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Journal of Cellular Physiology, ISSN 0021-9541, 12/2017, Volume 232, Issue 12, pp. 3409 - 3421
In this study, we examine whether an anti‐inflammatory thiourea derivative, compound #326, actions on ion channels. The effects of compound #326 on... 
large‐conductance Ca2+‐activated K+ channel | Na+ current | Ca2+‐activated K+ current | inflammation | thiourea derivative | large-conductance Ca | channel | current | activated K | POTENT ACTIVATION | PHYSIOLOGY | CA2+-ACTIVATED K+ CHANNELS | KB-R7943 | CURRENTS | GH CELLS | large-conductance Ca2+-activated K plus channel | CELL BIOLOGY | INHIBITION | PITUITARY-TUMOR CELLS | Ca2+-activated K+ current | Microglia - metabolism | Calcium - metabolism | Humans | Interleukin-1beta - genetics | Thiourea - pharmacology | Dose-Response Relationship, Drug | Transfection | Interleukin-1beta - metabolism | HEK293 Cells | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - agonists | Microglia - drug effects | Anti-Inflammatory Agents - pharmacology | Rats | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - metabolism | Pituitary Neoplasms - metabolism | Animals | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - genetics | Membrane Potentials | Nitric Oxide Synthase Type II - genetics | Calcium Signaling - drug effects | Lipopolysaccharides - pharmacology | Cell Line, Tumor | Calcium Channel Agonists - pharmacology | Mice, Inbred BALB C | Kinetics | Thiourea - analogs & derivatives | Ion Channel Gating - drug effects | Nitric Oxide Synthase Type II - metabolism | Nitric oxide | Potassium channels | Electric potential | Calcium | Interleukin | Potassium conductance | Immunity | Potassium channels (calcium-gated) | Interleukin 1 | Sodium channels (voltage-gated) | Thiourea | Ion channels | Potassium channels (voltage-gated) | Gating | Calcium channels | Channel gating | Tolbutamide | Inflammation | Free energy | Nitric-oxide synthase | Microglia | Resistance | Calcium conductance | Pituitary | Conductance | Potassium currents | Potassium | Index Medicus
Journal Article
by Yang, XY and Wang, GQ and Cao, T and Zhang, L and Ma, YZ and Jiang, SH and Teng, XC and Sun, XH
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 08/2019, Volume 294, Issue 35, pp. 12921 - 12932
Large-conductance calcium-activated potassium (BK) channels are ubiquitously expressed in most cell types where they regulate many cellular, organ, and... 
BK channel | cytokine | lipopolysaccharide (LPS) | MITOCHONDRIAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CA2+-ACTIVATED K+ CHANNELS | INVOLVEMENT | Toll-like receptor 4 (TLR4) | NF-kappa B (NF-KB) | ADHESION | SMOOTH-MUSCLE | innate immunity | neuroinflammation | BK CHANNELS | TRANSIENTS | ION CHANNELS | microglia | EXPRESSION | MAXIK
Journal Article
Neurochemical Research, ISSN 0364-3190, 12/2019, Volume 44, Issue 12, pp. 2733 - 2745
Journal Article
Experimental Neurology, ISSN 0014-4886, 05/2012, Volume 235, Issue 1, pp. 282 - 296
Journal Article
Glia, ISSN 0894-1491, 09/2018, Volume 66, Issue 9, pp. 1881 - 1895
Microglia show a rich repertoire of activation patterns regulated by a complex ensemble of surface ion channels, receptors, and transporters. We and others... 
PAP‐1 | potassium channel | pro‐inflammatory | Kv1.3 | microglia | Kir2.1 | PAP-1 | pro-inflammatory | TARGET | STIMULATION | SYNAPTIC PLASTICITY | NEUROSCIENCES | AMYLOID-BETA | MICE | LONG-TERM POTENTIATION | K+ CHANNEL | MEMORY T-CELLS | EXPRESSION | BLOCKER | Calcium-Binding Proteins - metabolism | Microglia - metabolism | Cytokines - metabolism | Immunity, Innate - drug effects | Microglia - drug effects | Tissue Culture Techniques | Escherichia coli | Mice, Inbred C57BL | Kv1.3 Potassium Channel - antagonists & inhibitors | Kv1.3 Potassium Channel - genetics | Long-Term Potentiation - immunology | Lipopolysaccharides | Mice, Knockout | Brain - drug effects | Brain - metabolism | Inflammation - metabolism | Animals | Kv1.3 Potassium Channel - metabolism | Microfilament Proteins - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Nervous system diseases | Analysis | Potassium channels | Potentiation | Cell culture | Brain | Animal models | Pathogenesis | Brain slice preparation | Activation | Neurotoxicity | Receptors | Rodents | Ion channels | Potassium channels (voltage-gated) | Immune response | Potassium channels (inwardly-rectifying) | Pharmacology | Inflammation | Gene expression | Ribonucleic acid--RNA | Nitric-oxide synthase | Microglia | Neurological diseases | Tumor necrosis factor | Immunoreactivity | Potassium | Hippocampus | Index Medicus
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2015, Volume 44, Issue 3, pp. 797 - 808
Journal Article
GLIA, ISSN 0894-1491, 10/2013, Volume 61, Issue 10, pp. 1620 - 1628
Microglia are the resident immune cells of the brain, which are important therapeutic targets for regulating the inflammatory responses particularly... 
MIGRATION | MEMBRANE | PI3-kinase | NEUROSCIENCES | Rho GTPases | RAC | potassium channel | RECEPTOR ACTIVATION | CROSSTALK | microglia | EXPRESSION | FILAMIN-A | BRAIN | MOTILITY | Potassium channels | G proteins | Cytoskeleton | Potassium
Journal Article
Journal Article
FRONTIERS IN CELLULAR NEUROSCIENCE, ISSN 1662-5102, 05/2018, Volume 12
The cytokine, transforming growth factor beta 1 (TGF beta 1), is up-regulated after central nervous system (CNS) injuries or diseases involving microglial... 
KV1.3 K+ CHANNELS | transcription profiling | TGF beta 1 | TGF-BETA | rat | CLASS-II TRANSACTIVATOR | NEUROSCIENCES | mouse | OPERATED CALCIUM-ENTRY | MEMBRANE-POTENTIALS | potassium channels | FUNCTIONAL EXPRESSION | anti-inflammatory | MHC GENE-EXPRESSION | INTRACEREBRAL HEMORRHAGE | NITRIC-OXIDE | ION CHANNELS | microglia | microglial activation
Journal Article
EUROPEAN JOURNAL OF PHARMACOLOGY, ISSN 0014-2999, 01/2017, Volume 795, pp. 1 - 7
Neuropathic pain is a debilitating, chronic condition with a significant unmet need for effective treatment options. Recent studies have demonstrated that in... 
Tactile allodynia | ACTIVATION | Chronic constriction injury | MECHANISMS | KCNN4/KCA3.1 | Neuropathic pain | Microglia | BLOCKERS | IMMUNE | Calcium-activated potassium channel | K(Ca)3.1 | Senicapoc | Von Frey | PHARMACOLOGY & PHARMACY | KCNN4 | ICA-17043 | K+ CHANNELS | BRAIN | MONONEUROPATHY
Journal Article
Journal Article