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Circulation, ISSN 0009-7322, 01/2005, Volume 111, Issue 2, pp. 194 - 197
Background - Brief periods of ischemia performed just at the time of reperfusion can reduce infarct size, a phenomenon called "postconditioning." After reflow,... 
Myocardial infarction | Ischemia | Reperfusion | ISCHEMIA-REPERFUSION INJURY | RAT HEARTS | CARDIAC & CARDIOVASCULAR SYSTEMS | CYCLOSPORINE-A | MYOCYTES | myocardial infarction | PORE | PERIPHERAL VASCULAR DISEASE | MYOCARDIAL REPERFUSION | CHANNELS | reperfusion | ischemia | MAP Kinase Signaling System - physiology | Oxidative Stress | Apoptosis - drug effects | Calcium - metabolism | Coronary Disease - physiopathology | Ion Channels - drug effects | Male | Ion Channels - physiology | Mitochondria, Heart - drug effects | Myocardial Reperfusion Injury - enzymology | Necrosis | Mitochondrial Membrane Transport Proteins | Ion Channel Gating - physiology | Time Factors | Myocardial Infarction - pathology | Ion Transport - drug effects | Extracellular Signal-Regulated MAP Kinases - physiology | Myocardial Infarction - etiology | Rabbits | Protein-Serine-Threonine Kinases - physiology | Random Allocation | Myocardial Ischemia - drug therapy | Myocardial Reperfusion Injury - physiopathology | Proto-Oncogene Proteins c-akt | Cyclosporine - therapeutic use | Animals | MAP Kinase Signaling System - drug effects | Membrane Potentials | Mitochondria, Heart - physiology | Myocardial Ischemia - complications | Myocardial Ischemia - therapy | Proto-Oncogene Proteins - physiology | Apoptosis - physiology | Ion Transport - physiology | Myocardial Reperfusion Injury - prevention & control | Ion Channel Gating - drug effects
Journal Article
Journal Article
FEBS Letters, ISSN 0014-5793, 2010, Volume 584, Issue 10, pp. 1989 - 1996
The permeability transition pore (PTP) is an inner mitochondrial membrane channel that has been thoroughly characterized functionally, yet remains an elusive... 
Cyclophilin D | Permeability transition pore | Kinase | Chaperone | Channel | Apoptosis | PTEN induced kinase 1 | cyclophilin D | inner mitochondrial membrane | outer mitochondrial membrane | cyclosporin A | adenine nucleotide translocator | GSK3 | permeability transition pore | extracellular signal regulated kinase | hexokinase | reperfusion injury salvage kinase | mitochondrial protein phosphatase 2C | IMM | PKC | PKG | amyloid beta | OMM | CsA | PP2Cm | receptor tyrosine kinases | G protein coupled receptors | ROS | ERK | permeability transition | ANT | PiC | SHP-1 | PTP | tumor necrosis factor receptor-associated protein 1 | Hsp90 | phosphate anion | GPCRs | protein phosphatase 2A | protein phosphatase 2B | reactive oxygen species | SH2-containing tyrosine phosphatase 1 | CyP-D | protein kinase G | heat shock protein 90 | protein kinase C | phosphate carrier | TRAP-1 | RISK | PP2A | RTKs | PINK1 | voltage-dependent anion channel | PP2B | VDAC | glycogen synthase kinase 3 | INNER MITOCHONDRIAL-MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-DEATH | CELL BIOLOGY | GLYCOGEN-SYNTHASE KINASE-3-BETA | HEXOKINASE-II | BRAIN MITOCHONDRIA | CYCLOPHILIN-D | BIOPHYSICS | THERAPEUTIC TARGET | CARDIOMYOCYTE PROTECTION | DEPENDENT ANION CHANNELS | PARKINSONS-DISEASE | Mitochondrial Membrane Transport Proteins - metabolism | Animals | Disease | Ion Channels - metabolism | Signal Transduction | Humans | Tyrosine | Phosphates | Synthesis | Phosphatases | Glycogen | Cytochrome P-450 | Heat shock proteins | Permeability | Protein kinases
Journal Article
Frontiers in Physiology, ISSN 1664-042X, 2013, Volume 4, pp. 95 - 95
Journal Article