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Journal Article
2008, 1. Aufl., ISBN 9780470111314, xvii, 616 p., [12] p. of plates
This is the definitive, one-stop resource on preclinical drug evaluation for potential mitochondrial toxicity, addressing the issue upfront in the drug... 
Drugs -- Toxicology | Mitochondrial pathology | Science | Chemistry | Industrial & Technical | Drugs | Toxicology
Book
1993, ISBN 9780124612051, Volume 2., xx, 502
Book
2002, International review of neurobiology, ISBN 9780123668547, Volume 53., xx, 559
Mitochondria are critical to the survival of cells, therefore, it is not surprising that abnormalities in mitochondrial function may lead to human disease.... 
Mitochondria | Mitochondrial pathology | Physiology
Book
Blood, ISSN 0006-4971, 02/2012, Volume 119, Issue 6, pp. 1490 - 1500
Ataxia-telangiectasia mutated (ATM) plays a central role in DNA damage responses, and its loss leads to development of T-cell malignancies. Here, we show that... 
OXIDATIVE STRESS | AUTOPHAGY GENE | BECLIN-1 | HEMATOPOIETIC STEM-CELLS | TUMOR-SUPPRESSOR | DJ-1 | IONIZING-RADIATION | HEMATOLOGY | CANCER | ATM-DEFICIENT MICE | PARKINSONS-DISEASE | Reactive Oxygen Species - metabolism | Thymocytes - metabolism | Humans | Immunoblotting | Autophagy | Lymphoma, T-Cell - metabolism | DNA-Binding Proteins - metabolism | Mitochondria - genetics | RNA Interference | Adenosine Triphosphate - metabolism | Tumor Suppressor Proteins - genetics | Membrane Potential, Mitochondrial | Apoptosis Regulatory Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Ataxia Telangiectasia - physiopathology | Beclin-1 | Microscopy, Electron, Transmission | Thymocytes - ultrastructure | Gene Expression | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Cells, Cultured | Kaplan-Meier Estimate | Oxygen Consumption | Protein-Serine-Threonine Kinases - genetics | Ataxia Telangiectasia - metabolism | Mitochondria - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Lymphoma, T-Cell - genetics | Reverse Transcriptase Polymerase Chain Reaction | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Ataxia Telangiectasia - genetics | Fibroblasts - cytology | Mice | Mitochondria - physiology | Index Medicus | Abridged Index Medicus | Lymphoid Neoplasia
Journal Article
2016, Second edition., ISBN 9783319286358
Web Resource
Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7334, pp. 359 - 365
Telomere dysfunction activates p53-mediated cellular growth arrest, senescence and apoptosis to drive progressive atrophy and functional decline in... 
STEM-CELLS | HEPATOCELLULAR-CARCINOMA | BIOGENESIS | PGC-1-ALPHA | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | LEADS | MICE | DAMAGE | P53 | COACTIVATORS | Cell Proliferation | Reactive Oxygen Species - metabolism | Hematopoietic Stem Cells - pathology | DNA, Mitochondrial - analysis | Adenosine Triphosphate - biosynthesis | Tumor Suppressor Protein p53 - genetics | RNA - genetics | Telomerase - deficiency | Cardiomyopathies - physiopathology | Telomerase - genetics | Myocardium - metabolism | Telomere - metabolism | Gluconeogenesis | Telomere - genetics | Liver - metabolism | Tumor Suppressor Protein p53 - metabolism | Cardiomyopathies - pathology | Transcription Factors - antagonists & inhibitors | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Tumor Suppressor Protein p53 - deficiency | Doxorubicin - toxicity | Aging - pathology | Myocardium - cytology | Transcription Factors - metabolism | Telomere - pathology | Animals | Cardiomyopathies - metabolism | Telomere - enzymology | Liver - cytology | Mice | Aging - metabolism | Cardiomyopathies - chemically induced | Telomeres | Mitochondria | Genetic aspects | Chemical properties | Abnormalities | Liver | Cell division | Mitochondrial DNA | Biosynthesis | Biology | DNA repair | Proteins | Studies | Aging | Telomerase | Binding sites | Apoptosis | Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 10/2016, Volume 17, Issue 3, pp. 684 - 696
Journal Article
Circulation, ISSN 0009-7322, 09/2011, Volume 124, Issue 12, pp. 1370 - 1381
Background-Angiotensin II (ATII), a potent vasoconstrictor, causes hypertension, promotes infiltration of myelomonocytic cells into the vessel wall, and... 
blood vessels | inflammation | hypertension | angiotensin II | oxidative stress | MACROPHAGE INFILTRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOTHELIAL DYSFUNCTION | SUPEROXIDE-PRODUCTION | MITOCHONDRIAL ALDEHYDE DEHYDROGENASE | CELL-LINEAGE-ABLATION | BLOOD-PRESSURE | IN-VITRO | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | MICE | Vasculitis - metabolism | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Oxidative Stress - physiology | Muscle, Smooth, Vascular - immunology | Male | Monocytes - metabolism | Monocytes - immunology | Muramidase - immunology | Vasculitis - immunology | Respiratory Burst - physiology | Hypertension - chemically induced | Gene Expression - immunology | Neutrophils - metabolism | Vasculitis - chemically induced | Macrophages - immunology | Endothelium, Vascular - immunology | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Mice, Inbred C57BL | Muramidase - genetics | Receptors, Chemokine - metabolism | Neutrophils - immunology | Mice, Transgenic | Hypertension - immunology | Muramidase - metabolism | Hypertension - metabolism | Macrophages - metabolism | Animals | Endothelium, Vascular - metabolism | Mice | CD11b Antigen - metabolism | Nitric Oxide - metabolism | Hypertension | Blood circulation disorders | Oxidative stress | Angiotensin | Physiological aspects | Development and progression | Research | Lysozyme | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article