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PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0128998
A variety of cytokine/cytokine receptor systems affect the biological behavior of acute leukemia cells. However, little is known about the clinical relevance... 
MAINTENANCE INTENSIFICATION THERAPY | SOLUBLE INTERLEUKIN-2-RECEPTOR | CELLS | INDIVIDUALIZED INDUCTION THERAPY | GM-CSF | MULTIDISCIPLINARY SCIENCES | ACUTE MYELOGENOUS LEUKEMIA | CONSOLIDATION THERAPY | PROLIFERATION | CLINICAL IMPORTANCE | HEMATOPOIETIC GROWTH-FACTORS | Prognosis | Leukemia, Myeloid, Acute - pathology | Humans | Leukemia, Myeloid, Acute - metabolism | Middle Aged | Male | Risk | Phenotype | Leukemia, Myeloid, Acute - diagnosis | Interleukin-2 Receptor alpha Subunit - metabolism | Adult | Female | Gene Expression Regulation, Neoplastic - drug effects | Cytokines - pharmacology | Karyotype | Leukemia, Myeloid, Acute - genetics | Flow cytometry | Leukemia | Interleukin | Cytokine receptors | Chains | Oncology | Kinases | Macrophages | Interleukin 6 | Receptors | Interleukin 2 | Granulocyte colony-stimulating factor | Risk assessment | Cell cycle | Bone marrow | CD25 antigen | Colony-stimulating factor | Granulocyte colony-stimulating factor receptors | University graduates | Glycoprotein gp130 | Interleukin 2 receptors | Cytokines | Internal medicine | Hematology | Granulocyte-macrophage colony-stimulating factor | Myeloid leukemia | Patients | c-Kit protein | Medicine | Cytometry | Chemotherapy | Leukocytosis | Medical prognosis | Stem cells | Cytogenetics | Lymphomas | Mutation | Acute myeloid leukemia | Cancer
Journal Article
Journal Article
by Lee, JJ and Yeh, CY and Jung, CJ and Chen, CW and Du, MK and Yu, HM and Yang, CJ and Lin, HY and Sun, A and Ko, JY and Cheng, SJ and Chang, YL and Chia, JS
PLOS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e85521
CD8(+) T cells play important roles in anti-tumor immunity but distribution profile or functional characteristics of effector memory subsets during tumor... 
HEAD | IMMUNE CELLS | IMMUNOTHERAPY | MULTIDISCIPLINARY SCIENCES | INDOLEAMINE 2,3-DIOXYGENASE | GENERATION | PROLIFERATION | IDENTIFICATION | CANCER | INTERLEUKIN-7 | LYMPHOCYTES | Interleukin-2 - metabolism | Cell Proliferation | Leukocyte Common Antigens - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Mouth Neoplasms - metabolism | Interferon-gamma - metabolism | Carcinoma, Squamous Cell - immunology | Case-Control Studies | Disease Progression | Mouth Neoplasms - immunology | Receptors, CCR7 - metabolism | Interleukin-7 Receptor alpha Subunit - metabolism | CD8-Positive T-Lymphocytes - metabolism | Mouth Neoplasms - pathology | Immunologic Memory | Tumor Cells, Cultured | Apoptosis | CC chemokine receptors | Laboratories | CD8 antigen | CD45RA antigen | Otolaryngology | Effector cells | Cytotoxicity | Lymphocytes T | Metastasis | Immunity | Lymph nodes | Molecular weight | Maxillofacial surgery | Interleukin 7 | Cell activation | Granzyme B | Immunology | Interleukin 2 | Lymphocytes | Immunotherapy | Surgery | Peripheral blood | Productivity | Antigens | Immunological memory | Memory cells | Squamous cell carcinoma | Cytokines | Tumor cells | CCR7 protein | Skewed distributions | Oral squamous cell carcinoma | Medicine | Cell death | γ-Interferon | Proteomics | Interferon | Head & neck cancer | Binding sites
Journal Article
Immunology, ISSN 0019-2805, 10/2010, Volume 131, Issue 2, pp. 289 - 299
Summary Invariant natural killer T (iNKT) cells are known to constitutively express the high affinity interlukin‐2 receptor α chain (CD25) in neonates, but the... 
T‐cell proliferation | cord blood | invariant natural killer T cells | interleukin‐2 receptor α chain | interleukin-2 receptor α chain | T-cell proliferation | SUBSETS | PRECURSOR FREQUENCIES | RECEPTOR | PROLIFERATION | IMMUNOLOGY | interleukin-2 receptor alpha chain | IN-VITRO | LYMPHOCYTE SUBPOPULATIONS | MEMORY | INVARIANT NKT CELLS | CD4(+) | V-ALPHA-24 | Infant, Newborn - immunology | Labor, Obstetric - immunology | Leukocyte Common Antigens - metabolism | Gene Expression - genetics | Humans | Male | Antigens, CD - metabolism | L-Selectin - metabolism | Lectins, C-Type - metabolism | Aging - immunology | Lymphocyte Activation - immunology | Interleukin-2 - immunology | Forkhead Transcription Factors - metabolism | Immune Tolerance - immunology | Natural Killer T-Cells - metabolism | Adult | Female | Receptors, Antigen, T-Cell - immunology | Antibodies, Monoclonal - immunology | Interleukin-2 Receptor alpha Subunit - immunology | Antibodies, Monoclonal - pharmacology | Antigens, Differentiation, T-Lymphocyte - metabolism | Natural Killer T-Cells - cytology | CD28 Antigens - immunology | Fetal Blood - cytology | Pregnancy | Interleukin-2 Receptor alpha Subunit - metabolism | Lymphocyte Activation - drug effects | Interleukin-7 Receptor alpha Subunit - metabolism | Interleukin-2 - pharmacology | Lymphocyte Count | Antigens - immunology | T-Lymphocytes - immunology | Cell Proliferation - drug effects | Kruppel-Like Transcription Factors - genetics | Natural Killer T-Cells - immunology | Original
Journal Article
Biotechnology and Applied Biochemistry, ISSN 0885-4513, 06/2008, Volume 50, Issue 2, pp. 97 - 112
Journal Article
Journal of International Medical Research, ISSN 0300-0605, 10/2011, Volume 39, Issue 5, pp. 1625 - 1635
Journal Article
BMC Cancer, ISSN 1471-2407, 08/2017, Volume 17, Issue 1, pp. 551 - 17
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e36316
Journal Article
Immunology, ISSN 0019-2805, 11/2010, Volume 131, Issue 3, pp. 371 - 376
Summary Interleukin‐2 (IL‐2) is one of the most studied cytokines driving T‐cell proliferation, activation and survival. It binds to the IL‐2 receptor... 
T regulatory cell | immune escape mechanism | CD25 | interleukin‐2 receptor α | B‐cell malignancy | Interleukin-2 receptor α | Immune escape mechanism | B-cell malignancy | INTERLEUKIN-2-RECEPTOR | TUMOR | RECEPTOR | IL-2 | LEVEL | IMMUNOLOGY | CANCER | interleukin-2 receptor alpha | NON-HODGKINS-LYMPHOMA | T-Lymphocyte Subsets - immunology | Tumor Escape - drug effects | T-Lymphocytes, Regulatory - metabolism | Lymphoma, B-Cell - blood | Humans | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Flow Cytometry | Interleukin-2 Receptor alpha Subunit - genetics | Lymphoma, B-Cell - immunology | Interleukin-2 Receptor alpha Subunit - immunology | Tumor Escape - immunology | Recombinant Proteins - metabolism | Forkhead Transcription Factors - biosynthesis | Enzyme-Linked Immunosorbent Assay | Interleukin-2 Receptor alpha Subunit - blood | Cell Separation | Cells, Cultured | Recombinant Proteins - genetics | T-Lymphocyte Subsets - pathology | Interleukin-2 Receptor alpha Subunit - metabolism | Recombinant Proteins - immunology | T-Lymphocyte Subsets - metabolism | Lymphoma, B-Cell - pathology | Cell Proliferation - drug effects | Care and treatment | Non-Hodgkin's lymphomas | T cells | Enzyme-linked immunosorbent assay | Cancer | Cell growth | Medical prognosis | T cell receptors | Original | interleukin-2 receptor α | Immunologi | Immunology | Medical and Health Sciences | Medicin och hälsovetenskap | MEDICIN | MEDICINE
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 10/1998, Volume 188, Issue 7, pp. 1369 - 1373
The expression of the murine interleukin (IL)-2 receptor or chain/CD25 is strongly induced at the transcriptional level after T cell activation. We show here... 
NF-AT factors | Transcription factors | T cells | Interleukin 2 receptor | Nuclear factor of activated T cells | MEDICINE, RESEARCH & EXPERIMENTAL | HMG-I(Y) | transcription factors | ENHANCER | IL-2 | INDUCTION | IMMUNOLOGY | ELF-1 | MICE LACKING | nuclear factor of activated T cells | FAMILY | GENE | interleukin 2 receptor | EXPRESSION
Journal Article