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Toxicology and Applied Pharmacology, ISSN 0041-008X, 11/2013, Volume 272, Issue 3, pp. 697 - 702
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9313 - e9313
Background: Lewy body disease is a heterogeneous group of neurodegenerative disorders characterized by alpha-synuclein accumulation that includes dementia with... 
WILD-TYPE | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | MOUSE MODEL | FRONTOTEMPORAL DEMENTIA | BIOLOGY | AGGREGATE-PRONE PROTEINS | NEURONAL EXPRESSION | PARKINSONS-DISEASE | TRANSGENIC MICE | GAUCHER-DISEASE | Pyramidal Cells - metabolism | Humans | Nerve Degeneration - genetics | Male | Autophagy | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Lewy Body Disease - genetics | RNA Interference | Brain - ultrastructure | Aged, 80 and over | Female | alpha-Synuclein - genetics | Pyramidal Cells - ultrastructure | Disease Models, Animal | Cell Line | Ubiquitin-Activating Enzymes - genetics | Signal Transduction | Lewy Body Disease - pathology | Mice, Transgenic | Microscopy, Electron | Ubiquitin-Activating Enzymes - metabolism | Blotting, Western | Nerve Degeneration - pathology | Lewy Body Disease - metabolism | Microscopy, Confocal | Autophagy-Related Protein 7 | Animals | Alzheimer Disease - metabolism | Brain - pathology | Cell Line, Tumor | Pyramidal Cells - pathology | Aged | Mice | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | TOR protein | Neurosciences | Animal models | Parkinson's disease | Pathogenesis | Toxicity | Parkinsons disease | Phagosomes | Nervous system | Synuclein | Neuromodulation | Accumulation | Modulators | Proteins | Infusion | Alterations | Neurodegeneration | Dementia disorders | Alzheimer's disease | Movement disorders | Enzymes | Neurodegenerative diseases | Neurons | Immunomodulation | Lewy body disease | Transgenic mice | Rapamycin | Patients | Lewy bodies | Studies | Pathology | Mutation | Alzheimers disease | Phagocytosis | Apoptosis | Dementia | Index Medicus
Journal Article
NEW ENGLAND JOURNAL OF MEDICINE, ISSN 0028-4793, 04/2004, Volume 350, Issue 16, pp. 1655 - 1664
Bone metastases are a frequent complication of cancer, occurring in up to 70 percent of patients with advanced breast or prostate cancer. The consequences of... 
MULTIPLE-MYELOMA | BREAST-CANCER | MEDICINE, GENERAL & INTERNAL | HORMONE-RELATED PROTEIN | OSTEOCLASTOGENESIS-INHIBITORY FACTOR | PROSTATE-CANCER | PARATHYROID-HORMONE | DIFFERENTIATION FACTOR | TUMOR-CELLS | MACROPHAGE INFLAMMATORY PROTEIN-1-ALPHA | MOLECULAR-MECHANISMS | Bone diseases | Diagnosis | Complications | Risk factors | Cancer
Journal Article
New England Journal of Medicine, ISSN 0028-4793, 04/2004, Volume 350, Issue 15, pp. 1535 - 1548
Journal Article
2015, ISBN 9781466580039, xx, 320
Antioxidants in Health and Disease discusses the effects of dietary antioxidants and antioxidant supplementation in humans. It reviews the latest... 
Antioxidants | Medicine, Preventive | Physiological effect | Health aspects | Therapeutic use | Medicine: General Issues | Nutrition | Nutraceuticals & Functional Foods | Free radicals | Adverse effects | Oxidants
Book
by Wang, CY and He, YH and Yang, M and Sun, HL and Zhang, SP and Wang, CH
TOXICOLOGY AND APPLIED PHARMACOLOGY, ISSN 0041-008X, 11/2013, Volume 273, Issue 1, pp. 59 - 67
Intracellular reactive oxygen species (ROS) are derived from nicotinamide adenine dinucleotide phosphate (NADPH) oxidase. Angiotensin II (Ang II) can cause... 
Bcl-2 | SYSTEM | OXIDATIVE STRESS | SUPEROXIDE | Safflor yellow B | MECHANISMS | NADPH OXIDASE ACTIVATION | p22(phox) | NAD(P)H OXIDASE | REACTIVE OXYGEN | NADPH oxidase | INFLAMMATION | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | TOXICOLOGY | VASCULAR ENDOTHELIAL-CELLS | Angiotensin II | Mitochondrial membrane potential | Up-Regulation | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | NADPH Oxidases - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Receptor, Angiotensin, Type 1 - genetics | Chalcone - pharmacology | Chalcone - analogs & derivatives | Umbilical Veins - drug effects | NADPH Oxidases - genetics | Vascular Cell Adhesion Molecule-1 - genetics | Angiotensin II - adverse effects | Umbilical Veins - cytology | bcl-2-Associated X Protein - genetics | Cell Survival - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Intercellular Adhesion Molecule-1 - metabolism | Intercellular Adhesion Molecule-1 - genetics | Receptor, Angiotensin, Type 1 - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Umbilical Veins - metabolism | Index Medicus | OXYGEN | APOPTOSIS | ENZYME ACTIVITY | INJURIES | VEINS | ANTIOXIDANTS | OXIDASES | MITOCHONDRIA | MEMBRANES | NADP | 60 APPLIED LIFE SCIENCES | ANGIOTENSIN
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 06/2017, Volume 118, Issue 6, pp. 1471 - 1479
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2012, Volume 129, Issue 3, pp. 717 - 725.e5
Journal Article