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Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 09/2017, Volume 140, Issue 3, pp. 809 - 821.e3
Hypertension is considered an immunologic disorder. However, the role of the IL-17 family in genetic hypertension in the spontaneously hypertensive rat (SHR)... 
Hypertension | CD161 | IL-17 | spontaneously hypertensive rat | digoxin | T cells | retinoic acid receptor–related orphan receptor γt | TH17 | Toll-like receptor | gene expression | innate immune system | T | 17 | INTERLEUKIN 17 | ACTIVATION | ENDOTHELIAL DYSFUNCTION | ORGAN DAMAGE | IMMUNOLOGY | NICOTINIC ACETYLCHOLINE-RECEPTORS | RESPONSES | ALLERGY | INFLAMMATION | T(H)17 | retinoic acid receptor-related orphan receptor gamma t | DIFFERENTIATION | TH17 CELLS | LYMPHOCYTES | Animals, Newborn | Rats, Inbred SHR | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - antagonists & inhibitors | Poly I-C - pharmacology | Cells, Cultured | Interleukin-17 - immunology | Interleukin-17 - physiology | Interleukin-17 - genetics | Rats, Inbred WKY | Male | Hypertension - immunology | Spleen - cytology | Toll-Like Receptor 3 - agonists | NK Cell Lectin-Like Receptor Subfamily B - immunology | Digoxin - pharmacology | Hypertension - physiopathology | Vasodilation | Aging - immunology | Animals | Aorta, Thoracic - physiology | Blood Pressure - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | RNA - metabolism | Flow cytometry | Transcription factors | Digoxin | Disease | Helper cells | Nervous system | Lymphocytes T | Birth | Kinases | Signal transduction | Splenocytes | Lymphocytes | Rodents | Measurement methods | Blood pressure | Immune system | Spleen | Immune response | Cytokines | Rats | Pharmacology | Inflammation | T cell receptors | Interleukin 17 | Endothelium | Immune systems | Acids | Prostate | Surface markers | Retinoic acid | Nicotine | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14751 - 14756
In several murine models of autoimmune arthritis, Th17 cells are the dominant initiators of inflammation. In human arthritis the majority of IL-17–secreting... 
T lymphocytes | Synovial fluid | Phenotypes | Cytokines | Juvenile rheumatoid arthritis | Cultured cells | Arthritis | Joint diseases | Joints | T cell antigen receptors | Juvenile | CD161 | RORC2 | RHEUMATOID-ARTHRITIS | GAMMA | MULTIDISCIPLINARY SCIENCES | JUVENILE IDIOPATHIC ARTHRITIS | CD4(+) T-CELLS | INDUCTION | MEMORY CELLS | IL-17 | juvenile | INFLAMED JOINTS | T-H-17 CELLS | LINEAGE | Receptors, CCR6 - immunology | Humans | Interleukin-17 - immunology | Molecular Sequence Data | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Base Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Interferon-gamma - genetics | Child | Cell Lineage - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Transforming Growth Factor beta - immunology | Amino Acid Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Interleukin-17 - genetics | Interleukin-12 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Arthritis, Juvenile - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Arthritis, Juvenile - metabolism | Interleukin-17 - metabolism | NK Cell Lectin-Like Receptor Subfamily B - genetics | Arthritis, Juvenile - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Receptors, CCR6 - genetics | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Autoimmunity | Research | Adaptation (Physiology) | Genotype & phenotype | Rodents | T cell receptors | Gene expression | Inflammatory diseases | Children & youth | Index Medicus | Animal models | Transcription factors | Helper cells | Interleukin 12 | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | double prime T-cell receptor | CCR6 protein | Interleukin 17 | Plasticity | Children | Biological Sciences
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 3965 - 3976
Psoriasis is a chronic inflammatory disorder of the skin affecting approximately 2% of the world's population. Accumulating evidence has revealed that the... 
GENERALIZED PUSTULAR PSORIASIS | MEDICINE, RESEARCH & EXPERIMENTAL | INFLAMMATORY SKIN-DISEASE | FAMILY-MEMBERS | MOUSE MODEL | DELTA-T-CELLS | NF-KAPPA-B | AUTOINFLAMMATORY DISEASE | INTERLEUKIN-12/23 MONOCLONAL-ANTIBODY | CUTTING EDGE | TRANSGENIC MICE | Cell Communication - immunology | Interleukin-1 - genetics | Membrane Glycoproteins - metabolism | Dermatitis - immunology | Dendritic Cells - immunology | Receptors, Interleukin-1 - genetics | Interleukin-17 - immunology | Dendritic Cells - pathology | Interleukins - metabolism | Interleukin-23 - genetics | Interleukins - genetics | Psoriasis - genetics | Interleukin-23 - metabolism | Psoriasis - pathology | Interleukins - immunology | Toll-Like Receptor 7 - immunology | Interleukin-1 - immunology | Psoriasis - metabolism | Membrane Glycoproteins - immunology | Dendritic Cells - metabolism | Psoriasis - immunology | Toll-Like Receptor 7 - genetics | Interleukin-1 - metabolism | Toll-Like Receptor 7 - metabolism | Dermatitis - genetics | Receptors, Interleukin-1 - immunology | Interleukin-17 - genetics | Cell Communication - genetics | Interleukin-23 - immunology | Keratinocytes - immunology | Membrane Glycoproteins - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Dermatitis - pathology | Interleukin-17 - metabolism | Keratinocytes - pathology | Animals | Keratinocytes - metabolism | Mice | Dermatitis - metabolism | Psoriasis | Interleukins | Keratinocytes | Genetic aspects | Dosage and administration | Diagnosis | Drug therapy | Index Medicus | Abridged Index Medicus | TLR7 protein | Dendritic cells | Mesenchyme | Interleukin 22 | Interleukin 23 | Leukocytes (neutrophilic) | Interleukin 1 receptors | Dermatitis | Inflammatory diseases | Interleukin 17 | Toll-like receptors | Skin
Journal Article
2012, Cytokines : intercellular mediators regulating host defense
Web Resource
Journal of Experimental Medicine, ISSN 0022-1007, 09/2009, Volume 206, Issue 10, pp. 2079 - 2089
Journal Article
The Journal of Immunology, ISSN 0022-1767, 10/2007, Volume 179, Issue 8, pp. 5462 - 5473
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2012, Volume 287, Issue 45, pp. 38168 - 38177
Th17 lymphocytes play a key role during immune responses against bacteria and fungi and are involved in the pathophysiology of multiple autoimmune disorders.... 
SYSTEMIC-LUPUS-ERYTHEMATOSUS | EFFECTOR FUNCTIONS | ACTIVATION | SAP | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | IL-17 | MECHANISMS | DIFFERENTIATION | TH17 | NFAT | Antigens, CD - immunology | Humans | Receptor-CD3 Complex, Antigen, T-Cell - genetics | Interleukin-17 - immunology | Lupus Erythematosus, Systemic - metabolism | Immunoblotting | Antigens, CD - genetics | Promoter Regions, Genetic - genetics | Antigens, CD - metabolism | CD28 Antigens - metabolism | Gene Knockdown Techniques | Receptor-CD3 Complex, Antigen, T-Cell - metabolism | NFATC Transcription Factors - immunology | CD28 Antigens - genetics | Chromatin Immunoprecipitation | Signaling Lymphocytic Activation Molecule Family Member 1 | T-Lymphocytes - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Lupus Erythematosus, Systemic - immunology | Antibodies - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | NFATC Transcription Factors - metabolism | Cells, Cultured | Interleukin-17 - genetics | Receptors, Cell Surface - metabolism | CD28 Antigens - immunology | Signaling Lymphocytic Activation Molecule Family | Receptors, Cell Surface - immunology | Antibodies - pharmacology | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Lupus Erythematosus, Systemic - genetics | Protein Binding | T-Lymphocytes - immunology | Mutation | Receptor-CD3 Complex, Antigen, T-Cell - immunology | NFATC Transcription Factors - genetics | Receptors, Cell Surface - genetics | Index Medicus | Gene Regulation | NFAT Transcription Factor | Signal Transduction | T Cell | SLAMF | Interleukin | RORgammat | T Cell Receptor
Journal Article
Blood, ISSN 0006-4971, 08/2014, Volume 124, Issue 7, pp. 1070 - 1080
Journal Article
Nature Immunology, ISSN 1529-2908, 01/2014, Volume 15, Issue 1, pp. 80 - 87
Journal Article
Immunity, ISSN 1074-7613, 2008, Volume 28, Issue 1, pp. 29 - 39
Journal Article