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AUTOPHAGY, ISSN 1554-8627, 05/2012, Volume 8, Issue 5, pp. 826 - 837
Autophagy is responsible for the degradation of protein aggregates and damaged organelles. Several studies have reported increased autophagic activity in... 
SYSTEM | APOPTOSIS | autophagy | GELATINASE-ASSOCIATED LIPOCALIN | p62 | CELL BIOLOGY | EARLY URINARY BIOMARKER | CISPLATIN INJURY | DISEASE | acute kidney injury | BETA-CELL MASS | Atg5 | kidney tubule system | TRANSGENIC MICE
Journal Article
Kidney International, ISSN 0085-2538, 05/2008, Volume 73, Issue 10, pp. 1187 - 1192
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, pp. 42296 - 42296
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e87667 - e87667
Various bioactive peptides have been implicated in the homeostasis of organs and tissues. Adrenomedullin (AM) is a peptide with various bioactivities.... 
UNFOLDED PROTEIN RESPONSE | HYPOTENSIVE PEPTIDE | GENE | INDUCED APOPTOSIS | PLASMA-LEVELS | MULTIDISCIPLINARY SCIENCES | ENDOGENOUS ADRENOMEDULLIN | VASCULAR INTEGRITY | ENDOPLASMIC-RETICULUM STRESS | RENAL INJURY | CISPLATIN NEPHROTOXICITY | Antibiotics, Antineoplastic - pharmacology | eIF-2 Kinase - metabolism | Humans | Antibiotics, Antineoplastic - adverse effects | Kidney Glomerulus - injuries | Kidney Diseases - genetics | Cell Death - genetics | Kidney Diseases - chemically induced | Kidney Glomerulus - metabolism | Cell Death - drug effects | Streptozocin - adverse effects | Kidney Diseases - metabolism | eIF-2 Kinase - genetics | Cell Line | Kidney Tubules, Proximal - pathology | Kidney Diseases - pathology | Kidney Tubules, Proximal - injuries | Receptor Activity-Modifying Protein 2 - metabolism | Adrenomedullin - metabolism | Kidney Glomerulus - pathology | Streptozocin - pharmacology | Adrenomedullin - genetics | Mice, Knockout | Animals | Endoplasmic Reticulum Stress | Kidney Tubules, Proximal - metabolism | Receptor Activity-Modifying Protein 2 - genetics | Mice | Proteins | Blood sugar | Cell death | Genetically modified organisms | Biochemistry | Kidney diseases | Protein binding | Peptides | Syngeneic grafts | Epithelial cells | Streptozocin | Tunicamycin | Homeostasis | Adrenomedullin | Homology | Lethality | Glucose | Receptor activity modifying proteins | Ischemia | Rodents | Animal tissues | Calcitonin | Damage | Genetic modification | Injuries | University graduates | Chronic illnesses | Glucose transporter | Stresses | Stroke | Kidneys | Mortality | Organs | Tubules | Ribonucleic acid--RNA | Insulin | Embryos | Biological activity | Stress | Medicine | Ramps | Signaling | Protein kinase | Diabetes | Endoplasmic reticulum | Transporter | Kidney transplantation | Apoptosis | Index Medicus | RNA | Ribonucleic acid
Journal Article
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 03/2012, Volume 302, Issue 5, pp. R494 - R509
ANG II is the most potent and important member of the classical renin-angiotensin system (RAS). ANG II, once considered to be an endocrine hormone, is now... 
Blood pressure | Kidneys | ACE inhibitors | Rodents | Cells | Special Topic | Intracrine Renin-Angiotensin System | signaling | ANG I-converting enzyme | renin | intracrine | hypertension | A New Paradigm in Cardiovascular and Renal Control | AT1 receptor
Journal Article
CPT: Pharmacometrics & Systems Pharmacology, ISSN 2163-8306, 05/2019, Volume 8, Issue 5, pp. 316 - 325
Drug‐induced kidney injury, a major cause of acute kidney injury, results in progressive kidney disease and is linked to increased mortality in hospitalized... 
Urine | Production increases | Studies | Parameter estimation | Kidneys | Simulation | Morphology | Biomarkers | Pharmacology | Inflammation | Patients | Drug dosages | Research
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 14882 - 14
The adoption of a new technology into basic research, and industrial and clinical settings requires rigorous testing to build confidence in the... 
BIOMARKERS | ON-A-CHIP | VITAMIN-D | CELLS | METABOLITES | MULTIDISCIPLINARY SCIENCES | DISEASE | KIDNEY INJURY MOLECULE-1 | MECHANISMS | DEHYDROGENASE | NEPHROTOXICITY | Cell culture | Vitamin D | Laboratories | Epithelial cells | Toxicity | Gene expression | Clear cell-type renal cell carcinoma
Journal Article
Journal Article