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PAIN, ISSN 0304-3959, 06/2019, Volume 160, Issue 6, pp. 1431 - 1447
Inflammation or injuries of the trigeminal nerve are often associated with persistent facial pain and its sequelae. A number of models have been described to... 
Headache | TRIGEMINAL NEUROPATHIC PAIN | GANGLION NEURONS | Memory | BEHAVIOR | MOUSE | Nerve injury | RATS | Trigeminal pain | Cognition | ACTIVATING TRANSCRIPTION FACTOR-3 | NEUROSCIENCES | SENSORY NEURONS | CLINICAL NEUROLOGY | Learning | RESPONSES | Optogenetic place preference | SENSITIZATION | Infraorbital nerve | CHRONIC CONSTRICTION INJURY | ANESTHESIOLOGY | IntelliCage
Journal Article
Journal of Pain, ISSN 1526-5900, 2008, Volume 9, Issue 11, pp. 1036 - 1048
Abstract Trigeminal nerve damage often leads to chronic pain syndromes including trigeminal neuralgia, a severely debilitating chronic orofacial pain syndrome.... 
Anesthesia & Perioperative Care | Pain Medicine | neuron | caudal medulla | allodynia | glia | Infraorbital nerve | RAT SPINAL-CORD | ACTIVATED PROTEIN-KINASE | GENE-RELATED PEPTIDE | PERSISTENT PAIN | BRAIN-STEM | NEUROSCIENCES | DORSAL-HORN | CLINICAL NEUROLOGY | NEUROPATHIC PAIN | SCIATIC-NERVE | CHRONIC CONSTRICTION INJURY | SUBSTANCE-P | Immunohistochemistry | Microglia - metabolism | Neurons - pathology | Cell Proliferation | Trigeminal Nerve Injuries | Astrocytes - pathology | Male | Trigeminal Neuralgia - etiology | Ligation - methods | Satellite Cells, Perineuronal - metabolism | Medulla Oblongata - physiopathology | Facial Nerve Injuries - physiopathology | Microglia - pathology | Medulla Oblongata - pathology | Receptors, Neurokinin-1 - metabolism | Neurons - metabolism | Calcitonin Gene-Related Peptide - metabolism | Disease Models, Animal | Grooming - physiology | Trigeminal Ganglion - pathology | Mice, Inbred C57BL | Medulla Oblongata - metabolism | Trigeminal Ganglion - metabolism | Trigeminal Neuralgia - physiopathology | Behavior, Animal - physiology | Activating Transcription Factor 3 - metabolism | Animals | Analysis of Variance | Facial Nerve Injuries - etiology | Trigeminal Neuralgia - pathology | Facial Nerve Injuries - pathology | Substance P - metabolism | Mice | Satellite Cells, Perineuronal - pathology | Astrocytes - metabolism | Care and treatment | Pain | Peptide hormones | Analysis | infraorbital nerve
Journal Article
Annals of Plastic Surgery, ISSN 0148-7043, 02/2018, Volume 80, Issue 2, pp. 141 - 144
BACKGROUNDPatients with facial fracture or head and neck surgery sometimes suffer from infraorbital nerve injury. This injury results in severe hemilateral... 
head and neck | facial trauma | nerve transfer | peripheral nerve | Infraorbital nerve | SURGERY | TUBE | RECONSTRUCTION | FACIAL DISMASKING FLAP | BASE | REPAIR | SQUAMOUS-CELL CARCINOMA | SPREAD
Journal Article
Journal Article
Journal of Veterinary Internal Medicine, ISSN 0891-6640, 11/2013, Volume 27, Issue 6, pp. 1571 - 1580
Journal Article
Cytokine, ISSN 1043-4666, 04/2018, Volume 104, pp. 124 - 129
The present study evaluated the effects of systemic pregabalin (PG) and diclofenac (Dic) on neuropathic orofacial pain induced by chronic constriction injury... 
Pain | Orofacial | Infraorbital nerve | Pregabalin | IL-1β | IL-6 | Neuropathic | BIOCHEMISTRY & MOLECULAR BIOLOGY | GUIDELINES | OROFACIAL PAIN | LIGATION | IL-1 beta | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | DAMAGE | DEGENERATION | CELL BIOLOGY | TRIAL | NEUROPATHIC PAIN | MODELS | NEURONS
Journal Article
Pain, ISSN 0304-3959, 2006, Volume 121, Issue 3, pp. 219 - 231
Glial activation is known to contribute to pain hypersensitivity following spinal sensory nerve injury. In this study, we investigated mechanisms by which... 
OX-42 | Medullary dorsal horn | Mental nerve | c-Fos | Minocycline | p38 mitogen-activated protein kinase | Glia | Inferior alveolar nerve | Tactile hypersensitivity | GFAP | MECHANICAL ALLODYNIA | DIFFERENTIAL EXPRESSION | mental nerve | SPINAL-CORD | NECROSIS-FACTOR-ALPHA | tactile hypersensitivity | FOS-LIKE IMMUNOREACTIVITY | PATHOLOGICAL PAIN | glia | NEUROSCIENCES | CLINICAL NEUROLOGY | medullary dorsal horn | CONSTRICTION INJURY | NEUROPATHIC PAIN | minocycline | NUCLEUS CAUDALIS | inferior alveolar nerve | ANESTHESIOLOGY | INFRAORBITAL NERVE | Immunohistochemistry | Microglia - metabolism | Trigeminal Nerve Injuries | Wallerian Degeneration - pathology | Minocycline - pharmacology | Trigeminal Caudal Nucleus - metabolism | Male | CD11b Antigen | Gliosis - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Trigeminal Caudal Nucleus - cytology | Up-Regulation - physiology | Neurons, Afferent - pathology | Trigeminal Nerve Diseases - metabolism | Trigeminal Nerve - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Trigeminal Nerve Diseases - complications | Gliosis - etiology | Disease Models, Animal | Astrocytes - cytology | Biomarkers - metabolism | Microglia - cytology | Trigeminal Nerve Diseases - physiopathology | Hyperalgesia - metabolism | Trigeminal Ganglion - pathology | Gliosis - metabolism | Wallerian Degeneration - physiopathology | Rats | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Animals | Trigeminal Ganglion - physiopathology | Hyperalgesia - etiology | Enzyme Activation - physiology | Astrocytes - metabolism | Antigen-antibody reactions | Allergy | Neurosciences | Analysis | Allergic reaction | Intermediate filament proteins | Protein kinases
Journal Article