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Nature Immunology, ISSN 1529-2908, 12/2013, Volume 14, Issue 12, pp. 1294 - 1301
Tissue-resident memory T cells (TRM cells) provide superior protection against infection in extralymphoid tissues. Here we found that CD103(+)CD8(+) TRM cells... 
MIGRATION | EFFECTOR | INTESTINAL EPITHELIUM | NONLYMPHOID TISSUE | RM CELLS | PERIPHERAL-TISSUES | DIFFERENTIATION | IMMUNOLOGY | LYMPHOCYTE EGRESS | CD103 EXPRESSION | CUTTING EDGE | Receptors, Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - immunology | Oligonucleotide Array Sequence Analysis | Skin - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Host-Pathogen Interactions - immunology | Lectins, C-Type - metabolism | Herpesvirus 1, Human - physiology | Integrin alpha Chains - immunology | CD8-Positive T-Lymphocytes - metabolism | Protein-Serine-Threonine Kinases - metabolism | Antigens, Differentiation, T-Lymphocyte - metabolism | Mice, Transgenic | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Herpes Simplex - immunology | Mice, Knockout | Cell Differentiation - immunology | Herpes Simplex - virology | Interleukin-15 - metabolism | Mice | CD8-Positive T-Lymphocytes - immunology | Receptors, Immunologic - genetics | Antigens, CD - immunology | Lectins, C-Type - immunology | Transcriptome - immunology | Lectins, C-Type - genetics | Interleukin-15 - immunology | Cell Differentiation - genetics | Signal Transduction - immunology | Flow Cytometry | Herpesvirus 1, Human - immunology | Receptors, Immunologic - immunology | Skin - virology | Immunologic Memory - immunology | Skin - immunology | Antigens, Differentiation, T-Lymphocyte - immunology | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Integrin alpha Chains - metabolism | Transcriptome - genetics | Mice, Inbred Strains | Animals | Interleukin-15 - genetics | Receptors, Transforming Growth Factor beta - metabolism | CD8-Positive T-Lymphocytes - virology | Antigens, Differentiation, T-Lymphocyte - genetics | Protein-Serine-Threonine Kinases - immunology | Integrin alpha Chains - genetics | Receptors, Immunologic - metabolism
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2012, Volume 189, Issue 8, pp. 3957 - 3969
The lectin pathway of complement is an important component of innate immunity. Its activation has been thought to occur via recognition of pathogens by... 
MALPUECH | PATTERN-RECOGNITION MOLECULES | MECHANISM | COMPLEMENT FACTOR-D | MBL | STOICHIOMETRY | MUTATIONS | IMMUNOLOGY | IDENTIFICATION | CARNEVALE | DEFICIENCY | Craniofacial Abnormalities - immunology | Transcriptional Activation - genetics | Complement Pathway, Mannose-Binding Lectin - immunology | Blepharoptosis - genetics | Eye Abnormalities - enzymology | Mannose-Binding Protein-Associated Serine Proteases - physiology | Eye Abnormalities - immunology | Humans | Heart Defects, Congenital - immunology | Craniosynostoses - enzymology | Blepharoptosis - immunology | Developmental Disabilities - genetics | Transcriptional Activation - immunology | Strabismus - genetics | Abdominal Muscles - abnormalities | Abnormalities, Multiple - immunology | Heart Defects, Congenital - genetics | Developmental Disabilities - enzymology | Blepharoptosis - enzymology | Heart Defects, Congenital - enzymology | Mannose-Binding Protein-Associated Serine Proteases - genetics | Complement Pathway, Alternative - genetics | Abnormalities, Multiple - genetics | Craniofacial Abnormalities - genetics | Strabismus - enzymology | Abdominal Muscles - immunology | Craniosynostoses - genetics | Complement Pathway, Mannose-Binding Lectin - genetics | Cryptorchidism - enzymology | Abdominal Muscles - enzymology | Hip Dislocation, Congenital - genetics | Codon, Nonsense | Eye Abnormalities - genetics | Animals | Craniofacial Abnormalities - enzymology | Craniosynostoses - immunology | Cryptorchidism - immunology | Hip Dislocation, Congenital - immunology | Complement Pathway, Alternative - immunology | Strabismus - immunology | Abnormalities, Multiple - enzymology | Developmental Disabilities - immunology | Hip Dislocation, Congenital - enzymology | Cryptorchidism - genetics
Journal Article
Immunity, ISSN 1074-7613, 2007, Volume 26, Issue 5, pp. 593 - 604
The ability of natural-killer cells to regulate adaptive immunity is not well understood. Here we define an interaction between the class Ib major... 
HUMDISEASE | CELLIMMUNO | NATURAL-KILLER-CELLS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RECEPTOR CD94/NKG2A | MULTIPLE-SCLEROSIS | DENDRITIC CELLS | IFN-GAMMA | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | IN-VIVO | IMMUNOLOGY | CLASS IB MOLECULES | SELF-TOLERANCE | Bone Marrow - immunology | Cell Proliferation | Encephalomyelitis, Autoimmune, Experimental - metabolism | Encephalomyelitis, Autoimmune, Experimental - immunology | DNA-Binding Proteins - deficiency | CD4-Positive T-Lymphocytes - immunology | DNA-Binding Proteins - metabolism | Histocompatibility Antigens Class I - metabolism | Signal Transduction - immunology | Lymphocyte Activation - immunology | Bone Marrow - metabolism | Receptors, Natural Killer Cell | NK Cell Lectin-Like Receptor Subfamily D - genetics | Killer Cells, Natural - immunology | Lentivirus - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Chimerin Proteins - immunology | Immunologic Memory - immunology | Genetic Predisposition to Disease | Encephalomyelitis, Autoimmune, Experimental - pathology | Gene Expression Regulation - immunology | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Histocompatibility Antigens Class I - genetics | DNA-Binding Proteins - genetics | Mice, Knockout | Killer Cells, Natural - cytology | Phenotype | Animals | Chimerin Proteins - metabolism | Mice | NK Cell Lectin-Like Receptor Subfamily C | Killer Cells, Natural - metabolism | Receptors, Immunologic - genetics | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Interferon-gamma - biosynthesis | Receptors, Immunologic - metabolism | Chimerin Proteins - genetics | Hypotheses | Peptides | Lymphocytes | Rodents | Ligands | T cell receptors | Immune system
Journal Article
by Li, Y and Liu, MF and Zuo, ZY and Liu, J and Yu, X and Guan, Y and Zhan, RH and Han, QJ and Zhang, J and Zhou, RB and Sun, R and Tian, ZG and Zhang, C
JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 07/2017, Volume 199, Issue 2, pp. 761 - 773
TLRs are key sensors for conserved bacterial molecules and play a critical role in host defense against invading pathogens. Although the roles of TLRs in... 
MOLECULAR-PATTERNS | RESPONSES | ACTIVATION | IMMUNE HOMEOSTASIS | EPITHELIAL-CELLS | RECOGNITION | SEROVAR TYPHIMURIUM INFECTION | TOLL-LIKE RECEPTORS | IMMUNOLOGY | EXPRESSION | INTRAEPITHELIAL LYMPHOCYTES | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Toll-Like Receptor 9 - genetics | Gastroenteritis - microbiology | NK Cell Lectin-Like Receptor Subfamily K - immunology | NF-kappa B - metabolism | Intestines - immunology | Salmonella Infections, Animal - immunology | Toll-Like Receptor 9 - immunology | Toll-Like Receptor 2 - deficiency | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Interleukin-1beta - metabolism | Toll-Like Receptor 3 - deficiency | Gastroenteritis - immunology | Interleukin-1beta - secretion | Intestines - pathology | Salmonella typhimurium - immunology | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Toll-Like Receptor 9 - deficiency | Gene Expression Regulation | Interleukin-1beta - immunology | Toll-Like Receptor 3 - immunology | Inflammation | NK Cell Lectin-Like Receptor Subfamily K - genetics | Animals | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Epithelial Cells - immunology | Salmonella Infections, Animal - microbiology | Toll-Like Receptor 2 - immunology | Mice | Toll-Like Receptor 9 - metabolism | Intestines - cytology | Pathogens | Salmonella | NF-κB protein | Toxicity | Digestive system | Epithelial cells | Homeostasis | Cytotoxicity | Infections | TLR4 protein | IL-1β | TLR9 protein | Proteins | Signaling | Lymphocytes | Intestine | Rodents | TLR2 protein | Toll-like receptors | Bacteria | Digestive tract | Immune system
Journal Article