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Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2009, Volume 284, Issue 16, pp. 10774 - 10782
The innate immune system provides an initial defense system against microbial infections and contributes to the development of adaptive immune response. Type I... 
DOUBLE-STRANDED-RNA | SIGNALING PATHWAYS | IMMUNE-RESPONSES | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | GENE-EXPRESSION | INNATE | TNF-ALPHA | HELICASE ANTIVIRAL PATHWAY | NF-KAPPA-B | ADAPTER PROTEIN | Fibroblasts - physiology | TNF Receptor-Associated Factor 2 - genetics | Immunity, Innate - physiology | p38 Mitogen-Activated Protein Kinases - metabolism | Interferons - genetics | DEAD-box RNA Helicases - metabolism | Virus Activation | Dendritic Cells - metabolism | Sendai virus - physiology | DEAD Box Protein 58 | MAP Kinase Kinase Kinases - genetics | Mice, Inbred C57BL | Cells, Cultured | TNF Receptor-Associated Factor 2 - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | MAP Kinase Kinase Kinases - metabolism | Mice, Knockout | DEAD-box RNA Helicases - genetics | Animals | Adaptor Proteins, Signal Transducing - genetics | Interferons - metabolism | Mitogen-Activated Protein Kinases - genetics | Signal Transduction - physiology | Dendritic Cells - cytology | Fibroblasts - cytology | Mice | Enzyme Activation | Adaptor Proteins, Signal Transducing - metabolism | Mitogen-Activated Protein Kinases - metabolism | Pathogens | RNA viruses | Phosphorylation | Dendritic cells | TAK1 protein | MKK3 protein | Interleukin 12 | MAP kinase | a-Interferon | Infection | TRAF2 protein | Cell activation | Enzymatic activity | c-Jun amino-terminal kinase | Protein kinase | Fibroblasts | Replication | Interferon
Journal Article
The Journal of Infectious Diseases, ISSN 0022-1899, 7/2013, Volume 208, Issue 1, pp. 50 - 56
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 665 - 680
Cellular lipid metabolism has been linked to immune responses; however, the precise mechanisms by which de novo fatty acid synthesis can regulate inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | GLUCOSE-METABOLISM | ATP-CITRATE LYASE | UCP2 | SEPTIC SHOCK | CELL-GROWTH | CRITICALLY-ILL PATIENTS | INTENSIVE INSULIN THERAPY | CANCER | MITOCHONDRIAL UNCOUPLING PROTEINS | Fatty Acid Synthase, Type I - genetics | Fatty Acid Synthase, Type I - biosynthesis | Lipids - genetics | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Interleukin-1beta - genetics | Lipids - biosynthesis | Sepsis - pathology | Interleukin-1beta - metabolism | Mitochondrial Proteins - metabolism | Sepsis - metabolism | Macrophages - pathology | Down-Regulation - genetics | Inflammasomes - genetics | Sepsis - genetics | Mice, Knockout | Carrier Proteins - genetics | Enzyme Induction - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Sepsis - chemically induced | Ion Channels - metabolism | Caspase 1 - genetics | Sepsis - therapy | Mice | Uncoupling Protein 2 | Interleukin-18 - genetics | Interleukin-18 - metabolism | Cellular proteins | Immune response | Synthesis | Sepsis | Development and progression | Lipids | Genetic aspects | Properties | Enzymes | Cytokines | Pathogenesis | Mortality | Glucose | Metabolism | Gene expression | Fatty acids | Proteins | Studies | Metabolites | Consent | Rodents
Journal Article