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Journal Article
Gut, ISSN 0017-5749, 06/2010, Volume 59, Issue 6, pp. 775 - 784
BackgroundThe molecular mechanisms underlying the pathophysiology of irritable bowel syndrome (IBS) are poorly understood. One mechanism may involve increased... 
CELLS | LACTULOSE | GLUTAMINE | INFLAMMATION | CYTOSKELETON-FREE VESICLES | GUT | DISORDERS | MICROPARTICLES | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | HYPERSENSITIVITY | Cytoplasmic Vesicles - enzymology | Humans | Irritable Bowel Syndrome - enzymology | Male | Tissue Array Analysis - methods | Young Adult | Base Sequence | Adult | Female | Glutamate-Ammonia Ligase - metabolism | Intestinal Absorption - physiology | Cells, Cultured | Signal Transduction - genetics | Irritable Bowel Syndrome - physiopathology | Permeability | Colon - metabolism | Irritable Bowel Syndrome - genetics | Intestinal Absorption - genetics | Gene Expression Regulation, Enzymologic - genetics | Glutamate-Ammonia Ligase - genetics | Irritable Bowel Syndrome - pathology | Signal Transduction - physiology | Colon - ultrastructure | MicroRNAs - physiology | Enzyme Activation - genetics | MicroRNA | Irritable bowel syndrome | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Cells | Urine | Excretory system | Inflammation | Gene expression | Abdomen | Studies | Pain | Biopsy | MicroRNAs | DNA methylation | Colon | Index Medicus | Abridged Index Medicus | intestinal membrane permeability | glutamine synthetase | miRNA | glutamine synthetase gene (GLUL) | Irritable bowel syndrome (IBS) | miR-29a | blood microvesicles
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 02/2005, Volume 48, Issue 3, pp. 805 - 811
Journal Article
Journal Article
Digestive and Liver Disease, ISSN 1590-8658, 11/2017, Volume 49, Issue 11, pp. 1201 - 1210
Necroptosis is an inflammatory form of programmed cell death requiring receptor-interacting protein kinase 3 (RIP3) and mixed lineage kinase domain-like... 
Intestinal inflammation | Adhesion molecules | Inflammasome | Necroptosis | CROHNS-DISEASE | DOMAIN-LIKE PROTEIN | ULCERATIVE-COLITIS | MIXED LINEAGE KINASE | DEATH | NLRP3 INFLAMMASOME | RECEPTOR-INTERACTING PROTEIN | MEDIATES NECROPTOSIS | TNF-ALPHA | ACTIVITY INDEX | GASTROENTEROLOGY & HEPATOLOGY | Protein Kinases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Intestinal Mucosa - metabolism | Phosphorylation | Protein Kinases - genetics | Inflammasomes - metabolism | Cadherins - metabolism | Humans | Cell Membrane Permeability | Child, Preschool | Crohn Disease - metabolism | Caspase 1 - metabolism | Protein Transport - drug effects | Necrosis | Epithelial Cells - physiology | Inflammation - metabolism | Amino Acid Chloromethyl Ketones - pharmacology | Interleukin-1beta - metabolism | HMGB1 Protein - metabolism | Indoles - pharmacology | Interleukin-8 - metabolism | Child | Cell Survival - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Colitis, Ulcerative - metabolism | HCT116 Cells | Imidazoles - pharmacology | Cell Adhesion | Tumor Necrosis Factor-alpha - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Transcription Factor RelA - metabolism | Adolescent | RNA, Messenger | Apoptosis | Permeability | Inflammation | Chromosomal proteins | Cell death | Protein kinases | Ulcerative colitis | Index Medicus
Journal Article
Oncotarget, ISSN 1949-2553, 10/2017, Volume 8, Issue 49, pp. 85828 - 85837
Journal Article
European Journal of Pharmaceutics and Biopharmaceutics, ISSN 0939-6411, 10/2015, Volume 96, pp. 329 - 337
Journal Article
Journal of Pharmaceutical Sciences, ISSN 0022-3549, 01/1999, Volume 88, Issue 1, pp. 28 - 33
Journal Article