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2015, ISBN 1408187337, viii, 296
"In a previous book published in 1985 as 'Thinkers of the New Left,' [the author] brought together a series of articles from The Salisbury review. [He has]... 
Socialists | Communism | Communists | Socialism | New Left
Book
2011, Cambridge studies in contentious politics, ISBN 0521145449, cm.
This book is an eye-opening account of transnational advocacy, not by environmental and rights groups, but by conservative activists. Mobilizing around diverse... 
Politics & government | Conservatism | Political activity | 21st century | Social conflict | Right and left (Political science) | Culture and globalization | History | Political activists | Political aspects | Non-governmental organizations | World politics | Non-govenrmental organizations
Book
2004, 1, ISBN 9780520241190, xiii, 394
In this comparison of left-wing violence in the United States and West Germany, Jeremy Varon focuses on America's Weather Underground and Germany's Red Army... 
Radicalism | History | Weather Underground Organization | 20th century | Germany (West) | Weatherman (Organization) | United States | Political violence | Baader-Meinhof gang | New Left
Book
2012, Cambridge studies in contentious politics., ISBN 9780521193818, xiv, 225
"Human rights, environmentalism, and global justice: these transnational movements today face fierce opposition from networks of conservative activists... 
Social conflict | Political activity | Right and left (Political science) | Culture and globalization | History | Political activists | Political aspects | Non-governmental organizations | Politics & government | Conservatism | 21st century | World politics | Non-govenrmental organizations
Book
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2016, Volume 36, Issue 4, pp. 636 - 646
OBJECTIVE—Basigin (Bsg) is a transmembrane glycoprotein that activates matrix metalloproteinases and promotes inflammation. However, the role of Bsg in the... 
heart failure | cytokines | hypertrophy | inflammation | angiotensin II | INHIBITS EMMPRIN | CHRONIC HEART-FAILURE | RECEPTOR | CANCER | METALLOPROTEINASE INDUCER EMMPRIN | PMA-INDUCED MACROPHAGES | ACTIVATES PLATELETS | PERIPHERAL VASCULAR DISEASE | CD147 | HEMATOLOGY | EXPRESSION | Oxidative Stress | Heart Failure - physiopathology | Male | Aortic Diseases - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Basigin - genetics | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Failure - etiology | Disease Models, Animal | Fibroblasts - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Hypertrophy, Left Ventricular - prevention & control | Rats | Heart Failure - genetics | Heart Failure - metabolism | Fibroblasts - pathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Aortic Diseases - genetics | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Fibrosis | Myocytes, Cardiac - metabolism | Hypertrophy, Left Ventricular - physiopathology | Rats, Wistar | Ventricular Function, Left | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Blood Proteins - metabolism | Hypertrophy, Left Ventricular - pathology | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Cells, Cultured | Ventricular Dysfunction, Left - etiology | Myocardium - pathology | Heart Failure - pathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Mechanotransduction, Cellular | Aortic Diseases - physiopathology | Animals | Aortic Diseases - complications | Basigin - metabolism | Matrix Metalloproteinases - metabolism
Journal Article
1990, Garland reference library of the social sciences, ISBN 0824037138, Volume 502, xxxix, 928
Book
2014, Routledge studies in extremism and democracy, ISBN 0415661501, xvii, 318
"Since 2009, the UK Independence Party (UKIP) has become the most successful challenger to the main parties in postwar English politics. Yet with the radical... 
Great Britain Politics and government | UK Independence Party | Right and left (Political science)
Book
Cardiovascular Research, ISSN 0008-6363, 12/2012, Volume 96, Issue 3, pp. 456 - 465
Active autophagy has recently been reported in doxorubicin-induced cardiotoxicity; here we investigated its pathophysiological role. Acute cardiotoxicity was... 
Heart failure | Autophagy | Doxorubicin | MAMMALIAN TARGET | CARDIAC & CARDIOVASCULAR SYSTEMS | ACTIVATED PROTEIN-KINASE | INDUCED APOPTOSIS | PHOSPHORYLATION | MECHANISMS | CELL-DEATH | DEGENERATION | HEART | INHIBITION | 3-METHYLADENINE | AMP-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - genetics | Ventricular Function, Left | Microtubule-Associated Proteins - metabolism | Heart Failure - physiopathology | Green Fluorescent Proteins - genetics | Ventricular Dysfunction, Left - chemically induced | Autophagy - drug effects | Autophagy-Related Protein-1 Homolog | Starvation - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - chemically induced | Time Factors | Hypertrophy, Left Ventricular - pathology | Adenosine Triphosphate - metabolism | Ventricular Dysfunction, Left - pathology | Protein-Serine-Threonine Kinases - metabolism | Green Fluorescent Proteins - metabolism | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Cells, Cultured | Rats | Mice, Transgenic | Heart Failure - metabolism | Antibiotics, Antineoplastic | Heart Failure - pathology | Cathepsin D - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Energy Metabolism | Myocytes, Cardiac - metabolism | Mice | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart Failure - chemically induced | Starvation - complications | Index Medicus
Journal Article
2008, ISBN 9780199286935, Volume 9780199286942, xii, 318
Neoliberalism. Neoconservatism. Postmarxism. Postmodernism. Is there really something genuinely new about today's isms? Have we moved past our traditional... 
Right and left (Political science) | Political science | History | Ideology | political theory | Nation | Political belief systems | Global imaginary | Ideology and religion | Globality | Community
Book
Journal of the American Heart Association, ISSN 2047-9980, 01/2016, Volume 5, Issue 1, p. n/a
Background Mitochondrial reactive oxygen species (ROS) are associated with metabolic heart disease (MHD). However, the mechanism by which ROS cause MHD is... 
metabolic heart disease | mitochondria | obesity | oxidative protein modifications | oxidative stress | Obesity | Oxidative stress | Metabolic heart disease | Mitochondria | Oxidative protein modifications | EXTREME OBESITY | CARDIAC & CARDIOVASCULAR SYSTEMS | UNCOUPLING PROTEINS | MECHANISMS | DIABETIC CARDIOMYOPATHY | SUCCINATE-DEHYDROGENASE | COMPLEX-II | PROTEIN S-GLUTATHIONYLATION | MICE | DYSFUNCTION | Mitochondrial Diseases - pathology | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Ventricular Function, Left | Mitochondria, Heart - pathology | Mitochondrial Diseases - metabolism | Electron Transport Complex I - metabolism | Mitochondrial Diseases - etiology | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Hypertrophy, Left Ventricular - pathology | Adenosine Triphosphate - metabolism | Diet, High-Fat | Ventricular Dysfunction, Left - pathology | Dietary Sucrose | Disease Models, Animal | Mitochondrial Diseases - genetics | Mitochondrial Diseases - prevention & control | Electron Transport Complex II - genetics | Oxidation-Reduction | Catalase - genetics | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Mice, Inbred C57BL | Ventricular Dysfunction, Left - etiology | Mice, Transgenic | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Catalase - metabolism | Ventricular Dysfunction, Left - metabolism | Animals | Energy Metabolism | Electron Transport Complex II - metabolism | Protein Processing, Post-Translational | Hypertrophy, Left Ventricular - physiopathology | Mutation | Mitochondrial Diseases - physiopathology
Journal Article
1992, ISBN 9780393030754, 432
Book