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by Zhou, Q and Wang, X and Yu, Z and Wu, X and Chen, X and Li, J and Zhu, Z and Liu, B and Su, L
Oncogene, ISSN 0950-9232, 03/2017, Volume 36, Issue 13, pp. 1873 - 1886
Gastric cancer (GC) is one of the most common types of cancer worldwide, and it involves extensive local tumour invasion, metastasis and poor prognosis.... 
ACTIVATION | TBLR1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | INTELLECTUAL DISABILITY | ONCOLOGY | TBL1XR1 | FREQUENT GAIN | GENES | GENETICS & HEREDITY | SQUAMOUS-CELL CARCINOMA | COREPRESSOR COMPLEX | Immunohistochemistry | Prognosis | Humans | Middle Aged | Stomach Neoplasms - metabolism | Male | Stomach Neoplasms - pathology | Cell Movement - genetics | MAP Kinase Signaling System | Neoplasm Metastasis | Receptor, Epidermal Growth Factor - metabolism | Heterografts | Neoplasm Grading | Matrix Metalloproteinase 7 - metabolism | Epithelial-Mesenchymal Transition | Female | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Disease Models, Animal | Stomach Neoplasms - genetics | Cell Proliferation - genetics | Gene Expression | Signal Transduction | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Tumor Burden | beta Catenin - metabolism | Disease Progression | Animals | Cell Line, Tumor | Aged | Mice | Neoplasm Staging | Stomach Neoplasms - mortality | Receptors, Cytoplasmic and Nuclear - metabolism | Transducin | Physiological aspects | Development and progression | Genetic aspects | Research | Stomach cancer | Cancer | Oncology | Tumorigenesis | Metastasis | Medical prognosis | Index Medicus | Original
Journal Article
Journal Article
Fertility and Sterility, ISSN 0015-0282, 2016, Volume 107, Issue 2, pp. 439 - 447
Objective To investigate whether G protein-coupled estrogen receptor (GPER, also known as GPR30 and GPER1) stabilizes hypoxia-inducible factor 1α (HIF-1α) in... 
Internal Medicine | Obstetrics and Gynecology | hypoxia-inducible factor 1α (HIF-1α) | Endometriosis | G protein-coupled estrogen receptor (GPER) | Estrogen | TARGET | ACTIVATION | hypoxia-inducible factor 1 alpha (HIF-1 alpha) | ANGIOGENESIS | OBSTETRICS & GYNECOLOGY | PATHOGENESIS | BREAST-CANCER CELLS | INVASION | REPRODUCTIVE BIOLOGY | GPER | HIF-1-ALPHA | STROMAL CELLS | EXPRESSION | Neovascularization, Physiologic - drug effects | Receptors, Estrogen - metabolism | Endometriosis - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, G-Protein-Coupled - metabolism | Stromal Cells - pathology | Humans | Endometrium - drug effects | Vascular Endothelial Growth Factor A - metabolism | Receptors, G-Protein-Coupled - agonists | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Case-Control Studies | Young Adult | Matrix Metalloproteinase 9 - metabolism | Transfection | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Matrix Metalloproteinase 9 - genetics | Stromal Cells - drug effects | Adult | Female | Protein Stability | Estradiol - pharmacology | Endometrium - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Receptors, Estrogen - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | RNA, Messenger - genetics | Stromal Cells - metabolism | Cells, Cultured | Endometriosis - pathology | Cell Movement - drug effects | Signal Transduction - drug effects | Endometriosis - metabolism | Receptors, G-Protein-Coupled - genetics | Endometrium - pathology | Index Medicus | endometriosis | Hypoxia inducible factor 1 α (HIF-1α)
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, pp. e115596 - e115596
Background: Systemic sclerosis (SSc) is an autoimmune connective tissue disease characterised by skin and internal organs fibrosis due to accumulation of extra... 
PATHOGENESIS | MATRIX METALLOPROTEINASES | COLLAGEN EXPRESSION | MYOCARDIAL-INFARCTION | TAK1 | MULTIDISCIPLINARY SCIENCES | LIVER FIBROSIS | GENE-EXPRESSION | TISSUE INHIBITOR | GROWTH-FACTOR | MICRORNAS | Dermis - metabolism | Extracellular Matrix Proteins - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Extracellular Matrix Proteins - genetics | Humans | Scleroderma, Systemic - metabolism | Gene Expression Regulation | Scleroderma, Systemic - genetics | Scleroderma, Systemic - pathology | 3' Untranslated Regions - genetics | Male | MicroRNAs - biosynthesis | Fibroblasts - pathology | Tissue Inhibitor of Metalloproteinase-1 - genetics | Proteolysis | Adaptor Proteins, Signal Transducing - genetics | Dermis - pathology | Female | Adaptor Proteins, Signal Transducing - biosynthesis | MicroRNAs - genetics | HeLa Cells | Fibroblasts - metabolism | Protectors | Control methods | Pathogenesis | Matrix metalloproteinase | Kinases | Tissue inhibitor of metalloproteinase 1 | Sclerosis | Western blotting | Proteins | Connective tissues | Immunology | Transfection | Rodents | Fibroblasts | Skin diseases | Extracellular matrix | Growth factors | Enzyme-linked immunosorbent assay | Interstitial collagenase | Binding | Secretion | Organs | Blocking | Pharmacology | Gene expression | Medicine | Biopsy | Systemic sclerosis | Collagen | Fibrosis | Transduction | Genetic engineering | Skin | Binding sites | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 138, Issue 1, pp. 61 - 75
Background Disease heterogeneity in patients with severe asthma and its relationship to inflammatory mechanisms remain poorly understood. Objective We aimed to... 
Allergy and Immunology | phenotype | endotype | topological data analysis | chitinase 3–like protein 1 | eosinophils | matrix metalloproteinase | cytokines | neutrophils | heterogeneity | Asthma | SPUTUM | LUNG-FUNCTION | PHENOTYPES | RESEARCH-PROGRAM | IMMUNOLOGY | IDENTIFICATION | YKL-40 | CLUSTER-ANALYSIS | chitinase 3-like protein 1 | OBSTRUCTIVE PULMONARY-DISEASE | ALLERGY | MICE | EXPRESSION | Asthma - diagnosis | Severity of Illness Index | Asthma - metabolism | Cytokines - metabolism | Humans | Middle Aged | Risk Factors | Male | Asthma - drug therapy | Matrix Metalloproteinase Inhibitors - metabolism | Case-Control Studies | Sputum - metabolism | Chitinase-3-Like Protein 1 - metabolism | Young Adult | Bayes Theorem | Inflammation Mediators - metabolism | Biomarkers | Adult | Female | Aged | Matrix Metalloproteinases - metabolism | Respiratory Function Tests | Medical colleges | Care and treatment | Hospitals | Respiratory agents | Research institutes | Pharmaceutical industry | Medicine, Experimental | Medical research | Information management | Analysis | Datasets | Biomedical research | Questionnaires | Neutrophils | Software | Patients | Methods | Quality of life | Index Medicus | Abridged Index Medicus | ECP, Eosinophil cationic protein | K-S, Kolmogorov-Smirnov | MMP, Matrix metalloproteinase | ACQ, Asthma Control Questionnaire | Feno, Fraction of exhaled nitric oxide | HAD, Hospital Anxiety and Depression | YKL-40, Chitinase 3–like protein 1 | Asthma and Lower Airway Disease | TDA, Topological data analysis | FGF, Fibroblast growth factor | GINA, Global Initiative for Asthma | ICS, Inhaled corticosteroid
Journal Article
Diabetologia, ISSN 0012-186X, 5/2011, Volume 54, Issue 5, pp. 1227 - 1241
Journal Article
Basic & Clinical Pharmacology & Toxicology, ISSN 1742-7835, 10/2011, Volume 109, Issue 4, pp. 292 - 299
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2014, Volume 357, Issue 2, pp. 527 - 534
Highlights • Treatment of CRC cells with HMBG1 contributes to induction of EMT. • HMBG1 activated RAGE/Snail/NF-κB pathways in HMBG1-induced EMT. • The... 
Hematology, Oncology and Palliative Medicine | High-mobility group box 1 | Epithelial–mesenchymal transition | Matrix metalloproteinase-7 | Colorectal carcinoma | Epithelial-mesenchymal transition | CANCER CELLS | RECEPTOR | HMGB1 | INVASION | ONCOLOGY | PATHWAY | NF-KAPPA-B | EXPRESSION | PROGRESSION | GLYCATION END-PRODUCTS | SNAIL | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - drug effects | NF-kappa B - metabolism | Epithelial-Mesenchymal Transition - genetics | Cell Movement - genetics | HMGB1 Protein - genetics | HMGB1 Protein - pharmacology | Matrix Metalloproteinase 7 - metabolism | RNA Interference | Receptor for Advanced Glycation End Products | Snail Family Transcription Factors | Colorectal Neoplasms - metabolism | HCT116 Cells | Signal Transduction - genetics | Recombinant Proteins - pharmacology | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Transcription Factors - metabolism | Cell Movement - drug effects | Matrix Metalloproteinase 7 - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Colorectal Neoplasms - pathology | Receptors, Immunologic - genetics | Microscopy, Fluorescence | Receptors, Immunologic - metabolism | Stem cells | Colorectal cancer | Proteins | Metastasis | Cytokines | Kinases | Gene expression | Cancer | Index Medicus
Journal Article