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1. Human papilloma virus infection
: a clinical atlas
by Barrasso, Renzo and Gross, G. (Gerd)
1997, ISBN 3861261170, 432
Papillomaviruses | Papillomavirus, Human | pathology | therapy | diagnosis | Papillomavirus diseases | Papovaviridae Infections
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2. Human papilloma viruses
by McCance, Dennis J
2002, 1st ed., Perspectives in medical virology, ISBN 9780444506269, Volume 8, vii, 194
Papillomaviruses | physiology | Papillomavirus, Human | Health aspects | immunology | Papillomavirus Infections
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3. Full Text Human papillomavirus and cervical cancer
by Crosbie, Emma J, PhD and Einstein, Mark H, MD and Franceschi, Silvia, MD and Kitchener, Henry C, Prof
Lancet, The, ISSN 0140-6736, 2013, Volume 382, Issue 9895, pp. 889 - 899
Summary Cervical cancer is caused by human papillomavirus infection. Most human papillomavirus infection is harmless and clears spontaneously but persistent... 
Internal Medicine | WOMEN | POPULATION | MEDICINE, GENERAL & INTERNAL | PERSISTENCE | DNA | VACCINE | HPV INFECTION | FOLLOW-UP | RISK | CYTOLOGY | INTRAEPITHELIAL NEOPLASIA | Human papillomavirus 18 - immunology | Human papillomavirus 6 - immunology | Humans | Uterine Cervical Neoplasms - epidemiology | Human papillomavirus 11 - ultrastructure | Uterine Cervical Neoplasms - pathology | Human papillomavirus 16 - immunology | Human papillomavirus 6 - ultrastructure | Papillomavirus Infections - pathology | Papillomavirus Infections - immunology | Female | Papillomavirus Infections - virology | Human papillomavirus 11 - immunology | Human papillomavirus 16 - ultrastructure | Human papillomavirus 11 - pathogenicity | Papillomavirus Vaccines - administration & dosage | Human papillomavirus 6 - pathogenicity | Human papillomavirus 16 - pathogenicity | Cross-Sectional Studies | Papillomavirus Infections - epidemiology | Uterine Cervical Neoplasms - prevention & control | Uterine Cervical Neoplasms - immunology | Vaginal Smears | Mass Screening | Virus Replication | Human papillomavirus 18 - pathogenicity | Papillomavirus Infections - prevention & control | Uterine Cervical Neoplasms - virology | Human papillomavirus 18 - ultrastructure | Papillomavirus Vaccines - immunology | Developing Countries | Cell Transformation, Neoplastic - pathology | Cell Transformation, Viral | Prevention | Papillomaviruses | Usage | Vaccines | Properties | Papillomavirus infections | Risk factors | Cervical cancer | Studies | Human papillomavirus | Immunization | Cellular biology | Womens health | Medical screening | Epidemiology | Age
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4. Full Text Human papilloma virus and autophagy
by Mattoscio, Domenico and Medda, Alessandro and Chiocca, Susanna
International Journal of Molecular Sciences, ISSN 1661-6596, 06/2018, Volume 19, Issue 6, p. 1775
Human papilloma viruses (HPVs) are a group of double-stranded DNA viruses known to be the primary cause of cervical cancer. In addition, evidence has now... 
Oncoviral proteins | Head and neck cancer | Viral tumorigenesis | Autophagy | Cervical cancer | HPV | GLOBAL BURDEN | autophagy | HEPATITIS-B-VIRUS | BECLIN 1 | PROTEIN | head and neck cancer | oncoviral proteins | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | cervical cancer | CHEMISTRY, MULTIDISCIPLINARY | CELL-DEATH | viral tumorigenesis | PATHWAY | DEGRADATION | INFECTION | EXPRESSION | Antiviral agents | Head | Melanoma | Viruses | Lipids | Vaccines | Organelles | DNA viruses | Anogenital | Proteins | Papilloma viruses | Human papillomavirus | Uterus | Infectivity | Human papilloma viruses | Vacuoles | Phagocytosis | Viral infections | Deoxyribonucleic acid--DNA | Cancer
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5. Full Text Papillomavirus E6 oncoproteins
by Vande Pol, Scott B and Klingelhutz, Aloysius J
Virology, ISSN 0042-6822, 2013, Volume 445, Issue 1, pp. 115 - 137
Abstract Papillomaviruses induce benign and malignant epithelial tumors, and the viral E6 oncoprotein is essential for full transformation. E6 contributes to... 
Infectious Disease | Papillomavirus | Ubiquitin | Transformation | Viral oncogenes | Notch | Telomerase | Cervical cancer | P53 | CERVICAL-CARCINOMA CELLS | PDZ-BINDING MOTIF | UBIQUITIN LIGASE E6-AP | VIROLOGY | TYPE-16 E7 ONCOPROTEIN | CALCIUM-INDUCED DIFFERENTIATION | TUMOR-SUPPRESSOR PROTEIN | ANCHORAGE-INDEPENDENT GROWTH | FOCAL ADHESION PROTEIN | NF-KAPPA-B | MAMMARY EPITHELIAL-CELLS | Paxillin - metabolism | Papillomaviridae - metabolism | Humans | Ubiquitin-Protein Ligases - metabolism | Uterine Cervical Neoplasms - pathology | Papillomaviridae - genetics | Protein Folding | Amino Acid Motifs | Papillomavirus Infections - pathology | Ubiquitination | Animals | Papillomavirus Infections - metabolism | Protein Binding | Uterine Cervical Neoplasms - virology | Female | Oncogene Proteins, Viral - genetics | Oncogene Proteins, Viral - metabolism | Peptides | Cellular proteins | Protein binding
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6. Full Text Papillomavirus E6 proteins
by Howie, Heather L and Katzenellenbogen, Rachel A and Galloway, Denise A
Virology, ISSN 0042-6822, 2008, Volume 384, Issue 2, pp. 324 - 334
Abstract The papillomaviruses are small DNA viruses that encode approximately eight genes, and require the host cell DNA replication machinery for their viral... 
Infectious Disease | E6 oncogene | hTERT | HPV | p53 | Apoptosis | P53 DNA-BINDING | PRIMARY HUMAN KERATINOCYTES | HPV E6 | UBIQUITIN LIGASE E6-AP | POLY(A) BINDING-PROTEINS | VIROLOGY | CATALYTIC SUBUNIT HTERT | ANCHORAGE-INDEPENDENT GROWTH | TYPE-16 E6 | TELOMERASE REVERSE-TRANSCRIPTASE | MAMMARY EPITHELIAL-CELLS | Cell Polarity | Papillomaviridae - metabolism | Tumor Suppressor Protein p53 - metabolism | Cell Adhesion | Virus Replication | Papillomavirus Infections - immunology | Papillomavirus Infections - virology | Telomerase - metabolism | Cell Differentiation | Papillomaviridae - physiology | Chromosomal Instability | Cell Transformation, Viral | GTP-Binding Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Papillomaviruses | DNA replication | Oncology, Experimental | DNA | DNA synthesis | Research | Tumor proteins | Telomerase | Health aspects | Papillomavirus infections | Cervical cancer | Cancer | ONCOGENES | APOPTOSIS | NEOPLASMS | TRANSCRIPTION | CELL PROLIFERATION | HOST | 60 APPLIED LIFE SCIENCES | ADHESION | DNA REPLICATION | SYNTHESIS | CELL CYCLE | PROTEINS | VIRUSES
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7. Full Text The papillomavirus E7 proteins
by Roman, Ann and Munger, Karl
Virology, ISSN 0042-6822, 2013, Volume 445, Issue 1, pp. 138 - 168
Abstract E7 is an accessory protein that is not encoded by all papillomaviruses. The E7 amino terminus contains two regions of similarity to conserved regions... 
Infectious Disease | Viral life cycle | Transformation | Regulation of cell cycle checkpoints | Viral oncology | Regulation of transcription | Cervical cancer | OPEN READING FRAME | HUMAN EPITHELIAL-CELLS | VIRAL LIFE-CYCLE | CERVICAL-CARCINOMA CELLS | RETINOBLASTOMA TUMOR-SUPPRESSOR | NECROSIS-FACTOR-ALPHA | CELL-CYCLE ARREST | KINASE-II PHOSPHORYLATION | VIROLOGY | RISK HUMAN PAPILLOMAVIRUSES | NORMAL HUMAN KERATINOCYTES | Amino Acid Sequence | Humans | Open Reading Frames | Molecular Sequence Data | Repressor Proteins - genetics | Uterine Cervical Neoplasms - pathology | Human papillomavirus 16 - metabolism | Papillomavirus E7 Proteins - genetics | Papillomavirus Infections - pathology | Papillomavirus E7 Proteins - metabolism | Ubiquitination | Animals | Cell Transformation, Neoplastic | Uterine Cervical Neoplasms - virology | Female | Oncogene Proteins, Viral - genetics | Papillomavirus Infections - virology | Transcription, Genetic | Mutation | Human papillomavirus 16 - genetics | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Papillomaviruses | Proteins | Papillomavirus infections
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