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2008, 1. Aufl., Advances in experimental medicine and biology, ISBN 0387690786, Volume 617, xlvi, 658
Information gathered from cell-free systems, cell cultures, animal models, and human studies, together will (1) provide important insights to our understanding... 
Carcinogenicity | Hormones | Congresses | Human Genetics | Medical Microbiology | Biomedicine | Microbiology | Cancer Research | Developmental Biology | Cell Biology
Book
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, pp. e45767 - e45767
Introduction: Estrogens regulate the proliferation of normal and neoplastic breast epithelium. Although the intracellular mechanisms of estrogens in the breast... 
MAMMARY EPITHELIAL ACINI | GROWTH-FACTOR RECEPTOR | ACTIVATION | CANCER CELLS | E-SCREEN ASSAY | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | ENDOCRINE DISRUPTION | BISPHENOL-A | PROLIFERATION | LUMEN FORMATION | Estradiol - analogs & derivatives | Receptors, Estrogen - metabolism | Epithelial Cells - metabolism | Estrogens - pharmacology | Gene Expression - drug effects | Receptors, G-Protein-Coupled - metabolism | Epithelial Cells - drug effects | Humans | Estrogen Antagonists - pharmacology | Immunoblotting | Receptors, Progesterone - genetics | Mammary Glands, Human - metabolism | Quinolines - pharmacology | Fulvestrant | Estrogen Receptor beta - metabolism | Estrogen Receptor beta - genetics | Cell Transformation, Neoplastic - genetics | Tumor Suppressor Proteins - genetics | Estrogen Receptor alpha - metabolism | Female | Trefoil Factor-1 | Cell Culture Techniques | Estrogen Receptor beta - antagonists & inhibitors | Estradiol - pharmacology | Mammary Glands, Human - drug effects | Phenols - pharmacology | Cell Line | Mammary Glands, Human - cytology | Receptors, Estrogen - genetics | Receptors, Estrogen - antagonists & inhibitors | Estrogen Receptor alpha - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Cell Transformation, Neoplastic - metabolism | Estrogen Receptor Modulators - pharmacology | Microscopy, Confocal | Estrogen Receptor alpha - genetics | Parabens - pharmacology | Receptors, G-Protein-Coupled - antagonists & inhibitors | Benzhydryl Compounds - pharmacology | Receptors, G-Protein-Coupled - genetics | Cell Transformation, Neoplastic - drug effects | Estrogens, Non-Steroidal - pharmacology | Benzodioxoles - pharmacology | Models | Bisphenol-A | Estradiol | Analysis | Transformation | Epithelial cells | Estrogens | Estrogen | Estrogen receptors | 17β-Estradiol | Carcinogenesis | Morphogenesis | Signal transduction | Carcinogens | Xenoestrogens | Mammary gland | Clearing | Deformation mechanisms | Cultures | Breast cancer | Gene expression | Epithelium | Studies | Bisphenol A | Sex hormones | Medical prognosis | Pharmacy | Morphology | Phenols | Breast | Disruption | Three dimensional models | Cancer | Apoptosis | Index Medicus
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 376, Issue 2, pp. 310 - 317
Cancer Letters: Highlights for Review • Blockade of the progestogen-PR is a untested strategy for breast cancer prevention • TPA opposed the pro-hyperplastic... 
Hematology, Oncology and Palliative Medicine | Prevention | Rats | Progesterone receptor | Telapristone acetate | Progestogen | Mammary carcinogenesis | MENSTRUAL-CYCLE | MEDROXYPROGESTERONE ACETATE | ULIPRISTAL ACETATE | BREAST-CANCER CELLS | MONODEMETHYLATED METABOLITE | AROMATASE INHIBITOR | MEGESTROL-ACETATE | UTERINE LEIOMYOMATA | ONCOLOGY | ESTROGEN PLUS PROGESTIN | POSTMENOPAUSAL WOMEN | Receptors, Estrogen - metabolism | Antigens, CD34 - metabolism | Neovascularization, Pathologic | Norpregnadienes - pharmacology | Mammary Neoplasms, Experimental - metabolism | Receptors, Progesterone - antagonists & inhibitors | Receptors, Progesterone - metabolism | Dose-Response Relationship, Drug | Time Factors | Methylnitrosourea | Mammary Neoplasms, Experimental - pathology | Female | Medroxyprogesterone Acetate | Mammary Neoplasms, Experimental - prevention & control | Antineoplastic Agents, Hormonal - pharmacology | Mammary Glands, Animal - pathology | Angiogenesis Inhibitors - pharmacology | Mammary Glands, Animal - drug effects | Hormone Antagonists - pharmacology | Mammary Neoplasms, Experimental - chemically induced | Animals | Mammary Glands, Animal - metabolism | Progestins | Cell Line, Tumor | 9,10-Dimethyl-1,2-benzanthracene | Cell Proliferation - drug effects | Mice | Progesterone | Mifepristone - pharmacology | Physiological aspects | Tumors | Carcinogenesis | Estrogen | Cancer | Medical research | Statistical analysis | Menstruation | Breasts | Breast cancer | Cancer therapies | Spheroids | Disease prevention | Angiogenesis | Cell growth | Rodents | Stem cells | Biomarkers | Index Medicus
Journal Article
Journal Article
Journal of Carcinogenesis, ISSN 0974-6773, 2011, Volume 10, Issue 1, pp. 33 - 33
Prostate cancer is the leading non-skin malignancy detected in US males and the second cause of death due to male cancer in the US. Androgenic hormones are... 
estrogens | prostate cancer | Adrogens | steroid hormones | hormonal carcinogenesis
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 11/2011, Volume 130, Issue 2, pp. 399 - 408
Clinical studies show that estrogen receptor-α (ER) expressing tumors tend to have better prognosis, respond to antiestrogen therapy and have wild-type p53.... 
Estrogen receptor | Medicine & Public Health | Mammary cancer | MMTV-Wnt-1 | Oncology | Breast cancer | Tamoxifen | p53 | ACTIVATION | WNT-1 TRANSGENIC MICE | TUMORS | BREAST-CANCER CELLS | GLAND HYPERPLASIA | ONCOLOGY | ESTROGEN-RECEPTOR-ALPHA | MOUSE MODEL | GENE-EXPRESSION | TUMORIGENESIS | IMMUNOHISTOCHEMISTRY | Mammary Neoplasms, Experimental - prevention & control | Wnt1 Protein - genetics | Gene Expression | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Genotype | Mice, Transgenic | Wnt1 Protein - metabolism | Loss of Heterozygosity | Mammary Neoplasms, Experimental - genetics | Tumor Suppressor Protein p53 - genetics | Animals | Estrogen Receptor alpha - genetics | Cell Transformation, Neoplastic - genetics | Antineoplastic Agents, Hormonal - therapeutic use | Tamoxifen - therapeutic use | Mammary Neoplasms, Experimental - pathology | Estrogen Receptor alpha - metabolism | Female | Heterozygote | Mice | Anthracyclines | Prognosis | RNA | Analysis | Estrogen | Genetic aspects | Research | Tumor proteins | Tumors | Index Medicus | Animal models | Transcription factors | Transcription | Estrogen receptors | Antiestrogens | Carcinogenesis | DNA-directed RNA polymerase | Doxorubicin | Transfection | Tumorigenesis | Mammary gland | Transgenes | Genotypes | Tumor cells | Transgenic mice | c-Jun protein | Data processing | Sp1 protein | Promoters | p53 protein | Mutation
Journal Article
Biochemistry (Moscow), ISSN 0006-2979, 10/2017, Volume 82, Issue 10, pp. 1118 - 1128
The insecticide dichlorodiphenyltrichloroethane (DDT) is a nonmutagenic xenobiotic compound able to exert estrogen-like effects resulting in activation of... 
Life Sciences | Biochemistry, general | Biomedicine, general | microRNAs | Microbiology | hormonal carcinogenesis | DDT | ERα | Bioorganic Chemistry | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INCREASES | MIR-205 | HUMAN BREAST-CANCER | TUMORS | ROLES | CLINICAL-APPLICATIONS | ESTROGEN-RECEPTOR-ALPHA | AROMATASE | ER alpha | Rats, Wistar | Uterus - drug effects | Uterus - metabolism | Gene Expression Regulation, Neoplastic | Aromatase - metabolism | RNA, Messenger - metabolism | DDT - toxicity | PTEN Phosphohydrolase - antagonists & inhibitors | Aromatase - genetics | DEAD-box RNA Helicases - antagonists & inhibitors | Estrogen Receptor alpha - metabolism | Female | Ovary - drug effects | DEAD-box RNA Helicases - metabolism | Ovary - metabolism | PTEN Phosphohydrolase - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | RNA, Messenger - genetics | Carcinogenesis - genetics | PTEN Phosphohydrolase - metabolism | Rats | Ribonuclease III - antagonists & inhibitors | DEAD-box RNA Helicases - genetics | Animals | Estrogen Receptor alpha - genetics | MicroRNAs - genetics | MicroRNA | Uterus | Genetic aspects | Ovaries | Gene expression | Genetic regulation | Health aspects | Carcinogenesis | Genes | Estrogens | Pesticides | MiRNA | Estrogen receptors | Activation | mRNA | Carcinogens | MicroRNAs | Rodents | Tumor suppressor genes | PTEN protein | Index Medicus
Journal Article
Hormones and Cancer, ISSN 1868-8497, 6/2019, Volume 10, Issue 2, pp. 77 - 88
Androgens are thought to cause prostate cancer, but the underlying mechanisms are unclear. Data from animal studies suggest that for androgens to cause... 
Hormonal carcinogenesis | Medicine & Public Health | Microbiology | Androgen | Estrogen | Oncology | Internal Medicine | Systems Biology | Prostate cancer | Endocrinology | Cell Biology
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e25125 - e25125
Epidemiological and experimental evidences strongly support the role of estrogens in breast tumor development. Both estrogen receptor (ER)-dependent and... 
OXIDATIVE STRESS | CATECHOL QUINONES | DNA-FRAGMENTATION-FACTOR | CONTAINING MONOOXYGENASE FMO | MULTIDISCIPLINARY SCIENCES | ANTIESTROGEN RESISTANCE | RECEPTOR MODULATORS | FEMALE ACI RATS | HUMAN LIVER-MICROSOMES | SYRIAN-HAMSTERS | DUCTAL BREAST-CANCER | Glutathione Peroxidase - metabolism | Rats | Estrogens - adverse effects | Mammary Neoplasms, Experimental - metabolism | Breast Neoplasms - drug therapy | Breast Neoplasms - metabolism | Catalase - metabolism | Dinoprostone - metabolism | Mammary Neoplasms, Experimental - chemically induced | Animals | Isoprostanes - metabolism | Antineoplastic Agents, Hormonal - therapeutic use | Tamoxifen - therapeutic use | Dinoprostone - analogs & derivatives | Estradiol - adverse effects | Deoxyguanosine - metabolism | Female | Oxidative Stress - drug effects | Breast Neoplasms - chemically induced | Deoxyguanosine - analogs & derivatives | Superoxide Dismutase - metabolism | Mammary Neoplasms, Experimental - drug therapy | Enzymes | Care and treatment | Phenols | Tamoxifen | Estradiol | Carcinogenesis | Epidemiology | Cancer | Cytochrome | Oxidative stress | Estrogens | Genes | DNA damage | Estrogen | Estrogen receptors | 17β-Estradiol | Superoxide dismutase | Kinases | Carcinogens | Catalase | Metabolites | 8-Hydroxydeoxyguanosine | Rodents | Animal tissues | Peroxidase | Deoxyribonucleic acid--DNA | Glutathione | Glutathione peroxidase | Kidneys | Breast cancer | Latency | Oxygenase | Sex hormones | Cell death | Breast | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article