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high fluence low-power laser irradiation (6) 6
apoptosis (4) 4
cells (3) 3
humans (3) 3
index medicus (3) 3
lasers (3) 3
oxidative stress (3) 3
proteins (3) 3
reactive oxygen species - metabolism (3) 3
activation (2) 2
animals (2) 2
apoptosis - radiation effects (2) 2
association (2) 2
bax activation (2) 2
biochemistry & molecular biology (2) 2
cancer (2) 2
cell line, tumor (2) 2
cellular biology (2) 2
cercopithecus aethiops (2) 2
cos cells (2) 2
cytochrome c (2) 2
dynamin-related protein 1 (2) 2
fission (2) 2
fluorescence resonance energy transfer (2) 2
high-fluence low-power laser irradiation (2) 2
high‐fluence low‐power laser irradiation (2) 2
microtubule-associated proteins - metabolism (2) 2
mitochondria (2) 2
phosphatases (2) 2
reactive oxygen species (2) 2
acetylcysteine (1) 1
adenocarcinoma (1) 1
analysis (1) 1
apoptosis - physiology (1) 1
bax (1) 1
bcl-2-associated x protein - metabolism (1) 1
biochemistry (1) 1
biomembranes (1) 1
calcineurin (1) 1
cancer therapies (1) 1
caspase 9 - metabolism (1) 1
caspase inhibitors (1) 1
caspase-9 (1) 1
caspases - metabolism (1) 1
cdc2 protein kinase - metabolism (1) 1
cdc25 phosphatases - metabolism (1) 1
cell survival - radiation effects (1) 1
cytochrome c group - antagonists & inhibitors (1) 1
cytochrome c group - metabolism (1) 1
cytochrome-c release (1) 1
cytochromes c - secretion (1) 1
dephosphorylation (1) 1
disease (1) 1
division (1) 1
dose-response relationship, radiation (1) 1
drp1 (1) 1
dynamics (1) 1
dynamin-related protein-1 (1) 1
dynamin‐related protein 1 (1) 1
feedback, physiological (1) 1
fluorescence (1) 1
fragmentation (1) 1
gene (1) 1
genes, reporter (1) 1
growth (1) 1
gtp phosphohydrolases - metabolism (1) 1
hafnium (1) 1
hematology, oncology and palliative medicine (1) 1
iap (1) 1
inhibition (1) 1
inhibitor of apoptosis proteins (1) 1
ir radiation (1) 1
kinases (1) 1
laser excitation (1) 1
laser modes (1) 1
laser theory (1) 1
living cells (1) 1
low-level light therapy (1) 1
low-level light therapy - methods (1) 1
lung neoplasms - metabolism (1) 1
lung neoplasms - pathology (1) 1
lung neoplasms - radiotherapy (1) 1
lungs (1) 1
membrane proteins - biosynthesis (1) 1
microscopy (1) 1
mitochondria - physiology (1) 1
mitochondria - radiation effects (1) 1
mitochondria - ultrastructure (1) 1
mitochondrial dna (1) 1
mitochondrial proteins - biosynthesis (1) 1
mitochondrial proteins - metabolism (1) 1
mitofusin 2 (1) 1
molecular biology (1) 1
negative feedback inhibition (1) 1
nf-κb (1) 1
oncology (1) 1
oxidation-reduction (1) 1
oxidative stress - radiation effects (1) 1
permeability (1) 1
phosphorylation (1) 1
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1. Full Text Mitochondrial oxidative stress causes mitochondrial fragmentation via differential modulation of mitochondrial fission–fusion proteins
by Wu, Shengnan and Zhou, Feifan and Zhang, Zhenzhen and Xing, Da
FEBS Journal, ISSN 1742-464X, 04/2011, Volume 278, Issue 6, pp. 941 - 954
Mitochondria are dynamic organelles that undergo continual fusion and fission to maintain their morphology and functions, but the mechanism involved is still... 
dynamin‐related protein 1 (Drp1) | high‐fluence low‐power laser irradiation (HF‐LPLI) | oxidative stress | fission | mitofusin 2 (Mfn2) | high-fluence low-power laser irradiation (HF-LPLI) | dynamin-related protein 1 (Drp1) | mitofusin 2 (Mfn2) | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IR RADIATION | DRP1 | DISEASE | DEPHOSPHORYLATION | CALCINEURIN | DYNAMIN-RELATED PROTEIN-1 | BAX | CYTOCHROME-C RELEASE | ASSOCIATION | Caspase 9 - metabolism | Cytochromes c - secretion | Reactive Oxygen Species - metabolism | Oxidative Stress | Microtubule-Associated Proteins - metabolism | Humans | Cercopithecus aethiops | Mitochondrial Proteins - biosynthesis | Mitochondria - ultrastructure | Membrane Proteins - biosynthesis | Animals | GTP Phosphohydrolases - metabolism | Lasers | Mitochondrial Proteins - metabolism | Cell Line, Tumor | Mitochondria - radiation effects | Apoptosis - physiology | Mitochondria - physiology | COS Cells | Cytochrome c | Oxidative stress | Mitochondrial DNA | Proteins | Mitochondria | Cellular biology | Molecular biology | Index Medicus
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2. Full Text Mechanism study on mitochondrial fragmentation under oxidative stress caused by high-fluence low-power laser irradiation
by Wu, Shengnan and Zhou, Feifan and Xing, Da
Proceedings of SPIE, ISSN 0277-786X, 03/2012, Volume 8211, Issue 1, pp. 82110K - 82110K-6
Mitochondria are dynamic organelles that undergo continual fusion and fission to maintain their morphology and functions, but the mechanism involved is still... 
Oxidative stress | High fluence low-power laser irradiation (HF-LPLI) | Dynamin-related protein 1 (Drp1) | Fission | Reactive oxygen species (ROS) | Proteins | Stresses | Mitochondria | Dynamics | Lasers | Hafnium | Activation | Fragmentation
Conference Proceeding
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3. Full Text Survivin mediates self-protection through ROS/cdc25c/CDK1 signaling pathway during tumor cell apoptosis induced by high fluence low-power laser irradiation
by Chu, Jiru and Wu, Shengnan and Xing, Da
Cancer Letters, ISSN 0304-3835, 2010, Volume 297, Issue 2, pp. 207 - 219
Abstract Survivin, an important member of inhibitor-of-apoptosis (IAP) family, can be up-regulated by various pro-apoptotic stimuli, such as UV, photodynamic... 
Hematology, Oncology and Palliative Medicine | High fluence low-power laser irradiation (HF-LPLI) | Self-protection | Survivin | Apoptosis | PHOTODYNAMIC THERAPY | PHOSPHORYLATION | PROLIFERATION | CANCER | RADIATION | DIVISION | INHIBITION | GENE | REPRESSION | ONCOLOGY | IAP | Up-Regulation | Low-Level Light Therapy - methods | Phosphorylation | Reactive Oxygen Species - metabolism | Inhibitor of Apoptosis Proteins | Apoptosis - radiation effects | Cytochrome c Group - antagonists & inhibitors | Microtubule-Associated Proteins - metabolism | Humans | Lung Neoplasms - metabolism | bcl-2-Associated X Protein - metabolism | Lung Neoplasms - radiotherapy | Lung Neoplasms - pathology | Caspase Inhibitors | Cell Survival - radiation effects | Cytochrome c Group - metabolism | CDC2 Protein Kinase - metabolism | Caspases - metabolism | Transfection | Cell Line, Tumor | Signal Transduction - radiation effects | cdc25 Phosphatases - metabolism | Cytochrome c | Phosphatases | Permeability | Photochemotherapy | Cells | Cancer | Tumors | Studies | Kinases | Cancer therapies | Index Medicus | Adenocarcinoma
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4. Phototherapy-treated apoptotic tumor cells induce pro-inflammatory cytokines production in macrophage
by Lu, Cuixia and Wei, Yanchun and Xing, Da
Progress in Biomedical Optics and Imaging - Proceedings of SPIE, ISSN 1605-7422, 2014, Volume 9230
TNF-α | NF-κB | High fluence low-power laser irradiation (HF-LPLI)
Conference Proceeding
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5. Full Text Measuring dynamics of Caspase-9 activity in living cells using FRET technique during apoptosis induced by high fluence low-power laser irradiation
by Wu, Shengnan and Huang, Lei and Sun, Xuegang and Chu, Jiru
Proceedings of SPIE, ISSN 0277-786X, 12/2008, Volume 7280, Issue 1, pp. 728023 - 728028
We investigated the activity of caspase-9 for its role in the regulation of apoptosis induced by high fluence Low-power laser irradiation (HF-LPLI). Using a... 
High fluence low-power laser irradiation (HF-LPLI) | Living cells | Fluorescence resonance energy transfer (FRET) | Caspase-9 | Apoptosis
Conference Proceeding
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6. High fluence low-power laser irradiation induced bax activation
by Wu, Shengnan and Xing, Da and Huang, Lei and Sun, Xuegang and Chu, Jiru
Optics InfoBase Conference Papers, 2009
High fluence low-power laser irradiation (HF-LPLI) | Bax activation
Conference Proceeding
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7. Full Text High fluence low-power laser irradiation induced Bax activation
by Shengnan Wu and Da Xing and Lei Huang and Xuegang Sun and Jiru Chu
2009 Conference on Lasers & Electro Optics & The Pacific Rim Conference on Lasers and Electro-Optics, 08/2009, pp. 1 - 2
We investigated Bax activity after high fluence low-power laser irradiation (HF-LPLI) treatment in human lung adenocarcinoma (ASTC-a-1) cells using single cell... 
Proteins | Laser theory | Microscopy | Lungs | Bax activation | Humans | Cells (biology) | Fluorescence | Laser modes | Laser excitation | Biomembranes | high fluence low-power laser irradiation (HF-LPLI) | High fluence low-power laser irradiation (HF-LPLI)
Conference Proceeding
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8. Full Text The reactive oxygen species–Src–Stat3 pathway provokes negative feedback inhibition of apoptosis induced by high‐fluence low‐power laser irradiation
by Sun, Xuegang and Wu, Shengnan and Xing, Da
FEBS Journal, ISSN 1742-464X, 11/2010, Volume 277, Issue 22, pp. 4789 - 4802
High‐fluence low‐power laser irradiation (HF‐LPLI) can induce apoptosis by triggering mitochondrial oxidative stress. Signal transducer and activator of... 
negative feedback inhibition | Stat3 | high‐fluence low‐power laser irradiation | reactive oxygen species (ROS) | Src | Negative feedback inhibition | High-fluence low-power laser irradiation | Reactive oxygen species (ROS) | CELLS | ACTIVATION | SRC TYROSINE KINASE | SURVIVIN EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | high-fluence low-power laser irradiation | CANCER | SIGNALING PATHWAY | GROWTH | ASSOCIATION | PHOSPHATASES | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Apoptosis - radiation effects | Cercopithecus aethiops | Feedback, Physiological | Animals | src-Family Kinases - metabolism | Fluorescence Resonance Energy Transfer | Signal Transduction - physiology | Oxidative Stress - radiation effects | src-Family Kinases - genetics | COS Cells | Dose-Response Relationship, Radiation | Genes, Reporter | Low-Level Light Therapy | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | RNA, Small Interfering - metabolism | Acetylcysteine | Analysis | Apoptosis | Oxidative stress | Biochemistry | Cellular biology | Lasers | Index Medicus
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