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humans (252) 252
membranoproliferative glomerulonephritis (222) 222
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urology & nephrology (144) 144
dense deposit disease (124) 124
hemolytic-uremic syndrome (106) 106
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complement (98) 98
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dense-deposit disease (36) 36
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disease (35) 35
c3 glomerulonephritis (34) 34
research (34) 34
glomerulonephritis, membranoproliferative - drug therapy (33) 33
kidney - pathology (33) 33
risk factors (33) 33
factor h (32) 32
factor-h (32) 32
glomerulonephritis, membranoproliferative - diagnosis (32) 32
complement c3 - metabolism (31) 31
activation (30) 30
kidney transplantation (30) 30
mice (29) 29
young adult (29) 29
kidney diseases (28) 28
nephritic factor (28) 28
glomerulonephritis, membranoproliferative - therapy (27) 27
complement c3 - analysis (26) 26
complement system proteins - immunology (26) 26
treatment outcome (26) 26
autoantibodies (25) 25
diagnosis (25) 25
kidney glomerulus - pathology (25) 25
mutations (25) 25
aged (24) 24
complement c3 - immunology (24) 24
monoclonal gammopathy (24) 24
biochemistry & molecular biology (23) 23
complement factor h (23) 23
glomerulonephritis - immunology (23) 23
glomerulopathy (23) 23
pathology (23) 23
glomerulonephritis, membranoproliferative - blood (22) 22
mesangiocapillary glomerulonephritis (22) 22
mpgn (22) 22
retrospective studies (22) 22
care and treatment (21) 21
child, preschool (21) 21
complement factor h - deficiency (21) 21
complement factor h - metabolism (21) 21
glomerulonephritis, membranoproliferative - etiology (21) 21
kidney glomerulus - immunology (21) 21
transplantation (21) 21
complement c3 nephritic factor - analysis (20) 20
complement pathway, alternative (20) 20
glomerulonephritis, membranoproliferative - complications (20) 20
health aspects (20) 20
kidneys (20) 20
atypical hemolytic uremic syndrome (19) 19
glomerulonephritis - pathology (19) 19
hemolytic-uremic syndrome - genetics (19) 19
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article (18) 18
autoimmunity (18) 18
membrane cofactor protein (18) 18
recurrence (18) 18
renal-transplantation (18) 18
analysis (17) 17
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deficiency (17) 17
genetic predisposition to disease (17) 17
nephrotic syndrome (17) 17
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antibodies, monoclonal, humanized - therapeutic use (16) 16
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1. Full Text Deletion of Lys224 in regulatory domain 4 of Factor H reveals a novel pathomechanism for dense deposit disease (MPGN II)
by Licht, C and Heinen, S and Józsi, M and Löschmann, I and Saunders, R.E and Perkins, S.J and Waldherr, R and Skerka, C and Kirschfink, M and Hoppe, B and Zipfel, P.F
Kidney International, ISSN 0085-2538, 07/2006, Volume 70, Issue 1, pp. 42 - 50
We report a novel pathomechanism for membranoproliferative glomerulonephritis type II (MPGN II) caused by a mutant Factor H protein expressed in the plasma.... 
complement | hemolytic uremic syndrome | membranoproliferative glomerulonephritis (MPGN), Factor H, alternative complement pathway | Hemolytic uremic syndrome | Complement | Membranoproliferative glomerulonephritis (MPGN), Factor H, alternative complement pathway | Factor H | HEMOLYTIC-UREMIC SYNDROME | PROTEIN | CRYSTAL-STRUCTURE | alternative complement pathway | DEFICIENCY | membranoproliferative glomerulonephritis (MPGN) | COMPLEMENT FACTOR-H | CONVERTASE | ALTERNATIVE PATHWAY | UROLOGY & NEPHROLOGY | MUTATIONS | HOMOZYGOUS FACTOR-H | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Amino Acid Sequence | Sequence Deletion | Humans | Molecular Sequence Data | Complement C3 Nephritic Factor - analysis | Protein Structure, Tertiary - genetics | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Lysine - genetics | Plasma - metabolism | Complement Factor H - metabolism | Pedigree | Plasma - chemistry | Complement C3 Nephritic Factor - metabolism | Complement Factor H - analysis | Female | Consanguinity | Complement Factor H - genetics | Complement Pathway, Alternative | Lysine - chemistry | Child
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2. Full Text Pathogenesis of the C3 glomerulopathies and reclassification of MPGN
by Bomback, Andrew S and Appel, Gerald B
Nature Reviews Nephrology, ISSN 1759-5061, 11/2012, Volume 8, Issue 11, pp. 634 - 642
Until recently, membranoproliferative glomerulonephritis (MPGN) was clinically classified as either primary, idiopathic MPGN or as secondary MPGN when an... 
COMPLEMENT FACTOR-H | DENSE DEPOSIT DISEASE | NEPHRITIC FACTOR | HYPOCOMPLEMENTEMIA | INHIBITOR ECULIZUMAB | ALTERNATIVE PATHWAY | ABNORMALITIES | DYSREGULATION | UROLOGY & NEPHROLOGY | MONOCLONAL GAMMOPATHY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - pathology | Glomerulonephritis, Membranoproliferative - immunology | Glomerulonephritis, Membranoproliferative - classification | Prognosis | Kidney - pathology | Complement Pathway, Alternative - immunology | Humans | Complement System Proteins - immunology | Glomerulonephritis, Membranoproliferative - diagnosis | Complement Activation - immunology | Complement C3 - immunology | Glomerulonephritis | Care and treatment | Diagnosis | Complement (Immunology) | Health aspects | Identification and classification
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3. Full Text Histopathology of MPGN and C3 glomerulopathies
by Cook, H. Terence and Pickering, Matthew C
Nature Reviews Nephrology, ISSN 1759-5061, 01/2015, Volume 11, Issue 1, pp. 14 - 22
'Membranoproliferative' describes glomerular injury characterized by capillary wall thickening and mesangial expansion owing to increased matrix deposition and... 
COMPLEMENT FACTOR-H | POSTINFECTIOUS GLOMERULONEPHRITIS | MESANGIOCAPILLARY GLOMERULONEPHRITIS | ALTERNATIVE PATHWAY | PROLIFERATIVE GLOMERULONEPHRITIS | GLOMERULONEPHRITIS TYPE-II | UROLOGY & NEPHROLOGY | CLINICAL-FEATURES | IDIOPATHIC MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | BASEMENT-MEMBRANE | Glomerulonephritis, Membranoproliferative - pathology | Glomerulonephritis, Membranoproliferative - immunology | Glomerulonephritis, Membranoproliferative - classification | Kidney - pathology | Glomerulonephritis - pathology | Humans | Glomerulonephritis - immunology | Complement C3 - immunology | Glomerulonephritis | Development and progression | Genetic aspects | Immune response | Gene mutations | Health aspects
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4. Full Text Acquired and genetic complement abnormalities play a critical role in dense deposit disease and other C3 glomerulopathies
by Servais, Aude and Noël, Laure-Hélène and Roumenina, Lubka T and Le Quintrec, Moglie and Ngo, Stephanie and Dragon-Durey, Marie-Agnès and Macher, Marie-Alice and Zuber, Julien and Karras, Alexandre and Provot, François and Moulin, Bruno and Grünfeld, Jean-Pierre and Niaudet, Patrick and Lesavre, Philippe and Frémeaux-Bacchi, Véronique
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 454 - 464
Dense deposit disease and glomerulonephritis with isolated C3 deposits are glomerulopathies characterized by deposits of C3 within or along the glomerular... 
complement | glomerulonephritis | clinical immunology | membranoproliferative glomerulonephritis (MPGN) | LONG-TERM | HEMOLYTIC-UREMIC SYNDROME | MESANGIOCAPILLARY GLOMERULONEPHRITIS | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | FACTOR-H DEFICIENCY | GLOMERULAR-FILTRATION-RATE | UROLOGY & NEPHROLOGY | MUTATIONS | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | MEMBRANE COFACTOR PROTEIN | Humans | Child, Preschool | Male | Complement System Proteins - metabolism | Young Adult | Time Factors | DNA Mutational Analysis | Renal Insufficiency - immunology | Child | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Risk Assessment | Complement C3 - metabolism | Gene Frequency | Risk Factors | Glomerulonephritis - genetics | Glomerulonephritis - immunology | Glomerulonephritis - mortality | Biomarkers - blood | Kidney Glomerulus - pathology | Disease Progression | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Phenotype | Complement Factor H - metabolism | Glomerulonephritis, Membranoproliferative - mortality | Glomerulonephritis - pathology | Adolescent | Age of Onset | Membrane Cofactor Protein - genetics | Kidney Glomerulus - immunology | Mutation | Complement Factor I - metabolism | Haplotypes | Complement C3 Nephritic Factor - genetics | Glomerulonephritis - therapy | Infant | Case-Control Studies | Adult | Complement System Proteins - genetics | Female | Membrane Cofactor Protein - metabolism | Complement Pathway, Alternative - genetics | France | Renal Insufficiency - genetics | Kaplan-Meier Estimate | Complement Factor I - genetics | Treatment Outcome | Chi-Square Distribution | Glomerulonephritis, Membranoproliferative - therapy | Biopsy | Complement C3 Nephritic Factor - metabolism | Complement Factor H - genetics
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5. Full Text Complement gene variants determine the risk of immunoglobulin-associated MPGN and C3 glomerulopathy and predict long-term renal outcome
by Iatropoulos, Paraskevas and Noris, Marina and Mele, Caterina and Piras, Rossella and Valoti, Elisabetta and Bresin, Elena and Curreri, Manuela and Mondo, Elena and Zito, Anna and Gamba, Sara and Bettoni, Serena and Murer, Luisa and Fremeaux-Bacchi, Veronique and Vivarelli, Marina and Emma, Francesco and Daina, Erica and Remuzzi, Giuseppe
Molecular Immunology, ISSN 0161-5890, 03/2016, Volume 71, pp. 131 - 142
Membranoproliferative glomerulonephritis (MPGN) is an uncommon cause of chronic nephropathy recently reclassified into immunoglobulin-associated MPGN (Ig-MPGN)... 
Rare diseases | Membranoproliferative glomerulonephritis | Dense-Deposit Disease | C3 glomerulopathy | C3 glomerulonephritis | ACTIVATION | HEMOLYTIC-UREMIC SYNDROME | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLASSIFICATION | FACTOR-H | IMMUNOLOGY | FACTOR B | THROMBOMODULIN | MUTATIONS | BINDING | Complement C3 Nephritic Factor - genetics | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Immunoglobulins | Glomerulonephritis, Membranoproliferative - classification | Humans | Risk Factors | Male | Multiplex Polymerase Chain Reaction | Genetic Variation | Young Adult | Glomerulonephritis, Membranoproliferative - genetics | Adolescent | Fluorescent Antibody Technique | Female | High-Throughput Nucleotide Sequencing | Polymorphism, Single Nucleotide | Complement Pathway, Alternative - genetics | Thrombomodulin - genetics | Kidney Failure, Chronic - etiology | Genetic research | Single nucleotide polymorphisms | Gene mutations | Children's hospitals | Genes | Autoimmunity | Medical research | Medicine, Experimental
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6. Full Text Membranoproliferative glomerulonephritis and C3 glomerulopathy: resolving the confusion
by Sethi, Sanjeev and Nester, Carla M and Smith, Richard J.H
Kidney International, ISSN 0085-2538, 03/2012, Volume 81, Issue 5, pp. 434 - 441
Membranoproliferative glomerulonephritis (MPGN) denotes a general pattern of glomerular injury that is easily recognized by light microscopy. With additional... 
pathology | glomerular disease | complement | immunology | membranoproliferative glomerulonephritis (MPGN) | DENSE DEPOSIT DISEASE | PROTEIN | ALTERNATIVE PATHWAY | MUTATION | UROLOGY & NEPHROLOGY | FACTOR-H | membrano-proliferative glomerulonephritis (MPGN) | REVEALS | Glomerulonephritis, Membranoproliferative - drug therapy | Glomerulonephritis, Membranoproliferative - pathology | Complement C3 - antagonists & inhibitors | Glomerulonephritis, Membranoproliferative - classification | Complement C3 - metabolism | Glomerulonephritis - pathology | Humans | Glomerulonephritis - immunology | Glomerulonephritis - classification | Microscopy, Electron | Antimitotic Agents - therapeutic use | Microscopy, Fluorescence
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7. Full Text C3 glomerulonephritis: clinicopathological findings, complement abnormalities, glomerular proteomic profile, treatment, and follow-up
by Sethi, Sanjeev and Fervenza, Fernando C and Zhang, Yuzhou and Zand, Ladan and Vrana, Julie A and Nasr, Samih H and Theis, Jason D and Dogan, Ahmet and Smith, Richard J.H
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 465 - 473
C3 glomerulonephritis (C3GN) is a recently described disorder that typically results from abnormalities in the alternative pathway (AP) of complement. Here, we... 
C3GN | proteomics | C3 glomerulopathy | MPGN | alternative pathway of complement | C3 glomerulonephritis | MACULAR DEGENERATION | CLASSIFICATION | RISK | PROTEIN 5 | DENSE DEPOSIT DISEASE | ALTERNATIVE PATHWAY | UROLOGY & NEPHROLOGY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Complement C3 - analysis | Predictive Value of Tests | Recurrence | Follow-Up Studies | Humans | Middle Aged | Complement System Proteins - immunology | Laser Capture Microdissection | Molecular Sequence Data | Male | Glomerulonephritis, Membranoproliferative - complications | Complement System Proteins - analysis | Young Adult | Time Factors | DNA Mutational Analysis | Mass Spectrometry | Adult | Autoantibodies - analysis | Complement System Proteins - genetics | Female | Complement Pathway, Alternative - genetics | Proteomics - methods | Child | Amino Acid Sequence | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Proteinuria - immunology | Treatment Outcome | Complement C3 Nephritic Factor - analysis | Hematuria - immunology | Kidney Transplantation | Minnesota | Kidney Glomerulus - pathology | Glomerulonephritis, Membranoproliferative - therapy | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Biopsy | Adolescent | Aged | Kidney Glomerulus - immunology | Mutation | Complement Factor H - genetics
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8. Full Text MPGN II – genetically determined by defective complement regulation?
by Licht, Christoph and Licht, Christoph and Schlötzer-Schrehardt, Ursula and Schlötzer-Schrehardt, Ursula and Kirschfink, Michael and Kirschfink, Michael and Zipfel, Peter F and Zipfel, Peter F and Hoppe, Bernd and Hoppe, Bernd
Pediatric Nephrology, ISSN 0931-041X, 1/2007, Volume 22, Issue 1, pp. 2 - 9
MPGN II is a rare disease which is characterized by complement containing deposits within the GBM. The disease is characterized by functional impairment of the... 
Factor H autoantibodies | Pediatrics | Factor H | Medicine & Public Health | Membranoproliferative glomerulonephritis type II (MPGN II) | C3 nephritic factor (C3NeF) | Alternative pathway of the complement system | ACTIVATION | HEMOLYTIC-UREMIC SYNDROME | GLOMERULONEPHRITIS TYPE-II | C3 CONVERTASE | IDIOPATHIC MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | alternative pathway of the complement system | FACTOR-H DEFICIENCY | DENSE DEPOSIT DISEASE | ALTERNATIVE PATHWAY | membranoproliferative glomerulonephritis type II (MPGN II) | UROLOGY & NEPHROLOGY | PEDIATRICS | MICE | MOLECULAR-BASIS | Complement C3 Nephritic Factor - genetics | Kidney - pathology | Complement Factor H - physiology | Humans | Mutation - genetics | Disease Progression | Glomerulonephritis, Membranoproliferative - drug therapy | Glomerulonephritis, Membranoproliferative - genetics | Animals | Glomerulonephritis, Membranoproliferative - physiopathology | Complement C3 Nephritic Factor - physiology | Complement Factor H - genetics | Kidney - physiopathology
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9. Full Text Unraveling the molecular mechanisms underlying complement dysregulation by nephritic factors in C3G and IC-MPGN
by Donadelli, Roberta and Pulieri, Patrizia and Piras, Rossella and Iatropoulos, Paraskevas and Valoti, Elisabetta and Benigni, Ariela and Remuzzi, Giuseppe and Noris, Marina
Frontiers in Immunology, ISSN 1664-3224, 10/2018, Volume 9, p. 2329
Membranoproliferative glomerulonephritis (MPGN) was recently classified as C3 glomerulopathies (C3G), and immune-complex (IC) mediated MPGN. Dysregulation of... 
Factor H | Membranoproliferative glomerulonephritis | C3 nephritic factors | C3 glomerulopathy | Complement alternative pathway | Terminal complement complex | C3 convertase | FACTOR-B | FLUID-PHASE | C-3 | FACTOR-H | IMMUNOLOGY | factor H | membranoproliferative glomerulonephritis | DENSE DEPOSIT DISEASE | ALTERNATIVE PATHWAY | IN-VIVO | DECAY-ACCELERATING FACTOR | complement alternative pathway | terminal complement complex | FACTOR C3NEF | Glomerulonephritis | Autoantibodies | Research
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10. Full Text Factor H autoantibodies in membranoproliferative glomerulonephritis
by Goodship, Timothy H.J and Pappworth, Isabel Y and Toth, Tibor and Denton, Mark and Houlberg, Kris and McCormick, Frances and Warland, David and Moore, Iain and Hunze, Eva-Maria and Staniforth, Scott J and Hayes, Christine and Cavalcante, Danielle Paixão and Kavanagh, David and Strain, Lisa and Herbert, Andrew P and Schmidt, Christoph Q and Barlow, Paul N and Harris, Claire L and Marchbank, Kevin J
Molecular Immunology, ISSN 0161-5890, 10/2012, Volume 52, Issue 3-4, pp. 200 - 206
Factor H autoantibodies are found in ∼10% of aHUS patients. Most are associated with complete deficiency of factor H related proteins 1/3 and bind to the C... 
Factor H | Complement | Autoantibodies | MPGN | HEMOLYTIC-UREMIC SYNDROME | FACTOR-B | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMMUNOLOGY | COMPLEMENT FACTOR-H | DENSE DEPOSIT DISEASE | NEPHRITIC FACTOR | MPGN-II | ALTERNATIVE PATHWAY | MUTATIONS | PREDISPOSE | Complement Factor H - immunology | Autoantibodies - blood | Complement C3 | Humans | Middle Aged | Male | Complement C3 Nephritic Factor - analysis | Young Adult | Glomerulonephritis, Membranoproliferative - genetics | Autoantibodies - immunology | Glomerulonephritis, Membranoproliferative - immunology | Adolescent | Adult | Female | Aged | Binding Sites, Antibody | Complement Factor H - chemistry | Proteins | Viral antibodies | Autoimmunity | Medical colleges | Immunoglobulins | Immunoglobulin G | Antibodies | Enzyme-linked immunosorbent assay | Protein binding
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11. Full Text Translational Mini-Review Series on Complement Factor H: Renal diseases associated with complement factor H: novel insights from humans and animals
by Pickering, M. C and Cook, H. T
Clinical & Experimental Immunology, ISSN 0009-9104, 02/2008, Volume 151, Issue 2, pp. 210 - 230
OTHER ARTICLES PUBLISHED IN THIS TRANSLATIONAL MINI-REVIEW SERIES ON COMPLEMENT FACTOR HGenetics and disease associations of human complement factor H. Clin... 
Factor H | thrombosis | complement | glomerulonephritis | Glomerulonephritis | Complement | Thrombosis | HEMOLYTIC-UREMIC SYNDROME | BETA-1H GLOBULIN | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | IMMUNOLOGY | factor H | DENSE DEPOSIT DISEASE | GLOMERULAR MESANGIAL CELLS | NEPHRITIC FACTOR | ALTERNATIVE PATHWAY | HOMOZYGOUS FACTOR-H | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Complement Factor H - immunology | Glomerulonephritis, Membranoproliferative - pathology | Humans | Middle Aged | Child, Preschool | Complement Factor H - deficiency | Infant | Male | Hemolytic-Uremic Syndrome - immunology | Hemolytic-Uremic Syndrome - pathology | Glomerulonephritis, Membranoproliferative - genetics | Hemolytic-Uremic Syndrome - genetics | Glomerulonephritis, Membranoproliferative - immunology | Animals | Adolescent | Adult | Autoantibodies - analysis | Female | Complement Factor H - genetics | Complement Pathway, Alternative | Child | Risk factors | Kidney diseases | Translational Mini-Review Series | Complement Factor H
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12. Full Text Complement and diseases: Defective alternative pathway control results in kidney and eye diseases
by Zipfel, Peter F and Heinen, Stefan and Józsi, Mihály and Skerka, Christine
Molecular Immunology, ISSN 0161-5890, 2006, Volume 43, Issue 1, pp. 97 - 106
The complement system is a central part of innate immunity and in its normal setting aimed to recognize and eliminate microbes. For elimination toxic... 
Factor H | Alternative pathway control | Membranoproliferative glomerulonephritis (MPGN) | Hemolytic uremic syndrome (HUS) | Age-related macular degeneration (ARMD) | Innate immunity | Complement | Pattern recognition | ACTIVATION | HEMOLYTIC-UREMIC SYNDROME | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | IMMUNOLOGY | factor H | membranoproliferative glomerulonephritis (MPGN) | TRANSPLANTATION | FACTOR-H DEFICIENCY | innate immunity | GENE | age-related macular degeneration (ARMD) | MUTATIONS | complement | alternative pathway control | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | hemolytic uremic syndrome (HUS) | pattern recognition | Glomerulonephritis, Membranoproliferative - pathology | Hemolytic-Uremic Syndrome - metabolism | Complement C3 Convertase, Alternative Pathway - metabolism | Humans | Complement Factor H - deficiency | Mice, Knockout | Hemolytic-Uremic Syndrome - pathology | Glomerulonephritis, Membranoproliferative - genetics | Hemolytic-Uremic Syndrome - genetics | Macular Degeneration - metabolism | Animals | Complement Factor H - metabolism | Macular Degeneration - genetics | Swine | Mice | Complement Pathway, Alternative - genetics | Mutation | Complement Factor H - genetics | Complement C3 Convertase, Alternative Pathway - deficiency | Complement C3 Convertase, Alternative Pathway - genetics | Glomerulonephritis, Membranoproliferative - metabolism | Disease Models, Animal | Macular Degeneration - pathology | Macular degeneration | Genetic research
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