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Acta Pharmaceutica Sinica B, ISSN 2211-3835, 01/2017, Volume 7, Issue 1, pp. 3 - 17
The intracellular calcium ions (Ca ) act as second messenger to regulate gene transcription, cell proliferation, migration and death. Accumulating evidences... 
Cell proliferation | entry | channels | Channel blockers | Cancer therapy | Migration | Store-operated Ca | Apoptosis | Store-operated Ca2+ entry | ACTIVATED CA2+ CHANNELS | POTENTIAL VANILLOID-1 TRPV1 | INOSITOL 1,4,5-TRISPHOSPHATE RECEPTOR | BREAST-CANCER | Ca2+ channels | HUMAN COLON-CANCER | PROSTATE-CANCER | SQUAMOUS-CELL CARCINOMA | ION-CHANNEL | MOLECULE 2 STIM2 | PHARMACOLOGY & PHARMACY | HUMAN OVARIAN-CANCER | PM, plasma membrane | CaMKII, calmodulin-dependent protein kinase II | mAb, monoclonal antibody | NFAT, nuclear factor of activated T cells | CaM, calmodulin | sarcoplasmic reticulum | CBG, cannabigerol | Ca2+ exchanger | RyR, ryanodine receptor | 20-GPPD, 20-O-β-D-glucopyranosyl-20(S)-protopanaxadiol | SPCA, secretory pathway Ca2+-ATPase | CYP3A4, cytochrome P450 3A4 | CPZ, capsazepine | SOCE, store-operated Ca2+ entry | CBD, cannabidiol | OSCC, oral squamous cell carcinoma cells | MLCK, myosin light-chain kinase | HCX, H | ER Ca2+-ATPase | Ca2+ exchangers | MCUR1, MCU uniporter regulator 1 | Review | TEA, tetraethylammonium | MICU (1, 2, 3), mitochondrial calcium uptake (type 1, type 2, type 3) | SR, endoplasmic | TG, thapsigargin | TRP (A, C, M, ML, N, P, V), transient receptor potential (ankyrin, canonical, melastatin, mucolipin, no mechanoreceptor potential C, polycystic, vanilloid) | IP3, inositol 1,4,5-trisphosphate | TPC2, two-pore channel 2 | ROS, reactive oxygen species | CRAC, Ca2+ release-activated Ca2+ channel | MCU, mitochondrial Ca2+ uniporter | PTP, permeability transition pore | NCX, Na+ | NSCLC, non-small cell lung cancer | CTL, cytotoxic T cells | PKC, protein kinase C | IP3R (1, 2, 3), IP3 receptor (type 1, type 2, type 3) | SERCA, SR | VGCC, voltage-gated Ca2+ channel | NF-κB, nuclear factor-κB | TRIM, 1-(2-(trifluoromethyl) phenyl) imidazole | PMCA, plasma membrane Ca2+-ATPase
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, p. e0148359
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 5/2015, Volume 93, Issue 5, pp. 499 - 509
Mitochondrial respiration is required for hypoxia-inducible factor (HIF)-1α stabilization, which is important for tumor cell survival, proliferation, and... 
Human Genetics | Angiogenesis | Mitochondria | Biomedicine | T-type Ca 2+ channel | Internal Medicine | NNC 55-0396 | Molecular Medicine | HIF-1α signal transduction | channel | T-type Ca | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | T-type Ca2+ channel | HIF-1 alpha signal transduction | TUMOR ANGIOGENESIS | HIF-1 | INTERMITTENT HYPOXIA | IN-VIVO | CALCIUM | GENETICS & HEREDITY | GENE-EXPRESSION | LYMPHANGIOGENESIS | MITOCHONDRIAL COMPLEX-III | PROLYL | Neoplasms - metabolism | Humans | Proteolysis - drug effects | Calcium Channels, T-Type - metabolism | Neoplasms - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Female | Calcium Channels, T-Type - genetics | Cyclopropanes - pharmacology | Disease Models, Animal | Cell Line | Angiogenesis Inhibitors - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Calcium Channel Blockers - pharmacology | Naphthalenes - pharmacology | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Neovascularization, Pathologic - drug therapy | Protein Stability - drug effects | Benzimidazoles - pharmacology | Protein Biosynthesis - drug effects | Mice | Neovascularization, Pathologic - metabolism | Proteasome Endopeptidase Complex - metabolism | Neoplasms - pathology
Journal Article
Journal Article
Journal Article
Cell Calcium, ISSN 0143-4160, 2008, Volume 44, Issue 4, pp. 339 - 352
Summary Altered calcium homeostasis and increased cytosolic calcium concentrations ([Ca2+ ]c ) are linked to neuronal apoptosis in epilepsy and in cerebral... 
Advanced Basic Science | Mitochondrial concentration of Ca2+ ([Ca2+]m) | Cytosolic aequorin (cyt_AEQ) | Bcl2 | Concentration of free Ca2+ ([Ca2+]) | Cytosolic concentration of Ca2+ ([Ca2+]c) | Mutated aequorin with a mitochondrial targeting signal (mitmut_AEQ) | AEQ: Aequorin | PC12 cells | Cytosolic concentration of Ca | Concentration of free Ca | Mitochondrial concentration of Ca | Ca | HOMEOSTASIS | Mitochondrial concentration of Ca2+ ([Ca2+](m)) | CHROMAFFIN CELLS | STIMULATION | INDUCED APOPTOSIS | CALCIUM CHANNELS | DEATH | CELL BIOLOGY | Cytosolic concentration of Ca2+ ([Ca2+](c)) | DEPOLARIZATION | AEQUORIN | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | Calcium - agonists | Nitriles - pharmacology | Calcium - metabolism | 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester - pharmacology | Rats | Mitochondria - metabolism | Nimodipine - pharmacology | Calcium Channel Blockers - pharmacology | Membrane Potentials - physiology | PC12 Cells | Down-Regulation - genetics | Calcium - antagonists & inhibitors | Patch-Clamp Techniques | Animals | Transfection | Calcium Channels, L-Type - drug effects | bcl-Associated Death Protein - drug effects | Cell Line, Tumor | Ionomycin - pharmacology | bcl-Associated Death Protein - metabolism | Calcium Channel Agonists - pharmacology | Calcium Channels, L-Type - metabolism | Benzopyrans - pharmacology | Ionophores - pharmacology
Journal Article
Biochemical Journal, ISSN 0264-6021, 12/2012, Volume 448, Issue 2, pp. 189 - 200
Journal Article