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Journal Article
The EMBO Journal, ISSN 0261-4189, 04/2015, Volume 34, Issue 8, pp. 1110 - 1125
Journal Article
The Journal of Physiology, ISSN 0022-3751, 06/2017, Volume 595, Issue 12, pp. 3765 - 3780
Pre‐ischaemic administration of aminooxiacetate (AOA), an inhibitor of the malate‐aspartate shuttle (MAS), provides cardioprotection against... 
malate‐aspartate shuttle | ischemia‐reperfusion | mitochondria | heart | oxidative stress | malate-aspartate shuttle | ischemia-reperfusion | GLYCOLYSIS | OXIDATIVE STRESS | PHYSIOLOGY | COMPLEX-I | PHOSPHORYLATION | INJURY | RAT-HEART | CARDIOPROTECTION | BLOCKADE | NEUROSCIENCES | ELECTRON-TRANSPORT | FREE-RADICALS | Myocardial Ischemia - metabolism | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Rats, Wistar | Citric Acid Cycle - drug effects | Rats | Male | Cardiotonic Agents - pharmacology | Mitochondria, Heart - drug effects | Myocardial Infarction - metabolism | Electron Transport Complex I - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Protective Agents - pharmacology | Myocardium - metabolism | Cell Respiration - drug effects | Malates - metabolism | Oxidation-Reduction - drug effects | Heart - drug effects | Aspartic Acid - metabolism | Aminooxyacetic Acid - pharmacology | Ischemia | Aspartate | Fatty acids | Heart | Tricarboxylic acid cycle | Oxidative stress | Reactive oxygen species | Emission | Damage prevention | Electron transport chain | Mitochondria | Control | Reperfusion | Rodents | Attenuation | Oxidation | Heart diseases | Injuries | Stress concentration | NADH-ubiquinone oxidoreductase | Oxygen | Preservation | Substrate inhibition | Metabolism | Substrates | Intermediates | Injury prevention | Inhibitors | Malate | Electron transport | Respiration | Preconditioning | Index Medicus | Mitochondrial Function and Myocardial Metabolism | Research Paper
Journal Article
by Wang, TX and Yao, WL and Li, J and He, QY and Shao, YF and Huang, FR
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM, ISSN 0193-1849, 10/2018, Volume 315, Issue 4, pp. E496 - E510
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2013, Volume 124, Issue 3, pp. 347 - 362
The mitochondrial transporter of aspartate‐glutamate Aralar/ AGC 1 is a regulatory component of the malate‐aspartate shuttle. Aralar deficiency in mouse and... 
B | VMAT | 2 | global cerebral hypomyelination | malate‐aspartate shuttle | mni | 1 deficiency | AGC | dopamine | ® | O | ank | malate-aspartate shuttle | AGC-1 deficiency | OmniBank | VMAT2 | NICOTINAMIDE NUCLEOTIDE TRANSHYDROGENASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RELEASE | NEUROSCIENCES | MICE LACKING | OmniBank (R) | METABOLISM | VESICULAR MONOAMINE TRANSPORTER-2 | MITOCHONDRIAL COMPLEX-I | MOUSE MODEL | NEURONS | RAT-BRAIN | EXPRESSION | Oxidative Stress - physiology | Amino Acid Transport Systems, Acidic - metabolism | Mitochondrial Diseases - metabolism | Dopamine - genetics | Amino Acid Transport Systems, Acidic - deficiency | Male | Hereditary Central Nervous System Demyelinating Diseases - physiopathology | Motor Skills Disorders - genetics | Mice, 129 Strain | Substantia Nigra - metabolism | Corpus Striatum - cytology | Corpus Striatum - metabolism | Substantia Nigra - cytology | Mitochondrial Membrane Transport Proteins - genetics | Dopamine - deficiency | Exploratory Behavior - physiology | Psychomotor Disorders - metabolism | Psychomotor Disorders - genetics | Mitochondrial Membrane Transport Proteins - deficiency | Antiporters - genetics | Malates - metabolism | Female | Neural Pathways - physiopathology | Psychomotor Disorders - physiopathology | Dopamine - metabolism | Hereditary Central Nervous System Demyelinating Diseases - genetics | Antiporters - deficiency | Emotions - physiology | Mitochondrial Diseases - genetics | Mitochondrial Membrane Transport Proteins - metabolism | Mice, Inbred C57BL | Antiporters - metabolism | Hereditary Central Nervous System Demyelinating Diseases - metabolism | Mice, Knockout | Pregnancy | Motor Skills Disorders - metabolism | Animals | Neural Pathways - cytology | Amino Acid Transport Systems, Acidic - genetics | Neural Pathways - metabolism | Aspartic Acid - metabolism | Mice | Aspartic Acid - physiology | Mitochondrial Diseases - physiopathology | Tyrosine | Phenols | Aspartate | Neurochemistry | Mitochondria | Dopamine | Brain | Reactive oxygen species | Hyperreactivity | Brain stem | Hyperactivity | Neurons | Substantia nigra | Cortex | Amino acids | Metabolism | Cytosol | Vesicular amine transporter | Amine oxidase (flavin-containing) | NADH | Neostriatum | Anxiety | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 11/2010, Volume 88, Issue 2, pp. 257 - 266
Preserved mitochondrial function is essential for protection against ischaemia-reperfusion (IR) injury. The malate-aspartate (MA) shuttle constitutes the... 
Ischaemia | Carbohydrate metabolism | Infarct size | Reperfusion | Malate-aspartate shuttle | CARDIAC & CARDIOVASCULAR SYSTEMS | REPERFUSION INJURY | MYOCARDIAL-ISCHEMIA | GUINEA-PIG HEART | RAT HEARTS | GLYCOLYSIS | MITOCHONDRIAL PERMEABILITY TRANSITION | CITRIC-ACID CYCLE | DOG HEART | GLUTAMATE | ANAEROBIC METABOLISM | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Rats, Wistar | Male | Mitochondria, Heart - drug effects | Myocardial Reperfusion Injury - pathology | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Cell Respiration - drug effects | Malates - metabolism | Rabbits | Oxidation-Reduction | Ventricular Function, Left - drug effects | Ventricular Pressure - drug effects | Rats | Myocardium - pathology | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Microdialysis | Ischemic Preconditioning, Myocardial | Oxygen Consumption - drug effects | Perfusion | Glucose - metabolism | Aspartic Acid - metabolism | Aminooxyacetic Acid - pharmacology | Hemodynamics - drug effects | Myocardial Infarction - prevention & control | In Vitro Techniques | Energy Metabolism - drug effects | Myocardial Reperfusion Injury - prevention & control | Index Medicus
Journal Article
Frontiers in Neuroscience, ISSN 1662-4548, 2014, Volume 8, pp. 366 - 366
Lactate, the conjugate base of lactic acid occurring in aqueous biological fluids, has been derided as a "dead-end" waste product of anaerobic metabolism.... 
Pyruvates | Lactate dehydrogenases | Mitochondria | Lactate | Malate aspartate | PYRUVATE | RAT-BRAIN ASTROCYTES | REDUCING EQUIVALENTS | mitochondria | malate aspartate | lactate dehydrogenases | NEUROSCIENCES | EXERCISE | DEHYDROGENASE | SKELETAL-MUSCLE MITOCHONDRIA | HEART | pyruvates | LIVER-MITOCHONDRIA | BLOOD LACTATE | lactate | ENERGY-METABOLISM | Lactate Dehydrogenases
Journal Article
Journal Article