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Blood, ISSN 0006-4971, 12/2012, Volume 120, Issue 24, pp. 4751 - 4760
Natural killer (NK) cells are lymphocytes that play an important role in the immune response to infection and malignancy. Recent studies in mice have shown... 
INNATE | NATURAL-KILLER-CELLS | INFECTION | HEMATOLOGY | EXPANSION | Receptors, Interleukin-12 - immunology | Antigens, CD - immunology | Interleukin-18 - immunology | Humans | Lectins, C-Type - immunology | Interferon-gamma - metabolism | Interleukin-15 - immunology | Antigens, CD - metabolism | NK Cell Lectin-Like Receptor Subfamily D - immunology | Lectins, C-Type - metabolism | NK Cell Lectin-Like Receptor Subfamily C - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Time Factors | Killer Cells, Natural - immunology | Receptors, Interleukin-12 - genetics | Interferon-gamma - genetics | CD56 Antigen - immunology | Immunologic Memory - immunology | Cytokines - immunology | Interleukin-12 - pharmacology | Receptors, Interleukin-18 - metabolism | CD56 Antigen - metabolism | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Antigens, Differentiation, T-Lymphocyte - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Interleukin-15 - pharmacology | Receptors, Interleukin-12 - metabolism | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Interleukin-12 - immunology | K562 Cells | Receptors, Interleukin-18 - genetics | Receptors, Interleukin-18 - immunology | Cell Proliferation - drug effects | Interleukin-18 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Cytokines - pharmacology | Index Medicus | Abridged Index Medicus | Immunobiology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2014, Volume 111, Issue 2, pp. 775 - 780
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0160321 - e0160321
In recent years, cytokine-mediated therapy has emerged as further advance alternative in cancer therapy. Interleukin-18 (IL-18) has exhibited interesting... 
HUMAN IL-18 | CELLS | MULTIDISCIPLINARY SCIENCES | BIOLOGICAL-ACTIVITY | GROWTH | BINDING-PROTEIN | DYNAMICS | CANCER | Interleukin-18 - immunology | Molecular Weight | Humans | Substrate Specificity | Structure-Activity Relationship | Pichia - metabolism | Cloning, Molecular | Protein Engineering | Killer Cells, Natural - immunology | Protein Interaction Domains and Motifs | Interleukin-18 - chemistry | Binding Sites | Amino Acid Sequence | Protein Conformation, alpha-Helical | Gene Expression | Interferon-gamma - secretion | Models, Molecular | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Killer Cells, Natural - cytology | Sequence Alignment | Protein Conformation, beta-Strand | Recombinant Proteins - immunology | Lymphocyte Activation - drug effects | Pichia - genetics | Cell Line, Tumor | Protein Binding | Receptors, Interleukin-18 - genetics | Receptors, Interleukin-18 - immunology | Interleukin-18 - pharmacology | Killer Cells, Natural - drug effects | Kinetics | Receptors, Interleukin-18 - chemistry | Interleukin-18 - genetics | Interferon-gamma - biosynthesis | Care and treatment | Research | Mutagenesis | Interleukin-18 | Cancer | Therapy | Laboratories | Interleukin | Science | Molecular dynamics | Pichia pastoris | Synergism | Flexibility | Anticancer properties | Proteins | Immunotherapy | Polarity | Recombinant | Cytokines | Cloning | Interleukin 12 | Biological activity | Mutants | Interleukin 18 | Medicine | γ-Interferon | Interferon | Genetic engineering | Mutation | Protein structure | Binding sites | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2013, Volume 123, Issue 11, pp. 4695 - 4705
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 06/2014, Volume 192, Issue 12, pp. 5924 - 5932
Fine-tuning of cytokine-inducing pathways is essential for immune homeostasis. Consistently, a dysregulated increase or decrease in pattern-recognition... 
INFLAMMATORY-BOWEL-DISEASE | COLITIS | MURAMYL DIPEPTIDE | IL-18 | KINASE | SUSCEPTIBILITY LOCI | PROTEIN ABUNDANCE | MICE | NOD2 | IMMUNOLOGY | CELIAC-DISEASE | Interleukin-18 - immunology | Receptors, Interleukin-1 Type I - immunology | Humans | Inflammatory Bowel Diseases - immunology | NF-kappa B - immunology | Male | Extracellular Signal-Regulated MAP Kinases - genetics | MAP Kinase Signaling System - immunology | Extracellular Signal-Regulated MAP Kinases - immunology | Phosphatidylinositol 3-Kinases - immunology | Chromosomes, Human, Pair 2 - genetics | Interleukin-18 Receptor beta Subunit - genetics | MAP Kinase Signaling System - genetics | Autocrine Communication - immunology | Inflammatory Bowel Diseases - genetics | Interleukin-18 Receptor alpha Subunit - genetics | Calcium Signaling - immunology | Female | Polymorphism, Single Nucleotide - immunology | Macrophages - immunology | Interleukin-18 Receptor alpha Subunit - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Autocrine Communication - genetics | Phosphatidylinositol 3-Kinases - genetics | Interleukin-18 Receptor beta Subunit - immunology | Chromosomes, Human, Pair 2 - immunology | NF-kappa B - genetics | Receptors, Interleukin-1 Type I - genetics | Calcium Signaling - genetics | Interleukin-18 - genetics | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
Nature Genetics, ISSN 1061-4036, 09/2014, Volume 46, Issue 10, pp. 1140 - 1146
Journal Article
ANNALS OF THE RHEUMATIC DISEASES, ISSN 0003-4967, 06/2019, Volume 78, pp. 292 - 292
Conference Proceeding
International Immunology, ISSN 0953-8178, 6/2008, Volume 20, Issue 6, pp. 791 - 800
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 04/2015, Volume 459, Issue 3, pp. 379 - 386
Interleukin-18 (IL-18) was recently reported to have a pro-tumor effect in various cancers. Increased IL-18 levels in the serum of cancer patients correlated... 
Tight junction | Breast cancer | Claudin-12 | Migration | Interleukin-18 (IL-18) | METASTASIS | SERUM INTERLEUKIN-18 | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMMUNE ESCAPE | HUMAN GASTRIC-CANCER | BIOPHYSICS | MESSENGER-RNA | MURINE MELANOMA-CELLS | TIGHT JUNCTION PROTEINS | EXPRESSION | CARCINOMA | N-CADHERIN | Claudin-1 - antagonists & inhibitors | RNA, Small Interfering - genetics | Interleukin-18 - physiology | Claudin-4 - genetics | Humans | Interleukin-18 - antagonists & inhibitors | Claudin-3 - physiology | Breast Neoplasms - physiopathology | Cell Movement - genetics | Cell Movement - physiology | Claudin-4 - physiology | Gene Knockdown Techniques | Tight Junctions - physiology | MCF-7 Cells | Claudin-3 - genetics | Claudin-1 - physiology | Female | Flavonoids - pharmacology | Tight Junctions - drug effects | Claudins - genetics | Claudins - physiology | Imidazoles - pharmacology | Recombinant Proteins - pharmacology | Down-Regulation - drug effects | Claudin-4 - antagonists & inhibitors | Cell Movement - drug effects | Breast Neoplasms - genetics | MAP Kinase Signaling System - drug effects | Breast Neoplasms - pathology | Claudin-1 - genetics | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Claudins - antagonists & inhibitors | Claudin-3 - antagonists & inhibitors | Interleukin-18 - genetics | Proteins | Development and progression | Metastasis | Interleukins | Melanoma | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 10/2012, Volume 82, Issue 8, pp. 892 - 902
Interleukin (IL)-18 is produced by leukocytes and renal parenchymal cells (tubular epithelial cells, podocytes, and mesangial cells). The IL-18 receptor... 
cisplatin nephrotoxicity | acute kidney injury | cytokines | CELLS | ACTIVATION | ACUTE-RENAL-FAILURE | TRANS-RETINOIC ACID | RESPONSES | INTERFERON-GAMMA | COLITIS | IL-18 | UROLOGY & NEPHROLOGY | NF-KAPPA-B | Tumor Necrosis Factor-alpha - blood | Caspase 3 - metabolism | Suppressor of Cytokine Signaling 1 Protein | Interleukin-18 - urine | Male | Membrane Proteins - urine | Adoptive Transfer | RNA, Messenger - metabolism | Antibodies, Blocking - administration & dosage | Signal Transduction - immunology | Cisplatin - toxicity | Base Sequence | Interleukin-18 Receptor alpha Subunit - genetics | Acute Kidney Injury - chemically induced | Membrane Proteins - metabolism | Acute Kidney Injury - immunology | Cytokines - genetics | Neutrophils - pathology | Biomarkers - metabolism | Chemokines - biosynthesis | Interleukin-18 - blood | Macrophages - pathology | Acute Kidney Injury - pathology | Lymphocyte Activation | Mice, Inbred C57BL | RNA, Messenger - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Inflammation - immunology | Hepatitis A Virus Cellular Receptor 1 | Acute Kidney Injury - physiopathology | Chemokines - genetics | Interleukin-18 Receptor alpha Subunit - metabolism | Interleukin-18 Receptor alpha Subunit - antagonists & inhibitors | Mice, Knockout | Suppressor of Cytokine Signaling 3 Protein | Animals | T-Lymphocyte Subsets - pathology | Transcription Factor RelA - metabolism | Interleukin-18 Receptor alpha Subunit - deficiency | Inflammation - prevention & control | Mice | Suppressor of Cytokine Signaling Proteins - metabolism | Cytokines - biosynthesis | Apoptosis | Index Medicus | Adoptive transfer
Journal Article
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, ISSN 0091-6749, 07/2017, Volume 140, Issue 1, pp. 316 - 317
Journal Article