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by Shi, L and Li, M and Zhang, F and Wang, B and Yi, X and Ding, Y and Wang, X
Clinical and Experimental Dermatology, ISSN 0307-6938, 04/2018, Volume 43, Issue 3, pp. 326 - 327
Journal Article
12/2012
Purpose: To investigate the anti-fatigue property of methazolamide (MTZ) in high-altitude anoxic mice. Methods: Mice fatigued by high-altitude hypoxia were... 
Methazolamide | Exhaustive exercise | High-altitude hypoxia | Anti-fatigue
Web Resource
Aging Cell, ISSN 1474-9718, 08/2018, Volume 17, Issue 4
Mounting evidence suggests that mitochondrial dysfunction plays a causal role in the etiology and progression of Alzheimer's disease (AD). We recently showed... 
Prevention | Methazolamide | Alzheimer's disease | Drug approval
Journal Article
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2015, Volume 11, Issue 7, pp. P860 - P861
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2008, Volume 28, Issue 38, pp. 9473 - 9485
Release of mitochondrial cytochrome c resulting in downstream activation of cell death pathways has been suggested to play a role in neurologic diseases... 
Methazolamide | Inhibitors of cytochrome c release | Mutant-htt ST14A cells | Drug screen | Huntington's disease | R6/2 mice | inhibitors of cytochrome c release | methazolamide | APOPTOSIS | mutant-htt ST14A cells | MINOCYCLINE | MICROGLIAL ACTIVATION | MITOCHONDRIA | NEURONAL CELL-DEATH | NEUROSCIENCES | PERMEABILITY TRANSITION | MOUSE MODEL | PARKINSON-DISEASE | drug screen | CASPASE-1 ACTIVATION | CEREBRAL-ISCHEMIA | Neuroprotective Agents - therapeutic use | Longevity - drug effects | Membrane Potential, Mitochondrial - drug effects | Brain - metabolism | Caspases - metabolism | Neuroprotective Agents - pharmacology | Membrane Potential, Mitochondrial - physiology | Caspases - drug effects | Cell Death - drug effects | Drug Evaluation, Preclinical | Huntington Disease - drug therapy | Huntington Disease - physiopathology | Carbonic Anhydrase Inhibitors - therapeutic use | Disease Models, Animal | Cytochromes c - antagonists & inhibitors | Brain - physiopathology | Cytochromes c - metabolism | Mice, Transgenic | Treatment Outcome | Mitochondria - metabolism | Carbonic Anhydrase Inhibitors - pharmacology | Mitochondria - drug effects | Huntington Disease - metabolism | Brain - drug effects | Animals | Cell Death - physiology | Methazolamide - therapeutic use | Methazolamide - pharmacology | Mice | Longevity - physiology | Cell Line, Transformed | Index Medicus
Journal Article
Drug Metabolism Letters, ISSN 1872-3128, 12/2016, Volume 10, Issue 4, pp. 295 - 305
Drug therapy is occasionally accompanied by an idiosyncratic severe toxicity, which occurs very rarely, but can lead to patient mortality. Methazolamide, an... 
Methazolamide | Stevens-Johnson syndrome | Toxic epidermal necrolysis | N-[3-methyl-5-sulfo-1,3,4-thiadiazo-2(3H)-ylidene]acetamide | Cytochrome P450 | Sulfenic acid | Sulfonic acid | Glutathione | Isoniazid - pharmacology | Chromatography, High Pressure Liquid - methods | Humans | Acetylcysteine - metabolism | Cytochrome P-450 Enzyme System - metabolism | Methazolamide - toxicity | Sulfenic Acids - metabolism | Lyases - metabolism | Oxygenases - antagonists & inhibitors | Carbonic Anhydrase Inhibitors - toxicity | Dexamethasone - pharmacology | Glucuronides - metabolism | beta-Naphthoflavone - pharmacology | Cysteine - metabolism | Tandem Mass Spectrometry - methods | Carbonic Anhydrase Inhibitors - therapeutic use | Cell Line | Methimazole - pharmacology | Oxidation-Reduction | Stevens-Johnson Syndrome - etiology | Keratinocytes | Glaucoma - drug therapy | Sulfonic Acids - toxicity | Sulfonic Acids - metabolism | Methazolamide - metabolism | Methazolamide - therapeutic use | Carbonic Anhydrase Inhibitors - metabolism | Glaucoma | Cytochrome | Cell culture | Toxicity | Acetylcysteine | Dimethylaniline monooxygenase (N-oxide-forming) | Flavin | Metabolites | Etiology | Oxidation | Isoniazid | Naphthoflavone | Inducers | Dexamethasone | Mass spectroscopy | Liquid chromatography | Metabolism | High-performance liquid chromatography | Monooxygenase | Acids | Skin | Mass spectrometry
Journal Article
Journal Article
Journal Article
JOURNAL OF GLAUCOMA, ISSN 1057-0829, 10/2010, Volume 19, Issue 8, pp. 568 - 568
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2015, Volume 86, pp. 29 - 40
Abstract Mitochondrial dysfunction has been recognized as an early event in Alzheimer's disease (AD) pathology, preceding and inducing neurodegeneration and... 
Neurology | Methazolamide | Mitochondria | Caspase activation | Hydrogen peroxide | Neuron | Carbonic anhydrase inhibitor | Amyloid | Alzheimer's disease | Hippocampus | Microglia | MICROGLIAL ACTIVATION | 3-NITROPROPIONIC ACID | SYNAPTIC MITOCHONDRIA | NEUROSCIENCES | MUSCLE DIFFERENTIATION | INDUCED OXIDATIVE STRESS | CYTOCHROME-C RELEASE | ALZHEIMERS A-BETA | PHARMACOLOGICAL INHIBITION | HUNTINGTONS-DISEASE | MOLECULAR-MECHANISMS | Mitochondria - enzymology | Caspase 9 - metabolism | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Brain - enzymology | Neuroglia - drug effects | Caspases - metabolism | Amyloid beta-Peptides - metabolism | Amyloid beta-Peptides - administration & dosage | DNA Fragmentation - drug effects | Neurons - drug effects | Cytochromes c - metabolism | Mice, Inbred C57BL | Neuroglia - enzymology | Cells, Cultured | Carbonic Anhydrase Inhibitors - administration & dosage | Alzheimer Disease - enzymology | Mitochondria - drug effects | Hydrogen Peroxide - metabolism | Brain - drug effects | Animals | Neurons - enzymology | Mice | Methazolamide - administration & dosage | In Vitro Techniques | Prevention | Mitochondrial DNA | Neurons | Analysis | Index Medicus | Life Sciences | Neurons and Cognition | Neurobiology | methazolamide | amyloid | hippocampus | carbonic anhydrase inhibitor | caspase activation | hydrogen peroxide | neuron | microglia
Journal Article
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