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by Wu, J and Ru, N.-Y and Zhang, Y and Li, Y and Wei, D and Ren, Z and Huang, X.-F and Chen, Z.-N and Bian, H
Oncogene, ISSN 0950-9232, 10/2011, Volume 30, Issue 43, pp. 4410 - 4427
Journal Article
Cancer Letters, ISSN 0304-3835, 2017, Volume 392, pp. 26 - 38
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 11/2010, Volume 124, Issue 2, pp. 317 - 326
Journal Article
Oncogene, ISSN 0950-9232, 04/2016, Volume 35, Issue 15, pp. 1888 - 1898
The process of epithelial-mesenchymal transition (EMT), in addition to being an initiating event for tumor metastasis, is implicated in conferring several... 
CARCINOMA-CELLS | LUNG-CARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-CELLS | CANCER-IMMUNOTHERAPY | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | RESISTANCE | CD59 | PROTEINS | EXPRESSION | NEUTRALIZATION | MODULATION | RNA, Small Interfering - genetics | Adenocarcinoma - pathology | Complement Activation | Tumor Escape - genetics | CD59 Antigens - immunology | Epithelial-Mesenchymal Transition - physiology | Humans | Gene Expression Regulation, Neoplastic | Neoplasm Proteins - immunology | Transcriptome | Lung Neoplasms - pathology | Male | Neoplasm Proteins - antagonists & inhibitors | Complement Membrane Attack Complex - immunology | Genetic Vectors - therapeutic use | Proteins | RNA, Messenger - biosynthesis | RNA Interference | Mice, Mutant Strains | Neoplasm Proteins - genetics | Tumor Escape - immunology | Promoter Regions, Genetic | Neoplasm Proteins - biosynthesis | CD59 Antigens - genetics | Cetuximab - pharmacology | Mice, SCID | CD59 Antigens - biosynthesis | Xenograft Model Antitumor Assays | Transforming Growth Factor beta - pharmacology | Animals | RNA, Neoplasm - biosynthesis | Epithelial-Mesenchymal Transition - immunology | Cell Line, Tumor | Mice | Smad3 Protein - physiology | Cytotoxicity, Immunologic | Index Medicus | lung cancer | Complement regulatory proteins | immune evasion | metastasis
Journal Article
The EMBO Journal, ISSN 0261-4189, 02/2011, Volume 30, Issue 4, pp. 783 - 795
The epithelial–mesenchymal transition (EMT) is a crucial event in wound healing, tissue repair, and cancer progression in adult tissues. Here, we demonstrate... 
FGF‐2 | TGF‐β | δEF1 | FGF receptor | EMT | FGF-2 | TGF-β | dEF1 | DELTA-EF1 | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | SUPPRESSION | FIBROBLASTS | CELL BIOLOGY | PROSTATE ADENOCARCINOMA | COREPRESSORS | TGF-beta | SQUAMOUS-CELL CARCINOMA | CANCER PROGRESSION | TUMOR-GROWTH | Neoplasms - metabolism | Protein Binding - genetics | Myofibroblasts - physiology | Epithelial-Mesenchymal Transition - physiology | Homeodomain Proteins - metabolism | Humans | Actins - metabolism | Fibroblast Growth Factor 2 - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Actins - genetics | DNA-Binding Proteins - metabolism | Myofibroblasts - metabolism | Cell Differentiation - genetics | Protein Isoforms - metabolism | Neoplasms - genetics | Protein Binding - drug effects | Receptors, Fibroblast Growth Factor - genetics | Fibroblast Growth Factor 2 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Cell Differentiation - physiology | Neoplasm Invasiveness | Alternative Splicing - genetics | Cells, Cultured | Transforming Growth Factor beta - physiology | Alcohol Oxidoreductases - metabolism | Signal Transduction - genetics | Myofibroblasts - drug effects | Protein Isoforms - physiology | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Alternative Splicing - drug effects | Cell Differentiation - drug effects | Models, Biological | Receptors, Fibroblast Growth Factor - metabolism | Neoplasms - pathology | Protein Isoforms - genetics | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2015, Volume 17, Issue 5, pp. 678 - 688
Journal Article
Circulation, ISSN 0009-7322, 03/2015, Volume 131, Issue 11, pp. 1006 - 1018
BACKGROUND—The vascular remodeling responsible for pulmonary arterial hypertension (PAH) involves predominantly the accumulation of α-smooth muscle... 
Human | Hypertension | Vimentin | Sirolimus | Twist1 protein | Vascular remodeling | Epithelial-mesenchymal transition | Neointima | Animal | Type ii | Models | Bone morphogenetic protein receptors | Cardiovascular diseases | Pulmonary | RAPAMYCIN | MIGRATION | FIBROSIS | models, animal | CARDIAC & CARDIOVASCULAR SYSTEMS | neointima | hypertension, pulmonary | vimentin | cardiovascular diseases | vascular remodeling | DISCOVERY | bone morphogenetic protein receptors, type II | IN-VITRO | sirolimus | TWIST1 protein, human | ARTERIAL-HYPERTENSION | PERIPHERAL VASCULAR DISEASE | epithelial-mesenchymal transition | SMOOTH-MUSCLE-CELLS | KNOCKOUT RATS | TRANSDIFFERENTIATION | Vascular Remodeling | Humans | Gene Expression Profiling | Hypertension, Pulmonary - chemically induced | Actins - genetics | RNA, Messenger - biosynthesis | Vimentin - genetics | Cell Transdifferentiation | Lung - metabolism | Disease Models, Animal | Lung - pathology | Actins - biosynthesis | Bone Morphogenetic Protein Receptors, Type II - genetics | Vimentin - biosynthesis | Cells, Cultured | Rats | Hypertension, Pulmonary - genetics | Hypoxia - complications | Monocrotaline - toxicity | Sirolimus - pharmacology | Animals | Biomarkers | Mutation | Endothelial Cells - pathology | Lung - blood supply | Hypertension, Pulmonary - pathology | Mesoderm - pathology | Bone Morphogenetic Protein Receptors, Type II - biosynthesis | Cell Movement | Index Medicus | Abridged Index Medicus | Life Sciences
Journal Article
Oncogene, ISSN 0950-9232, 05/2014, Volume 33, Issue 18, pp. 2307 - 2316
Signals from the tumor microenvironment trigger cancer cells to adopt an invasive phenotype through epithelial-mesenchymal transition (EMT). Relatively little... 
Vimentin | Epithelial-mesenchymal transition | Signal transduction | Breast cancer | Calcium | STAT3 | MIGRATION | ACTIVATION | calcium | METASTASIS | signal transduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHENOTYPE | breast cancer | vimentin | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | epithelial-mesenchymal transition | TRPM7 | CHANNELS | UP-REGULATION | CONTRIBUTES | PROGRESSION | RNA, Small Interfering - genetics | Phosphorylation | Vimentin - biosynthesis | Calcium - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Protein-Serine-Threonine Kinases | Epidermal Growth Factor - metabolism | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Breast Neoplasms - metabolism | Cell Hypoxia | TRPM Cation Channels - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | Cell Line, Tumor | TRPM Cation Channels - genetics | Female | TRPM Cation Channels - metabolism | Epidermal Growth Factor - pharmacology | Calcium Signaling | STAT3 Transcription Factor - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Care and treatment | Calcium channels | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Risk factors | Cellular biology | Index Medicus | Epithelial-mesenchymal transition (EMT)
Journal Article