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American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 06/2014, Volume 306, Issue 12, pp. E1378 - E1387
Incomplete beta-oxidation of fatty acids in mitochondria is a feature of insulin resistance and type 2 diabetes mellitus (T2DM). Previous studies revealed that... 
Acylcarnitine | β-oxidation | Inflammation | TLR | Pattern recognition receptors | OXIDATION | PHYSIOLOGY | acylcarnitine | SATURATED FATTY-ACIDS | INDUCED INSULIN-RESISTANCE | pattern recognition receptors | TOLL-LIKE RECEPTOR-2 | OBESITY | inflammation | beta-oxidation | ENDOCRINOLOGY & METABOLISM | EXPRESSION | Phosphorylation | Toll-Like Receptor 2 - agonists | Toll-Like Receptor 2 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | Humans | Carnitine - metabolism | Diabetes Mellitus, Type 2 - metabolism | MAP Kinase Signaling System | Receptors, Pattern Recognition - antagonists & inhibitors | Receptors, Pattern Recognition - metabolism | Macrophages - secretion | Cyclooxygenase 2 - genetics | Diabetes Mellitus, Type 2 - immunology | Myeloid Differentiation Factor 88 - antagonists & inhibitors | Receptors, Pattern Recognition - agonists | Carnitine - analogs & derivatives | Macrophages - immunology | Recombinant Proteins - metabolism | Myristic Acids - metabolism | Cyclooxygenase 2 - chemistry | Cytokines - secretion | Gene Silencing | Myeloid Differentiation Factor 88 - genetics | Enzyme Induction | Recombinant Proteins - chemistry | Toll-Like Receptor 2 - metabolism | Macrophage Activation | Myeloid Differentiation Factor 88 - agonists | Macrophages - metabolism | Animals | Cyclooxygenase 2 - metabolism | Cell Line, Tumor | Receptors, Pattern Recognition - genetics | Mice | Protein Processing, Post-Translational | Myeloid Differentiation Factor 88 - metabolism | Cell Line, Transformed | Docosahexaenoic Acids - metabolism | Type 2 diabetes | Physiological aspects | Insulin resistance | Cellular signal transduction | Research | Diabetes | Oxidation | T cell receptors | Cytokines | Index Medicus
Journal Article
Journal Article
Science, ISSN 0036-8075, 4/2012, Volume 336, Issue 6080, pp. 477 - 481
In metazoans, cells depend on extracellular growth factors for energy homeostasis. We found that glycogen synthase kinase-3 (GSK3), when deinhibited by default... 
T lymphocytes | Phosphorylation | Cell growth | HCT116 cells | REPORTS | Cell lines | Antibodies | Homeostasis | Inductive reasoning | Acetylation | Cells | INACTIVATION | INSULIN | INHIBITION | PHOSPHORYLATION | GSK3 | MULTIDISCIPLINARY SCIENCES | GLYCOGEN-SYNTHASE KINASE-3 | MACHINERY | ULK1 | CELL-SURVIVAL | MTOR | Humans | Histone Acetyltransferases - genetics | Culture Media, Serum-Free | Glycogen Synthase Kinase 3 beta | Intracellular Signaling Peptides and Proteins - metabolism | Autophagy | Autophagy-Related Protein-1 Homolog | Intercellular Signaling Peptides and Proteins - metabolism | Culture Media | Histone Acetyltransferases - metabolism | HEK293 Cells | Trans-Activators - genetics | Lysine Acetyltransferase 5 | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Cell Line | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Rats | Glycogen Synthase Kinase 3 - metabolism | Animals | Glycogen Synthase Kinase 3 - genetics | Cell Line, Tumor | Glucose - metabolism | Trans-Activators - metabolism | Mice | Synthesis | Glycogen | Transferases | Physiological aspects | Research | Health aspects | Growth factors | Proteins | Signal transduction | Yeast | Cellular biology | Microbiology | Index Medicus | Pathways | Histones | Kinases | Mammals | Serums
Journal Article
Current Pharmaceutical Design, ISSN 1381-6128, 01/2018, Volume 24, Issue 2, pp. 158 - 170
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, pp. e47713 - e47713
Background & Aims: While it is widely accepted that obesity is associated with low-grade systemic inflammation, the molecular origin of the inflammation... 
SYSTEM | ACTIVATION | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | DISEASE | ECOLOGY | MODEL | NF-KAPPA-B | ADIPOSE-TISSUE | INFILTRATION | Endotoxins - blood | Diet, High-Fat - adverse effects | Male | Adipose Tissue - metabolism | Inflammation - metabolism | Forkhead Transcription Factors - metabolism | Inflammation Mediators - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Cytokines - blood | Endotoxins - metabolism | Cytokines - metabolism | Macrophages - pathology | Signal Transduction | Adipose Tissue - pathology | Inflammation Mediators - blood | Gastrointestinal Tract - microbiology | Toll-Like Receptor 4 - genetics | Adiposity - genetics | Toll-Like Receptor 4 - metabolism | Gastrointestinal Tract - metabolism | Mice, Knockout | Obesity - metabolism | Colitis - etiology | Animals | Mice | Forkhead Box Protein O3 | Metagenome | Obesity | Microbiota (Symbiotic organisms) | Causes of | Physiological aspects | Toll-like receptors | Development and progression | Inflammation | Genetic aspects | Research | Ketogenic diet | Phosphorylation | Adipose tissue | Body weight | AKT protein | Kinases | Macrophages | High fat diet | Proteins | Signal transduction | Microbiota | Intestine | Rodents | Colon | Digestive tract | FOXO3 protein | NF-κB protein | Cytokines | Digestive system | Permeability | TLR4 protein | Nitric-oxide synthase | Immune systems | Signaling | Insulin resistance | Infiltration | Cyclooxygenase-2 | Index Medicus
Journal Article
2001, Progress in molecular and subcellular biology, ISBN 3540417095, Volume 26., x, 186
Book
PARASITES & VECTORS, ISSN 1756-3305, 04/2019, Volume 12, Issue 1, pp. 187 - 187
BackgroundSignalling pathways have been extensively investigated in the free-living nematode Caenorhabditis elegans, but very little is known about these... 
Haemonchus contortus | PARASITIC NEMATODES | TGF-BETA | INSULIN-LIKE PEPTIDE | PHYSIOLOGICAL-FUNCTION | Proteomic analysis | Transcriptomic analysis | TROPICAL MEDICINE | NUCLEAR RECEPTOR | ARRESTED DEVELOPMENT | Phospho-proteomic analysis | CAENORHABDITIS-ELEGANS | GENE | Dauer signalling pathway | DIAPAUSE | EXPRESSION | PARASITOLOGY | Signal Transduction | Genomics | Gene Expression Regulation | Transcriptome | Models, Molecular | Haemonchus - metabolism | Gene Expression Profiling | Transcription Factors - genetics | Haemonchus - genetics | Animals | Metabolic Networks and Pathways | Proteomics | Caenorhabditis elegans Proteins - genetics | Caenorhabditis elegans | Analysis | Genes | Bone morphogenetic proteins | Genetic transcription | G proteins | Transforming growth factors | Nematoda | Cyclic guanylic acid | Steroid hormones | Transcription factors | Phosphorylation | Target recognition | G protein-coupled receptors | Transcription | Peptides | Insulin-like growth factor I | Crosstalk | Biological evolution | Transforming growth factor | Guanosine | Lipids | Homology | Genomes | Insulin-like growth factors | Hormones | Parasites | Proteins | Signal transduction | Receptors | Pathways | Parasitic diseases | Cyclic GMP | Profiles | Post-translation | Growth factors | Worms | Economic models | Therapeutic applications | MiRNA | Insulin | Biological activity | Signaling | MicroRNAs | Regulatory mechanisms (biology) | Nematodes | Ligands | Steroids | Index Medicus
Journal Article