Hereditary gelsolin amyloidosis | Elastic fibers | Arterial wall structure | Collagen | P COMPONENT | GSN GENE | MICROBIOLOGY | IMMUNOLOGY | P